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by Lundqvist, Andreas and van Hoef, Vincent and Zhang, Xiaonan and Wennerberg, Erik and Lorent, Julie and Witt, Kristina and Sanz, Laia Masvidal and Liang, Shuo and Murray, Shannon and Larsson, Ola and Kiessling, Rolf and Mao, Yumeng and Sidhom, John-William and Bessell, Catherine A and Havel, Jonathan and Schneck, Jonathan and Chan, Timothy A and Sachsenmeier, Eliot and Woods, David and Berglund, Anders and Ramakrishnan, Rupal and Sodre, Andressa and Weber, Jeffrey and Zappasodi, Roberta and Li, Yanyun and Qi, Jingjing and Wong, Philip and Sirard, Cynthia and Postow, Michael and Newman, Walter and Koon, Henry and Velcheti, Vamsidhar and Callahan, Margaret K and Wolchok, Jedd D and Merghoub, Taha and Lum, Lawrence G and Choi, Minsig and Thakur, Archana and Deol, Abhinav and Dyson, Gregory and Shields, Anthony and Haymaker, Cara and Uemura, Marc and Murthy, Ravi and James, Marihella and Wang, Daqing and Brevard, Julie and Monaghan, Catherine and Swann, Suzanne and Geib, James and Cornfeld, Mark and Chunduru, Srinivas and Agrawal, Sudhir and Yee, Cassian and Wargo, Jennifer and Patel, Sapna P and Amaria, Rodabe and Tawbi, Hussein and Glitza, Isabella and Woodman, Scott and Hwu, Wen-Jen and Davies, Michael A and Hwu, Patrick and Overwijk, Willem W and Bernatchez, Chantale and Diab, Adi and Massarelli, Erminia and Segal, Neil H and Ribrag, Vincent and Melero, Ignacio and Gangadhar, Tara C and Urba, Walter and Schadendorf, Dirk and Ferris, Robert L and Houot, Roch and Morschhauser, Franck and Logan, Theodore and Luke, Jason J and Sharfman, William and Barlesi, Fabrice and Ott, Patrick A and Mansi, Laura and Kummar, Shivaani and Salles, Gilles and Carpio, Cecilia and Meier, Roland and Krishnan, Suba and McDonald, Dan and Maurer, Matthew and Gu, Xuemin and Neely, Jaclyn and Suryawanshi, Satyendra and Levy, Ronald and Khushalani, Nikhil and Wu, Jennifer and Zhang, Jinyu and Basher, Fahmin and Rubinstein, Mark and Bucsek, Mark and Qiao, Guanxi and ...
Journal for ImmunoTherapy of Cancer, ISSN 2051-1426, 11/2016, Volume 4, Issue S1
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2016, Volume 126, Issue 6, pp. 2334 - 2340
Journal Article
Nature communications, ISSN 2041-1723, 11/2018, Volume 9, Issue 1, pp. 4821 - 13
Fusions involving the oncogenic gene RET have been observed in thyroid and lung cancers. Here we report RET gene alterations, including amplification, missense... 
