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1. Full Text Abstract IA15: Rarer, yet both interesting and relevant: Other ovarian cancers
by Huntsman, David G
Clinical Cancer Research, ISSN 1078-0432, 01/2016, Volume 22, Issue 2 Supplement, pp. IA15 - IA15
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2. Full Text Distinct developmental trajectories of endometriotic epithelium and stroma: implications for the origins of endometriosis
: Distinct developmental patterns of endometriotic epithelium and stroma
by Lac, Vivian and Huntsman, David G
The Journal of Pathology, ISSN 0022-3417, 11/2018, Volume 246, Issue 3, pp. 257 - 260
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3. Full Text Abstract 5610: Circulating tumor DNA: FOXL2 402C-G mutation can be identified in plasma from adult granulosa cell tumor patients with recurrent disease
by McConechy, Melissa K and Farkkila, Anniina and Yang, Winnie and Andersson, Noora and Ng, Ying and Unkila-Kallio, Leila and McAlpine, Jessica N and Gilks, Blake and Anttonen, Mikko and Huntsman, David G
Cancer Research, ISSN 0008-5472, 10/2014, Volume 74, Issue 19 Supplement, pp. 5610 - 5610
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4. Full Text Abstract IA8: Discovering drivers in rare ovarian cancer subtypes
by Huntsman, David G
Clinical Cancer Research, ISSN 1078-0432, 10/2013, Volume 19, Issue 19 Supplement, pp. IA8 - IA8
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5. Full Text Boveri at 100: Theodor Boveri and genetic predisposition to cancer
by Hansford, Samantha and Huntsman, David G
The Journal of Pathology, ISSN 0022-3417, 10/2014, Volume 234, Issue 2, pp. 142 - 145
One hundred years have passed since the publication of Theodore Boveri's Zur Frage der Entstehung maligner Tumouren [Concerning the Origin of Malignant... 
genetics | chromosomal theory | chromosome | tumour suppressor gene | inheritance | cancer | hereditary | genomic instability | Tumour suppressor gene | Inheritance | Chromosome | Genetics | Hereditary | Cancer | Chromosomal theory | Genomic instability | RETINOBLASTOMA | ONCOLOGY | MUTATIONS | PATHOLOGY | Genetic Predisposition to Disease - genetics | Neoplasms - genetics | History, 20th Century | Humans | Chromosome Aberrations | History, 19th Century | Genetic Predisposition to Disease - history | Medical Oncology - history | Germany | Genetic research | Development and progression | Genetic aspects | Index Medicus
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6. Full Text Identification of 12 new susceptibility loci for different histotypes of epithelial ovarian cancer
by Phelan, Catherine M and Kuchenbaecker, Karoline B and Tyrer, Jonathan P and Kar, Siddhartha P and Lawrenson, Kate and Winham, Stacey J and Dennis, Joe and Pirie, Ailith and Riggan, Marjorie J and Chornokur, Ganna and Earp, Madalene A and Lyra, Paulo C and Lee, Janet M and Coetzee, Simon and Beesley, Jonathan and McGuffog, Lesley and Soucy, Penny and Dicks, Ed and Lee, Anew and Barrowdale, Daniel and Lecarpentier, Julie and Leslie, Goska and Aalfs, Cora M and Aben, Katja K. H and Adams, Marcia and Adlard, Julian and Anulis, Irene L and Anton-Culver, Hoda and Antonenkova, Natalia and Aravantinos, Gerasimos and Arnold, Norbert and Arun, Banu K and Arver, Brita and Azzollini, Jacopo and Balmaña, Judith and Banerjee, Susana N and Barjhoux, Laure and Barkardottir, Rosa B and Bean, Yukie and Beckmann, Matthias W and Beeghly-Fadiel, Alicia and Benitez, Javier and Bermisheva, Marina and Bernardini, Marcus Q and Birrer, Michael J and Bjorge, Line and Black, Amanda and Blankstein, Kenneth and Blok, Marinus J and Bodelon, Clara