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1. Full Text Predicting clinical outcome of therapy-resistant prostate cancer
by Ma, Xin and Huang, Jiaoti
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 06/2019, Volume 116, Issue 23, pp. 11090 - 11092
Antigens | Medical colleges | Care and treatment | Prognosis | Patient outcomes | Prostate cancer | Cancer | Prostate
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2. Full Text Identification of a Cell of Origin for Human Prostate Cancer
by Andrew S. Goldstein and Jiaoti Huang and Changyong Guo and Isla P. Garraway and Owen N. Witte
Science, ISSN 0036-8075, 7/2010, Volume 329, Issue 5991, pp. 568 - 571
Luminal cells are believed to be the cells of origin for human prostate cancer, because the disease is characterized by luminal cell expansion and the absence... 
Epithelial cells | Neurons | Stromal cells | REPORTS | Stem cells | Basal cell neoplasms | Prostate cancer | Prostate | Cells | Endothelial cells | Cell transplantation | PROGENITORS | STEM-CELLS | EPITHELIAL-CELLS | SUBPOPULATION | MULTIDISCIPLINARY SCIENCES | PTEN | LEADS | INTRAEPITHELIAL NEOPLASIA | EXPRESSION | CARCINOMA | BASAL | Transcriptional Regulator ERG | Prostatic Neoplasms - metabolism | Prostatic Intraepithelial Neoplasia - pathology | Epithelial Cells - metabolism | Humans | Receptors, Androgen - metabolism | Male | Prostate - metabolism | Prostate - pathology | Keratins - analysis | Flow Cytometry | Biomarkers, Tumor - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Prostatic Intraepithelial Neoplasia - metabolism | Prostatic Neoplasms - pathology | Epithelium - pathology | Transduction, Genetic | Cell Separation | Epithelial Cells - pathology | Mice, SCID | Animals | Cell Transformation, Neoplastic | Prostate - cytology | Mice, Inbred NOD | Trans-Activators - metabolism | Mice | Usage | Cancer cells | Physiological aspects | Causes of | Genetic aspects | Histology | Gene expression | Cellular biology | Pathogenesis | Human | Antigens | Origins | Biomedical materials | In vivo testing | In vivo tests | Cancer | Index Medicus
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3. Full Text Prostate cancer: molecular and cellular mechanisms and their implications in therapy resistance and disease progression
by Feng, NH and Huang, JT
ASIAN JOURNAL OF ANDROLOGY, ISSN 1008-682X, 05/2019, Volume 21, Issue 3, pp. 213 - 214
Prostate cancer is among the most common malignancies in Western countries, and its incidence is rapidly rising in Asia where it was traditionally considered... 
ANDROLOGY | UROLOGY & NEPHROLOGY | Epigenetics | Androgens | Disease | Cancer therapies | Prostate cancer | Tumors
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4. Full Text Basal Epithelial Stem Cells Are Efficient Targets for Prostate Cancer Initiation
by Devon A. Lawson and Yang Zong and Sanaz Memarzadeh and Li Xin and Jiaoti Huang and Owen N. Witte
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2010, Volume 107, Issue 6, pp. 2610 - 2615
Prevailing theories suggest that luminal cells are the origin of prostate cancer because it is histologically defined by basal cell loss and malignant luminal... 
Epithelial cells | Stromal cells | Stem cells | Basal cell neoplasms | Lesions | Magnification | Prostate | Prostate cancer | Cells | Tissue grafting | AKT | ERG | ETS | CD49F | PROGENITOR CELLS | TRANSFORMATION | ANDROGEN RECEPTOR | MURINE PROSTATE | MULTIDISCIPLINARY SCIENCES | PTEN | MODEL | CD49f | VENTRAL PROSTATE | SUBPOPULATION | EXPRESSION | Fibroblast Growth Factor 10 - pharmacology | Prostatic Neoplasms - metabolism | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | Receptors, Androgen - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | Prostatic Neoplasms - physiopathology | Stem Cells - metabolism | Proto-Oncogene Proteins c-akt - genetics | Flow Cytometry | Proto-Oncogene Protein c-fli-1 - metabolism | Female | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Prostatic Neoplasms - pathology | Proto-Oncogene Protein c-fli-1 - genetics | Signal Transduction | Mice, Inbred C57BL | Epithelial Cells - pathology | Mice, Transgenic | Mice, Inbred Strains | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Phosphatidylinositol 3-Kinases - genetics | Regeneration | Animals | Receptors, Androgen - genetics | Stem Cells - drug effects | Stem Cells - pathology | Mice | Development and progression | Genetic aspects | Risk factors | Signal transduction | T cell receptors | Gene expression | Index Medicus | Biological Sciences
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5. Full Text 国民党推动世代交替
by 黄涛
两岸关系, ISSN 1006-5679, 2014, Issue 6, pp. 49 - 51
... 
