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PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e43721
Oncogenic signaling promotes tumor invasion and metastasis, in part, by increasing the expression of tri- and tetra-branched N-glycans. The branched N-glycans... 
ADVANCED MALIGNANCIES | ENZYMATIC BASIS | INHIBITION | MULTIDISCIPLINARY SCIENCES | GROWTH | SWAINSONINE | N-ACETYLGLUCOSAMINYLTRANSFERASE V | TUMOR-CELLS | MICE | ASPARAGINE-LINKED OLIGOSACCHARIDES | TRANSCRIPTIONAL REGULATION | Neoplasm Transplantation | Signal Transduction | Humans | Enzyme Inhibitors - pharmacology | N-Acetylglucosaminyltransferases - genetics | Male | Disease Progression | N-Acetylglucosaminyltransferases - metabolism | Prostatic Neoplasms - therapy | Neoplasm Metastasis | Neoplasms - therapy | Phenotype | Animals | Prostatic Neoplasms - genetics | Neoplasms - genetics | Cell Line, Tumor | Polysaccharides - chemistry | Female | Cell Membrane - metabolism | Mice | HeLa Cells | Swainsonine - pharmacology | RNA, Small Interfering - metabolism | Enzymes | Polysaccharides | Metastasis | Health aspects | Prostate cancer | Cancer | Cell proliferation | Enzyme activity | Transcription | N-Acetylglucosinyldiphosphodolichol N-acetylglucosaminyltransferase | Xenotransplantation | Drug resistance | Cell surface | Metastases | Receptors | Enzymatic activity | Metabolites | Cell cycle | Xenografts | Genetics | Inhibition | Medical research | Phenotypes | Tumor cells | Biophysics | Growth factor receptors | N-glycans | Tumor cell lines | Cervix | Metabolism | Adhesion | Mannosidase | Signaling | Lungs | Medical prognosis | Cell lines | Prostate | Cell migration
Journal Article
Blood, ISSN 0006-4971, 03/2010, Volume 115, Issue 11, pp. 2251 - 2259
Journal Article
Cancer Research, ISSN 0008-5472, 08/2008, Volume 68, Issue 16, pp. 6688 - 6697
Journal Article
Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 15 Supplement, pp. 3039 - 3039
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e95281
Journal Article
Journal Article
Leukemia, ISSN 0887-6924, 01/2019, Volume 33, Issue 1, pp. 37 - 51
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 1, pp. 315 - 328
Journal Article
Blood, ISSN 0006-4971, 11/2018, Volume 132, Issue Supplement 1, pp. 1350 - 1350
Abstract UBA1 is the major ubiquitin-activating enzyme that initiates the ubiquitylation cascade whereby proteins are tagged with mono- or polyubiquitin to... 
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 11/2008, Volume 7, Issue 11, pp. 3546 - 3555
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e60253
We have successfully delivered a reactive alkylating agent, chlorambucil (Cbl), to the mitochondria of mammalian cells. Here, we characterize the mechanism of... 
APOPTOSIS | METABOLISM | PHARMACOKINETICS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | NITROGEN-MUSTARD | RESISTANCE | CHRONIC LYMPHOCYTIC-LEUKEMIA | INDUCTION | CHLORAMBUCIL | PENETRATING PEPTIDES | Antineoplastic Agents, Alkylating - chemical synthesis | Cross-Linking Reagents - chemistry | Leukemia - pathology | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Leukemia - drug therapy | Antineoplastic Agents, Alkylating - pharmacology | DNA, Mitochondrial | Leukemia - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Chlorambucil - pharmacology | Mice, SCID | Necrosis - pathology | Xenograft Model Antitumor Assays | Animals | Chlorambucil - chemical synthesis | Mice, Inbred NOD | Mice | HeLa Cells | Drug Delivery Systems - methods | Cell-Penetrating Peptides - chemistry | Biochemistry | Mitochondrial DNA | Analysis | Leukemia | Cells | Apoptosis | Drug delivery systems | Peptides | Toxicity | DNA damage | Drug resistance | Alkylation | Mammalian cells | Exploitation | Chlorambucil | Proteins | Mitochondria | Cell cycle | Xenografts | Biocompatibility | Pharmaceutical sciences | Deoxyribonucleic acid--DNA | Cbl protein | Medical research | Mortality | Pharmacology | Metabolism | Hospitals | Microscopy | Cell death | Pharmacy | Cancer | Deoxyribonucleic acid | DNA
Journal Article
Blood, ISSN 0006-4971, 11/2019, Volume 134, Issue Supplement_1, pp. 2530 - 2530
Patients with relapsed AML (Acute Myeloid Leukemia) have a poor prognosis and few therapeutic options. Therefore, it remains critical to identify biological... 
