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Frontiers in Pharmacology, ISSN 1663-9812, 06/2017, Volume 8, pp. 368 - 368
Endometrial cancer (EC) is the most common estrogen-dependent gynecological malignancy in the developed World. To investigate the local formation of estradiol... 
Sulfotransferases | Sulfatase | Aldo-keto reductase 1C3 | 17β-hydroxysteroid dehydrogenases | Aromatase | aromatase | sulfotransferases | sulfatase | aldo-keto reductase 1C3
Journal Article
Chemico-Biological Interactions, ISSN 0009-2797, 06/2015, Volume 234, pp. 309 - 319
Estrogens have important roles in the pathogenesis of endometrial cancer. They can have carcinogenic effects through stimulation of cell proliferation or... 
Aromatase pathway | Sulfatase pathway | Phase I and phase II estrogen metabolism | Aldo–keto reductase 1C3 (AKR1C3) | 17β-Hydroxysteroid dehydrogenases (17β-HSDs, HSD17B) | Catechol-O-methyl transferase (COMT) | 17b-Hydroxysteroid dehydrogenases | (17b-HSDs HSD17B)Aldoketo reductase 1C3 (AKR1C3) | 17 beta-Hydroxysteroid dehydrogenases (17 beta-HSDs, HSD17B) | DEHYDROGENASES | QUINONES | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROGESTERONE ACTION | 17-BETA-ESTRADIOL | CARCINOGENESIS | OVARIAN ENDOMETRIOSIS | Aldo-keto reductase 1C3 (AKR1C3) | ENZYMES | PHARMACOLOGY & PHARMACY | TOXICOLOGY | PHASE-I | CARCINOMA | PRE-RECEPTOR REGULATION | Sulfatases - genetics | Progesterone Reductase - metabolism | Humans | Endometrial Neoplasms - metabolism | Aromatase - metabolism | Catechol O-Methyltransferase - genetics | Hydroxyprostaglandin Dehydrogenases - metabolism | Androstenedione - metabolism | Catechol O-Methyltransferase - metabolism | Aromatase - genetics | Endometrial Neoplasms - genetics | Sulfatases - metabolism | Estrogens - genetics | 3-Hydroxysteroid Dehydrogenases - metabolism | Female | Sulfotransferases - metabolism | Estrogens - metabolism | Quinones - pharmacology | Glutathione S-Transferase pi - genetics | Sulfotransferases - genetics | Estrone - genetics | Quinone Reductases - metabolism | Androstenedione - genetics | Glutathione S-Transferase pi - metabolism | Estrogens - biosynthesis | RNA, Messenger - genetics | Transcriptome - genetics | Progesterone Reductase - genetics | Arylsulfotransferase - genetics | Hydroxyprostaglandin Dehydrogenases - genetics | Estrone - analogs & derivatives | Aldo-Keto Reductase Family 1 Member C3 | Estrone - metabolism | Cell Line, Tumor | Quinone Reductases - genetics | 3-Hydroxysteroid Dehydrogenases - genetics | Arylsulfotransferase - metabolism | Physiological aspects | Endometrial cancer | Sulfates | Genes | Estrogen | Steroids | Index Medicus
Journal Article
Chemico-Biological Interactions, ISSN 0009-2797, 06/2015, Volume 234, p. 320
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 2011, Volume 331, Issue 1, pp. 158 - 167
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 03/2013, Volume 367, Issue 1-2, pp. 74 - 84
► Expression of , , and MB-COMT levels were higher in endometriosis. ► Expression of , , , , and was decreased. ► Three NQO1 isoforms were detected; NQO1c... 