and Bogdanova, Natalia and Bojesen, Anders and Bonanni, Bernardo and Borg, Åke and Bradbury, Angela R and Brenton, James D and Brewer, Carole and Brinton, Louise and Broberg, Per and Brooks-Wilson, Angela and Bruinsma, Fiona and Brunet, Joan and Buecher, Bruno and Butzow, Ralf and Buys, Sauna S and Caldes, Trinidad and Caligo, Maria A and Campbell, Ian and Cannioto, Rikki and Carney, Michael E and Cescon, Terence and Chan, Salina B and Chang-Claude, Jenny and Chanock, Stephen and Chen, Xiao Qing and Chiew, Yoke-Eng and Chiquette, Jocelyne and Chung, Wendy K and Claes, Kathleen B. M and Conner, Thomas and Cook, Linda S and Cook, Jackie and Cramer, Daniel W and Cunningham, Julie M and D'Aloisio, Aimee A and Daly, Mary B and Damiola, Francesca and Damirovna, Sakaeva Dina and Dansonka-Mieszkowska, Agnieszka and Dao, Fanny and Davidson, Rosemarie and Defazio, Anna and Delnatte, Capucine and Doheny, Kimberly F and Diez, Orland and Ding, Yuan Chun and Doherty, Jennifer Anne and Domchek, Susan M and Dorfling, Cecilia M and Dörk, Thilo and ... and KConFab Investigators and EMEMBRACE Study and GEMO Study Collaborators and OPAL Study Grp and AOCS Study Grp and HEBON Study and AOCS study group and EMBRACE Study and OPAL study group and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Ortopedi and Uppsala universitet and Institutionen för kirurgiska vetenskaper
Nature genetics, ISSN 1061-4036, 2017, Volume 49, Issue 5, pp. 680 - 691
To identify common alleles associated with different histotypes of epithelial ovarian cancer (EOC), we pooled data from multiple genome-wide genotyping... 
BREAST-CANCER | COMMON VARIANTS | POPULATION | METAANALYSIS | GENETICS & HEREDITY | RISK | MUTATIONS | CLEAR-CELL CARCINOMA | ASSOCIATION | ENDOMETRIOSIS | TELOMERE LENGTH | Neoplasms, Glandular and Epithelial - pathology | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Meta-Analysis as Topic | Humans | Risk Factors | Genetic Loci - genetics | Ovarian Neoplasms - pathology | Genotype | Ovarian Neoplasms - genetics | BRCA1 Protein - genetics | Neoplasms, Glandular and Epithelial - genetics | Telomere-Binding Proteins - genetics | Alleles | Carcinoma, Ovarian Epithelial | Female | Polymorphism, Single Nucleotide | Mutation | BRCA2 Protein - genetics | Genetic susceptibility | Physiological aspects | Genetic aspects | Single nucleotide polymorphisms | Identification and classification | Health aspects | Risk factors | Ovarian cancer | Carriers | Medical research | Ovarian carcinoma | Genes | Gene regulation | Genomes | Breast cancer | Loci | Cancer | Index Medicus | Life Sciences | Human health and pathology | Genetics | Medical and Health Sciences | Medicin och hälsovetenskap
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7. Full Text Shared heritability and functional enrichment across six solid cancers
by Jiang, Xia and Jiang, Xia and Finucane, Hilary K and Finucane, Hilary K and Schumacher, Fredrick R and Schumacher, Fredrick R and Schmit, Stephanie L and Tyrer, Jonathan P and Tyrer, Jonathan P and Han, Younghun and Han, Younghun and Michailidou, Kyriaki and Michailidou, Kyriaki and Lesseur, Corina and Kuchenbaecker, Karoline B and Kuchenbaecker, Karoline B and Dennis, Joe and Conti, David V and Casey, Graham and Gaudet, Mia M and Huyghe, Jeroen R and Albanes, Demetrius and Aldrich, Melinda C and Aldrich, Melinda C and Andrew, Angeline S and Andrew, Angeline S and Andrulis, Irene L and Anton-Culver, Hoda and Anton-Culver, Hoda and Antoniou, Antonis C and Antoniou, Antonis C and Antonenkova, Natalia N and Antonenkova, Natalia N and Arnold, Susanne M and Aronson, Kristan J and Aronson, Kristan J and Arun, Banu