立法院 | 社会运动 | 世代 | 国民党 | 党务改革 | 台湾地区 | 马英九 | 朱立伦
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6. Full Text Highly efficient capture of circulating tumor cells by using nanostructured silicon substrates with integrated chaotic micromixers
by Wang, Shutao and Liu, Kan and Liu, Jian and Yu, Zeta T.-F and Xu, Xiaowen and Zhao, Libo and Lee, Tom and Lee, Eun Kyung and Reiss, Jean and Lee, Yi-Kuen and Chung, Leland W. K and Huang, Jiaoti and Rettig, Matthew and Seligson, David and Duraiswamy, Kumaran N and Shen, Clifton K.-F and Tseng, Hsian-Rong
Angewandte Chemie - International Edition, ISSN 1433-7851, 03/2011, Volume 50, Issue 13, pp. 3084 - 3088
Cell capture | Circulating tumor cell | Cancer diagnosis | Nanostructured materials | Microfluidics | cancer diagnosis | CASCADE | CHEMISTRY, MULTIDISCIPLINARY | MICROCHANNELS | nanostructured materials | BREAST-CANCER | circulating tumor cell | RARE CELLS | cell capture | CANCER METASTASIS | SYSTEMS | microfluidics | EXPRESSION | PROGRESSION | BLOOD | Prostatic Neoplasms - pathology | Prostatic Neoplasms - chemistry | Neoplastic Cells, Circulating - pathology | Humans | Male | Molecular Imaging | Neoplastic Cells, Circulating - chemistry | Complex Mixtures - chemistry | Equipment Design | Microfluidics - instrumentation | Breast Neoplasms - chemistry | Breast Neoplasms - pathology | Surface Properties | Breast Neoplasms - blood | Nanostructures - chemistry | Prostatic Neoplasms - blood | Female | Microfluidics - methods | Silicon - chemistry | Index Medicus
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7. Full Text MP53-07 CANCER DETECTION RATE OF CONVENTIONAL VS 3D TEMPLATE PROSTATE BIOPSY IN A MRI/US FUSION DEVICE
by Natarajan, Shyam and Huang, Jiaoti and Marks, Leonard
Journal of Urology, The, ISSN 0022-5347, 2016, Volume 195, Issue 4, pp. e699 - e699
Urology | Magnetic resonance imaging | Diagnosis | Cancer
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8. Function of PTP1B in Neuroendocrine Differentiation of Prostate Cancer
by Huang, Jiaoti
2008, AD-a482 246.
The goal of the project is to identify the molecular mechanisms responsible for therapeutic failure in prostate cancer patients receiving hormonal therapy... 
stem cells | neuroendocrine cells | androgens | hormones | cxcr2 | cxcr1 | igf-1 receptor | sense organs | cells(biology) | patients | cultures(biology) | hormonal therapy | prostate cancer | in vitro analysis | androgen receptor | cancer stem cells | endocrine glands | ptp1b | neurology | il-8
Government Document
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9. Full Text FOXA2 is a sensitive and specific marker for small cell neuroendocrine carcinoma of the prostate
by Park, Jung Wook and Lee, John K and Witte, Owen N and Huang, Jiaoti
Modern Pathology, ISSN 0893-3952, 09/2017, Volume 30, Issue 9, pp. 1262 - 1272
The median survival of patients with small cell neuroendocrine carcinoma is significantly shorter than that of patients with classic acinar-type... 