Glutathione S-transferase pi 1 (GSTP1) | (NRH):quinone oxidoreductase 2 (NQO2) | Sulfotransferases (SULTs) | NAD(P)H:quinone oxidoreductase 1 (NQO1) | Cytochrome P450 (CYP) | Catechol-O-methyltransferase (COMT) | ORTHO-QUINONES | CELLS | OXIDATIVE STRESS | HUMAN-BREAST-CANCER | RECEPTOR | CYTOCHROME-P450 ISOFORMS | ESTRONE | 17-BETA-ESTRADIOL | CELL BIOLOGY | CATECHOL-O-METHYLTRANSFERASE | ENDOCRINOLOGY & METABOLISM | EXPRESSION | Immunohistochemistry | Cytochrome P-450 CYP1A1 - genetics | Steroid Hydroxylases - metabolism | Reactive Oxygen Species - metabolism | Endometriosis - genetics | Humans | Aryl Hydrocarbon Hydroxylases - genetics | Gene Expression Profiling | NAD(P)H Dehydrogenase (Quinone) - genetics | Steroid 16-alpha-Hydroxylase - metabolism | Catechol O-Methyltransferase - genetics | Case-Control Studies | Young Adult | Catechol O-Methyltransferase - metabolism | Endometriosis - enzymology | Estrogens - chemistry | Isoenzymes - metabolism | Metabolic Detoxication, Phase II - genetics | Adult | Female | Metabolic Detoxication, Phase I - genetics | Estrogens - metabolism | Steroid Hydroxylases - genetics | Glutathione S-Transferase pi - genetics | Glutathione S-Transferase pi - metabolism | Ovary - pathology | Up-Regulation - genetics | Cytochrome P-450 CYP1A1 - metabolism | Blotting, Western | Aryl Hydrocarbon Hydroxylases - metabolism | Gene Expression Regulation, Enzymologic | Ovary - enzymology | Cytochrome P-450 CYP1B1 | NAD(P)H Dehydrogenase (Quinone) - metabolism | Steroid 16-alpha-Hydroxylase - genetics | Endometriosis | Thiols | Glutathione transferase | Quinone | Cytochrome P-450 | Index Medicus
Journal Article
Journal Article
Zdravniski Vestnik, ISSN 1318-0347, 2014, Volume 83, Issue 7, pp. 529 - 537
  Increased local concentrations of estrogens are a well-known risk factor for endometrial cancer development. Estrogens can act as mitogens via estrogen... 
Catechol estrogens | Steroid sulfatase | Estrogen quinones | Aromatase | Estrogen metabolism | Endometrial cancer | Metabolites | Deoxyribonucleic acid--DNA | Estrogen
Journal Article
Chemico-Biological Interactions, ISSN 0009-2797, 02/2013, Volume 202, Issue 1-3, pp. 210 - 217
► In endometrial cancer progesterone biosynthesis genes are down-regulated. ► Expression of most progesterone metabolism genes is not altered in cancerous... 
Steroidogenic acute regulatory protein | 5α-Reductase | Pre-receptor metabolism | Side-chain cleavage enzyme | 3-Keto/20-keto-reductase | 3-ALPHA-HYDROXYSTEROID DEHYDROGENASES | ADENOCARCINOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | STEROID 5-ALPHA-REDUCTASE | RECEPTOR-A | 5 alpha-Reductase | ER-ALPHA | 17-BETA-HYDROXYSTEROID-DEHYDROGENASE | KETO REDUCTASE SUPERFAMILY | ESTROGEN | PHARMACOLOGY & PHARMACY | TOXICOLOGY | YOUNG-WOMEN | TYPE-2 | Cholesterol Side-Chain Cleavage Enzyme - metabolism | Progesterone Reductase - metabolism | Humans | Middle Aged | Endometrial Neoplasms - metabolism | Multienzyme Complexes - metabolism | Receptors, Progesterone - genetics | Hydroxyprostaglandin Dehydrogenases - metabolism | Receptors, Progesterone - metabolism | Cholesterol Side-Chain Cleavage Enzyme - genetics | Endometrial Neoplasms - genetics | Phosphoproteins | 3-Oxo-5-alpha-Steroid 4-Dehydrogenase - metabolism | Aged, 80 and over | 3-Hydroxysteroid Dehydrogenases - metabolism | Adult | Female | Progesterone - biosynthesis | Membrane Proteins - metabolism | Steroid Isomerases - genetics | Endometrium - metabolism | 20-Hydroxysteroid Dehydrogenases - genetics | Membrane Proteins - genetics | Steroid Isomerases - metabolism | RNA, Messenger - genetics | Multienzyme Complexes - genetics | Progesterone Reductase - genetics | 20-Hydroxysteroid Dehydrogenases - metabolism | Down-Regulation - genetics | Hydroxyprostaglandin Dehydrogenases - genetics | Aldo-Keto Reductase Family 1 Member C3 | Estradiol Dehydrogenases - genetics | 17-Hydroxysteroid Dehydrogenases - genetics | 3-Oxo-5-alpha-Steroid 4-Dehydrogenase - genetics | Estradiol Dehydrogenases - metabolism | Progesterone - metabolism | Aged | Progesterone - genetics | 3-Hydroxysteroid Dehydrogenases - genetics | 17-Hydroxysteroid Dehydrogenases - metabolism | Endometrial cancer | RNA | Genes | Cytochrome P-450 | Physiological aspects | Genetic aspects | Progesterone | Cancer | Index Medicus
Journal Article
Zdravniški Vestnik, ISSN 1318-0347, 08/2014, Volume 83, Issue 7-8
Increased local concentrations of estrogens are a well-known risk factor for endometrial cancer. Estrogens can act as mitogens via the estrogen receptors, and... 
aromatase, steroid sulfatase, catechol estrogens, estrogen quinones, estrogen metabolism
Journal Article
Journal Article