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Nature communications, ISSN 2041-1723, 06/2019, Volume 10, Issue 1, pp. 2402 - 13
Platelet-leukocyte interactions amplify inflammatory reactions, but the underlying mechanism is still unclear. CLEC5A and CLEC2 are spleen tyrosine kinase... 
Viral diseases | Viruses | Inflammatory response | Lethality | Leukocytes | Exosomes | Macrophages | Kinases | Receptors | Vesicles | Toll-like receptors | Stat1 protein | Protein-tyrosine kinase | Spleen | Tyrosine | Vector-borne diseases | Dengue | Dengue fever | Leukocytes (neutrophilic) | Inflammation | Pattern recognition | CLEC5A protein | White blood cells | TLR2 protein | Venom | Syk protein | Platelets
Journal Article
Journal of Biomedical Science, ISSN 1021-7770, 06/2017, Volume 24, Issue 1, pp. 39 - 9
Decoy receptor 3 (DcR3), also known as tumor necrosis factor receptor (TNFR) superfamily member 6b (TNFRSF6B), is a soluble decoy receptor which can neutralize... 
M2 macrophages | TNFR6B | Biomarker | Decoy receptor 3 (DcR3) | MEDICINE, RESEARCH & EXPERIMENTAL | FAS LIGAND | TNF-LIKE LIGAND | DENDRITIC CELLS | DCR3 LEVELS | TH17 IMMUNE-RESPONSES | DISEASE-ACTIVITY | CELL BIOLOGY | NECROSIS-FACTOR SUPERFAMILY | UP-REGULATION | MACROPHAGE DIFFERENTIATION | EXPRESSION | Mice - genetics | Neoplasms - etiology | Receptors, Tumor Necrosis Factor, Member 6b - metabolism | Humans | Gene Expression Regulation | Immunologic Factors - genetics | Inflammation - etiology | Animals | Neoplasms - genetics | Animals, Genetically Modified - genetics | Inflammation - genetics | Immunologic Factors - metabolism | Receptors, Tumor Necrosis Factor, Member 6b - genetics | Apoptosis | Macrophages | Tumor necrosis factor | Health aspects | Pathogenesis | Differentiation (biology) | Metastases | Proteins | Cell activation | Light | Fibroblasts | Physiology | Fusion protein | Lupus | Wound healing | Rheumatic diseases | Maintenance | Studies | Inflammatory bowel disease | Rheumatoid arthritis | Ligands | Mice | In vitro methods and tests | Tumor necrosis factor receptors | Physiological effects | Fas antigen | Medical services | Lymphocytes T | Genomes | Tissues | Inflammatory diseases | Epidermal growth factor | Transgenic animals | Feedback | Rodents | Attenuation | Tumor necrosis factor-TNF | Recombinant | Psoriasis | Dendritic cells | Tumor cells | Transgenic mice | Inflammation | Cell differentiation | Fc receptors | Gene amplification | FasL protein | In vivo methods and tests | Autoimmune diseases | Cancer
Journal Article
FRONTIERS IN IMMUNOLOGY, ISSN 1664-3224, 12/2019, Volume 10, p. 2867
The protective roles of endosomal toll-like receptors (TLRs) and cytosolic nucleic acid sensors are well elucidated, but the pathogenic host factors during... 
CELLS | microvesicle | Syk | THROMBOSIS | MACROPHAGES | IMMUNOLOGY | DENGUE-VIRUS-INFECTION | NEUTROPHIL EXTRACELLULAR TRAPS | DC-SIGN | INFLAMMATION | CLEC5A | CLEC2 | TOLL-LIKE RECEPTORS | TLR | PLATELET ACTIVATION | extracellular vesicle | exosome | Infection | Lectins | Health aspects | Snakes
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2008, Volume 105, Issue 10, pp. 3690 - 3695
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 05/2015, Volume 97, Issue 5, pp. 825 - 835
NLRP3 is the most crucial member of the NLR family, as it detects the existence of pathogen invasion and self-derived molecules associated with cellular... 
Tyrosine kinase | IL-1β | NLR | ATP | UBIQUITIN | tyrosine kinase | IL-1 beta | IMMUNOLOGY | CELL BIOLOGY | TYROSINE KINASE SYK | RESPONSES | INHIBITORS | RECEPTORS | HEMATOLOGY | DEUBIQUITINATION
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 299 - 13
Journal Article
PLoS Pathogens, ISSN 1553-7366, 04/2012, Volume 8, Issue 4, p. e1002655
CLEC5A/MDL-1, a member of the myeloid C-type lectin family expressed on macrophages and neutrophils, is critical for dengue virus (DV)-induced hemorrhagic... 
ACTIVATION | MICROBIOLOGY | MYELOID CELLS | DAP12-ASSOCIATING LECTIN (MDL)-1 | DENGUE | MDL-1 CLEC5A | VIROLOGY | MICROGLIA | NEURONAL DEATH | CENTRAL-NERVOUS-SYSTEM | MICE | PARASITOLOGY | WEST-NILE | Lectins, C-Type - immunology | Encephalitis, Japanese - genetics | Lectins, C-Type - genetics | Lectins, C-Type - metabolism | STAT1 Transcription Factor - metabolism | Macrophages - virology | Astrocytes - immunology | Encephalitis Virus, Japanese - metabolism | Adaptor Proteins, Signal Transducing - immunology | Blood-Brain Barrier - virology | Neurons - metabolism | Encephalitis, Japanese - metabolism | Cytokines - genetics | Astrocytes - virology | Macrophages - immunology | Cytokines - immunology | Neurons - virology | Blood-Brain Barrier - immunology | Immunity, Cellular - genetics | Cytokines - metabolism | Immunity, Humoral - genetics | Neurons - immunology | Receptors, Cell Surface - metabolism | Inflammation | STAT1 Transcription Factor - genetics | Receptors, Cell Surface - immunology | STAT1 Transcription Factor - immunology | Blood-Brain Barrier - metabolism | Encephalitis Virus, Japanese - genetics | Mice, Knockout | Macrophages - metabolism | Animals | Encephalitis, Japanese - immunology | Adaptor Proteins, Signal Transducing - genetics | Mice | Adaptor Proteins, Signal Transducing - metabolism | Antibodies, Monoclonal, Murine-Derived - pharmacology | Encephalitis Virus, Japanese - immunology | Astrocytes - metabolism | Receptors, Cell Surface - genetics | Encephalitis | Physiological aspects | Development and progression | Virulence (Microbiology) | Research | Macrophages | Health aspects | Flow cytometry | Phosphorylation | Cytokines | Pathogenesis | Dengue fever | Viruses | Infections | Experiments | Proteins | Studies | Brain research | Rodents | Labeling
Journal Article
Blood, ISSN 0006-4971, 01/2013, Volume 121, Issue 1, pp. 95 - 106
Journal Article