MOLECULAR CHARACTERIZATION | THERAPY | ADENOCARCINOMA | GROWTH | PHENOTYPE | DIFFERENTIATION | PATHOLOGY | CASTRATION | CANCER | EXPRESSION | PROGRESSION | Immunohistochemistry | Carcinoma, Small Cell - secondary | Neoplasm Transplantation | Predictive Value of Tests | Prostatic Neoplasms - metabolism | Carcinoma, Small Cell - genetics | Cell Proliferation | Hepatocyte Nuclear Factor 3-beta - genetics | Tissue Array Analysis | Humans | Gene Expression Regulation, Neoplastic | Male | Heterografts | Prostatic Neoplasms - genetics | Biomarkers, Tumor - metabolism | Carcinoma, Neuroendocrine - metabolism | Hepatocyte Nuclear Factor 3-beta - metabolism | Prostatic Neoplasms - pathology | Diagnosis, Differential | Carcinoma, Small Cell - metabolism | Carcinoma, Neuroendocrine - genetics | Carcinoma, Neuroendocrine - secondary | Tumor Burden | Animals | Biopsy | Cell Line, Tumor | Mice, Inbred NOD | Biomarkers, Tumor - genetics | Adenocarcinoma | Androgens | Cell survival | Autopsy | Xenografts | Tumor cell lines | Prostate | Prostate cancer | Lymph nodes | Metastases | Index Medicus | biomarker | small cell neuroendocrine cancer | FOXA2
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10. Full Text Functional expression of sodium-glucose transporters in cancer
by Claudio Scafoglio and Bruce A. Hirayama and Vladimir Kepe and Jie Liu and (刘捷) and Chiara Ghezzi and Nagichettiar Satyamurthy and Neda A. Moatamed and Jiaoti Huang and Hermann Koepsell and Jorge R. Barrio and Ernest M. Wright
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2015, Volume 112, Issue 30, pp. E4111 - E4119
Glucose is a major metabolic substrate required for cancer cell survival and growth. It is mainly imported into cells by facilitated glucose transporters... 
SGLT2-inhibitors | SGLT2 | Prostate cancer | Pancreatic cancer | CELLS | MULTIDISCIPLINARY SCIENCES | PANCREATIC-CANCER | GEMCITABINE | pancreatic cancer | prostate cancer | POSITRON-EMISSION-TOMOGRAPHY | THERAPY | METABOLISM | PROSTATE-CANCER | LACTATE | MICE | RAT-BRAIN | Glucose Transport Proteins, Facilitative - metabolism | Immunohistochemistry | Neoplasm Transplantation | Prostatic Neoplasms - metabolism | Pancreatic Neoplasms - metabolism | Sodium-Glucose Transporter 2 - metabolism | Humans | Gene Expression Regulation, Neoplastic | Male | Gene Expression Profiling | Positron-Emission Tomography | Sodium-Glucose Transporter 2 - antagonists & inhibitors | Necrosis | Pancreatic Neoplasms - drug therapy | Kidney - metabolism | Adenocarcinoma - metabolism | Animals | Biological Transport | Glucosides - chemistry | Female | Mice | Fluorine Radioisotopes - chemistry | Prostatic Neoplasms - drug therapy | Glucose metabolism | Biological transport, Active | Genetic aspects | Gene expression | Observations | Survival analysis | Glucose | Rodents | Tumors | Index Medicus | Biological Sciences | PNAS Plus
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11. Full Text Prostate epithelial cell of origin determines cancer differentiation state in an organoid transformation assay
by Jung Wook Park and John K. Lee and John W. Phillips and Patrick Huang and Donghui Cheng and Jiaoti Huang and Owen N. Witte
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2016, Volume 113, Issue 16, pp. 4482 - 4487
The cell of origin for prostate cancer remains a subject of debate. Genetically engineered mouse models have demonstrated that both basal and luminal cells can... 
Oragnoid culture | Luminal cell | Cancer differentiation | Cells of origin | Human prostate cancer | Neoplasm Transplantation | Prostatic Neoplasms - metabolism | Epithelial Cells - metabolism | Kallikreins - biosynthesis | Humans | Male | Prostate - metabolism | Proto-Oncogene Proteins c-akt - genetics | Prostate - pathology | Prostate-Specific Antigen - biosynthesis | Proto-Oncogene Proteins c-myc - biosynthesis | Heterografts | Prostatic Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Receptors, Androgen - biosynthesis | Organoids - metabolism | Cell Differentiation | Prostate-Specific Antigen - genetics | PTEN Phosphohydrolase - genetics | Prostatic Neoplasms - pathology | Organoids - pathology | Transduction, Genetic | Kallikreins - genetics | PTEN Phosphohydrolase - biosynthesis | Epithelial Cells - pathology | Mice, SCID | Cell Transformation, Neoplastic - metabolism | Lentivirus | Proto-Oncogene Proteins c-akt - biosynthesis | Animals | Receptors, Androgen - genetics | Cell Line, Tumor | Mice | Proto-Oncogene Proteins c-myc - genetics | Causes of | Cell differentiation | Epithelial cells | Prostate cancer | Health aspects | Antigens | Genetic engineering | Biological assays | Rodents | Cells | Cancer | Index Medicus | Biological Sciences | cells of origin | cancer differentiation | human prostate cancer | luminal cell | oragnoid culture
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12. Full Text Monoclonal antibody targeting of N-cadherin inhibits prostate cancer growth, metastasis and castration resistance
by Yamashiro, Joyce and Wada, Robert and An, Jaibin and Wainberg, Zev A and Tran, Chau P and Gleave, Martin and Vessella, Robert L and Shimomura, Tatsuya and Tanaka, Hiroshi and Fazli, Ladan and Horvath, Steven and Rettig, Matthew B and Huang, Jiaoti and Kono, Evelyn and Miyazaki, Hideyo and Reiter, Robert E
Nature Medicine, ISSN 1078-8956, 12/2010, Volume 16, Issue 12, pp. 1414 - 1420
The transition from androgen-dependent to castration-resistant prostate cancer (CRPC) is a lethal event of uncertain molecular etiology. Comparing gene... 
