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TRENDS IN CELL BIOLOGY, ISSN 0962-8924, 07/2019, Volume 29, Issue 7, pp. 569 - 579
Most human tumors are composed of genetically and phenotypically heterogeneous cancer cell populations, which poses a major challenge for the clinical... 
TARGETED THERAPY | CELLS | STOCHASTIC GENE-EXPRESSION | EVOLUTION | DNA METHYLATION | EPIGENETIC MECHANISM | RESISTANCE | HYPOXIA | CANCER | TUMOR HETEROGENEITY | CELL BIOLOGY | Epigenetic inheritance | Enzymes | Medical colleges | Chemotherapy | Lysine | Evolution | Genetic aspects | Cancer | Heterogeneity | Regulators | Chromatin | Profiling | Computer applications | Epigenetics | Software | Mutation | Tumors
Journal Article
Nature, ISSN 0028-0836, 08/2018, Volume 560, Issue 7718, pp. 325 - 330
Journal Article
Cancer Research, ISSN 0008-5472, 01/2018, Volume 78, Issue 1, pp. 205 - 215
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2016, Volume 113, Issue 22, pp. 6259 - 6264
Journal Article
Scientific Reports, ISSN 2045-2322, 05/2016, Volume 6, Issue 1, p. 26643
Aberrant expression of myeloid cell leukemia-1 (MCL-1) is a major cause of drug resistance in triple-negative breast cancer (TNBC) cells. Mucin 1 (MUC1) is a... 
Oxidative stress | Reactive oxygen species | Cell survival | Bcl-2 protein | Extracellular signal-regulated kinase | AKT protein | Breast cancer | Drug resistance | Mcl-1 protein | 1-Phosphatidylinositol 3-kinase | Mucin | Bcl-x protein | Bcl protein
Journal Article
Journal Article
PLOS GENETICS, ISSN 1553-7404, 03/2019, Volume 15, Issue 3, p. e1008002
Mammary epithelial progenitors are the normal cell-of-origin of breast cancer. We previously defined a population of p27(+) quiescent hormone-responsive... 
EPITHELIAL-CELLS | P27 | BREAST-CANCER RISK | PATHWAY | SUSCEPTIBILITY | GENETICS & HEREDITY | ESTROGEN | DEPENDENT KINASE INHIBITOR | REVEAL | TUMORIGENESIS | P27(KIP1) | Endocrine Cells - physiology | Mammary Glands, Animal | Estrogens | Humans | Stem Cells | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Estrogen Receptor alpha | Female | Cell Differentiation | Cell Proliferation - genetics | Genetic Predisposition to Disease - genetics | Receptors, Estrogen | Signal Transduction | Risk Factors | Cyclin-Dependent Kinase Inhibitor p27 - physiology | Rats | Epithelial Cells | Rats, Sprague-Dawley | Rats, Inbred ACI | Pregnancy | Mammary Glands, Human - growth & development | Animals | Breast Neoplasms - genetics | Animals, Genetically Modified - genetics | Receptors, Progesterone | Progesterone | Cyclin-Dependent Kinase Inhibitor p27 - genetics | Integrin alpha1 | Complications and side effects | Enzyme inhibitors | Physiological aspects | Mammary glands | Genetic aspects | Breast cancer | Chromosome deletion | Risk factors | Chromatin | Prolactin | Leptin | Genomics | Estrogen | Gene expression | Health aspects | Transcription | Lactation | Laboratories | Epithelial cells | Oncology | Systematic review | Genomes | Transplantation | Gene deletion | Kinases | Medical schools | Cyclin-dependent kinase | Carcinogens | Clonal deletion | Quorum sensing | Mammary gland | Bioinformatics | ErbB protein | Cyclin-dependent kinases | Stat5 protein | Progenitor cells | Medicine | Pathology | Womens health | Stem cells | Cyclin-dependent kinase inhibitor p27 | Software | Mutation | Health risk assessment | Females | Prostate | Tumors
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2018, Volume 128, Issue 7, pp. 2979 - 2995
Journal Article
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 08/2019, Volume 133, pp. 26 - 35
Journal Article
Oncogene, ISSN 0950-9232, 05/2017, Volume 36, Issue 20, pp. 2791 - 2801
Journal Article