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Journal of Neuropathology and Experimental Neurology, ISSN 0022-3069, 10/2010, Volume 69, Issue 10, pp. 1017 - 1033
The complex manifestations of chronic multiple sclerosis (MS)are due in part to widespread axonal abnormalities that affect lesional and nonlesional areas in... 
Neurofascin | Node of Ranvier | Neuroinflammation | Multiple sclerosis | Sodium channel | Demyelination | CHANNEL EXPRESSION | MINOCYCLINE | SODIUM-CHANNELS | PATHOLOGY | NEUROSCIENCES | AUTOIMMUNE ENCEPHALOMYELITIS | CLINICAL NEUROLOGY | DEGENERATION | APPEARING WHITE-MATTER | JUNCTIONS | GLUTAMATE EXCITOTOXICITY | PARKINSONS-DISEASE | Peptide Fragments | Minocycline - pharmacology | Humans | Middle Aged | Encephalomyelitis, Autoimmune, Experimental - immunology | Caspase 1 - metabolism | Male | Ranvier's Nodes - metabolism | Brain - metabolism | DNA-Binding Proteins - metabolism | Anti-Bacterial Agents - therapeutic use | Microglia - immunology | Microglia - physiology | NAV1.6 Voltage-Gated Sodium Channel | Encephalomyelitis, Autoimmune, Experimental - chemically induced | Minocycline - therapeutic use | Sodium Channels - metabolism | Microglia - pathology | Adult | Female | Indoles | HLA-DR Antigens - metabolism | Encephalomyelitis, Autoimmune, Experimental - pathology | Gene Expression Regulation - immunology | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Mice, Inbred C57BL | Myelin-Oligodendrocyte Glycoprotein | Glycoproteins | Ranvier's Nodes - drug effects | CD3 Complex - metabolism | Toll-Like Receptor 4 - metabolism | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Microscopy, Confocal | Animals | Analysis of Variance | Kv1.2 Potassium Channel - metabolism | Axons - pathology | Brain - pathology | Multiple Sclerosis - pathology | Aged | Anti-Bacterial Agents - pharmacology | Mice | Ranvier's Nodes - pathology | Neurofilament Proteins - metabolism | Postmortem Changes | Nitric Oxide Synthase Type II - metabolism
Journal Article
Annals of Neurology, ISSN 0364-5134, 12/2018, Volume 84, Issue 6, pp. 829 - 842
Objective Cortical gray matter (GM) pathology, involving demyelination and neurodegeneration, associated with meningeal inflammation, could be important in... 
COMPLEMENT-SYSTEM | MATTER | ATROPHY | ASSOCIATE | DISEASE | NEURODEGENERATION | REMYELINATION | PATHOLOGY | NEURONAL LOSS | NEUROSCIENCES | CLINICAL NEUROLOGY | B-CELL FOLLICLES | Immunohistochemistry | Multiple sclerosis | Cortex | Inflammation | Hybridization | Macrophages | Substantia grisea | Disease control | Microglia | Pathology | Cell activation | Neurodegeneration | Demyelination | Biomarkers | Lesions | Immunofluorescence
Journal Article
Annals of Neurology, ISSN 0364-5134, 04/2018, Volume 83, Issue 4, pp. 739 - 755
Journal Article
Journal Article
Journal Article
Journal Article
Journal Article
Brain Pathology, ISSN 1015-6305, 07/2018, Volume 28, Issue 4, pp. 507 - 520
The complement pathway has potential contributions to both white (WM) and grey matter (GM) pathology in Multiple Sclerosis (MS). A quantitative assessment of... 
immunohistochemistry | complement | tissue microarray | multiple sclerosis | SYSTEM | FACTOR-H | PATHOLOGY | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | PATHOGENESIS | MICROGLIA | INFLAMMATION | DISEASE | EXPRESSION | DEMYELINATING LESIONS | Immunohistochemistry | Neurons - pathology | Complement Activation | Kidney - pathology | Humans | Middle Aged | Astrocytes - pathology | Kidney - immunology | Male | Tissue Array Analysis - methods | White Matter - immunology | Microglia - immunology | Astrocytes - immunology | Microglia - pathology | Adult | Female | Gray Matter - immunology | Neurons - immunology | White Matter - pathology | Gray Matter - pathology | Brain - pathology | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Aged | Brain - immunology | Viral antibodies | Fibrin | Multiple sclerosis | Analysis | Fibrinogen | Antibodies | Methods | Complement receptors | Regulators | Complement component C3b | Methodology | Complement component C5a | Activation | Complement | Substantia grisea | Clusterin | Proteins | Complement factor H | Lesions | Localization | Quantitative analysis | Neurons | Glial fibrillary acidic protein | Cortex | Complement regulatory proteins | Complement component C1 | Complement component C1q | Pathology | Complement activation | Biomarkers | Digitization | Histocompatibility antigen HLA
Journal Article