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1. Full Text 2132-P: Islet Amyloid Deposition Is Cytotoxic to Islet Endothelial Cells
by CASTILLO, JOSEPH J and HOGAN, MEGHAN F and APLIN, ALFRED and HACKNEY, DARYL J and HULL, REBECCA L
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 2132
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2. Full Text 170-OR: Tamoxifen Treatment Causes Insulin Resistance in Obese Mice
by SCALZO, REBECCA L and HULL, SARA E and REUSCH, JANE E.B and MACLEAN, PAUL and WELLBERG, ELIZABETH
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 170
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3. Full Text 2001-P: Thermoneutrality Dissociates Glucose Tolerance from Insulin Sensitivity in Rats
by KELLER, AMY C and KNAUB, LESLIE and HULL, SARA E and SCALZO, REBECCA L and WALKER, LORI A and REUSCH, JANE E.B
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 2001
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4. Full Text Age-Related Attenuation of Aerobic Exercise Training Adaptation in 24-Week Old Rats
by SCALZO, REBECCA L and EVANS, GRAHAME F and HULL, SARA E and KNAUB, LESLIE and WALKER, LORI A and REUSCH, JANE E
Diabetes, ISSN 0012-1797, 05/2018, Volume 67, Issue Supplement 1, p. 1916
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5. Full Text Progressive Loss of β-Cell Function Leads to Worsening Glucose Tolerance in First-Degree Relatives of Subjects With Type 2 Diabetes
by Miriam Cnop and Josep Vidal and Rebecca L. Hull and Kristina M. Utzschneider and Darcy B. Carr and Todd Schraw and Philipp E. Scherer and Edward J. Boyko and Wilfred Y. Fujimoto and Steven E. Kahn
Diabetes Care, ISSN 0149-5992, 03/2007, Volume 30, Issue 3, pp. 677 - 682
Progressive Loss of β-Cell Function Leads to Worsening Glucose Tolerance in First-Degree Relatives of Subjects With Type 2 Diabetes Miriam Cnop , MD, PHD 1 ,... 
POLYCYSTIC-OVARY-SYNDROME | FASTING GLUCOSE | LIFE-STYLE INTERVENTION | INSULIN-RESISTANCE | HIGH-RISK | ENDOCRINOLOGY & METABOLISM | SENSITIVITY | FOLLOW-UP | MELLITUS | SECRETION | DYSFUNCTION | Body Mass Index | Insulin-Secreting Cells - physiology | Glucose Tolerance Test | Glucose Intolerance - epidemiology | Prediabetic State - genetics | Blood Glucose - analysis | Follow-Up Studies | Body Size | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | Risk Factors | Insulin Resistance | Insulin - blood | Adiponectin - blood | Diabetes Mellitus, Type 2 - physiopathology | Sensitivity and Specificity | Adult | Family | Weight Gain
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6. Full Text Mechanisms linking obesity to insulin resistance and type 2 diabetes
by Utzschneider, Kristina M and Kahn, Steven E and Hull, Rebecca L
Nature, ISSN 0028-0836, 12/2006, Volume 444, Issue 7121, pp. 840 - 846
Obesity is associated with an increased risk of developing insulin resistance and type 2 diabetes. In obese individuals, adipose tissue releases increased... 
