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Pancreatology, ISSN 1424-3903, 2016, Volume 16, Issue 3, pp. S5 - S5
  Specifically, we have shown that * pancreatic cancer is hierarchical organised and cancer stem cells as the root of this disease drive tumour progression,... 
Endocrinology & Metabolism | Gastroenterology and Hepatology | Metastasis | Metabolism | Pancreatic cancer | Stem cells
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2007, Volume 49, Issue 24, pp. 2341 - 2349
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e76518 - e76518
Journal Article
Nature Methods, ISSN 1548-7091, 10/2014, Volume 11, Issue 11, pp. 1161 - 1169
Journal Article
Gut, ISSN 0017-5749, 12/2015, Volume 64, Issue 12, pp. 1936 - 1948
ObjectiveCancer stem cells (CSCs) represent the root of many solid cancers including pancreatic ductal adenocarcinoma, are highly chemoresistant and represent... 
DOWNSTREAM TARGET | SOLID TUMORS | PATHWAY | SELF-RENEWAL | PROLIFERATION | TUMORIGENICITY | IDENTIFICATION | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | MICRORNA CLUSTER | TBX3 | Cyclin-Dependent Kinase Inhibitor p57 - metabolism | MicroRNAs - antagonists & inhibitors | Pancreatic Neoplasms - metabolism | Neoplastic Stem Cells - drug effects | Epigenesis, Genetic | Humans | Transforming Growth Factor beta1 - metabolism | Carcinoma, Pancreatic Ductal - metabolism | Gene Expression Regulation, Neoplastic | Transcriptome | Activins - metabolism | Deoxycytidine - pharmacology | MicroRNAs - metabolism | Nodal Protein - metabolism | Carcinoma, Pancreatic Ductal - genetics | Gene Knockdown Techniques | Pancreatic Neoplasms - drug therapy | Cell Self Renewal | Deoxycytidine - therapeutic use | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Neoplastic Stem Cells - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Antimetabolites, Antineoplastic - pharmacology | Female | Signal Transduction | Down-Regulation | Pancreatic Neoplasms - genetics | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Carcinoma, Pancreatic Ductal - drug therapy | Antimetabolites, Antineoplastic - therapeutic use | Drug Resistance, Neoplasm - genetics | Animals | Cell Cycle Checkpoints - drug effects | Cell Transformation, Neoplastic | Mice, Nude | Mice | MicroRNAs - genetics | Deoxycytidine - analogs & derivatives | Care and treatment | MicroRNA | Pancreatic cancer | Patient outcomes | Analysis | Stem cells | Research | Metastasis | Risk factors | International organizations | Medical research | Biomedical research | Cell cycle | Tumors | Index Medicus | Abridged Index Medicus | CELL CYCLE CONTROL | PANCREATIC CANCER | DRUG RESISTANCE | STEM CELLS | Pancreas | 1506 | INTRACELLULAR SIGNALING
Journal Article
Circulation, ISSN 0009-7322, 02/2002, Volume 105, Issue 6, pp. 739 - 745
Background-Statins inhibit HMG-CoA reductase to reduce the synthesis of cholesterol and isoprenoids that modulate diverse cell functions. We investigated the... 
