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1. Full Text Self-report data as a tool for subtype identification in genetically-defined Parkinson’s Disease
by Winslow, Ashley R and Hyde, Craig L and Wilk, Jemma B and Eriksson, Nicholas and Cannon, Paul and Miller, Melissa R and Hirst, Warren D
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 1 - 7
Through a targeted recruitment 23andMe has collected DNA and patient-reported symptoms from more than 10,000 subjects reporting a physician-verified diagnosis... 
Parkinson's disease | Data processing | Diagnosis | Mutation | LRRK2 protein | Population genetics | Age | Movement disorders | Deoxyribonucleic acid--DNA
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2. Full Text Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression
by Wray, Naomi R and Ripke, Stephan and Mattheisen, Manuel and Trzaskowski, MacIej and Byrne, Enda M and Abdellaoui, Abdel and Adams, Mark J and Agerbo, Esben and Air, Tracy M and Andlauer, Till M. F and Bacanu, Silviu-Alin and Bækvad-Hansen, Marie and Beekman, Aartjan F. T and Bigdeli, Tim B and Binder, Elisabeth B and Blackwood, Douglas R. H and Bryois, Julien and Buttenschøn, Henriette N and Bybjerg-Grauholm, Jonas and Cai, Na and Castelao, Enrique and Christensen, Jane Hvarregaard and Clarke, Toni-Kim and Coleman, Jonathan I. R and Colodro-Conde, Lucía and Couvy-Duchesne, Baptiste and Craddock, Nick and Crawford, Gregory E and Crowley, Cheynna A and Dashti, Hassan S and Davies, Gail and Deary, Ian J and Degenhardt, Franziska and Derks, Eske M and DIrek, Nese and Dolan, Conor V and Dunn, Erin C and Eley, Thalia C and Eriksson, Nicholas and Escott-Price, Valentina and Kiadeh, Farnush Hassan Farhadi and Finucane, Hilary K and Forstner, Andreas J and Frank, Josef and Gaspar, Héléna A and Gill, Michael and Giusti-Rodríguez, Paola and Goes, Fernando S and Gordon, Scott D and Grove, Jakob and Hall, Lynsey S and Hannon, Eilis and Hansen, Christine Søholm and Hansen, Thomas F and Herms, Stefan and Hickie, Ian B and Hoffmann, Per and Homuth, Georg and Horn, Carsten and Hottenga, Jouke-Jan and Hougaard, David M and Hu, Ming and Hyde, Craig L and Ising, Marcus and Jansen, Rick and Jin, Fulai and Jorgenson, Eric and Knowles, James A and Kohane, Isaac S and Kraft, Julia and Kretzschmar, Warren W and Krogh, Jesper and Kutalik, Zoltán and Lane, Jacqueline M and Li, Yihan and Li, Yun and Lind, Penelope A and Liu, Xiaoxiao and Lu, Leina and MacIntyre, Donald J and MacKinnon, Dean F and Maier, Robert M and Maier, Wolfgang and Marchini, Jonathan and Mbarek, Hamdi and McGrath, Patrick and McGuffin, Peter and Medland, Sarah E and Mehta, DIvya and Middeldorp, Christel M and Mihailov, Evelin and Milaneschi, Yuri and Milani, Lili and Mill, Jonathan and Mondimore, Francis M and Montgomery, Grant W and Mostafavi, Sara and Mullins, Niamh and Nauck, Matthias and Ng, Bernard and ... and Major Depressive Disorder Working and EQTLGEN and eQTLGen and Major Depressive Disorder Working Group of the Psychiatric Genomics Consortium and 23andMe and the Major Depressive Disorder Working Group of the Psychiatric Genomics Consortium
Nature Genetics, ISSN 1061-4036, 05/2018, Volume 50, Issue 5, pp. 668 - 681
Major depressive disorder (MDD) is a common illness accompanied by considerable morbidity, mortality, costs, and heightened risk of suicide. We conducted a... 