SECRETORY CAPACITY | FREE FATTY-ACIDS | HIGH-RISK | MULTIDISCIPLINARY SCIENCES | ADAPTATION | SENSITIVITY | IMPAIRED GLUCOSE-TOLERANCE | HYPERINSULINEMIA | DYSFUNCTION | BETA-CELL FUNCTION | ADIPOSE-TISSUE | Diabetes Mellitus, Type 2 - physiopathology | Islets of Langerhans - pathology | Obesity - complications | Diabetes Mellitus, Type 2 - genetics | Humans | Insulin Resistance - genetics | Islets of Langerhans - physiopathology | Diabetes Mellitus, Type 2 - etiology | Obesity - physiopathology | Diabetes Mellitus, Type 2 - pathology | Obesity - genetics | Obesity | Genetics | Diabetes | Molecular biology | Drug resistance | Insulin
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7. Full Text Activation of the NLRP3 inflammasome by islet amyloid polypeptide provides a mechanism for enhanced IL-1β in type 2 diabetes
by Mullooly, Niamh and Mills, Kingston H G and O'Neill, Luke A J and Kahn, Steven E and Mok, K Hun and Chen, Zhe and Tannahill, Gillian M and Dunne, Aisling and Becker, Christine and Sharp, Fiona A and Lavelle, Ed C and Yoshihara, Eiji and Mielke, Lisa A and Franchi, Luigi and Yodoi, Junji and Subramanian, Shoba L and Harris, James and Coll, Rebecca C and Newsholme, Philip and Masters, Seth L and Hull, Rebecca L and Nuñez, Gabriel
Nature Immunology, ISSN 1529-2908, 10/2010, Volume 11, Issue 10, pp. 897 - 904
Interleukin 1 beta (IL-1 beta) is an important inflammatory mediator of type 2 diabetes. Here we show that oligomers of islet amyloid polypeptide (IAPP), a... 
HUMAN PANCREATIC-ISLETS | IN-VITRO | AMYLIN | INSULIN-RESISTANCE | MOUSE MODEL | LOW-DENSITY-LIPOPROTEIN | BETA-CELL MASS | MELLITUS | IMMUNOLOGY | NALP3 INFLAMMASOME | TRANSGENIC MICE | NLR Family, Pyrin Domain-Containing 3 Protein | Dendritic Cells - immunology | Humans | Diabetes Mellitus, Type 2 - metabolism | Amyloid - metabolism | Glyburide - pharmacology | Interleukin-1beta - metabolism | Islets of Langerhans - metabolism | Diabetes Mellitus, Type 2 - immunology | Dendritic Cells - metabolism | Macrophages - immunology | Mice, Inbred C57BL | Carrier Proteins - antagonists & inhibitors | Cells, Cultured | Rats | Mice, Transgenic | Receptors, Cytoplasmic and Nuclear - genetics | Hypoglycemic Agents - pharmacology | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Islet Amyloid Polypeptide | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Mice | Receptors, Cytoplasmic and Nuclear - metabolism
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8. Full Text The islet endothelial cell: a novel contributor to beta cell secretory dysfunction in diabetes
by Hogan, Meghan F and Hull, Rebecca L
Diabetologia, ISSN 0012-186X, 6/2017, Volume 60, Issue 6, pp. 952 - 959
The pancreatic islet is highly vascularised, with an extensive capillary network. In addition to providing nutrients and oxygen to islet endocrine cells and... 
Endothelial cell | Human Physiology | Metabolic Diseases | Internal Medicine | Inflammation | Islet | Review | Insulin secretion | Medicine & Public Health | Beta cell | Capillary | Microvasculature | Diabetes | OXIDATIVE STRESS | INSULIN-SECRETION | GROWTH FACTOR-A | ANGIOTENSIN-II | BLOOD-FLOW | ENDOCRINE PANCREAS | GLUCOSE | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | MICE | BASEMENT-MEMBRANE | Feces - microbiology | Diabetes Mellitus, Type 2 - blood | Animals | Diabetes Mellitus, Type 2 - microbiology | Islets of Langerhans - metabolism | Humans | Islets of Langerhans - physiopathology | Gastrointestinal Microbiome - physiology | Diabetes Mellitus, Type 2 - metabolism | Blood Glucose - metabolism | Type 2 diabetes | Physiological aspects | Pancreatic beta cells | Analysis | Endothelium | Beta cells | Glucose | Pancreas | Cells | Endocrinology
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9. Full Text Impact of Intra-Abdominal Fat and Age on Insulin Sensitivity and β-Cell Function
by Kristina M. Utzschneider and Darcy B. Carr and Rebecca L. Hull and Keiichi Kodama and Jane B. Shofer and Barbara M. Retzlaff and Robert H. Knopp and Steven E. Kahn
Diabetes, ISSN 0012-1797, 11/2004, Volume 53, Issue 11, pp. 2867 - 2872
Impact of Intra-Abdominal Fat and Age on Insulin Sensitivity and β-Cell Function Kristina M. Utzschneider 1 , Darcy B. Carr 2 , Rebecca L. Hull 1 , Keiichi... 
