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Journal of Oncology, ISSN 1687-8450, 2014, Volume 2014, pp. 743181 - 6
Insertion of central venous port (CVP) catheter in the cancer population is associated with increased incidence of venous thromboembolic events (VTE). However,... 
Complications and side effects | Cancer patients | Care and treatment | Usage | Demographic aspects | Diagnosis | Research | Thromboembolism | Risk factors | Intravenous catheterization | Clinical Study
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2017, Volume 23, Issue 10, pp. 1226 - 1233
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 11/2003, Volume 12, Issue 22, pp. 3017 - 3024
Mal de Meleda is an autosomal recessive inflammatory and keratotic palmoplantar skin disorder due to mutations in the ARS B gene, encoding for SLURP-1... 
GENE-ENCODING SLURP-1 | LY-6 SUPERFAMILY | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | CELL-CELL-ADHESION | MUTATIONS | NICOTINIC ACETYLCHOLINE-RECEPTORS | PLASMINOGEN-ACTIVATOR RECEPTOR | HUMAN KERATINOCYTES | VENOM ALPHA-NEUROTOXINS | MEMBER | ANTIGEN | Moths - cytology | Keratoderma, Palmoplantar - genetics | DNA, Complementary - administration & dosage | Humans | Antigens, Ly - genetics | Peptides - genetics | Acetylcholine - metabolism | Dose-Response Relationship, Drug | Recombinant Proteins - isolation & purification | Antigens, Ly - pharmacology | Cell Nucleus - metabolism | Peptides - metabolism | Keratoderma, Palmoplantar - metabolism | Keratoderma, Palmoplantar - pathology | Female | Urokinase-Type Plasminogen Activator - chemistry | Clone Cells | Protein Structure, Tertiary | Recombinant Proteins - metabolism | Amino Acid Sequence | Cell Line | Epidermis - metabolism | Microinjections | Peptides - chemistry | Receptors, Cholinergic - metabolism | Oocytes - metabolism | Receptors, Cholinergic - drug effects | Neurotransmitter Agents - metabolism | Models, Molecular | Antigens, Ly - chemistry | Urokinase-Type Plasminogen Activator - genetics | Genes, Recessive | Urokinase-Type Plasminogen Activator - isolation & purification | Patch-Clamp Techniques | Phenotype | Animals | Antigens, Ly - isolation & purification | Mutation | Xenopus laevis - physiology | DNA, Complementary - metabolism | Urokinase-Type Plasminogen Activator - pharmacology | Index Medicus
Journal Article
Molecular Therapy, ISSN 1525-0016, 01/2018, Volume 26, Issue 1, pp. 8 - 9
In our opinion, few have yet fully appreciated this problem. [...]the new findings described in this issue of Molecular Therapy are important, because they... 
MEDICINE, RESEARCH & EXPERIMENTAL | SQUAMOUS-CELL CARCINOMA | METASTASIS | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENETICS & HEREDITY | Autoantigens | Skin | Humans | Skin Neoplasms | Immunoglobulins | Transplants & implants | Oral cancer | Collagen | Immunotherapy | Surgery | Melanoma | Metastasis | Radiation therapy | Cancer therapies | Skin cancer | Metastases | Index Medicus
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 137, Issue 4, pp. 1189 - 1196.e2
Background Data on patients affected by chronic mucocutaneous candidiasis underscore the preponderant role of IL-17 receptor A (IL-17RA) in preserving... 
Allergy and Immunology | CECR1 | Adenosine deaminase 2 | whole-genome analyses | vasculitis | immunodeficiency | chronic mucocutaneous candidiasis | chronic inflammation | IL17RA | INTERLEUKIN-1-BETA | CANDIDIASIS | ADA2 | IMMUNOLOGY | T-HELPER-CELLS | FAMILY | ALLERGY | DIFFERENTIATION | VASCULOPATHY | TH17 CELLS | Sequence Deletion | Humans | Receptors, Interleukin-17 - immunology | Child, Preschool | Receptors, Interleukin-17 - genetics | Vasculitis - complications | Vasculitis - immunology | Receptors, Interleukin-17 - deficiency | Inflammation - complications | Vasculitis - genetics | Fatal Outcome | Female | Intercellular Signaling Peptides and Proteins - deficiency | Adenosine Deaminase - deficiency | Child | Siblings | Adenosine Deaminase - genetics | Candidiasis, Chronic Mucocutaneous - genetics | Candidiasis, Chronic Mucocutaneous - immunology | Intercellular Signaling Peptides and Proteins - genetics | Inflammation - immunology | Comparative Genomic Hybridization | Adolescent | Candidiasis, Chronic Mucocutaneous - complications | Inflammation - genetics | Chronic Disease | Adenosine Deaminase - immunology | Intercellular Signaling Peptides and Proteins - immunology | Physiological aspects | Health aspects | Medical genetics | Diseases | Genomics | Analysis | Adenosine | Genealogy | Bacterial infections | Cytokines | Laboratories | Cloning | Neutrophils | Infections | Inflammation | Patients | Dermatitis | Blood | Granulocytes | Lymphocytes | Biopsy | Bone marrow | Mutation | Age | Immune system | Neutropenia | Index Medicus | Abridged Index Medicus
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2010, Volume 55, Issue 5, pp. 469 - 480
Objectives We studied the signal transduction of atrial structural remodeling that contributes to the pathogenesis of atrial fibrillation (AF). Background... 