SYSTEMATIC ANALYSIS | EUROPE 2010 | METAANALYSIS | MENTAL-DISORDERS | GENETICS & HEREDITY | BODY-MASS INDEX | LOCI | MENDELIAN RANDOMIZATION | LD SCORE REGRESSION | EDUCATIONAL-ATTAINMENT | POLYGENIC RISK | Genetic Predisposition to Disease | Phenotype | Schizophrenia - genetics | Depressive Disorder, Major - genetics | Humans | Risk Factors | Female | Male | Polymorphism, Single Nucleotide | Genome-Wide Association Study - methods | Multifactorial Inheritance | Case-Control Studies | Brain | Phenotypes | Splicing | Mental disorders | Genomics | Schizophrenia | Genomes | Mental depression | Risk analysis | Gene expression | Suicide | Morbidity | Risk factors | Educational attainment | Meta-analysis | Consortia | Liability | Body mass | Etiology | Education | Body size | DNA methylation | Gene loci | Deoxyribonucleic acid--DNA | Index Medicus
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3. Full Text Genetic risk, coronary heart disease events, and the clinical benefit of statin therapy: an analysis of primary and secondary prevention trials
by Mega, Jessica L, Dr and Stitziel, Nathan O, Dr and Smith, J Gustav, MD and Chasman, Daniel I, PhD and Caulfield, Mark J, Prof and Devlin, James J, PhD and Nordio, Francesco, PhD and Hyde, Craig L, PhD and Cannon, Christopher P, Prof and Sacks, Frank M, Prof and Poulter, Neil R, Prof and Sever, Peter S, Prof and Ridker, Paul M, Prof and Braunwald, Eugene, Prof and Melander, Olle, Prof and Kathiresan, Sekar, MD and Sabatine, Marc S, Prof and Kardiovaskulär forskning - hypertoni and Cardiovascular Research - Hypertension and Kardiologi and Lund University and EpiHealth: Epidemiology for Health and Cardiology and Lunds universitet and EXODIAB: Excellence in Diabetes Research in Sweden
Lancet, The, ISSN 0140-6736, 2015, Volume 385, Issue 9984, pp. 2264 - 2271
Summary Background Genetic variants have been associated with the risk of coronary heart disease. In this study, we tested whether or not a composite of these... 
Internal Medicine | LDL CHOLESTEROL | MEDICINE, GENERAL & INTERNAL | POLYMORPHISMS | VARIANTS | RECURRENT MYOCARDIAL-INFARCTION | CARDIOVASCULAR-DISEASE | SUSCEPTIBILITY LOCI | CHROMOSOME 9P21 | PRAVASTATIN | CARDIAC DEATH | ARTERY-DISEASE | Recurrence | Primary Prevention | Numbers Needed To Treat | Coronary Disease - genetics | Risk Assessment | Humans | Hydroxymethylglutaryl-CoA Reductase Inhibitors - therapeutic use | Coronary Disease - drug therapy | Treatment Outcome | Secondary Prevention | Prevention | Medical colleges | Genetic research | Clinical trials | Coronary heart disease | Population genetics | Statins | Cardiovascular agents | Heart attacks | Cardiovascular disease | Genetics | Health risk assessment | Drug therapy | Cholesterol | Index Medicus | Abridged Index Medicus | Kardiologi | Clinical Medicine | Cardiac and Cardiovascular Systems | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
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4. Full Text An atlas of genetic influences on human blood metabolites
by Shin, So-Youn and Fauman, Eric B and Petersen, Ann-Kristin and Krumsiek, Jan and Santos, Rita and Huang, Jie and Arnold, Matthias and Erte, Idil and Forgetta, Vincenzo and Yang, Tsun-Po and Walter, Klaudia and Menni, Cristina and Chen, Lu and Vasquez, Louella and Valdes, Ana M and Hyde, Craig L and Wang, Vicky and Ziemek, Daniel and Roberts, Phoebe and Xi, Li and Grundberg, Elin and Waldenberger, Melanie and Richards, J. Brent and Mohney, Robert P and Milburn, Michael V and John, Sally L and Trimmer, Jeff and Theis, Fabian J and Overington, John P and Suhre, Karsten and Brosnan, M Julia and Gieger, Christian and Kastenmüller, Gabi and Spector, Tim D and Soranzo, Nicole and Multiple Tissue Human Expression R and Multiple Tissue Human Expression Resource (MuTHER) Consortium and The Multiple Tissue Human Expression Resource (MuTHER) Consortium
Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 6, pp. 543 - 550
Genome-wide association scans with high-throughput metabolic profiling provide unprecedented insights into how genetic variation influences metabolism and... 