BODY-FAT | MINIMAL MODEL | MEN | ENDOCRINOLOGY & METABOLISM | RESISTANCE | VISCERAL ADIPOSITY | INTOLERANCE | PANCREATIC B-CELL | JAPANESE-AMERICANS | SECRETION | GLUCOSE-TOLERANCE | Body Composition | Body Mass Index | Insulin - pharmacology | Fasting | Islets of Langerhans - physiology | Age Factors | Cross-Sectional Studies | Insulin - physiology | Humans | Middle Aged | Insulin - blood | Abdomen | Blood Glucose - drug effects | Aging - physiology | Adult | Female | Aged | Blood Glucose - metabolism | Adipose Tissue - anatomy & histology
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10. Full Text Activation of the NLRP3 inflammasome by islet amyloid polypeptide provides a mechanism for enhanced IL-1β 2 in type 2 diabetes
by Masters, Seth L and Dunne, Aisling and Subramanian, Shoba L and Hull, Rebecca L and Tannahill, Gillian M and Sharp, Fiona A and Becker, Christine and Franchi, Luigi and Yoshihara, Eiji and Chen, Zhe and Mullooly, Niamh and Mielke, Lisa A and Harris, James and Coll, Rebecca C and Mills, Kingston H. G and Mok, K Hun and Newsholme, Philip and Nuñez, Gabriel and Yodoi, Junji and Kahn, Steven E and Lavelle, Ed C and O'Neill, Luke A. J
Nature Immunology, ISSN 1529-2908, 10/2010, Volume 11, Issue 10, pp. 897 - 904
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11. Full Text Neprilysin Deficiency Is Associated With Expansion of Islet β-Cell Mass in High Fat-Fed Mice
by Parilla, Jacqueline H and Hull, Rebecca L and Zraika, Sakeneh
Journal of Histochemistry & Cytochemistry, ISSN 0022-1554, 7/2018, Volume 66, Issue 7, pp. 523 - 530
Neprilysin (NEP) is an endopeptidase known to modulate nervous, cardiovascular, and immune systems via inactivation of regulatory peptides. In addition, it may... 
β-cell mass | type 2 diabetes | neprilysin | α-cell mass | high-fat diet | metalloendopeptidase | islet
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12. Full Text Glucagon‐like peptide‐1 receptor antagonism impairs basal exercise capacity and vascular adaptation to aerobic exercise training in rats
by Scalzo, Rebecca L and Knaub, Leslie A and Hull, Sara E and Keller, Amy C and Hunter, Kendall and Walker, Lori A and Reusch, Jane E. B
Physiological Reports, 07/2018, Volume 6, Issue 13, pp. e13754 - n/a
Cardiorespiratory fitness (CRF) inversely predicts cardiovascular (CV) mortality and CRF is impaired in people with type 2 diabetes (T2D). Aerobic exercise... 
Aortic strain | vascular stiffness | mitochondrial respiration | Heart | Glucagon | Diabetes mellitus | Mortality | Vasoconstriction | Aerobics | Corticotropin-releasing hormone | Vasodilation | Physical fitness | Mitochondria | Exercise | Rodents | Phenylephrine | Aorta | Aerobic capacity | Acetylcholine | Ventricle | Electron transport | Cardiovascular diseases | Respiration | Diabetes mellitus (non-insulin dependent) | Fitness training programs | Adaptation | Fitness
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13. One year of sitagliptin treatment protects against islet amyloid-associated [Beta]-cell loss and does not induce pancreatitis or pancreatic neoplasia in mice
by Kathryn Aston-Mourney and Shoba L Subramanian and Sakeneh Zraika and Thanya Samarasekera and Daniel T Meier and Lynn C Goldstein and Rebecca L Hull
American Journal of Physiology, ISSN 0193-1849, 08/2013, Volume 305, Issue 4, p. E475
  The dipeptidyl peptidase-4 (DPP-4) inhibitor sitagliptin is an attractive therapy for diabetes, as it increases insulin release and may preserve β-cell mass.... 
