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Pancreas, ISSN 0885-3177, 2016, Volume 45, Issue 3, pp. 443 - 457
Journal Article
Theranostics, ISSN 1838-7640, 2019, Volume 9, Issue 2, pp. 324 - 336
K-ras mutation and p53 loss are the most prevalent genetic alterations in pancreatic cancer. In addition to these two alterations, pancreatic tumors frequently... 
Platelet-derived growth factor (PDGF) | β-catenin | Src kinase | Metastasis | Pancreatic cancer | beta-catenin | TRANSFORMATION | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | RNF43 | PHASE-II | DASATINIB | platelet-derived growth factor (PDGF) | P53 | pancreatic cancer | APC | metastasis | KRAS | WNT
Journal Article
Pancreatology, ISSN 1424-3903, 2015, Volume 15, Issue 3, pp. S35 - S36
  Introduction: Foretinib is a multiple kinase inhibitor undergoing different phase clinical trials and exhibits potent inhibitory effect on hepatocyte growth... 
Endocrinology & Metabolism | Gastroenterology and Hepatology | Studies | Angiogenesis | Cell growth | Vascular endothelial growth factor | Pancreatic cancer | Apoptosis
Journal Article
Cellular Signalling, ISSN 0898-6568, 2008, Volume 20, Issue 6, pp. 1134 - 1141
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 2013, Volume 12, Issue 11, pp. 2505 - 2516
Journal Article
PLoS ONE, ISSN 1932-6203, 2012, Volume 7, Issue 4, p. e35078
Background: Oral cancer, which is the fourth most common cancer in Taiwanese men, is associated with environmental carcinogens. The possibility that genetic... 
FUNCTIONAL POLYMORPHISM | PROMOTER POLYMORPHISMS | DIFFERENTIAL EXPRESSION | TAIWAN | BIOLOGY | TUMOR | RISK | SQUAMOUS-CELL CARCINOMAS | NF-KAPPA-B | ASSOCIATION | PROGRESSION | Smoking - adverse effects | Promoter Regions, Genetic | NF-KappaB Inhibitor alpha | Disease Susceptibility | Genetic Association Studies | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Male | NF-kappa B p50 Subunit - genetics | Alcohol Drinking - adverse effects | I-kappa B Proteins - genetics | Polymorphism, Genetic | Areca - adverse effects | Asian Continental Ancestry Group | Taiwan | Mouth Neoplasms - pathology | Adult | Female | Aged | Mouth Neoplasms - genetics | Squamous cell carcinoma | Analysis | Genes | Development and progression | Genetic aspects | Disease susceptibility | B cells | Mouth cancer | Genetic polymorphisms | Cancer | Carriages | Oxidative stress | Oral cancer | DNA damage | Biochemistry | Kinases | Gene polymorphism | Carcinogenesis | Metastases | Proteins | Mens health | Carcinogens | Cell growth | Fibroblasts | Genotypes | Medical research | NF-κB protein | Sarcoidosis | Health risks | Environmental factors | Oral squamous cell carcinoma | Circadian rhythm | Metabolism | Gene expression | Real time | Patients | Homozygotes | Medicine | Polymerase chain reaction | Signaling | Tobacco | Hospitals | Medical prognosis | Alleles | Hypoxia | Mutation | Tumors | Apoptosis | Polymorphism | Nicotine
Journal Article
Journal of Intelligent and Fuzzy Systems, ISSN 1064-1246, 2018, Volume 35, Issue 6, pp. 6229 - 6239
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 05/2010, Volume 223, Issue 2, pp. 492 - 499
Aberrant sialylation catalyzed by sialyltransferases (STs) is frequently found in cancer cells and is associated with increased cancer metastasis. However, ST... 
