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Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 12/2013, Volume 17, Issue 12, pp. 1543 - 1553
The enhancement of endogenous angiogenesis after stroke will be critical in neurorepair therapies where endothelial progenitor cells (EPCs) might be key... 
endothelial progenitor cell | matrix metalloproteinase‐9 | neurorepair | cerebral ischaemia | stroke | angiogenesis | Matrix metalloproteinase-9 | Angiogenesis | Stroke | Cerebral ischaemia | Endothelial progenitor cell | Neurorepair | MEDICINE, RESEARCH & EXPERIMENTAL | matrix metalloproteinase-9 | MOBILIZATION | THERAPEUTIC NEOVASCULARIZATION | TRANSPLANTATION | STEM | CELL BIOLOGY | MATRIX METALLOPROTEINASES | RESPONSES | NEUROGENESIS | TEMPORAL PROFILE | EXPRESSION | Neovascularization, Physiologic - drug effects | Endothelial Cells - metabolism | Cell Count | Humans | Proteoglycans - metabolism | Immunophenotyping | Male | Brain Ischemia - physiopathology | Matrix Metalloproteinase 9 - deficiency | Stem Cells - metabolism | Brain Ischemia - enzymology | Collagen - metabolism | Matrix Metalloproteinase Inhibitors - pharmacology | Matrix Metalloproteinase 9 - metabolism | Animals | Brain Ischemia - pathology | Stem Cells - drug effects | Stem Cells - pathology | Mice | Laminin - metabolism | Endothelial Cells - pathology | Drug Combinations | Endothelial Cells - drug effects | Fibronectins | Ischemia | Endothelium | Occlusion | Cell culture | Networks | Network formation | Matrix metalloproteinases | Blood vessels | Matrix metalloproteinase | Progenitor cells | Endothelial cells | Gelatinase B | Studies | Hypotheses | Inhibitors | Cerebral blood flow | Rodents | Morphology | Stem cells | Metalloproteinase | Veins & arteries | Original
Journal Article
by Al-Shahi Salman, Rustam and Frantzias, Joseph and Lee, Robert J and Lyden, Patrick D and Battey, Thomas W K and Ayres, Alison M and Goldstein, Joshua N and Mayer, Stephan A and Steiner, Thorsten and Wang, Xia and Arima, Hisatomi and Hasegawa, Hitoshi and Oishi, Makoto and Godoy, Daniel A and Masotti, Luca and Dowlatshahi, Dar and Rodriguez-Luna, David and Molina, Carlos A and Jang, Dong-Kyu and Davalos, Antonio and Castillo, José and Yao, Xiaoying and Claassen, Jan and Volbers, Bastian and Kazui, Seiji and Okada, Yasushi and Fujimoto, Shigeru and Toyoda, Kazunori and Li, Qi and Khoury, Jane and Delgado, Pilar and Sabín, José Álvarez and Hernández-Guillamon, Mar and Prats-Sánchez, Luis and Cai, Chunyan and Kate, Mahesh P and McCourt, Rebecca and Venkatasubramanian, Chitra and Diringer, Michael N and Ikeda, Yukio and Worthmann, Hans and Ziai, Wendy C and d'Esterre, Christopher D and Aviv, Richard I and Raab, Peter and Murai, Yasuo and Zazulia, Allyson R and Butcher, Kenneth S and Seyedsaadat, Seyed Mohammad and Grotta, James C and Martí-Fàbregas, Joan and Montaner, Joan and Broderick, Joseph and Yamamoto, Haruko and Staykov, Dimitre and Connolly, E Sander and Selim, Magdy and Leira, Rogelio and Moon, Byung Hoo and Demchuk, Andrew M and Di Napoli, Mario and Fujii, Yukihiko and Anderson, Craig S and Rosand, Jonathan and Hanley, Daniel F and Butcher, Kenneth S and Davis, Stephen and Gregson, Barbara and Lees, Kennedy R and Lyden, Patrick D and Mayer, Stephan A and Muir, Keith W and Steiner, Thorsten and Xie, Peng and Bakhshayesh, Babak and McDonald, Mark and Brott, Thomas and Pennati, Paolo and Parry-Jones, Adrian R and Smith, Craig J and Hopkins, Stephen J and Slevin, Mark and Campi, Veronica and Singh, Puneetpal and Papa, Francesca and Popa-Wagner, Aurel and Tudorica, Valeria and Takagi, Ryo and Teramoto, Akira and Weissenborn, Karin and Lanfermann, Heinrich and ICH Growth Individual Patient Data Meta-analysis Collaborators and VISTA-ICH Collaboration
The Lancet Neurology, ISSN 1474-4422, 10/2018, Volume 17, Issue 10, pp. 885 - 894
Journal Article
Stroke, ISSN 0039-2499, 04/2008, Volume 39, Issue 4, pp. 1121 - 1126
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2013, Volume 8, Issue 9, p. e73244
Journal Article
Atherosclerosis, ISSN 0021-9150, 2011, Volume 216, Issue 1, pp. 205 - 211
Journal Article
International Journal of Nanomedicine, ISSN 1176-9114, 06/2016, Volume 11, pp. 3035 - 3048
Journal Article
Frontiers in Neurology, ISSN 1664-2295, 2019, Volume 10, p. 187
Cerebral amyloid angiopathy (CAA) is a common cause of lobar intracerebral hemorrhage (ICH) in elderly individuals and it is the result of the cerebrovascular... 
Cerebral amyloid angiopathy | ApoA-I | ApoJ | ApoE | Intracerebral hemorrhage | Clusterin | β-amyloid | clusterin | PROTEIN | HEMORRHAGE | ALZHEIMERS-DISEASE | DEPOSITION | CEREBROSPINAL-FLUID | NEUROSCIENCES | CLINICAL NEUROLOGY | BETA | beta-amyloid | intracerebral hemorrhage | TRANSPORT | cerebral amyloid angiopathy | EXPRESSION | APOLIPOPROTEIN-A-I | Amyloidosis | Genetic aspects | Research | Gene expression | Apolipoproteins
Journal Article