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Neurochemistry International, ISSN 0197-0186, 2010, Volume 56, Issue 3, pp. 394 - 403
Our previous work demonstrated the marked decrease of mitochondrial complex I activity in the cerebral cortex of immature rats during the acute phase of... 
(S)-3,4-DCPG | Long periods of survival following the seizures | ATP levels | 4-Hydroxynonenal | Immature rats | Complex I inhibition | 3-Nitrotyrosine | Protection | Protein carbonyls | Free radical scavengers | dl-Homocysteic acid-induced seizures | Dl-Homocysteic acid-induced seizures | OXIDATIVE STRESS | SUPEROXIDE-PRODUCTION | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | DL-Homocysteic acid-induced seizures | STATUS EPILEPTICUS | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | CEREBRAL-CORTEX | CELL-DEATH | BRAIN MITOCHONDRIA | ULTRASTRUCTURAL DAMAGE | PARKINSONS-DISEASE | Free Radical Scavengers - pharmacology | Rats, Wistar | Oxidative Stress - physiology | Cerebral Cortex - pathology | Mitochondrial Diseases - metabolism | Epilepsy - metabolism | Male | Seizures - metabolism | Epilepsy - physiopathology | Aldehydes - metabolism | Homocysteine - analogs & derivatives | Electron Transport Complex I - metabolism | Tyrosine - analogs & derivatives | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Seizures - chemically induced | Seizures - physiopathology | Time Factors | Mitochondrial Diseases - chemically induced | Metabolic Networks and Pathways - physiology | Energy Metabolism - physiology | Disease Models, Animal | Animals, Newborn | Homocysteine - toxicity | Rats | Survival Rate | Excitatory Amino Acid Agonists - pharmacology | Mitochondria - metabolism | Mitochondria - drug effects | Down-Regulation - drug effects | Electron Transport Complex I - drug effects | Down-Regulation - physiology | Tyrosine - metabolism | Animals | Convulsants - toxicity | Oxidative Stress - drug effects | Mitochondrial Diseases - physiopathology | Energy Metabolism - drug effects | Glutamate | Seizures (Medicine)
Journal Article