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Blood, ISSN 0006-4971, 01/2010, Volume 115, Issue 2, pp. 353 - 362
Alternatively activated macrophages play an important role in host defense in the context of a T helper type 2 (Th2) microenvironment such as parasitic... 
IN-VITRO | INTERLEUKIN (IL)-4 | FACTOR-ALPHA PRODUCTION | IFN-GAMMA | NEISSERIA-MENINGITIDIS | MURINE MACROPHAGES | MONONUCLEAR-CELLS | SCAVENGER RECEPTOR MARCO | MONOCLONAL-ANTIBODY | INFECTION | HEMATOLOGY | Interleukin-13 - immunology | Th2 Cells - immunology | Proto-Oncogene Proteins c-akt - genetics | Interleukin-13 - genetics | Interleukin-4 - pharmacology | Time Factors | Myeloid Differentiation Factor 88 - immunology | Neisseria meningitidis, Serogroup B | Phosphorylation - immunology | Phosphorylation - drug effects | Receptors, Immunologic - immunology | Proto-Oncogene Proteins c-akt - metabolism | Signal Transduction | Myeloid Differentiation Factor 88 - genetics | Macrophages, Peritoneal - immunology | Receptors, Cell Surface - metabolism | Interleukin-13 - metabolism | Receptors, Cell Surface - immunology | Th2 Cells - metabolism | Macrophage Activation | Mice, Knockout | Interleukin-4 - immunology | Interleukin-4 - secretion | Animals | Proto-Oncogene Proteins c-akt - immunology | Mice | Macrophages, Peritoneal - metabolism | Receptors, Immunologic - genetics | Myeloid Differentiation Factor 88 - metabolism | Phagocytosis | Receptors, Immunologic - metabolism | Receptors, Cell Surface - genetics | Index Medicus | Abridged Index Medicus | Interleukin-4 | Phosphorylation | Macrophages, Peritoneal | Interleukin-13 | Receptors, Cell Surface | Myeloid Differentiation Factor 88 | Th2 Cells | Proto-Oncogene Proteins c-akt | Life Sciences | Immunology | Receptors, Immunologic | Phagocytes, Granulocytes, and Myelopoiesis
Journal Article
Current Drug Targets, ISSN 1389-4501, 2016, Volume 17, Issue 1, pp. 15 - 22
Highly active antiretroviral therapy (HAART) has dramatically extended the lifespan and quality of life of individuals infected with human immunodeficiency... 
HIV-1 | TNF-alpha | Tat | Reservoirs | Vpr | Gp120 | Nef | HAART | RHEUMATOID-ARTHRITIS | CD4(+) T-LYMPHOCYTES | reservoirs | MONOCLONAL-ANTIBODY | TUMOR-NECROSIS-FACTOR | ACTIVE ANTIRETROVIRAL THERAPY | IMMUNODEFICIENCY-VIRUS TYPE-1 | EPIGENETIC REGULATION | gp120 | PHARMACOLOGY & PHARMACY | PRIMARY HUMAN MACROPHAGES | PERIPHERAL-BLOOD MONOCYTES | NF-KAPPA-B
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e59591 - e59591
Objectives: There has been increased interest in the possible role of human cytomegalovirus (HCMV) in carcinogenesis during the last decade. HCMV... 
