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Nature Communications, ISSN 2041-1723, 05/2016, Volume 7, Issue 1, p. 11523
ADAM17, a prominent member of the 'Disintegrin and Metalloproteinase' (ADAM) family, controls vital cellular functions through cleavage of transmembrane... 
L-SELECTIN | ACTIVATION | SHEDDING ACTIVITY | MULTIDISCIPLINARY SCIENCES | APOPTOTIC CELLS | ECTODOMAIN | RECEPTOR | NECROSIS-FACTOR-ALPHA | SUBSTRATE SELECTIVITY | CONVERTING-ENZYME | BLOOD-CELLS
Journal Article
BBA - Biomembranes, ISSN 0005-2736, 05/2016, Volume 1858, Issue 5, pp. 971 - 979
Bacterial infections, with the most severe form being sepsis, can often not be treated adequately leading to high morbidity and lethality of infected patients... 
Sepsis | Inflammation | Endotoxin | LPS-neutralization | Antimicrobial peptides | BIOPHYSICAL CHARACTERIZATION | LACTOFERRICIN | MECHANISM | NEUTRALIZING ACTIVITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | LIMULUS ANTILIPOPOLYSACCHARIDE FACTOR | POLYMYXIN-B | IN-VITRO | BIOPHYSICS | LIPID-A | LIPOPOLYSACCHARIDE | Prevention | Bacterial infections | Analysis | Lactoferrins | Bacteria | Mitogens | Antibacterial agents
Journal Article
Journal Article
Gut, ISSN 0017-5749, 05/2014, Volume 64, Issue 2, pp. 222 - 232
Objective Antimicrobial peptides (AMP) provide protection from infection by pathogenic microorganisms and restrict bacterial growth at epithelial surfaces to... 
INVASIVE BACTERIAL-INFECTION | INFLAMMATION | CELL ALPHA-DEFENSINS | HOST-DEFENSE | LECTIN REGIII-GAMMA | ANTIMICROBIAL PEPTIDES | PANETH CELLS | GASTROENTEROLOGY & HEPATOLOGY | ILEAL CROHNS-DISEASE | EXPRESSION | INNATE IMMUNITY | Intestinal Mucosa - metabolism | Humans | Microbiota - drug effects | Salmonella Infections - metabolism | Salmonella typhimurium - metabolism | Anti-Bacterial Agents - therapeutic use | Anti-Inflammatory Agents - administration & dosage | Anti-Inflammatory Agents - pharmacokinetics | Female | Mucus - metabolism | Salmonella typhimurium - drug effects | Drug Evaluation, Preclinical - methods | Salmonella Infections - drug therapy | Salmonella typhimurium - physiology | Administration, Oral | Mice, Inbred C57BL | Peptide Fragments - administration & dosage | Cells, Cultured | Peptide Fragments - pharmacokinetics | Intestinal Mucosa - microbiology | Mucus - microbiology | Animals | Proteins - metabolism | Anti-Bacterial Agents - pharmacokinetics | Salmonella Infections - microbiology | Peptide Fragments - therapeutic use | Anti-Bacterial Agents - administration & dosage | Host-Pathogen Interactions - physiology | Care and treatment | Bacterial infections | Anti-inflammatory drugs | Antimicrobial peptides | Homeostasis | Dosage and administration | Research | Risk factors | Salmonella | Peptides | Laboratories | Antimicrobial agents | Infections | Small intestine | Biological activity | E coli | Rodents | Bacteria | River ecology
Journal Article
Biophysical Journal, ISSN 0006-3495, 02/2016, Volume 110, Issue 3, pp. 79a - 79a
Journal Article
Journal Article
Antimicrobial Agents and Chemotherapy, ISSN 0066-4804, 03/2013, Volume 57, Issue 3, pp. 1480 - 1487
Classifications Services AAC Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit... 
