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Nature Reviews Cancer, ISSN 1474-175X, 06/2011, Volume 11, Issue 6, pp. 426 - 437
Journal Article
Cell, ISSN 0092-8674, 07/2012, Volume 150, Issue 2, pp. 251 - 263
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2015, Volume 10, Issue 10, p. e0140712
Journal Article
Oncotarget, ISSN 1949-2553, 2015, Volume 6, Issue 5, pp. 3211 - 3224
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e64159
The role of B cells in cancer and the underlying mechanisms remain to be further explored. Here, we show that tumor-associated B cells with activated STAT3... 
IMMUNE CELLS | THERAPY | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | PHENOTYPE | CANCER | EXPRESSION | T-CELLS | ENDOTHELIAL GROWTH-FACTOR | SIGNAL TRANSDUCER | LYMPHOCYTES | Humans | Gene Expression Regulation, Neoplastic | Mice, 129 Strain | Neoplasms, Experimental - pathology | Signal Transduction - immunology | Neoplasms, Experimental - immunology | Neoplasms, Experimental - genetics | Tumor Burden - genetics | Neovascularization, Pathologic - immunology | B-Lymphocytes - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Endothelial Cells - metabolism | Mice, Inbred C57BL | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Disease Progression | Mice, Knockout | Endothelial Cells - immunology | Microscopy, Confocal | Animals | B-Lymphocytes - immunology | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Mice | Neovascularization, Pathologic - metabolism | STAT3 Transcription Factor - immunology | Tumor Burden - immunology | Analysis | Lung cancer | Melanoma | Development and progression | B cells | Gene expression | Health aspects | Cancer | Lupus | Medical research | Antigens | Animal models | Review boards | Stat3 protein | Tissues | Patients | Clinical outcomes | Skin cancer | Studies | Angiogenesis | Signaling | Immunology | Lymphocytes B | Lymphocytes | Immunotherapy | Rodents | Prostate | Tumors
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2013, Volume 110, Issue 11, pp. 4321 - 4326
Although targeting oncogenic mutations in the BRAF serine/threonine kinase with small molecule inhibitors can lead to significant clinical responses in... 
Genomics | Melanoma | Cell lines | Small interfering RNA | Melanocytes | Genetic mutation | Oncogenes | Tumors | Apoptosis | Cancer | METASTATIC MELANOMA | SURVIVAL ONCOGENE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MUTATION | SENSITIVITY | MITF | MALIGNANT-MELANOMA | B-RAF | CELL-LINES | CANCER | Inhibitor of Apoptosis Proteins - genetics | Minor Histocompatibility Antigens | Humans | Gene Expression Regulation, Neoplastic | Drug Resistance, Neoplasm | Male | Melanocytes - metabolism | Antineoplastic Agents - therapeutic use | Proto-Oncogene Proteins c-bcl-2 - metabolism | Melanoma - genetics | Melanocytes - pathology | Inhibitor of Apoptosis Proteins - metabolism | Proto-Oncogene Proteins B-raf - metabolism | Melanoma - metabolism | Microphthalmia-Associated Transcription Factor - metabolism | Melanoma - pathology | Gene Amplification - drug effects | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Cell Transformation, Neoplastic | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Cell Line, Tumor | Microphthalmia-Associated Transcription Factor - genetics | Proto-Oncogene Proteins c-bcl-2 - genetics | Cell Line, Transformed | Gene mutations | Physiological aspects | Genetic aspects | Research | Gene expression | Computational biology | Biological Sciences | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Cancer and Oncology | Medicin och hälsovetenskap | Cancer och onkologi
Journal Article
Molecular Oncology, ISSN 1574-7891, 03/2016, Volume 10, Issue 3, pp. 450 - 463
Journal Article