Cardiovascular | Internal Medicine | atrial fibrillation | Rac1 | connexin 43 | oxidative stress | CTGF | FIBROSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | ADULT-RAT CARDIOMYOCYTES | HEART-FAILURE | ANGIOTENSIN-II | BINDING PROTEIN | CARDIAC FIBROBLASTS | HYPERTROPHY | ADHESION | GTPASES | Heart Atria - pathology | Cadherins - metabolism | Humans | Middle Aged | Transforming Growth Factor beta1 - metabolism | Male | rhoA GTP-Binding Protein - metabolism | Gene Expression Profiling | Myocardium - metabolism | Female | Hydroxymethylglutaryl-CoA Reductase Inhibitors | Fibroblasts - metabolism | Animals, Newborn | Atrial Fibrillation - pathology | Receptor Cross-Talk | Angiotensin II - metabolism | Connexin 43 - metabolism | Signal Transduction | Rats | Mice, Transgenic | Myocardium - pathology | Rats, Sprague-Dawley | Heart Atria - metabolism | Atrial Fibrillation - metabolism | Animals | Fibrosis | Myocytes, Cardiac - metabolism | Aged | Mice | Connective Tissue Growth Factor - metabolism | rac1 GTP-Binding Protein - metabolism | Phosphates | Niacinamide | Coenzymes | RNA | Atrial fibrillation | Adenine | Transforming growth factors | Cellular signal transduction | Cardiac arrhythmia | Biotechnology | Oxidative stress | Cell culture | Cardiomyocytes | Hybridization | RNA polymerase | Experiments | Studies | Signal transduction | Transgenic animals | Rodents | Software | Agreements | Laboratory animals | Arrays | Statins | Deoxyribonucleic acid--DNA | Index Medicus | Abridged Index Medicus
Journal Article
The Journal of investigative dermatology, ISSN 0022-202X, 06/2019
CYLD is a deubiquitylase with tumor suppressor functions, firstly identified in patients with familial cylindromatosis. Despite many molecular mechanisms in... 
Index Medicus
Journal Article
Revue Medicale Suisse, ISSN 1660-9379, 04/2015, Volume 11, Issue 468, pp. 763 - 767
Journal Article
Revue medicale suisse, ISSN 1660-9379, 04/2015, Volume 11, Issue 468, p. 763
Progress in paediatric dermatology is achieved in both research and therapeutic fields. In infectiology, scabies and bed bugs are a scourge for dermatologists,... 
Dermoscopy - methods | Skin Diseases - diagnosis | Skin Diseases - therapy | Dermatology - methods | Humans | Skin Diseases - pathology | Child | Switzerland
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, pp. e9258 - e9258
Background: The Notch pathway is essential for proper epidermal differentiation during embryonic skin development. Moreover, skin specific loss of Notch... 
B-CELL DEVELOPMENT | HUMAN EPITHELIAL-CELLS | STEM-CELLS | IN-VITRO | MOUSE KERATINOCYTES | BIOLOGY | MICE | T-CELL | GROWTH-FACTOR | EXPRESSION | THYMIC STROMAL LYMPHOPOIETIN | Dermatitis, Atopic - genetics | Skin - metabolism | Humans | Receptor, Notch2 - genetics | Receptors, Notch - genetics | Myeloproliferative Disorders - genetics | Granulocyte Colony-Stimulating Factor - genetics | Dermatitis, Atopic - mortality | Flow Cytometry | Receptors, Cytokine - metabolism | Receptors, Cytokine - genetics | Granulocyte Colony-Stimulating Factor - metabolism | Skin - pathology | Immunoglobulins | Cytokines - metabolism | Mice, Inbred C57BL | Receptors, Notch - physiology | Mice, Transgenic | Survival Rate | Signal Transduction - genetics | Dermatitis, Atopic - physiopathology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Receptor, Notch2 - physiology | Animals | Mice, Nude | Models, Biological | Survival Analysis | Myeloproliferative Disorders - physiopathology | Signal Transduction - physiology | Mice | Skin - physiopathology | Receptor, Notch1 - genetics | Myeloproliferative Disorders - mortality | Receptor, Notch1 - physiology | Embryonic development | Psoriasis | Atopic dermatitis | Bone marrow | Transplantation | Skin | B cells | Syngeneic grafts | Pathogenesis | Bone marrow transplantation | Homeostasis | Lichen planus | Inactivation | Dermatitis | Thymus | Signal transduction | Embryogenesis | Biomedical materials | Immunology | Granulocytes | Granulocyte colony-stimulating factor | Lymphocytes | Rodents | Biocompatibility | Drug dosages | Osteopenia | Neutropenia | Spleen | Repressing | Deactivation | Cytokines | Developmental biology | Dermatology | Leukocytes (eosinophilic) | Neutrophils | Keratinocytes | Breast cancer | Gene expression | Myeloproliferative diseases | Hemopoiesis | Signaling | Thymic stromal lymphopoietin | Stem cells | Mast cells | Infiltration | Notch protein | Bone | Hematopoietic system | Mutation | Eosinophils | Index Medicus
Journal Article