MEDICINE, RESEARCH & EXPERIMENTAL | ANDROGEN RECEPTOR | STEM-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INCREASES | MESENCHYMAL TRANSITION | ANTITUMOR-ACTIVITY | CELL BIOLOGY | THERAPY | INDEPENDENCE | PATHWAY | BIOLOGY | PROGRESSION | Immunohistochemistry | Immunotherapy - methods | Enzyme-Linked Immunosorbent Assay | Antibodies, Monoclonal - pharmacology | Humans | Antibodies, Monoclonal - therapeutic use | Male | Mice, SCID | Blotting, Western | Disease Progression | Gene Knockdown Techniques | Prostatic Neoplasms - immunology | Prostatic Neoplasms - therapy | Animals | Cadherins - immunology | Neoplasm Metastasis - prevention & control | Cell Line, Tumor | Interleukin-8 - secretion | Mice | Gene Expression Regulation, Neoplastic - drug effects | Cadherins - genetics | Antibodies, Monoclonal - immunology | Usage | Care and treatment | Monoclonal antibodies | Development and progression | Genetic aspects | Research | Metastasis | Gene expression | Health aspects | Prostate cancer | Risk factors | Proteins | Biomedical research | Cellular biology | Index Medicus
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13. Full Text Making a Tissue Microarray
by Koo, Matthew and Squires, Jill M and Ying, Daphne and Huang, Jiaoti and Huang, Jiaoti
Methods in molecular biology (Clifton, N.J.), ISSN 1064-3745, 2019, Volume 1897, pp. 313 - 323
Tissue microarray (TMA) is a widely used, high-throughput, cost-effective, and tissue and reagent-conserving method of performing molecular analysis. Multiple... 
Immunohistochemistry | Pathology | Tissue microarray | Molecular profiling | Research | Comparative analysis | Tissue Array Analysis - methods | Paraffin Embedding - methods | Humans
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14. Full Text Prostate cancer-Associated SPOP mutations confer resistance to BET inhibitors through stabilization of BRD4
by Dai, Xiangpeng and Gan, Wenjian and Li, Xiaoning and Wang, Shangqian and Zhang, Wei and Huang, Ling and Liu, Shengwu and Zhong, Qing and Guo, Jianping and Zhang, Jinfang and Chen, Ting and Shimizu, Kouhei and Beca, Francisco and Blattner, Mirjam and Vasudevan, Divya and Buckley, Dennis L and Qi, Jun and Buser, Lorenz and Liu, Pengda and Inuzuka, Hiroyuki and Beck, Andrew H and Wang, Liewei and Wild, Peter J and Garraway, Levi A and Rubin, Mark A and Barbieri, Christopher E and Wong, Kwok-Kin and Muthuswamy, Senthil K and Huang, Jiaoti and Chen, Yu and Bradner, James E and Wei, Wenyi
Nature Medicine, ISSN 1078-8956, 09/2017, Volume 23, Issue 9, pp. 1063 - 1071
The bromodomain and extraterminal (BET) family of proteins comprises four members-BRD2, BRD3, BRD4 and the testisspecific isoform BRDT-that largely function as... 