OVEREXPRESSION | ADHESION | PHYSIOLOGY | RAS ONCOGENE | IN-VIVO | FIBRONECTIN | PROTEIN-TYROSINE KINASE | E-CADHERIN | SIALIC ACIDS | EXPRESSION | CARCINOMA | CELL BIOLOGY | Lung Neoplasms - drug therapy | RNA-Binding Proteins - genetics | Membrane Glycoproteins - metabolism | Humans | Neoplasm Invasiveness - prevention & control | Extracellular Matrix - metabolism | Lithocholic Acid - chemical synthesis | Lung Neoplasms - physiopathology | Integrins - metabolism | Cell Movement - physiology | Extracellular Matrix - chemistry | Dose-Response Relationship, Drug | Lithocholic Acid - pharmacology | Neoplasm Metastasis - drug therapy | Neoplasm Metastasis - prevention & control | Carcinoma - enzymology | Sialyltransferases - antagonists & inhibitors | Female | Antineoplastic Agents - pharmacology | Carcinoma - drug therapy | Lung Neoplasms - enzymology | Extracellular Matrix - drug effects | Enzyme Inhibitors - pharmacology | RNA Splicing Factors | Treatment Outcome | Cell Adhesion - drug effects | Glycolipids - metabolism | Sialyltransferases - metabolism | N-Acetylneuraminic Acid - metabolism | Carcinoma - physiopathology | Mice, Knockout | Neoplasm Invasiveness - physiopathology | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Cell Adhesion - physiology | Neoplasm Metastasis - physiopathology | Cell Line, Tumor | Signal Transduction - physiology | Mice | Mice, Inbred BALB C | Integrins - antagonists & inhibitors | Lithocholic Acid - analogs & derivatives
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, p. e47298
Substantial evidence supports the oncogenic role of the E3 ubiquitin ligase S-phase kinase-associated protein 2 (Skp2) in many types of cancers through its... 
ANAPHASE-PROMOTING COMPLEX | DESTRUCTION | PROTEIN | UBIQUITIN LIGASE | BETA-TRCP | COP9 SIGNALOSOME | MULTIDISCIPLINARY SCIENCES | DEGRADATION | SCFSKP2 | PHOSPHORYLATION-DEPENDENT PROTEOLYSIS | CYCLE | Sirtuin 1 - metabolism | Prostatic Neoplasms - metabolism | S-Phase Kinase-Associated Proteins - physiology | Peptide Hydrolases - genetics | Humans | Gene Expression Regulation, Neoplastic | Male | Multiprotein Complexes - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Breast Neoplasms - metabolism | Multiprotein Complexes - metabolism | Breast Neoplasms - enzymology | beta-Transducin Repeat-Containing Proteins - metabolism | Female | Binding Sites | Intracellular Signaling Peptides and Proteins - genetics | Peptide Hydrolases - metabolism | Mutagenesis, Site-Directed | COP9 Signalosome Complex | S-Phase Kinase-Associated Proteins - metabolism | beta-Transducin Repeat-Containing Proteins - chemistry | Feedback, Physiological | Energy Metabolism | Sirtuin 1 - physiology | Cell Line, Tumor | Prostatic Neoplasms - enzymology | S-Phase Kinase-Associated Proteins - genetics | Ligases | Cancer cells | Physiological aspects | Genetic aspects | Research | Properties | Prostate cancer | Risk factors | Ubiquitin | Phosphorylation | Crosstalk | Transducin | Proline | Feedback loops | Glucose | Kinases | Accumulation | Deoxyglucose | Cullin | Proteins | Cyclin-dependent kinase | Ubiquitination | Energy | Feedback | Cell cycle | Pharmaceutical sciences | Ubiquitin-protein ligase | Medical research | Enzymes | F-box protein | Breast cancer | Gene expression | Sp1 protein | SIRT1 protein | Substrates | Cyclin-dependent kinase 2 | Signaling | Skp2 protein | Skp2 gene | Medical prognosis | Pharmacy | Aberration | Drug discovery | Destabilization | Prostate | Cancer
Journal Article