HOST-DEFENSE PEPTIDES | UNITED-STATES | GRAM-NEGATIVE SEPSIS | IMMUNITY | SURVIVING SEPSIS CAMPAIGN | ENDOTOXIN | SEPTIC SHOCK | MICROBIOLOGY | PHARMACOLOGY & PHARMACY | LIPOPOLYSACCHARIDES | EPIDEMIOLOGY | STRATEGIES | Humans | Molecular Sequence Data | Bacteremia - mortality | Lipopolysaccharides - antagonists & inhibitors | Bacteremia - microbiology | Sepsis - drug therapy | Peptides - chemical synthesis | Sepsis - metabolism | Female | Staphylococcal Infections - microbiology | Virulence Factors - antagonists & inhibitors | Disease Models, Animal | Endotoxemia - microbiology | Staphylococcal Infections - metabolism | Amino Acid Sequence | Staphylococcal Infections - drug therapy | Endotoxemia - drug therapy | Bacteremia - drug therapy | Staphylococcus aureus - pathogenicity | Peptides - pharmacology | Sepsis - mortality | Drug Synergism | Staphylococcal Infections - mortality | Bacteremia - metabolism | Animals | Lipopolysaccharides - biosynthesis | Survival Analysis | Anti-Bacterial Agents - pharmacology | Mice | Mice, Inbred BALB C | Endotoxemia - metabolism | Endotoxemia - mortality | Staphylococcus aureus - drug effects | Staphylococcus aureus - growth & development | Sepsis - microbiology | Virulence Factors - biosynthesis | Animal models | Antiseptics | Cecum | Endotoxins | Inflammation | Drug development | Bacteremia | Infection | Pathogenicity | Antibiotics | Antibiotic resistance | Sepsis | Endotoxemia | Experimental Therapeutics
Journal Article
Scientific Reports, ISSN 2045-2322, 2016, Volume 6, Issue 1, p. 34221
The pattern recognition receptor TLR4 is well known as a crucial receptor during infection and inflammation. Several TLR4 antagonists have been reported to... 
TOLL-LIKE-RECEPTOR-4 | STIMULATION | RECOGNITION | CELL ACTIVATION | INFLAMMATION | PERSISTENCE | MULTIDISCIPLINARY SCIENCES | TOLL-LIKE RECEPTORS | LIGAND | RECEPTOR 4 ACTIVATION | ENDOTOXINS | D-Galactosamine | E coli | Toll-like receptors | Amino acid sequence | Antagonists | TLR4 protein | Gram-negative bacteria | Pattern recognition | Polymyxin B | Lipid A | Lipopolysaccharides
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 09/2017, Volume 140, pp. 64 - 72
Toll-like receptor (TLR) 4-independent recognition of lipopolysaccharide (LPS) in the cytosol by inflammatory caspases leads to non-canonical inflammasome... 
Inflammatory caspases | Interleukin-1beta (IL-1β) | Synthetic anti-endotoxin peptides | Pyroptosis | Cytoplasmic lipopolysaccharide (LPS) | DEFENSE | RECEPTOR | NLRP3 INFLAMMASOME | RELEASE | NONCANONICAL INFLAMMASOME ACTIVATION | IMMUNE-RESPONSES | CASPASES | TARGETS | PHARMACOLOGY & PHARMACY | IL-1-BETA | Interleukin-1beta (IL-1 beta) | Inflammasomes - metabolism | Pyroptosis - drug effects | Monocytes - cytology | Humans | Anti-Inflammatory Agents, Non-Steroidal - chemistry | Cytoplasm - metabolism | Monocytes - metabolism | Monocytes - immunology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Macrophages - secretion | Inflammasomes - drug effects | Interleukin-1beta - metabolism | Myeloid Cells - immunology | Myeloid Cells - drug effects | Toll-Like Receptor 4 - antagonists & inhibitors | Macrophages - immunology | Interleukin-1beta - secretion | Cell Survival - drug effects | Lipopolysaccharides - toxicity | Peptides - chemistry | Cells, Cultured | Keratinocytes - secretion | Sulfonamides - pharmacology | Toll-Like Receptor 4 - metabolism | Keratinocytes - immunology | Peptides - pharmacology | Monocytes - drug effects | Endotoxins - antagonists & inhibitors | Macrophages - metabolism | Myeloid Cells - secretion | Keratinocytes - drug effects | Keratinocytes - metabolism | Inflammasomes - immunology | Cell Line, Tumor | Myeloid Cells - metabolism | Macrophages - drug effects | Macrophage Activation - drug effects | Cytoplasm - drug effects | Cell Line, Transformed | Endotoxins - toxicity | Chromosomal proteins | Peptides | Interleukins | Cell death | Pharmacy | Septic shock | Skin | Macrophages | Mitogens | Index Medicus
Journal Article