PROFILES | VARIANTS | GENETICS & HEREDITY | GENOME-WIDE ASSOCIATION | Metabolomics | Blood Chemical Analysis | Humans | Middle Aged | Genetic Loci - genetics | Data Mining | Male | Gene Expression Profiling | Blood - metabolism | Genetic Variation | Young Adult | Aged, 80 and over | Adult | Female | Genome-Wide Association Study | Europe | Computational Biology | Genotype | United Kingdom | Metabolism, Inborn Errors - genetics | Adolescent | Aged | Internet | Germany | Cohort Studies | Quantitative trait loci | Evaluation | Genome-wide association studies | Genetic aspects | Metabolism | Identification and classification | Studies | Medical research | Metabolites | Genes | Genomics | Index Medicus
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5. Full Text Identification of 15 genetic loci associated with risk of major depression in individuals of European descent
by Hyde, Craig L and Nagle, Michael W and Tian, Chao and Chen, Xing and Paciga, Sara A and Wendland, Jens R and Tung, Joyce Y and Hinds, David A and Perlis, Roy H and Winslow, Ashley R
Nature Genetics, ISSN 1061-4036, 09/2016, Volume 48, Issue 9, pp. 1031 - 1036
Despite strong evidence supporting the heritability of major depressive disorder (MDD), previous genome-wide studies were unable to identify risk loci among... 
TRIAL | MEF2C | MOOD DISORDERS | GENETICS & HEREDITY | SCHIZOPHRENIA | EPILEPSY | BURDEN | MEIS2 | DORSAL | BRAIN | GENOME-WIDE ASSOCIATION | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Meta-Analysis as Topic | Humans | Middle Aged | Risk Factors | Genetic Loci - genetics | Genotype | Male | Case-Control Studies | Phenotype | Depressive Disorder, Major - genetics | Polymorphism, Single Nucleotide - genetics | Adult | Female | Quantitative trait loci | Europeans | Genetic aspects | Health aspects | Identification and classification | Major depressive disorder | Risk factors | Neurosciences | Statistical analysis | Disease | Genomics | Schizophrenia | Genomes | Mental depression | Meta-analysis | Datasets | Studies | Consortia | Liability | Population | Index Medicus | genome-wide | GWAS | depression | SNP | MDD | single nucleotide polymorphism
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6. Full Text Genome-Wide Association of Lipid-Lowering Response to Statins in Combined Study Populations
by Barber, Mathew J and Mangravite, Lara M and Hyde, Craig L and Chasman, Daniel I and Smith, Joshua D and McCarty, Catherine A and Li, Xiaohui and Wilke, Russell A and Rieder, Mark J and Williams, Paul T and Ridker, Paul M and Chatterjee, Aurobindo and Rotter, Jerome I and Nickerson, Deborah A and Stephens, Matthew and Krauss, Ronald M
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 3, pp. e9763 - e9763
Background: Stains effectively lower total and plasma LDL-cholesterol, but the magnitude of decrease varies among individuals. To identify single nucleotide... 