Hypoxia | Lipids | Inflammation | Apoptosis | Endothelium | endothelium | hypoxia | lipids | CARDIAC & CARDIOVASCULAR SYSTEMS | HMG-COA REDUCTASE | apoptosis | ENDOTHELIAL-CELL MIGRATION | HYPERCHOLESTEROLEMIA | CANCER | inflammation | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | CERIVASTATIN | INHIBITORS | RHO-GTPASES | SIMVASTATIN | HEMATOLOGY | SMOOTH-MUSCLE CELLS | Endothelium, Vascular - cytology | Neovascularization, Physiologic - drug effects | Receptors, Vascular Endothelial Growth Factor | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Hyperlipidemias - genetics | Apolipoproteins E - deficiency | Apoptosis - drug effects | Vascular Endothelial Growth Factors | Humans | Receptor Protein-Tyrosine Kinases - biosynthesis | Endothelium, Vascular - drug effects | Carcinoma, Lewis Lung - blood supply | Neovascularization, Pathologic - pathology | Dose-Response Relationship, Drug | Lymphokines - metabolism | Female | Heptanoic Acids - pharmacology | Hyperlipidemias - drug therapy | Mice, Inbred C57BL | Cells, Cultured | Cell Division - drug effects | Carcinoma, Lewis Lung - drug therapy | Mice, Knockout | Atorvastatin Calcium | Cell Movement - drug effects | Pyrroles - pharmacology | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Signal Transduction - drug effects | Cell Differentiation - drug effects | Endothelium, Vascular - metabolism | Neovascularization, Pathologic - drug therapy | Apolipoproteins E - genetics | Receptors, Growth Factor - biosynthesis | Carcinoma, Lewis Lung - pathology | Mice | Polyisoprenyl Phosphates - pharmacology | Pyridines - pharmacology | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
STEM CELLS, ISSN 1066-5099, 10/2015, Volume 33, Issue 10, pp. 2893 - 2902
Journal Article
ONCOGENE, ISSN 0950-9232, 08/2019, Volume 38, Issue 34, pp. 6226 - 6239
Pancreatic ductal adenocarcinoma (PDAC) arises through accumulation of multiple genetic alterations. However, cancer cells also acquire and depend on... 
STEM-CELLS | INHIBITION | ONCOLOGY | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOT1L | GENETICS & HEREDITY | DEDIFFERENTIATION | PLURIPOTENCY | TECHNOLOGY | CELL BIOLOGY | Adenocarcinoma | Alkaline phosphatase | Immunomodulation | Pancreatic cancer | Oct-4 protein | MiRNA | Fibroblasts | Epigenetics | Transplantation | Cell differentiation | Pluripotency | Index Medicus
Journal Article
Gut, ISSN 0017-5749, 12/2015, Volume 64, Issue 12, pp. 1921 - 1935
ObjectivesThe tumour stroma/microenvironment not only provides structural support for tumour development, but more importantly it provides cues to cancer stem... 
PEPTIDE LL-37 | ANTIMICROBIAL PROTEIN HCAP18/LL-37 | BONE-MARROW | TUMOR-ASSOCIATED MACROPHAGES | VITAMIN-D-RECEPTOR | SELF-RENEWAL | GROWTH-FACTOR | MYELOID CELLS | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | CATHELICIDIN LL-37 | Neoplastic Stem Cells - secretion | Pancreatic Neoplasms - metabolism | Gene Expression - drug effects | Neoplastic Stem Cells - drug effects | Tissue Array Analysis | Humans | Receptors, Formyl Peptide - antagonists & inhibitors | Carcinoma, Pancreatic Ductal - metabolism | Tumor Microenvironment | Purinergic P2X Receptor Antagonists - pharmacology | Antimicrobial Cationic Peptides - metabolism | Carcinogenesis - metabolism | Carcinoma, Pancreatic Ductal - genetics | Cell Self Renewal - drug effects | Activins - secretion | Macrophages - secretion | Receptors, Formyl Peptide - metabolism | Transforming Growth Factor beta1 - pharmacology | Neoplasm Invasiveness | Pancreatic Neoplasms - pathology | Antimicrobial Cationic Peptides - pharmacology | Carcinogenesis - genetics | Pancreatic Neoplasms - genetics | Antimicrobial Cationic Peptides - secretion | Carcinoma, Pancreatic Ductal - pathology | Carcinogenesis - drug effects | Animals | Signal Transduction - drug effects | Mice, Nude | Protein Array Analysis | Receptors, Purinergic P2X7 - metabolism | Macrophages - drug effects | Mice | Care and treatment | Immune response | Analysis | Pancreatic cancer | Stem cells | Research | Risk factors | Proteins | Studies | Peptides | Metastasis | Kinases | Cancer therapies | Growth factors | Tumors | Ovarian cancer | Index Medicus | Abridged Index Medicus
Journal Article