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The Journal of Cell Biology, ISSN 0021-9525, 12/2009, Volume 187, Issue 7, pp. 1037 - 1054
Journal Article
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 06/2010, Volume 14, Issue 6b, pp. 1760 - 1776
Primary endothelial cells are fully resistant to TNF‐related apoptosis‐inducing ligand (TRAIL)‐mediated apoptosis. Here, we demonstrate that certain... 
endothelial cells | DISC | c‐FLIP | apoptosis | TRAIL receptor | contact hypersensitivity | nickel ions | Nickel ions | C-FLIP | Endothelial cells | Apoptosis | Contact hypersensitivity | MEDICINE, RESEARCH & EXPERIMENTAL | PHOSPHATIDYLINOSITOL 3-KINASE/AKT | DEATH | SIGNALING COMPLEX | CELL BIOLOGY | CASPASE-8 ACTIVATION | GENE-EXPRESSION | TUMORICIDAL ACTIVITY | c-FLIP | NF-KAPPA-B | UP-REGULATION | ECZEMATOUS DERMATITIS | Transcription, Genetic - drug effects | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Caspase 8 - metabolism | Cells, Cultured | Keratinocytes - cytology | Down-Regulation - drug effects | Nickel - immunology | TNF-Related Apoptosis-Inducing Ligand - pharmacology | Allergens - immunology | Keratinocytes - drug effects | Protein Processing, Post-Translational - drug effects | Endothelial Cells - cytology | Keratinocytes - metabolism | Allergens - pharmacology | Cell Membrane - metabolism | Nickel - pharmacology | CASP8 and FADD-Like Apoptosis Regulating Protein - metabolism | Endothelial Cells - enzymology | Umbilical Veins - cytology | Cell Membrane - drug effects | Endothelial Cells - drug effects | RNA, Small Interfering - metabolism | Allergens | Transcription | Gene regulation | Kinases | Dermatitis | Proteins | Signal transduction | Receptors | FLIP protein | Physiology | Deoxyribonucleic acid--DNA | Antigens | Cytokines | RNA-mediated interference | Keratinocytes | Caspase | Environmental factors | Allergies | Environmental conditions | Depletion | Tumor necrosis factor | Ligands | Nickel | Skin | TRAIL protein | Original
Journal Article
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 05/2008, Volume 128, Issue 5, pp. 1134 - 1147
Death ligands not only activate a death program but also regulate inflammatory signalling pathways, for example, through NF-κB induction. Although tumor... 
C-FLIP | NECROSIS-FACTOR-ALPHA | EPIDERMAL-KERATINOCYTES | MEDIATED APOPTOSIS | MAMMALIAN-CELLS | FACTOR RECEPTOR | DEATH DOMAIN PROTEIN | HUMAN KERATINOCYTES | CELL-DEATH | SIGNALING COMPLEX | DERMATOLOGY
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 12/2009, Volume 187, Issue 7, p. 1037
  A role for cellular inhibitors of apoptosis (IAPs [cIAPs]) in preventing CD95 death has been suspected but not previously explained mechanistically. In this... 
Proteins | Molecules | Signal transduction | Kinases | Cells | Apoptosis
Journal Article
Journal Article
The Journal of Investigative Dermatology, ISSN 0022-202X, 05/2008, Volume 128, Issue 5, p. 1134
  Death ligands not only activate a death program but also regulate inflammatory signalling pathways, for example, through NF-kappaB induction. Although tumor... 
Journal Article
Molecular Reproduction and Development, ISSN 1040-452X, 06/2006, Volume 73, Issue 6, pp. 784 - 794
Journal Article
Journal Article