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Journal Article
PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, p. e100777
Inhibition of sodium glucose cotransporter 2 (SGLT2) has been reported as a new therapeutic strategy for treating diabetes. However, the effect of SGLT2... 
OXIDATIVE STRESS | ACTIVATION | HYPERGLYCEMIA | SGLT2 INHIBITOR | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | RATS | COMPLICATIONS | MELLITUS | PANCREATIC-FUNCTION | RENAL INJURY | Inflammation - pathology | Oxidative Stress | Kidney Cortex - metabolism | Body Weight - drug effects | Diabetic Nephropathies - drug therapy | Male | Kidney Function Tests | Inflammation - metabolism | Kidney - metabolism | Glomerular Mesangium - metabolism | Glomerular Mesangium - drug effects | Homeostasis - drug effects | Kidney - physiopathology | Disease Models, Animal | Diabetic Nephropathies - pathology | Gene Expression | Glucosides - pharmacology | Kidney Tubules, Proximal - pathology | Kidney - drug effects | Macrophages - pathology | Diabetic Nephropathies - metabolism | Cells, Cultured | Diabetic Nephropathies - genetics | Kidney Cortex - pathology | Sodium-Glucose Transporter 2 - antagonists & inhibitors | Blood Glucose - drug effects | Glomerular Mesangium - pathology | Macrophages - metabolism | Animals | Insulin-Secreting Cells - drug effects | Kidney Tubules, Proximal - metabolism | Glucose - metabolism | Inflammation - genetics | Benzhydryl Compounds - pharmacology | Mice | Apoptosis | Oxidases | Type 2 diabetes | Blood sugar | Analysis | Body weight | Diabetic nephropathies | Hemoglobin | DNA polymerases | Gene expression | Health aspects | Oxidative stress | RNA-directed DNA polymerase | Syngeneic grafts | Homeostasis | Glucose | Macrophages | Blood | NAD(P)H oxidase | Biomedical materials | Hyperglycemia | Rodents | Biocompatibility | Blood pressure | Inhibition | Pancreas | Pharmaceutical sciences | Age | University graduates | Neurochemistry | Creatinine | Kidneys | Cytokines | Diabetes mellitus | Osteopontin | Inflammation | Dentistry | Metabolism | Medicine | Studies | Inhibitors | Nephropathy | Sodium | Monocyte chemoattractant protein | Fibrosis | Infiltration | Diabetes | Monocyte chemoattractant protein 1
Journal Article
Genes to Cells, ISSN 1356-9597, 09/2011, Volume 16, Issue 9, pp. 951 - 960
Journal Article
Nihon Geka Gakkai zasshi, ISSN 0301-4894, 09/2016, Volume 117, Issue 5, pp. 412 - 414
Journal Article
Nihon Geka Gakkai zasshi, ISSN 0301-4894, 09/2016, Volume 117, Issue 5, p. 412
Journal Article
Diabetes, ISSN 0012-1797, 11/2012, Volume 61, Issue 11, pp. 2823 - 2832
It is unknown whether adipokines derived from adipose tissues modulate endoplasmic reticulum (ER) stress induced in obesity. Here, we show that visceral... 