Polypeptides | Toxicity | Medical treatment | Rodents | Diabetes | Insulin
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14. Full Text Increased Dietary Fat Promotes Islet Amyloid Formation and β-Cell Secretory Dysfunction in a Transgenic Mouse Model of Islet Amyloid
by Rebecca L. Hull and Sofianos Andrikopoulos and C. Bruce Verchere and Josep Vidal and Feng Wang and Miriam Cnop and Ronald L. Prigeon and Steven E. Kahn
Diabetes, ISSN 0012-1797, 02/2003, Volume 52, Issue 2, pp. 372 - 379
Increased Dietary Fat Promotes Islet Amyloid Formation and β-Cell Secretory Dysfunction in a Transgenic Mouse Model of Islet Amyloid Rebecca L. Hull , Sofianos... 
POLYPEPTIDE AMYLIN | PERFUSED PANCREAS | OBESITY | PANCREATIC-ISLETS | C57BL/6J MICE | ACIDS | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | TYPE-2 DIABETES-MELLITUS | EXPRESSION | GLUCOSE-TOLERANCE | Amyloid - genetics | Islets of Langerhans - drug effects | Pancreatic Polypeptide - metabolism | Amyloid - physiology | Glucose Tolerance Test | Body Weight | Insulin - analysis | Humans | Male | Mice, Transgenic | Glucose Intolerance - blood | Animals | Islet Amyloid Polypeptide | Dietary Fats - pharmacology | Islets of Langerhans - metabolism | Islets of Langerhans - physiopathology | Glucagon - metabolism | Mice | Blood Glucose - metabolism | Somatostatin - metabolism | Glucose Intolerance - physiopathology
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15. Full Text Intra-Abdominal Fat Is a Major Determinant of the National Cholesterol Education Program Adult Treatment Panel III Criteria for the Metabolic Syndrome
by Darcy B. Carr and Kristina M. Utzschneider and Rebecca L. Hull and Keiichi Kodama and Barbara M. Retzlaff and John D. Brunzell and Jane B. Shofer and Brian E. Fish and Robert H. Knopp and Steven E. Kahn
Diabetes, ISSN 0012-1797, 08/2004, Volume 53, Issue 8, pp. 2087 - 2094
Intra-Abdominal Fat Is a Major Determinant of the National Cholesterol Education Program Adult Treatment Panel III Criteria for the Metabolic Syndrome Darcy B.... 
VISCERAL FAT | OBESITY | RISK-FACTORS | SYNDROME-X | INSULIN-RESISTANCE | SERUM-LEVELS | ENDOCRINOLOGY & METABOLISM | NECROSIS-FACTOR-ALPHA | CORONARY-HEART-DISEASE | ABDOMINAL FAT | ADIPOSE-TISSUE | Blood Pressure | Blood Glucose - analysis | Lipoproteins, HDL - blood | Cholesterol - blood | Humans | Middle Aged | Reference Values | Washington | Abdomen | Patient Selection | Metabolic Syndrome - blood | Triglycerides - blood | Adult | Adipose Tissue - anatomy & histology | Metabolic Syndrome - rehabilitation | Patient Education as Topic | Adipose tissues | Measurement | Obesity | Causes of | Insulin resistance | Health aspects | Risk factors
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16. Full Text Treatment of Hyperglycemia with Empagliflozin, With or Without Metformin, Improves Islet Endothelial Cell Health and Insulin Secretion in db/db Diabetic Mice
by HOGAN, MEGHAN F and HACKNEY, DARYL J and APLIN, ALFRED and MUNDINGER, THOMAS O and ZRAIKA, SAKENEH and HULL, REBECCA L
Diabetes, ISSN 0012-1797, 05/2018, Volume 67, Issue Supplement 1, p. 1815
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