MEDICINE, RESEARCH & EXPERIMENTAL | RECRUITMENT | ONCOPROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | DRUG-RESISTANCE | UBIQUITIN LIGASES | P-TEFB | CELL BIOLOGY | BREAST-CANCER | DEGRADATION | PROMOTES | BROMODOMAIN PROTEIN BRD4 | Prostatic Neoplasms - metabolism | Cell Proliferation | Immunoprecipitation | Humans | Protein-Serine-Threonine Kinases | Immunoblotting | Male | RNA-Binding Proteins | Molecular Targeted Therapy | Azepines | Thalidomide - analogs & derivatives | Ubiquitination | Prostatic Neoplasms - genetics | Triazoles | HEK293 Cells | Nuclear Proteins - genetics | Prostatic Neoplasms - drug therapy | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Benzodiazepines | Transcription Factors - metabolism | Drug Resistance, Neoplasm - genetics | Cullin Proteins | HeLa Cells | Apoptosis | Gene mutations | Physiological aspects | Genetic aspects | Research | Drug therapy | Drug resistance | Prostate cancer | Inhibitor drugs | Transcription | Leukocyte migration | Leukemia | c-Myc protein | Clinical trials | Myc protein | Cullin | Degradation | Proteins | Organoids | Cell cycle | Medical research | Abundance | Tumor cell lines | Mutants | Molecular modelling | Inhibitors | Bet protein | Mutation | Prostate | Cell migration | Cancer | Index Medicus
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15. Full Text Clinical application of a 3D ultrasound-guided prostate biopsy system
by Natarajan, Shyam, M.S and Marks, Leonard S., M.D and Margolis, Daniel J.A., M.D and Huang, Jiaoti, M.D., Ph.D and Macairan, Maria Luz, M.D and Lieu, Patricia and Fenster, Aaron, Ph.D
Urologic Oncology: Seminars and Original Investigations, ISSN 1078-1439, 2011, Volume 29, Issue 3, pp. 334 - 342
Abstract Objectives Prostate biopsy (Bx) has for 3 decades been performed in a systematic, but blind fashion using 2D ultrasound (US). Herein is described the... 
Urology | Artemis | Biopsy | MRI | Prostate | Diffusion weighted imaging | Cancer | DIFFUSION-WEIGHTED MRI | LOCALIZATION | CONTRAST-ENHANCED MRI | RADICAL PROSTATECTOMY | PERIPHERAL ZONE | ONCOLOGY | NEGATIVE BIOPSIES | ACTIVE SURVEILLANCE | CANCER DETECTION | UROLOGY & NEPHROLOGY | ENDORECTAL COIL | DIAGNOSTIC-CRITERIA | Prostatic Neoplasms - pathology | Adenocarcinoma - pathology | Prostatic Neoplasms - surgery | Humans | Middle Aged | Male | Magnetic Resonance Imaging | Pilot Projects | Ultrasonography | Aged, 80 and over | Prostatic Neoplasms - diagnostic imaging | Adult | Aged | Adenocarcinoma - diagnostic imaging | Imaging, Three-Dimensional | Neoplasm Staging | Adenocarcinoma - surgery | Medical colleges | Prostate cancer | Index Medicus
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16. Full Text Regulated proteolysis of Trop2 drives epithelial hyperplasia and stem cell self-renewal via β-catenin signaling
by Stoyanova, Tanya and Goldstein, Andrew S and Cai, Houjian and Drake, Justin M and Huang, Jiaoti and Witte, Owen N
Genes and Development, ISSN 0890-9369, 10/2012, Volume 26, Issue 20, pp. 2271 - 2285
The cell surface protein Trop2 is expressed on immature stem/progenitor-like cells and is overexpressed in many epithelial cancers. However the biological... 
β-catenin | Self-renewal | Transformation | Regulated intramembrane proteolysis | Trop2 | beta-catenin | MURINE PROSTATE | INTRAMEMBRANE PROTEOLYSIS | MONOCLONAL-ANTIBODY | DEVELOPMENTAL BIOLOGY | E-CADHERIN | transformation | self-renewal | CELL BIOLOGY | BREAST-CANCER | IN-VITRO | POOR-PROGNOSIS | PROSTATE-CANCER | GENETICS & HEREDITY | MAMMARY STEM/PROGENITOR CELLS | regulated intramembrane proteolysis | INTRACELLULAR DOMAIN | Antigens, Neoplasm - genetics | Prostatic Neoplasms - pathology | Cell Adhesion Molecules - genetics | Cell Proliferation | Epithelial Cells - metabolism | Signal Transduction | Humans | Gene Expression Regulation | Male | Stem Cells - cytology | Cell Adhesion Molecules - metabolism | beta Catenin - metabolism | Hyperplasia - pathology | Animals | Proteolysis | L-Selectin | Antigens, Neoplasm - metabolism | Mice | Cell Transformation, Neoplastic - pathology | Epithelial Cells - cytology | Cellular proteins | Care and treatment | Hyperplasia | Physiological aspects | Genetic aspects | Research | Carcinogenesis | Index Medicus | Research Paper
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