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Science, ISSN 0036-8075, 2/2007, Volume 315, Issue 5813, pp. 856 - 859
A central issue in the regulation of apoptosis by the Bcl-2 family is whether its BH3-only members initiate apoptosis by directly binding to the essential... 
Research fellowships | Protein isoforms | Medical research | Myeloid cells | Antibodies | Cytochromes | Ligands | Reports | Grants | Viability | Apoptosis | RESPONSES | FAMILY-MEMBERS | X-L | MULTIDISCIPLINARY SCIENCES | BIM | HELIX | MITOCHONDRIAL-MEMBRANE | PUMA | DOMAINS | BH3-ONLY PROTEINS | CELL-DEATH | bcl-2-Associated X Protein - chemistry | Humans | BH3 Interacting Domain Death Agonist Protein - genetics | Proto-Oncogene Proteins - chemistry | Neoplasm Proteins - metabolism | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Bcl-2-Like Protein 11 | Tumor Suppressor Proteins - genetics | BH3 Interacting Domain Death Agonist Protein - chemistry | Apoptosis Regulatory Proteins - genetics | bcl-Associated Death Protein - metabolism | Membrane Proteins - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | Protein Structure, Tertiary | Proto-Oncogene Proteins - metabolism | Cell Line | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Apoptosis Regulatory Proteins - chemistry | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Proteins - metabolism | Membrane Proteins - chemistry | Models, Biological | Myeloid Cell Leukemia Sequence 1 Protein | Mice | Mutation | bcl-X Protein - metabolism | Research | Peptides | Analysis | Biochemistry | Cellular biology | Molecular biology | Binding sites | Index Medicus
Journal Article
American Journal of Kidney Diseases, ISSN 0272-6386, 2015, Volume 67, Issue 6, pp. 902 - 911
Journal Article
PLoS Genetics, ISSN 1553-7390, 05/2015, Volume 11, Issue 5, pp. e1005211 - e1005211
Journal Article
PLoS Genetics, ISSN 1553-7390, 05/2015, Volume 11, Issue 5
  Down syndrome (DS), with trisomy of chromosome 21 (HSA21), is the commonest human aneuploidy. Pre-leukemic myeloproliferative changes in DS foetal livers... 
Medical research | Transcription factors | Leukemia | Colleges & universities | Down syndrome | Gene expression | Scholarships & fellowships | Councils | Stem cells | Bone marrow | Mutation | Chromosomes | Acquisitions & mergers | Cancer
Journal Article
Human Pathology, ISSN 0046-8177, 2013, Volume 44, Issue 7, pp. 1262 - 1270
Journal Article
Kidney & Blood Pressure Research, ISSN 1420-4096, 01/2018, Volume 42, Issue 6, pp. 1033 - 1044
Background/Aims: Cardiac biomarkers are associated with cardiac abnormalities and adverse outcomes in dialysis patients. Our aim was to report the effect of... 
Heart | Brain natriuretic peptide | Echocardiography | Laboratories | Mortality | Abnormalities | 2,4,6-Trinitrotoluene | Feasibility studies | Galectin-3 | Patients | Beta blockers | Peritoneal dialysis | Randomization | Biomarkers | Dialysis | Bioindicators | Secondary analysis | Ventricle
Journal Article
Nephrology, ISSN 1320-5358, 03/2015, Volume 20, Issue 3, pp. 140 - 147
Journal Article
American Journal of Kidney Diseases, ISSN 0272-6386, 2012, Volume 60, Issue 3, pp. 463 - 472
Journal Article
Diabetes, ISSN 0012-1797, 03/2010, Volume 59, Issue 3, pp. 644 - 652
OBJECTIVE-High concentrations of circulating glucose are believed to contribute to defective insulin secretion and beta-cell function in diabetes and at least... 
BCL-2 | OXIDATIVE STRESS | INSULIN-RESISTANCE | BETA-CELLS | ENDOCRINOLOGY & METABOLISM | RECEPTOR | DEATH | TOXICITY | ENDOPLASMIC-RETICULUM STRESS | DEFICIENT MICE | FAILURE | bcl-2-Associated X Protein - chemistry | Apoptosis - drug effects | Receptors, Interleukin-1 - genetics | bcl-2 Homologous Antagonist-Killer Protein - genetics | Proto-Oncogene Proteins - chemistry | Diabetes Mellitus, Type 2 - metabolism | RNA, Messenger - metabolism | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Insulin-Secreting Cells - metabolism | Bcl-2-Like Protein 11 | Mice, Mutant Strains | Tumor Suppressor Proteins - chemistry | Tumor Suppressor Proteins - genetics | fas Receptor - genetics | Apoptosis Regulatory Proteins - genetics | Glucose - toxicity | Insulin-Secreting Cells - cytology | Membrane Proteins - metabolism | Stress, Physiological - drug effects | DNA Fragmentation - drug effects | bcl-2-Associated X Protein - genetics | Protein Structure, Tertiary | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Apoptosis Regulatory Proteins - chemistry | Cytochromes c - metabolism | Mice, Inbred C57BL | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Ribose - toxicity | Mitochondria - metabolism | Mitochondria - drug effects | Apoptosis Regulatory Proteins - metabolism | Animals | Insulin-Secreting Cells - drug effects | Membrane Proteins - chemistry | Mice | Apoptosis - physiology | Diabetes Mellitus, Type 2 - pathology | Proto-Oncogene Proteins c-bcl-2 - genetics | bcl-2 Homologous Antagonist-Killer Protein - chemistry | Cellular proteins | Care and treatment | Physiological aspects | Islands of Langerhans | Research | Diabetes | Health aspects | Apoptosis | Index Medicus | Abridged Index Medicus | Original
Journal Article
Journal Article
New Zealand Medical Journal, ISSN 0028-8446, 11/2016, Volume 129, Issue 1445, pp. 23 - 34
Characterises and evaluates the changes in high sensitivity cardiac troponin T (hsTnT) in incident haemodialysis patients. Source: National Library of New... 
Troponin I | Hemodialysis | Complications | Coronary Disease - blood | Prognosis | Prospective Studies | Humans | Middle Aged | Coronary Disease - etiology | Male | Biomarkers - blood | Kidney Failure, Chronic - therapy | Troponin T - blood | Time Factors | Renal Dialysis - adverse effects | Female | Aged | Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 05/2008, Volume 57, Issue 5, pp. 1284 - 1292
OBJECTIVE--Apoptosis of pancreatic [beta]-cells is critical in both diabetes development and failure of islet transplantation. The role in these processes of... 
Cellular proteins | Pancreatic beta cells | Influence | Research | Diabetes | Properties | Apoptosis
Journal Article