C-REACTIVE PROTEIN | DENSITY-LIPOPROTEIN CHOLESTEROL | TREATMENT PANEL-III | THERAPY | GENE | PHARMACOGENOMICS | BIOLOGY | RISK | LOCI | CORONARY-ARTERY-DISEASE | STRATIFICATION | Simvastatin - therapeutic use | Genome-Wide Association Study | Heptanoic Acids - therapeutic use | Humans | Middle Aged | Genotype | Male | Inflammation | Cholesterol - metabolism | Atorvastatin Calcium | Lipids - chemistry | Phenotype | Hydroxymethylglutaryl-CoA Reductase Inhibitors - therapeutic use | Bayes Theorem | Adult | Female | Aged | Polymorphism, Single Nucleotide | Pyrroles - therapeutic use | Pravastatin - therapeutic use | Pharmacogenetics | Blood cholesterol | Analysis | Low density lipoproteins | Simvastatin | Genes | Genomes | Genetic aspects | Single nucleotide polymorphisms | Pravastatin | Apolipoproteins | Medical research | Lipoproteins (low density) | Statistical analysis | Clinical trials | Cardiovascular disease | Population studies | Pharmacology | Single-nucleotide polymorphism | Conditional probability | Low density lipoprotein | Cholesterol | Disease prevention | Apolipoprotein E | Rodents | Atorvastatin | Chromosome 14 | Bayesian analysis | Lipoproteins (high density) | Statins | Index Medicus
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7. Full Text An integrated epigenomic analysis for type 2 diabetes susceptibility loci in monozygotic twins
by Yuan, Wei and Xia, Yudong and Bell, Christopher G and Yet, Idil and Ferreira, Teresa and Ward, Kirsten J and Gao, Fei and Loomis, A. Katrina and Hyde, Craig L and Wu, Honglong and Lu, Hanlin and Liu, Yuan and Small, Kerrin S and Viñuela, Ana and Morris, Andrew P and Berdasco, María and Esteller, Manel and Brosnan, M. Julia and Deloukas, Panos and McCarthy, Mark I and John, Sally L and Bell, Jordana T and Wang, Jun and Spector, Tim D
Nature Communications, ISSN 2041-1723, 2014, Volume 5, Issue 1, pp. 5719 - 5719
DNA methylation has a great potential for understanding the aetiology of common complex traits such as Type 2 diabetes (T2D). Here we perform genome-wide... 
OBESITY | WIDE DNA METHYLATION | HUMAN-DISEASE | HUMAN GENOME | PROTEIN | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | RECEPTOR | REGULATORY ELEMENTS | ASSOCIATION | ADIPOSE-TISSUE | Nerve Tissue Proteins - blood | Diabetes Mellitus, Type 2 - genetics | Epigenesis, Genetic | Humans | Protein Kinase C beta - genetics | Taurocholic Acid - blood | Genetic Loci | Insulin - blood | Case-Control Studies | DNA Methylation | Insulin - genetics | Neoplasm Proteins - genetics | Protein Kinase C beta - blood | Promoter Regions, Genetic | Genetic Predisposition to Disease | Genome-Wide Association Study | Epigenomics | Receptors, G-Protein-Coupled - blood | Caspases - genetics | Biomarkers - blood | Twins, Monozygotic | Nerve Tissue Proteins - genetics | Diabetes Mellitus, Type 2 - blood | CpG Islands | Neoplasm Proteins - blood | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein | Receptors, G-Protein-Coupled - genetics | Caspases - blood | Genome, Human | Index Medicus | Epigenetics | Diabetes | Twins | Bessons | Diabetis | Epigenètica
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8. Full Text Genome-wide scan identifies variation in MLXIPL associated with plasma triglycerides
by Thompson, John F and Chambers, John C and Elliott, Paul and Gómez Pérez, Francisco J and Warnes, Gregory R and Aguilar-Salinas, Carlos A and Milos, Patrice M and Frazer, Kelly A and Hinds, David A and Scott, James and Kooner, Jaspal S and Cox, David R and Hyde, Craig L
Nature Genetics, ISSN 1061-4036, 02/2008, Volume 40, Issue 2, pp. 149 - 151
We tested over 267,000 SNPs in 1,005 Northern Europeans and 248,000 in 1,006 Indian Asians for association with triglycerides and HDL cholesterol, with... 