K and Arun, Banu K and Bandera, Elisa V and Bandera, Elisa V and Barkardottir, Rosa B and Barnes, Daniel R and Batra, Jyotsna and Beckmann, Matthias W and Benitez, Javier and Benitez, Javier and Benlloch, Sara and Benlloch, Sara and Berchuck, Andrew and Berchuck, Andrew and Berndt, Sonja I and Bickeböller, Heike and Bien, Stephanie A and Bien, Stephanie A and Blomqvist, Carl and Blomqvist, Carl and Boccia, Stefania and Bogdanova, Natalia V and Bogdanova, Natalia V and Bojesen, Stig E and Bolla, Manjeet K and Bolla, Manjeet K and Brauch, Hiltrud and Brenner, Hermann and Brenton, James D and Brenton, James D and Brook, Mark N and Brook, Mark N and Brunet, Joan and Brunet, Joan and Brunnström, Hans and Buchanan, Daniel D and Buchanan, Daniel D and Burwinkel, Barbara and Burwinkel, Barbara and Butzow, Ralf and Cadoni, Gabriella and Caldés, Trinidad and Caldés, Trinidad and Caligo, Maria A and Caligo, Maria A and Campbell, Ian and Campbell, Ian and Campbell, Peter T and Cancel-Tassin, Géraldine and Cancel-Tassin, Géraldine and Cannon-Albright, Lisa and Cannon-Albright, Lisa and Campa, Daniele and Campa, Daniele and Caporaso, Neil and Carvalho, André L and Carvalho, André L and Chan, Andrew T and Chan, Andrew T and Chang-Claude, Jenny and Chang-Claude, Jenny and Chanock, Stephen J and Chanock, Stephen J and Chen, Chu and ...
Nature Communications, ISSN 2041-1723, 01/2019, Volume 10, Issue 1, pp. 431 - 23
Quantifying the genetic correlation between cancers can provide important insights into the mechanisms driving cancer etiology. Using genome-wide association... 
BREAST-CANCER | LUNG-CANCER | RISK-FACTORS | PARTITIONING HERITABILITY | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY LOCI | GENETIC ARCHITECTURE | ANALYSES IDENTIFY | MENDELIAN RANDOMIZATION | CELL-TYPES | GENOME-WIDE ASSOCIATION | Lung Neoplasms - ethnology | Colorectal Neoplasms - genetics | Humans | Ovarian Neoplasms - pathology | Lung Neoplasms - pathology | Male | Prostatic Neoplasms - diagnosis | Case-Control Studies | Colorectal Neoplasms - diagnosis | Ovarian Neoplasms - ethnology | Inheritance Patterns | Ovarian Neoplasms - genetics | Mental Disorders - genetics | Prostatic Neoplasms - genetics | Smoking - physiopathology | Head and Neck Neoplasms - ethnology | Female | Neoplasm Proteins - genetics | Breast Neoplasms - ethnology | Lung Neoplasms - genetics | Prostatic Neoplasms - pathology | Genetic Predisposition to Disease | Genome-Wide Association Study | Colorectal Neoplasms - ethnology | Ovarian Neoplasms - diagnosis | European Continental Ancestry Group | Smoking - genetics | Prostatic Neoplasms - ethnology | Head and Neck Neoplasms - pathology | Mental Disorders - physiopathology | Smoking - ethnology | Phenotype | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Head and Neck Neoplasms - diagnosis | Head and Neck Neoplasms - genetics | Breast Neoplasms - diagnosis | Mental Disorders - ethnology | Polymorphism, Single Nucleotide | Colorectal Neoplasms - pathology | Lung Neoplasms - diagnosis | Enrichment | Correlation | Statistical analysis | Ovarian carcinoma | Mental disorders | Lung cancer | Lung | Colorectal carcinoma | Genomes | Breast cancer | Regulatory sequences | Ovarian cancer | Etiology | Breast | Heritability | Neck | Solid tumors | Prostate cancer | Prostate | Cancer | Smoking | Index Medicus
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8. Full Text A recurrent germline PAX5 mutation confers susceptibility to pre-B cell acute lymphoblastic leukemia