ACTIVATION | INSULIN-RESISTANCE | PROSTATE-CANCER CELLS | ENDOCRINOLOGY & METABOLISM | GRP78 | ALPHA-MACROGLOBULIN | RECEPTOR | SERUM | MICE | ENDOPLASMIC-RETICULUM STRESS | ADIPOSE-TISSUE | Up-Regulation | Humans | Heat-Shock Proteins - antagonists & inhibitors | Hepatocytes - pathology | Male | Neoplasm Proteins - antagonists & inhibitors | Hepatocytes - metabolism | Neoplasm Proteins - metabolism | Serpins - chemistry | Recombinant Proteins - isolation & purification | Heat-Shock Proteins - genetics | Adipokines - genetics | Serpins - isolation & purification | Adipokines - chemistry | Cell Membrane - metabolism | Neoplasm Proteins - genetics | Recombinant Proteins - metabolism | HSP40 Heat-Shock Proteins - metabolism | Recombinant Proteins - antagonists & inhibitors | Serpins - genetics | HSP40 Heat-Shock Proteins - genetics | Serpins - metabolism | Signal Transduction | Heat-Shock Proteins - metabolism | Rats | Mice, Transgenic | Adipokines - metabolism | HSP40 Heat-Shock Proteins - antagonists & inhibitors | Intra-Abdominal Fat - metabolism | Mice, Knockout | Obesity - metabolism | Obesity - pathology | Protein Transport | Animals | Endoplasmic Reticulum Stress | Cell Line, Tumor | Ligands | Mice | Adipokines - isolation & purification | Physiological aspects | Obesity | Research | Protease inhibitors | Endoplasmic reticulum | Analysis | Obesity Studies
Journal Article
Journal of Cardiology, ISSN 0914-5087, 2016, Volume 69, Issue 3, pp. 591 - 595
Journal Article
DNA and Cell Biology, ISSN 1044-5498, 1995, Volume 14, Issue 2, pp. 95 - 101
The adenovirus (Ad) El region genes, EIA and E1B, are well known cooperatively to transform primary rodent cells and activate a number of cellular promoters,... 
CELLS | HUMAN RENIN GENE | ACID | TRANSACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | GENETICS & HEREDITY | CLASS-I ANTIGEN | CDNA | EXPRESSION | AP-1 | CELL BIOLOGY
Journal Article
Journal of Electrocardiology, ISSN 0022-0736, 11/2018, Volume 51, Issue 6, pp. 1172 - 1172
Journal Article
Journal of Arrhythmia, ISSN 1880-4276, 12/2017, Volume 33, Issue 6, pp. 637 - 639
Ventricular capture management is an automatic pacing threshold adjustment algorithm that automatically measures pacing threshold through detection of the... 
Automatic pacing threshold adjustment algorithm | Pacing failure | Lupus | Algorithms | Case Report
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e85594
Nuclear hormone receptors (NHRs) are transcription factors that regulate carbohydrate and lipid metabolism, immune responses, and inflammation. Although... 
GAMMA | ACTIVATION | LIPID-METABOLISM | ERR-ALPHA | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | DISEASE | PROTEINURIA | DIABETIC-NEPHROPATHY | MICE | PPAR-ALPHA AGONIST | Male | Receptors, Cytoplasmic and Nuclear - classification | Kidney - metabolism | Mice, Mutant Strains | COUP Transcription Factors - genetics | Kidney Tubules - metabolism | Cell Line | Gene Expression | Nuclear Receptor Subfamily 4, Group A, Member 1 - genetics | Podocytes - metabolism | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Cells, Cultured | Diabetic Nephropathies - genetics | Kidney - cytology | Receptors, Cytoplasmic and Nuclear - genetics | Mesangial Cells - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Kidney Tubules - cytology | Animals | COUP Transcription Factor II - genetics | COUP Transcription Factors - metabolism | Nuclear Receptor Subfamily 4, Group A, Member 1 - metabolism | Mice | COUP Transcription Factor II - metabolism | Microscopy, Fluorescence | Receptors, Cytoplasmic and Nuclear - metabolism | Type 2 diabetes | Diabetic nephropathies | Physiological aspects | Albumin | Carbohydrate metabolism | Glucose | Hormones | Dextrose | Biotechnology | Transcription factors | Syngeneic grafts | Epithelial cells | Pathogenesis | Liver | Nur77 protein | Homeostasis | Kinases | Proteins | Receptors | Hyperglycemia | Rodents | liver X receptors | Lipid metabolism | Pharmaceutical sciences | University graduates | Animal care | Carbohydrates | Ovalbumin | Immune response | Diabetes mellitus | Dentistry | Metabolism | Nuclear receptors | Polymerase chain reaction | Medicine | Nephropathy | Hepatocytes | Cell lines | Diabetes | Kidney diseases | Collecting duct | Clear cell-type renal cell carcinoma
Journal Article