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Clinical Chemistry, ISSN 0009-9147, 12/2015, Volume 61, Issue 12, p. 1444
  Author Contributions: All authors confirmed they have contributed to the intellectual content of this paper and have met the following 3 requirements: (a)... 
Pediatrics | Conflicts of interest | Metabolism
Journal Article
Clinical Chemistry, ISSN 0009-9147, 12/2015, Volume 61, Issue 12, p. 1444
Journal Article
Diabetes, ISSN 0012-1797, 05/2018, Volume 67, Issue Supplement 1, p. 1779
Journal Article
The Linacre Quarterly, ISSN 0024-3639, 09/2019, p. 2436391987376
Individuals who experience a gender identity that is discordant with biological sex are increasingly presenting to physicians for assistance in alleviating... 
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 03/2019, Volume 104, Issue 3, pp. 686 - 687
Journal Article
Clinical chemistry, 12/2015, Volume 61, Issue 12, p. 1444
Journal Article
Journal Article
American Journal of Physiology, ISSN 0193-1849, 12/2018, Volume 315, Issue 6, p. E1121
Gain-of-function (GOF) mutations in the ATP-sensitive potassium (KATP) channels cause neonatal diabetes. Despite the well-established genetic root of the... 
Neonates | Divergence | Glucagon | Liver | Glucose | Genotype & phenotype | Remission | Glibenclamide | Glucagon-like peptide 1 | Meloxicam | Phenotypes | Cytokines | Medical treatment | Diabetes mellitus | Inflammation | Potassium channels | Insulin | Babies | Sensitivity | Animals | Leptin | Mice | Sensitivity enhancement | Diabetes | Mutation | Potassium | ATP
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2016, Volume 150, Issue 4, pp. S715 - S715
Journal Article
Protein Science, ISSN 0961-8368, 12/2015, Volume 24, Issue 12, pp. 2008 - 2019
Journal Article
The New Atlantis, ISSN 1543-1215, 04/2017, Issue 52
Public controversies about how institutions should treat individuals who identify as a gender that does not correspond to their biological sex have recently... 
Talk shows | Clinics | Psychotherapy | Identification | Gender | Males | Gender identity | Girls | Neutralism | Disputes | Sex role identity | Teachers | Television | Repression (Political) | Childhood | Boys | Adolescents | Females
Journal Article
The Journal of clinical endocrinology and metabolism, 03/2019, Volume 104, Issue 3, pp. 686 - 687
Journal Article
Journal Article
Best Practice & Research: Clinical Endocrinology & Metabolism, ISSN 1521-690X, 2010, Volume 25, Issue 3, pp. 459 - 468
Identification and characterization of the molecular mechanisms contributing to the high incidence of insulin resistance in HIV infected patients treated with... 
Endocrinology & Metabolism | glucose transport | insulin sensitivity | nucleoside reverse transcriptase inhibitors | HIV protease inhibitors | in vitro models | lipotoxicity | ER stress | adipocytokines | METABOLIC SYNDROME | OXIDATIVE STRESS | FATTY LIVER-DISEASE | ANTIRETROVIRAL THERAPY | ENDOPLASMIC-RETICULUM STRESS | DIABETES-MELLITUS | SKELETAL-MUSCLE | PROTEASE INHIBITORS BLOCK | ENDOCRINOLOGY & METABOLISM | ADIPOCYTE DIFFERENTIATION | GLUCOSE DISPOSAL | HIV Protease Inhibitors - adverse effects | Insulin-Secreting Cells - secretion | Anti-HIV Agents - adverse effects | Humans | Liver - metabolism | Insulin Resistance | Adipose Tissue, White - metabolism | Muscle, Skeletal - metabolism | HIV Protease Inhibitors - administration & dosage | Reverse Transcriptase Inhibitors - administration & dosage | HIV Protease Inhibitors - therapeutic use | Anti-HIV Agents - administration & dosage | Reverse Transcriptase Inhibitors - adverse effects | Insulin-Secreting Cells - metabolism | Animals | Insulin-Secreting Cells - drug effects | Liver - drug effects | Muscle, Skeletal - drug effects | Anti-HIV Agents - therapeutic use | HIV Infections - drug therapy | Reverse Transcriptase Inhibitors - therapeutic use | Drug Therapy, Combination | Adipose Tissue, White - drug effects | HIV Infections - metabolism | Protease inhibitors | Proteases | Analysis | Physiological aspects | HIV patients | Insulin resistance | Glucose | Dextrose | Insulin sensitivity
Journal Article
ISSN 0012-1797, 10/2011, Volume 60, Issue 10, p. 2515
  Neonatal diabetes mellitus (NDM) can be caused by gain-of-function ATP-sensitive K(+) (K(ATP)) channel mutations. This realization has led to sulfonylurea... 
Journal Article
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