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PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, pp. e0130624 - e0130624
Neuroinflammation is the local reaction of the brain to infection, trauma, toxic molecules or protein aggregates. The brain resident macrophages, microglia,... 
INTERLEUKIN-1 | ACTIVATION | MOLECULAR PLATFORM | INHIBITION | DISTINCT PATHWAYS | NEUROINFLAMMATION | MULTIDISCIPLINARY SCIENCES | IL-1-BETA | MECHANISMS | RECEPTORS | NALP3 INFLAMMASOME | Microglia - metabolism | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Peptide Fragments - toxicity | Caspase 1 - metabolism | Interleukin-1alpha - metabolism | alpha-Synuclein - pharmacology | Interleukin-1beta - metabolism | Interleukin-1beta - secretion | Microglia - cytology | Brain - cytology | Interleukin-1beta - analysis | Enzyme-Linked Immunosorbent Assay | Microglia - drug effects | Amyloid beta-Peptides - toxicity | Mice, Inbred C57BL | Cells, Cultured | Mice, Knockout | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Caspase 1 - genetics | Caspase 1 - deficiency | Receptors, Purinergic P2X7 - metabolism | Mice | Interleukin-18 - metabolism | Astrocytes - metabolism | Cytokines | Health aspects | Nervous system diseases | Brain | Cell culture | Traumatic brain injury | Peptides | Aluminum sulfate | Homeostasis | Nervous system | Activation | Macrophages | Synuclein | Proteins | Rodents | Amyloid | Life sciences | Communication | Immune system | Neurodegenerative diseases | Astrocytes | Inflammation | Trauma | Molecular chains | Microglia | Interleukin 18 | Mode of action | Neurological diseases | Nigericin | Brain research | Ligands | Alum | Laboratory animals | Alzheimers disease | Chemokines | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2016, Volume 11, Issue 9, p. e0162717
Parkinson's disease (PD) is histologically described by the deposition of alpha-synuclein, whose accumulation in Lewy bodies causes dopaminergic neuronal... 
SIGNALING PATHWAYS | OXIDATIVE STRESS | ACTIVATES MICROGLIA | THP-1 CELLS | MULTIDISCIPLINARY SCIENCES | THERAPEUTIC TARGET | MUTATION | NORMAL MOUSE-BRAIN | FAMILIAL PARKINSONS-DISEASE | NF-KAPPA-B | NEURODEGENERATIVE DISEASES | Microglia - metabolism | Gene Expression | Parkinson Disease - pathology | Reactive Oxygen Species - metabolism | Signal Transduction | Humans | Cells, Cultured | Mutant Proteins - genetics | Mutant Proteins - metabolism | Parkinson Disease - genetics | Point Mutation | Inflammation - metabolism | Animals | Inflammation Mediators - metabolism | Inflammation - genetics | Microglia - pathology | Mice | Parkinson Disease - metabolism | alpha-Synuclein - genetics | alpha-Synuclein - metabolism | Amino Acid Substitution | Gene mutations | Genetic aspects | Cell culture | Oxidative stress | Phosphorylation | Neurosciences | Transcription factors | Parkinson's disease | Disease | Laboratories | Neurobiology | Activation | Kinases | Synuclein | Proteins | Antioxidants | Stimulators | Neurodegeneration | Cascades | Tumor necrosis factor-TNF | Physiology | Life sciences | Degeneration | Movement disorders | Communication | Dopamine receptors | NF-κB protein | Dopamine | Neurodegenerative diseases | Neurons | Inflammation | Mutants | Microglia | Lewy bodies | Signaling | Brain research | Reactivity | Mutation
Journal Article
Journal of Neuroimmunology, ISSN 0165-5728, 2009, Volume 210, Issue 1, pp. 3 - 12
Abstract M1 and M2 are the extremes of the differentiation spectrum of activated macrophages. Since microglia are members of the same cell lineage, we have... 
Neurology | Allergy and Immunology | | Signature genes | Alternative activation | Phagocytosis | Notch | Microglia | CELLS | ADULT-MOUSE BRAIN | ALZHEIMERS-DISEASE | IFN-GAMMA | AGED MICE | IMMUNOLOGY | HUMAN MONOCYTES | NEUROSCIENCES | MACROPHAGE ACTIVATION | A beta | IN-VIVO | PPAR-GAMMA | Microglia - metabolism | Molecular Weight | Receptors, Notch - metabolism | Gliosis - chemically induced | Gene Expression Profiling | Gliosis - physiopathology | Cell Lineage - drug effects | Alzheimer Disease - pathology | Gliosis - pathology | Chemotaxis - physiology | Encephalitis - physiopathology | Cytokines - toxicity | Amyloid beta-Peptides - metabolism | Inflammation Mediators - metabolism | Cell Differentiation - physiology | Cytokines - genetics | Receptors, Notch - drug effects | Microglia - cytology | Cell Line | Alzheimer Disease - physiopathology | Cytokines - metabolism | Microglia - drug effects | Amyloid beta-Peptides - toxicity | Mice, Inbred C57BL | Cells, Cultured | Inflammation Mediators - toxicity | Encephalitis - pathology | Gene Expression Regulation - physiology | Chemotaxis - drug effects | Cell Lineage - physiology | Gene Expression Regulation - drug effects | Phenotype | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Alzheimer Disease - metabolism | Signal Transduction - physiology | Mice | Encephalitis - chemically induced | Index Medicus
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 07/2010, Volume 114, Issue 2, pp. 576 - 586
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 10/2016, Volume 53, Issue 8, pp. 5041 - 5055
Journal Article
Genomics Data, ISSN 2213-5960, 03/2016, Volume 7, pp. 7 - 11
Astrocytes, the most abundant glial cell population in the central nervous system, have important functional roles in the brain as blood brain barrier... 
Primary astrocytes | Inflammation | Gene expression | Microarrays
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 10/2016, Volume 53, Issue 8, p. 5041
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s12035-015-9428-3 Brain... 
Glucose metabolism | Nervous system diseases | Neurons | Glycogen | Stem cells | Inflammation | Intermediate filament proteins
Journal Article
Journal Article
Glia, ISSN 0894-1491, 12/2009, Volume 57, Issue 16, pp. 1741 - 1753
The Notch pathway is implicated in many aspects of the central nervous system (CNS) development and functions. Recently, we and others identified the Notch... 
Hes1 | gliosis | inflammation | Notch | Inflammation | Gliosis | PROGENITOR CELLS | BINDING PROTEIN | NEUROSCIENCES | CROSS-TALK | IMMUNOREACTIVE PHENOTYPE | FIBRILLARY ACIDIC PROTEIN | GENE-EXPRESSION | NOTCH SIGNALING PATHWAY | REACTIVE GLIOSIS | REGIONAL HETEROGENEITY | BRAIN
Journal Article
Journal Article
Journal of Neuroimmunology, ISSN 0165-5728, 05/2009, Volume 210, Issue 1-2, p. 3
M1 and M2 are the extremes of the differentiation spectrum of activated macrophages. Since microglia are members of the same cell lineage, we have... 
Peptides | Anti-inflammatory drugs | Analysis | Genetic aspects
Journal Article