by Shah, S and Schrader, K.A and Waanders, E and Timms, A.E and Vijai, J and Miething, C and Wechsler, J and Yang, J and Hayes, J and Klein, R.J and Zhang, Jinghui and Wei, L and Wu, G and Rusch, M and Nagahawatte, P and Ma, J and Chen, S.C and Song, G and Cheng, J and Meyers, P and Bhojwani, D and Jhanwar, S and Maslak, P and Fleisher, M and Littman, J and Offit, L and Rau-Murthy, R and Fleischut, M.H and Corines, M and Murali, R and Gao, X and Manschreck, C and Kitzing, T and Murty, V.V and Raimondi, S.C and Kuiper, R.P and Simons, A and Schiffman, J.D and Onel, K and Plon, S.E and Wheeler, D.A and Ritter, D and Ziegler, D.S and Tucker, K and Sutton, R and Chenevix-Trench, G and Li, J and Huntsman, D.G and Hansford, S and Senz, J and Walsh, T and Lee, M. van der and Hahn, C.N and Roberts, K.G and King, M.C and Lo, S.M and Levine, R.L and Viale, A and Socci, N.D and Nathanson, K.L and Scott, H.S and Daly, M and Lipkin, S.M and Lowe, S.W and Downing, J.R and Altshuler, D and Sandlund, J.T and Horwitz, M.S and Mullighan, C.G and Offit, K
Nature Genetics, ISSN 1061-4036, 2013, Volume 45, Issue 10, pp. 1226 - 1231
Somatic alterations of the lymphoid transcription factor gene PAX5 (also known as BSAP) are a hallmark of B cell precursor acute lymphoblastic leukemia... 
TRANSCRIPTION FACTOR PAX5 | TRANSACTIVATION | GENETICS & HEREDITY | DIFFERENTIATION | DNA-SEQUENCING DATA | IDENTIFICATION | EXPRESSION | CHILDHOOD | CIRCULAR BINARY SEGMENTATION | GENETIC ALTERATIONS | BSAP | Precursor B-Cell Lymphoblastic Leukemia-Lymphoma - genetics | Germ-Line Mutation | Genetic Predisposition to Disease | Polymorphism, Single Nucleotide | Humans | PAX5 Transcription Factor - genetics | Gene mutations | Genetic aspects | Genetic transcription | Research | Acute lymphocytic leukemia | Health aspects | Risk factors | Haplotypes | Genealogy | Genomes | Mutation | Gene expression | Binding sites | Cancer | Index Medicus
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9. Full Text Clear cell carcinomas of the ovary and kidney: clarity through genomics
: Ovarian and renal clear cell carcinomas
by Ji, Jennifer X and Wang, Yi Kan and Cochrane, Dawn R and Huntsman, David G
The Journal of Pathology, ISSN 0022-3417, 04/2018, Volume 244, Issue 5, pp. 550 - 564
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10. Full Text Type Iγ phosphatidylinositol phosphate kinase modulates adherens junction and E-cadherin trafficking via a direct interaction with μ 1B adaptin
by Ling, Kun and Bairstow, Shawn F and Carbonara, Chateen and Turbin, Dmitry A and Huntsman, David G and Anderson, Richard A
Journal of Cell Biology, ISSN 0021-9525, 01/2007, Volume 176, Issue 3, pp. 343 - 353
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11. Full Text Abstract 4318: Identifying drivers of SMARCA4/BRG1-deficient SCCOHT tumorigenesis by integrative multi-omic analysis
by Orlando, Krystal A and Raab, Jesse R and Lang, Jessica D and Hendricks, William P and Wang, Yemin and Huntsman, David G and Trent, Jeffrey M and Parker, Joel S and Weissman, Bernard E
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 4318 - 4318
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12. Full Text Clinical and genetic analysis of recurrent adult-type granulosa cell tumor of the ovary: Persistent preservation of heterozygous c.402C>G FOXL2 mutation
by Yanagida, Satoshi and Anglesio, Michael S and Nazeran, Tayyebeh M and Lum, Amy and Inoue, Momoko and Iida, Yasushi and Takano, Hirokuni and Nikaido, Takashi and Okamoto, Aikou and Huntsman, David G
PLoS ONE, ISSN 1932-6203, 06/2017, Volume 12, Issue 6, pp. e0178989 - e0178989
Background Adult-type granulosa cell tumors of the ovary (aGCTs) are rare tumors that represent 2-5% of ovarian malignancies. The prognosis of this tumor is... 
