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Acta Neuropathologica, ISSN 0001-6322, 2013, Volume 126, Issue 4, pp. 461 - 477
  The identification of microglia-associated, neurological disease-causing mutations in patients, combined with studies in mouse models has highlighted... 
Activation | Alzheimer's disease | Phagocytosis | Cellular senescence | Microglia | Development and progression | Amyloid beta-protein
Journal Article
Journal Article
The EMBO Journal, ISSN 0261-4189, 02/2019, Volume 38, Issue 4
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2018, Volume 128, Issue 7, p. 2774
Journal Article
Alzheimer's & Dementia: The Journal of the Alzheimer's Association, ISSN 1552-5260, 2016, Volume 12, Issue 7, pp. P366 - P366
Journal Article
Alzheimer's & Dementia: The Journal of the Alzheimer's Association, ISSN 1552-5260, 07/2016, Volume 12, Issue 7, p. P366
Journal Article
Acta neuropathologica, 09/2016, Volume 132, Issue 3, p. 361
Diets high in fat (HFD) are known to cause an immune response in the periphery as well as the central nervous system. In peripheral adipose tissue, this immune... 
Microglia - metabolism | Animals | Central Nervous System - metabolism | Obesity - complications | Diet, High-Fat | Gliosis - metabolism | Mice, Inbred C57BL | Male | Neurons - metabolism | Animal Feed | Astrocytes - metabolism
Journal Article
Neuron, ISSN 0896-6273, 05/2013, Volume 78, Issue 4, pp. 575 - 577
Journal Article
Journal of Comparative Neurology, ISSN 0021-9967, 03/2016, Volume 524, Issue 4, pp. 783 - 806
Journal Article
Journal Article
Acta neuropathologica communications, ISSN 2051-5960, 06/2017, Volume 5, Issue 1, p. 52
Journal Article
Immunity, ISSN 1074-7613, 09/2017, Volume 47, Issue 3, pp. 566 - 581.e9
Microglia play a pivotal role in the maintenance of brain homeostasis but lose homeostatic function during neurodegenerative disorders. We identified a... 
Alzheimer’s disease | multiple sclerosis | transcriptional regulation | neurodegeneration | TREM2 | APOE | amyotrophic lateral sclerosis | microglia | Alzheimer's disease | GENE-EXPRESSION SIGNATURE | ACTIVATION | MULTIPLE-SCLEROSIS | ALZHEIMERS-DISEASE | MOUSE MODEL | MACROPHAGE | MICE | IMMUNOLOGY | DEFICIENCY | APOLIPOPROTEIN-E | CELL-DEATH | Microglia - metabolism | Apolipoproteins E - deficiency | Membrane Glycoproteins - metabolism | Humans | Cerebral Cortex - pathology | Transcriptome | Apoptosis - genetics | Monocytes - metabolism | Gene Expression Profiling | Monocytes - immunology | Phagocytosis - genetics | Neurodegenerative Diseases - immunology | Apolipoproteins E - metabolism | Cerebral Cortex - metabolism | Alzheimer Disease - pathology | Microglia - immunology | Superoxide Dismutase-1 - metabolism | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | Female | Neurons - metabolism | Disease Models, Animal | Gene Targeting | Plaque, Amyloid - pathology | Signal Transduction | Gene Expression Regulation | Phagocytosis - immunology | Immune Tolerance | Mice, Transgenic | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - metabolism | Mice, Knockout | Encephalomyelitis, Autoimmune, Experimental | Phenotype | Animals | Apoptosis - immunology | Apolipoproteins E - genetics | Alzheimer Disease - metabolism | Superoxide Dismutase-1 - genetics | Plaque, Amyloid - metabolism | Mice | Alzheimer Disease - genetics | Transforming Growth Factor beta - metabolism | Receptors, Immunologic - metabolism | Cluster Analysis | Nervous system diseases | Multiple sclerosis | Neurons | Analysis | Amyotrophic lateral sclerosis | Genetic aspects | Genetic transcription | Apolipoproteins | Brain | Animal models | Myeloid cells | Disease | Transcription | Neurodegenerative diseases | Genes | Homeostasis | Microglia | Neurological diseases | Proteins | Molecular modelling | Restoration | Neurodegeneration | Apolipoprotein E | Rodents | β-Amyloid | Plaques | Phagocytosis | Apoptosis
Journal Article
ACTA NEUROPATHOLOGICA COMMUNICATIONS, ISSN 2051-5960, 08/2019, Volume 7, Issue 1
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2009, Volume 119, Issue 1, pp. 61 - 69
The clear association of Th17 cells with autoimmune pathogenicity implicates Th17 cytokines as critical mediators of chronic autoimmune diseases such as EAE.... 
ENCEPHALOMYELITIS | MEDICINE, RESEARCH & EXPERIMENTAL | TGF-BETA | MULTIPLE-SCLEROSIS | INTERLEUKIN-17 FAMILY | IFN-GAMMA | RECEPTOR COMPLEX | CENTRAL-NERVOUS-SYSTEM | T-CELLS | TH17 CELLS | CUTTING EDGE | Interleukins | Physiological aspects | Development and progression | Research | Autoimmune diseases | T cells | Health aspects | Risk factors
Journal Article