CANCER-CELLS | HEPATOCELLULAR-CARCINOMA CELLS | LIVER INFLAMMATION | STAT3 ACTIVATION | CYCLIN D1 EXPRESSION | MULTIDISCIPLINARY SCIENCES | HUMAN CYTOMEGALOVIRUS | THERAPEUTIC TARGET | ENDOTHELIAL-CELLS | GENE-EXPRESSION | NF-KAPPA-B | Cyclin D1 - metabolism | Up-Regulation | Cell Proliferation | Humans | Hepatocytes - pathology | Janus Kinases - metabolism | Hepatocytes - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Liver Neoplasms - pathology | Inhibitor of Apoptosis Proteins - metabolism | Cytomegalovirus - physiology | Hepatocytes - secretion | Interleukin-6 - metabolism | STAT3 Transcription Factor - metabolism | Liver Neoplasms - virology | Tumor Suppressor Protein p53 - metabolism | Carcinoma, Hepatocellular - virology | Interleukin-6 - secretion | Hep G2 Cells | Animals | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Hepatocytes - virology | Mice | Enzyme Activation | Carcinoma, Hepatocellular - metabolism | Infection | Medical research | Medicine, Experimental | Development and progression | Cytomegalovirus infections | Tumor proteins | Health aspects | Enzyme-linked immunosorbent assay | Cell proliferation | Cell culture | GTP-binding protein | Transformation | Laboratories | p53 Protein | Hepatocellular carcinoma | Viruses | Infections | Cyclin D1 | Kinases | Carcinogenesis | Western blotting | Interleukin 6 | Liver cancer | Angiogenesis | Hepatitis | Carcinogens | Penicillin | Cell cycle | Fibroblasts | Pathogens | Cytomegalovirus | MDM2 protein | Immunoglobulins | Liver diseases | Colonies | Stat3 protein | Agar | Cultures | Inflammation | Survivin | Virology | Cyclin-dependent kinase inhibitor p21 | Hepatocytes | Stem cells | Transformations | Viral infections | Index Medicus
Journal Article
Viruses, ISSN 1999-4915, 04/2018, Volume 10, Issue 4, p. 190
Akt signaling plays a central role in many biological processes that are key players in human immunodeficiency virus 1 (HIV-1) pathogenesis. The persistence of... 
HIV-1 | Resting CD4+ T cells | Monocyte | NNRTI | Reservoir | Akt | cART | Protease inhibitor | protease inhibitor | MACROPHAGES | CD8(+) T-CELLS | NEF | NELFINAVIR | resting CD4+T cells | REPLICATION | VIROLOGY | CD4(+) | monocyte | INFECTION | reservoir | LATENT RESERVOIR | NF-KAPPA-B | EXPRESSION | Index Medicus | resting CD4+ T cells
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 12574 - 14
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2009, Volume 4, Issue 6, pp. e6093 - e6093
The persistence of transcriptionally silent but replication-competent HIV-1 reservoirs in Highly Active Anti-Retroviral Therapy (HAART)-treated infected... 
MULTIDISCIPLINARY SCIENCES | Anti-HIV Agents - pharmacology | HIV-1 - metabolism | NF-KappaB Inhibitor alpha | NF-kappa B - antagonists & inhibitors | Humans | Middle Aged | Enzyme Inhibitors - pharmacology | Nucleosomes - metabolism | Cytoplasm - metabolism | I-kappa B Proteins - metabolism | Virus Latency - drug effects | Drug Synergism | Monocytes - virology | Cell Nucleus - metabolism | CD8-Positive T-Lymphocytes - metabolism | Phorbol Esters - pharmacology | Adult | HIV Infections - drug therapy | HIV-1 - enzymology | Aged | Highly active antiretroviral therapy | Antiviral agents | Control | Physiological aspects | Genetic research | Genetic transcription | T cells | HIV (Viruses) | Health aspects | Therapy | Oxidative stress | Histone deacetylase | Transcription | Laboratories | CD8 antigen | Leukocytes (mononuclear) | Viruses | Lymphocytes T | Infections | Activation | Remodeling | DNA-directed RNA polymerase | Synergism | Recruitment | Long terminal repeat | Cell growth | Transfection | Acquired immune deficiency syndrome--AIDS | Lymphocytes | Human immunodeficiency virus--HIV | Peripheral blood mononuclear cells | Elongation | Deoxyribonucleic acid--DNA | NF-κB protein | Latent infection | Gene expression | Patients | Virology | CD4 antigen | Polymerase | Monocytes | Inhibitors | Ribonucleic acids | Replication | Reservoirs | Combined treatment | RNA polymerase II | Index Medicus | HIV Infections | HIV-1 | Enzyme Inhibitors | Virus Latency | I-kappa B Proteins | Anti-HIV Agents | CD8-Positive T-Lymphocytes | Life Sciences | Microbiology and Parasitology | NF-kappa B | Nucleosomes | Phorbol Esters | Cell Nucleus | Cytoplasm | Acquired immune deficiency syndrome | Deoxyribonucleic acid | AIDS | HIV | DNA | Human immunodeficiency virus
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, pp. e70939 - e70939
Journal Article
The open virology journal, ISSN 1874-3579, 2013, Volume 7, Issue 1, pp. 57 - 71
HIV exploits the T-cell signaling network to gain access to downstream cellular components, which serves as effective tools to break the cellular barriers.... 
HIV-1 | reservoirs | T cells | viral proteins | Nef | Apoptosis
Journal Article
Journal Article