HETEROGENEITY | THERAPY | MULTIDISCIPLINARY SCIENCES | TERM-FOLLOW-UP | FOXL2 | CANCER | EXPRESSION | CARCINOMA | CHEMOTHERAPY | Immunohistochemistry | Granulosa Cell Tumor - genetics | Humans | Middle Aged | Risk Factors | Genotype | Male | Forkhead Transcription Factors - genetics | Mutation - genetics | Aged, 80 and over | Adult | Female | Heterozygote | Aged | Forkhead Box Protein L2 | Genetic research | Development and progression | Ovaries | Research | Granulosa cell tumor | Data analysis | Prognosis | Preservation | Pathogenesis | Genes | Data processing | Assaying | Disease control | Patients | Survival | Ovarian cancer | Studies | Pathology | Discrimination | Surgery | Genetic analysis | Mutation | Tumors | Index Medicus
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13. Full Text ARID1A Mutations in Endometriosis-Associated Ovarian Carcinomas
by Wiegand, Kimberly C and Shah, Sohrab P and Al-Agha, Osama M and Zhao, Yongjun and Tse, Kane and Zeng, Thomas and Senz, Janine and McConechy, Melissa K and Anglesio, Michael S and Kalloger, Steve E and Yang, Winnie and Heravi-Moussavi, Alireza and Giuliany, Ryan and Chow, Christine and Fee, John and Zayed, Abdalnasser and Prentice, Leah and Melnyk, Nataliya and Turashvili, Gulisa and Delaney, Allen D and Madore, Jason and Yip, Stephen and McPherson, Andrew W and Ha, Gavin and Bell, Lynda and Fereday, Sian and Tam, Angela and Galletta, Laura and Tonin, Patricia N and Provencher, Diane and Miller, Dianne and Jones, Steven J.M and Moore, Richard A and Morin, Gregg B and Oloumi, Arusha and Boyd, Niki and Aparicio, Samuel A and Shih, Ie-Ming and Mes-Masson, Anne-Marie and Bowtell, David D and Hirst, Martin and Gilks, Blake and Marra, Marco A and Huntsman, David G
The New England Journal of Medicine, ISSN 0028-4793, 10/2010, Volume 363, Issue 16, pp. 1532 - 1543
In this study of ovarian clear-cell or endometrioid tumors, nearly half the samples had mutations in the ARID1A gene, which encodes a component of the SWI–SNF... 
MEDICINE, GENERAL & INTERNAL | GRADE SEROUS CARCINOMA | SWI/SNF COMPLEXES | DISTINCT-HISTOLOGIC-TYPE | POOR-PROGNOSIS | MECHANISMS | CLEAR-CELL CARCINOMA | COMPARATIVE GENOMIC HYBRIDIZATION | CANCER | TUMORS | EXPRESSION | Carcinoma, Endometrioid - genetics | Gene Expression | Adenocarcinoma, Clear Cell - metabolism | Humans | Ovarian Neoplasms - pathology | Nuclear Proteins - metabolism | Endometriosis - pathology | Gene Expression Profiling | Transcription Factors - genetics | Endometriosis - complications | Carcinoma, Endometrioid - metabolism | Ovarian Neoplasms - genetics | Transcription Factors - metabolism | Sequence Analysis, RNA | Cell Line, Tumor | Female | Adenocarcinoma, Clear Cell - pathology | Ovarian Neoplasms - metabolism | Mutation | Nuclear Proteins - genetics | Adenocarcinoma, Clear Cell - genetics | Genes, Tumor Suppressor | Carcinoma, Endometrioid - pathology | Care and treatment | Gene mutations | Causes of | Endometriosis | Tumor suppressor genes | Genetic aspects | Research | Ovarian cancer | Medical research | Cancer | Index Medicus | Abridged Index Medicus
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14. Full Text Categorization of cancer through genomic complexity could guide research and management strategies
: Categorization of cancer through genomic complexity
by Horlings, Hugo M and Flanagan, Adrienne M and Huntsman, David G
The Journal of Pathology, ISSN 0022-3417, 08/2015, Volume 236, Issue 4, pp. 397 - 402
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