CELL LUNG-CANCER | DOMAIN | MULTIDISCIPLINARY SCIENCES | TYROSINE KINASE INHIBITOR | TRUNCATED FORM | PHASE-II | OPEN-LABEL | MEDULLARY-THYROID CARCINOMA | GENE FUSION | EXPRESSION | ESTROGEN-RECEPTOR | NIH 3T3 Cells | Oncogene Proteins, Fusion - metabolism | Proto-Oncogene Proteins c-ret - metabolism | Receptor, ErbB-2 - genetics | Humans | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Breast Neoplasms - metabolism | MCF-7 Cells | Piperidines - pharmacology | Cell Transformation, Neoplastic - genetics | ras Guanine Nucleotide Exchange Factors - metabolism | Female | Antineoplastic Agents - pharmacology | Proto-Oncogene Proteins c-ret - antagonists & inhibitors | Nuclear Receptor Coactivators - metabolism | ras Guanine Nucleotide Exchange Factors - genetics | Signal Transduction | Breast Neoplasms - drug therapy | Cell Transformation, Neoplastic - metabolism | Phosphatidylinositol 3-Kinases - genetics | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Nude | Oncogene Proteins, Fusion - genetics | Anilides - pharmacology | Cell Line, Tumor | Mitogen-Activated Protein Kinases - genetics | Oncogene Proteins, Fusion - antagonists & inhibitors | Mice | Pyridines - pharmacology | Cell Transformation, Neoplastic - drug effects | Cell Transformation, Neoplastic - pathology | Quinazolines - pharmacology | Nuclear Receptor Coactivators - genetics | Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-ret - genetics | Transformation | Therapeutic applications | Xenotransplantation | MAP kinase | Breast cancer | ErbB-2 protein | 1-Phosphatidylinositol 3-kinase | Metastases | Amplification | Alterations | Missense mutation | Ret protein | Lungs | Xenografts | Breast | Mutation | Thyroid | Cancer
Journal Article
Journal Article
Frontiers in Oncology, ISSN 2234-943X, 2014, Volume 4, p. 277
Multi-modality cancer treatments that include chemotherapy, radiation therapy, and targeted agents are highly effective therapies. Their use, especially in... 
Chemotherapy | Breast cancer | Cardiotoxicity | Radiation therapy | Cardiac biomarkers | breast cancer | cardiotoxicity | Cardiac Biomarkers | chemotherapy | radiation therapy
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 09/2017, Volume 23, Issue 18, pp. 5631 - 5638
Purpose: Many patients with BRAFV600E mutant melanoma treated with BRAF inhibitors experience a rapid response, but ultimately develop resistance. Insight into... 
Sensitivity analysis | Inhibitors | Experimental design | MEK inhibitors | Melanoma | Cell lines | Mutation | Fusion protein | Gene expression | Cancer | Fitness | Metastases
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e69851
Several epidemiological studies have suggested a link between melanoma and breast cancer. Metabotropic glutamate receptor 1 (GRM1), which is involved in many... 
COMMON VARIANTS | SIGNALING PATHWAYS | GENETIC POLYMORPHISMS | MUTATION CARRIERS | GLUTAMATE-RECEPTOR-1 | MULTIDISCIPLINARY SCIENCES | RISK | PROLIFERATION | SUSCEPTIBILITY LOCUS | BRCA1 | GRM1 | Demography | Tissue Array Analysis | Humans | Middle Aged | Receptors, Metabotropic Glutamate - metabolism | Incidence | Neoplasm Recurrence, Local - pathology | Young Adult | Neoplasm Metastasis | Receptors, Metabotropic Glutamate - genetics | Aged, 80 and over | Adult | Estrogen Receptor alpha - metabolism | Female | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Estradiol - pharmacology | Breast Neoplasms - epidemiology | Breast Neoplasms - classification | Genetic Predisposition to Disease | Genetic Association Studies | Disease-Free Survival | Phenotype | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Tamoxifen - pharmacology | Polymorphism, Single Nucleotide - genetics | Tamoxifen - analogs & derivatives | Neoplasm Recurrence, Local - genetics | Aged | Cell Proliferation - drug effects | Cohort Studies | Oncology, Experimental | Genes | Estrogen | Melanoma | Amino acids | Breast cancer | Research | Tamoxifen | Glutamate | Gene expression | Epidemiology | Analysis | Genetic research | Genetic aspects | Single nucleotide polymorphisms | Progesterone | Cancer | Cell proliferation | Transformation | Correlation | Estrogens | Melanocytes | Glutamic acid receptors | Single-nucleotide polymorphism | Kinases | Medical diagnosis | Metastases | Genotypes | Deoxyribonucleic acid--DNA | Patients | Genotyping | Breast | Mutation | Health risk assessment | Glutamic acid receptors (metabotropic) | Tumors | Deoxyribonucleic acid | DNA
Journal Article
Cancer Causes & Control, ISSN 0957-5243, 8/2017, Volume 28, Issue 8, pp. 809 - 817
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 12/2015, Volume 14, Issue 12 Supplement 2, pp. C133 - C133
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 142, Issue 5, pp. S-777 - S-777
Journal Article
Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 2487 - 2487
Journal Article