Journal Article
Cancer Cell, ISSN 1535-6108, 2011, Volume 20, Issue 5, pp. 674 - 688
Journal Article
Cancer Research, ISSN 0008-5472, 07/2016, Volume 76, Issue 14_Supplement, pp. 210 - 210
A subset of AML cells display unique mitochondrial characteristics such as increased mitochondrial DNA (mtDNA), mt mass, and sensitivity to inhibition of mtDNA... 
Journal Article
Cancer Research, ISSN 0008-5472, 07/2016, Volume 76, Issue 14 Supplement, pp. 210 - 210
Journal Article
Blood, ISSN 0006-4971, 11/2018, Volume 132, Issue Supplement 1, pp. 2795 - 2795
Abstract Hematopoietic cells are arranged in a hierarchy where stem and progenitor cells differentiate into mature blood cells. Likewise, AML (Acute Myeloid... 
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, p. e58367
Recently, we demonstrated that the anti-bacterial agent tigecycline preferentially induces death in leukemia cells through the inhibition of mitochondrial... 
COMPLEX-III | MECHANISM | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | ANTICANCER-DRUG | DNA | MULTIDISCIPLINARY SCIENCES | CYTOTOXICITY ASSAY | LACTIC-ACIDOSIS | FACTOR-I | TISSUES | TRANSLATION | Protein Biosynthesis | Minocycline - pharmacology | Humans | Leukemia, Myeloid, Acute - metabolism | Minocycline - analogs & derivatives | Drug Resistance, Neoplasm | Mitochondrial Proteins - genetics | Glycolysis - drug effects | Gene Expression Regulation, Leukemic - genetics | Glycolysis - genetics | Leukemia, Myeloid, Acute - drug therapy | Hypoxia-Inducible Factor 1, alpha Subunit - biosynthesis | Neoplasm Proteins - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Leukemia, Myeloid, Acute - pathology | Neoplasm Proteins - biosynthesis | Gene Expression Regulation, Leukemic - drug effects | Electron Transport Complex IV - biosynthesis | Mitochondrial Proteins - biosynthesis | Electron Transport Complex IV - genetics | Oxygen Consumption - drug effects | Oxygen Consumption - genetics | Cell Line, Tumor | Anti-Bacterial Agents - pharmacology | Leukemia, Myeloid, Acute - genetics | Proteins | RNA sequencing | Glucose metabolism | RNA | Genes | Protein biosynthesis | Genetic transcription | Adaptations | Phosphorylation | Leukemia | Immunoblotting | Mitochondrial DNA | Antiinfectives and antibacterials | Gene sequencing | Signal transduction | Mitochondria | Cell growth | Health care networks | Rodents | Bacteria | Membrane potential | Inhibition | Deoxyribonucleic acid--DNA | Myeloid leukemia | Oxygen consumption | Metabolism | Electron microscopy | Ribonucleic acid--RNA | Cyclooxygenase-1 | Protein synthesis | Glycolysis | Cristae | Hypoxia | Cyclooxygenase-2 | Tigecycline | Acute myeloid leukemia | Binding sites | Cancer | Deoxyribonucleic acid | Ribonucleic acid
Journal Article