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Nature, ISSN 0028-0836, 02/2012, Volume 482, Issue 7384, pp. 216 - U107
Our understanding of Alzheimer's disease pathogenesis is currently limited by difficulties in obtaining live neurons from patients and the inability to model... 
AMYLOID BETA-PROTEIN | APP | MICROTUBULE-BINDING | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | DOWN-SYNDROME | MOUSE MODEL | TAU | DYSFUNCTION | FIBROBLASTS | SENILE PLAQUES | Neurons - pathology | Coculture Techniques | Humans | Middle Aged | Amyloid beta-Peptides - secretion | tau Proteins - metabolism | Male | Phosphoproteins - metabolism | Endosomes - metabolism | Synapsins - metabolism | Alzheimer Disease - pathology | Cellular Reprogramming | Protease Inhibitors - pharmacology | Amyloid beta-Protein Precursor - secretion | Proteolysis | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | Aged, 80 and over | Female | Neurons - metabolism | Phosphorylation - drug effects | Neurons - drug effects | Fibroblasts - metabolism | Induced Pluripotent Stem Cells - metabolism | Astrocytes - cytology | Biomarkers - metabolism | Induced Pluripotent Stem Cells - pathology | Peptide Fragments - metabolism | Cells, Cultured | Peptide Fragments - secretion | Glycogen Synthase Kinase 3 - metabolism | Amyloid Precursor Protein Secretases - metabolism | Amyloid beta-Protein Precursor - genetics | Models, Biological | Alzheimer Disease - metabolism | Fibroblasts - cytology | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Enzyme Activation | Messenger RNA | Synthesis | Glycogen | Stem cells | Physiological aspects | Research | Alzheimer's disease | Proteins | Phosphorylation | Neurons | Efficiency | Genomes | Mutation | Alzheimers disease | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2012, Volume 7, Issue 8, pp. e42614 - e42614
Journal Article
Human Mutation, ISSN 1059-7794, 06/2011, Volume 32, Issue 6, pp. 598 - 609
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 01/2009, Volume 29, Issue 4, pp. 1093 - 1104
Astrocytes in the CNS respond to tissue damage by becoming reactive. They migrate, undergo hypertrophy, and form a glial scar that inhibits axon regeneration.... 
Rapamycin | Akt | Ischemia | Tuberin | Spinal cord injury | Cell migration | spinal cord injury | CHONDROITIN SULFATE PROTEOGLYCANS | AXON REGENERATION | KINASE INHIBITORS | NEUROSCIENCES | ischemia | TUBEROUS SCLEROSIS COMPLEX | FIBRILLARY ACIDIC PROTEIN | CELL-MIGRATION | cell migration | CENTRAL-NERVOUS-SYSTEM | GROWTH-FACTOR | ACTIN CYTOSKELETON | PTEN-DEFICIENT | rapamycin | Protein Kinases - metabolism | Protein Kinases - genetics | Spinal Cord Injuries - drug therapy | Vimentin - metabolism | Immunosuppressive Agents - therapeutic use | ErbB Receptors - genetics | Epidermal Growth Factor - drug effects | Male | Ras Homolog Enriched in Brain Protein | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Spinal Cord Injuries - pathology | Transfection - methods | Vimentin - genetics | Up-Regulation - physiology | Excitatory Amino Acid Transporter 2 - genetics | Flavonoids - pharmacology | Neuropeptides - genetics | Chromones - pharmacology | Immunosuppressive Agents - pharmacology | Disease Models, Animal | Astrocytes - drug effects | Sirolimus - therapeutic use | ErbB Receptors - metabolism | Spinal Cord Injuries - metabolism | Epidermal Growth Factor - genetics | Cells, Cultured | Enzyme Inhibitors - pharmacology | Morpholines - pharmacology | Rats | Neuropeptides - metabolism | Monomeric GTP-Binding Proteins - genetics | Transcription Factors - genetics | Excitatory Amino Acid Transporter 2 - metabolism | Rats, Sprague-Dawley | Sirolimus - pharmacology | Transcription Factors - metabolism | Up-Regulation - drug effects | Animals | Analysis of Variance | Monomeric GTP-Binding Proteins - metabolism | Signal Transduction - physiology | TOR Serine-Threonine Kinases | Astrocytes - metabolism | Index Medicus
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 03/2001, Volume 296, Issue 3, pp. 756 - 761
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 06/2017, Volume 34, Issue 11, pp. 1981 - 1995
Penetrating traumatic brain injury (PTBI) is one of the major cause of death and disability worldwide. Previous studies with penetrating ballistic-like brain... 
Original Articles | cell transplantation | behavior deficit | TBI | PBBI | hNSC | neuronal differentiation | PROGENITOR CELLS | FUNCTIONAL RECOVERY | SYNAPTIC PROTEIN | AMYOTROPHIC-LATERAL-SCLEROSIS | NEUROSCIENCES | RAT MODEL | CLINICAL NEUROLOGY | PHASE-I TRIAL | GUNSHOT WOUNDS | STEM/PROGENITOR CELLS | STROMAL CELLS | RODENT MODEL | CRITICAL CARE MEDICINE | Brain Injuries, Traumatic - therapy | Humans | Rats, Inbred F344 | Rats | Random Allocation | Rats, Nude | Rats, Sprague-Dawley | Head Injuries, Penetrating - diagnosis | Cognition Disorders - diagnosis | Stem Cell Transplantation - methods | Animals | Neurons - physiology | Neural Stem Cells - transplantation | Cognition Disorders - therapy | Head Injuries, Penetrating - therapy | Brain Injuries, Traumatic - diagnosis | Cell Differentiation - physiology | Brain | Usage | Patient outcomes | Stem cells | Green fluorescent protein | Research | Injuries | Cell proliferation | Animal models | Traumatic brain injury | Mycophenolic acid | Transplants & implants | Leukocyte migration | Brain stem | Neurobiology | Cytology | Central nervous system | Cognitive ability | Parenchyma | Stem cell transplantation | Transplantation | Motor task performance | Methylprednisolone | Allografts | Neurodegeneration | Wounds | Transplants | Latency | Immunosuppression | Tacrolimus | Brain damage | Neural stem cells | Mycophenolate mofetil | Cell migration | Index Medicus
Journal Article
Journal Article
Neuroscience, ISSN 0306-4522, 2007, Volume 147, Issue 2, pp. 546 - 560
Journal Article
Journal Article