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Journal of the American College of Cardiology, ISSN 0735-1097, 2016, Volume 68, Issue 21, pp. 2348 - 2364
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2014, Volume 63, Issue 24, pp. 2734 - 2741
Objectives This study sought to investigate the effect of endothelial dysfunction on the development of cardiac hypertrophy and fibrosis. Background... 
Cardiovascular | Internal Medicine | endothelial-mesenchymal transition | diastolic dysfunction | endothelium | NADPH (nicotinamide adenine dinucleotide phosphate) oxidase | angiotensin II | CELLS | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | HEART-FAILURE | ATHEROSCLEROSIS | ANGIOTENSIN-II | PATHOPHYSIOLOGY | HYPERTROPHY | MICE | CONTRIBUTES | Heart Failure, Diastolic - genetics | Mesenchymal Stromal Cells - enzymology | Humans | Cardiomegaly - pathology | Male | Endothelium, Vascular - enzymology | Membrane Glycoproteins - physiology | Ventricular Dysfunction, Left - genetics | Ventricular Dysfunction, Left - pathology | NADPH Oxidases - genetics | Ventricular Dysfunction, Left - enzymology | Inflammation Mediators - physiology | Fibrosis - genetics | Fibrosis - enzymology | Cells, Cultured | Heart Failure, Diastolic - enzymology | Mice, Transgenic | Cardiomegaly - enzymology | Heart Failure, Diastolic - pathology | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Animals | Endothelium, Vascular - pathology | Mice | Fibrosis - pathology | Mesenchymal Stromal Cells - pathology | Cardiomegaly - genetics | NADPH Oxidases - physiology | Oxidases | Heart | Fibrosis | Stem cells | Endothelium | Hypertension | Heart failure | Genotype & phenotype | Stroke | Heart attacks | Pathogenesis | Rodents | Cardiomyocytes | Variance analysis
Journal Article
Scientific reports, ISSN 2045-2322, 2019, Volume 9, Issue 1, pp. 6055 - 15
..., Marc van Bilsen1,3 and stephane Heymans1,4,5 Heart failure (HF) is the leading cause of death in the Western world. Pathophysiological processes underlying HF... 
FIBROSIS | APOPTOSIS | ACTIVATION | MACROPHAGES | CARDIAC-HYPERTROPHY | THERAPEUTIC TARGET | EXPRESSION PROFILES | DYSFUNCTION | POLARIZATION | FAMILY | MULTIDISCIPLINARY SCIENCES | Heart failure | Polarization | Transcription | Cardiomyocytes | Inflammation | Macrophages | Chromosome 3 | Coronary artery disease | Inflammatory diseases | Cell activation | Collagen | MicroRNAs | Fibrosis | Fibroblasts | miRNA | Heart diseases | Hypertrophy
Journal Article
Journal Article
Journal Article
Basic research in cardiology, ISSN 1435-1803, 2017, Volume 112, Issue 4, pp. 1 - 13
... • Mark Schellings 1 • Maarten Vanhaverbeke 2 • Johan Verjans 4,5 • Ilona Cuijpers 1,2 • Stephane Heymans 1,2,3 • Anna Papageorgiou 1,2 Received: 20 January 2017... 
Myocardial infarction | Heart failure | ANGIOGENESIS | POSTMYOCARDIAL INFARCTION | MACROPHAGES | Semaphorin3A | PHENOTYPE | MONOCYTES | Inflammation | VASCULAR-PERMEABILITY | SEMAPHORIN 3A | REPAIR | THERAPY | STEM-CELL | Medicine & Public Health | Cardiology | CARDIAC & CARDIOVASCULAR SYSTEMS | Chemotaxis, Leukocyte | Myocardial Infarction - genetics | Male | Monocytes - metabolism | Semaphorin-3A - metabolism | Wound Healing | Recovery of Function | Semaphorin-3A - deficiency | Time Factors | Myocardial Infarction - pathology | Myocardium - metabolism | Female | Myocardial Infarction - physiopathology | Disease Models, Animal | Myocarditis - genetics | Semaphorin-3A - genetics | Macrophages - pathology | Signal Transduction | Cells, Cultured | Myocardium - pathology | Myocardial Infarction - metabolism | Macrophage Activation | Mice, Knockout | Macrophages - metabolism | Phenotype | Animals | Myocarditis - metabolism | Heterozygote | Myocarditis - pathology | Myocarditis - physiopathology | Apoptosis | Heart | Macrophages | Heart attack | Polarization | Heart attacks | Leukocyte migration | CX3CR1 protein | Myocardial ischemia | Leukocytes | Tissues | Optimization | Recruitment | Ischemia | Rodents | Bone marrow | Heart diseases | Genotypes | Deposition | Wound healing | Patients | Adhesion | Monocytes | Semaphorins | Collagen | Myocardium | Infarction | Mice | Surface area | In vitro methods and tests | Original Contribution
Journal Article
Nature immunology, ISSN 1529-2916, 2015, Volume 16, Issue 4, pp. 415 - 425
Journal Article