HDL | OBESITY | GENE | CHREBP | GENETICS & HEREDITY | Humans | Middle Aged | Male | Genetic Variation | Adult | Female | Odds Ratio | Principal Component Analysis | Europe | European Continental Ancestry Group | Linear Models | Genetic Markers | Chromosomes, Human, Pair 7 | India | Linkage Disequilibrium | Homozygote | Asian Continental Ancestry Group | Metabolic Syndrome - genetics | Alleles | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Triglycerides - blood | Mexico | Aged | Polymorphism, Single Nucleotide | Triglycerides - genetics | Genome, Human | Cohort Studies | Physiological aspects | Triglycerides | Genetic aspects | Research | Single nucleotide polymorphisms | Metabolic syndrome X | Diagnosis | Risk factors | Plasma | Genomics | Cholesterol | Polymorphism | Index Medicus
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9. Full Text EARLY DETECTION OF THE UNEXPECTED, HARMFUL, PHARMACOLOGIC EFFECTS OF TORCETRAPIB: A RETROSPECTIVE PROTEOMICS ANALYSIS OF PLASMA SAMPLES FROM THE ILLUMINATE TRIAL
by Williams, Stephen and DeLisle, Robert K and Hyde, Craig L and Malarstig, Anders and Ostroff, Rachel and Weiss, Sophie
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2017, Volume 69, Issue 11, pp. 152 - 152
  Background: Early detection of adverse effects of novel therapies and their mechanistic understanding prior to large-scale clinical trials could prevent harm... 
Cardiovascular | Internal Medicine | CARDIAC & CARDIOVASCULAR SYSTEMS | Analysis | Anticholesteremic agents | Proteins | Health risk assessment | Proteomics
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10. Full Text Comprehensive Whole-Genome and Candidate Gene Analysis for Response to Statin Therapy in the Treating to New Targets (TNT) Cohort
by Thompson, John F and Hyde, Craig L and Wood, Linda S and Paciga, Sara A and Hinds, David A and Cox, David R and Hovingh, G. Kees and Kastelein, John J.P
Circulation: Cardiovascular Genetics, ISSN 0016-6731, 04/2009, Volume 2, Issue 2, pp. 173 - U240
Comprehensive Whole-Genome and Candidate Gene Analysis for Response to Statin Therapy in the Treating to New Targets (TNT) Cohort John F. Thompson, PhD ; Craig... 
Myocardial infarction | Genetics | Hydroxymethylglutaryl coenzyme a reductase inhibitors | Hypercholesterolemia | CARDIAC & CARDIOVASCULAR SYSTEMS | COENZYME-A REDUCTASE | myocardial infarction | POLYMORPHISM | hydroxymethylglutaryl coenzyme A reductase inhibitors | hypercholesterolemia | ATORVASTATIN | DENSITY-LIPOPROTEIN CHOLESTEROL | genetics | GENETICS & HEREDITY | PHARMACOGENETICS | PRAVASTATIN | SIMVASTATIN | ASSOCIATION | PCSK9 GENE | Genome-Wide Association Study | Heptanoic Acids - therapeutic use | Humans | Middle Aged | Genotype | Male | Atorvastatin Calcium | Coronary Disease - genetics | Apolipoproteins E - genetics | Serine Endopeptidases - genetics | Coronary Disease - drug therapy | Adult | Female | Aged | Polymorphism, Single Nucleotide | Proprotein Convertases | Pyrroles - therapeutic use | Cohort Studies | Proprotein Convertase 9 | Index Medicus
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