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humans (5) 5
cancer (4) 4
index medicus (4) 4
antineoplastic agents - administration & dosage (3) 3
apoptosis (3) 3
cell biology (3) 3
cell line, tumor (3) 3
cell survival - drug effects (3) 3
genetic aspects (3) 3
mutation (3) 3
prognosis (3) 3
tumor suppressor protein p53 - genetics (3) 3
adult (2) 2
antibodies, monoclonal, humanized - administration & dosage (2) 2
apoptosis - drug effects (2) 2
blood glucose - analysis (2) 2
breast neoplasms - diagnosis (2) 2
breast neoplasms - epidemiology (2) 2
breast neoplasms - mortality (2) 2
carcinogenesis - drug effects (2) 2
carcinogenesis - genetics (2) 2
carcinoma - diagnosis (2) 2
carcinoma - epidemiology (2) 2
carcinoma - mortality (2) 2
care and treatment (2) 2
cell cycle - drug effects (2) 2
cell proliferation - drug effects (2) 2
chemotherapy (2) 2
deoxyribonucleic acid--dna (2) 2
diagnosis, differential (2) 2
disease progression (2) 2
dna mutational analysis (2) 2
dna repair (2) 2
drug synergism (2) 2
e3 ubiquitin ligase (2) 2
effect modifier, epidemiologic (2) 2
expression (2) 2
fasting glucose (2) 2
fbw7 (2) 2
female (2) 2
furans - pharmacology (2) 2
her2 positive breast cancer (2) 2
imidazoles - pharmacology (2) 2
mesothelioma (2) 2
middle aged (2) 2
mutant p53 (2) 2
neoplasm staging (2) 2
nono (2) 2
oncology (2) 2
p53 status (2) 2
papillary renal carcinoma (2) 2
physiology (2) 2
piperazines - pharmacology (2) 2
receptor, erbb-2 - metabolism (2) 2
research (2) 2
rita (2) 2
survival analysis (2) 2
tfe3 (2) 2
trastuzumab (2) 2
treatment outcome (2) 2
tumor suppressor protein p53 - metabolism (2) 2
adenoviruses (1) 1
analysis (1) 1
antineoplastic agents - pharmacology (1) 1
antitumor-activity (1) 1
asbestos (1) 1
biochemistry (1) 1
bioinformatics (1) 1
bone neoplasms - metabolism (1) 1
bone neoplasms - pathology (1) 1
brain neoplasms - drug therapy (1) 1
brain neoplasms - genetics (1) 1
brain neoplasms - metabolism (1) 1
c-myc (1) 1
cancer cells (1) 1
cancer patients (1) 1
cancer therapies (1) 1
cell death (1) 1
cell growth (1) 1
cell movement - drug effects (1) 1
cell-cycle (1) 1
cell-survival (1) 1
central nervous system (1) 1
childrens oncology group (1) 1
cisplatin - administration & dosage (1) 1
cisplatin - pharmacology (1) 1
clear cell-type renal cell carcinoma (1) 1
colon (1) 1
combination (1) 1
cyclin-dependent kinases (1) 1
death (1) 1
deregulation (1) 1
diagnosis (1) 1
differentiation (1) 1
dl922-947 (1) 1
dna damage (1) 1
dosage and administration (1) 1
down-regulation (1) 1
drug resistance (1) 1
drug resistance, neoplasm - drug effects (1) 1
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1. Full Text Abstract 3747: Oncolytic viruses as a possible therapeutic strategy against malignant mesothelioma
by Iannuzzi, Carmelina Antonella and Passaro, Carmela and Boffo, Silvia and Forte, Iris Maria and Indovina, Paola and Macaluso, Marcella and Portella, Giuseppe and Giordano, Antonio and Pentimalli, Francesca
Cancer Research, ISSN 0008-5472, 07/2016, Volume 76, Issue 14 Supplement, pp. 3747 - 3747
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2. Targeted therapy based on p53 reactivation reduces both glioblastoma cell growth and resistance to temozolomide
by Forte, Iris Maria and Indovina, Paola and Iannuzzi, Carmelina Antonella and Cirillo, Donatella and Di Marzo, Domenico and Barone, Daniela and Capone, Francesca and Pentimalli, Francesca and Giordano, Antonio
International journal of oncology, 06/2019, Volume 54, Issue 6, p. 2189
Glioblastoma (GB) is the most common and aggressive malignant tumor of the central nervous system. Despite current intensive treatment regimens, consisting of... 
Cell Survival - drug effects | Furans - pharmacology | Humans | Tumor Suppressor Protein p53 - metabolism | Brain Neoplasms - genetics | Imidazoles - pharmacology | Brain Neoplasms - drug therapy | Piperazines - pharmacology | Temozolomide - pharmacology | Brain Neoplasms - metabolism | Tumor Suppressor Protein p53 - genetics | Drug Synergism | Glioblastoma - genetics | Protein Binding - drug effects | Cell Line, Tumor | Glioblastoma - metabolism | Cell Proliferation - drug effects | Mutation | Cell Cycle - drug effects | Glioblastoma - drug therapy | Proto-Oncogene Proteins c-mdm2 - metabolism | Drug Resistance, Neoplasm - drug effects
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3. Full Text Abstract LB-080: Reactivating RBL2/p130 oncosuppressive function as a new, possible antitumoral strategy
by Pentimalli, Francesca and Esposito, Luca and Forte, Iris Maria and Iannuzzi, Carmelina Antonella and Rizzolio, Flavio and Tuccinardi, Tiziano and Indovina, Paola and Boffo, Silvia and Giordano, Antonio
Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 15 Supplement, pp. LB-080 - LB-080
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4. Targeted therapy based on p53 reactivation reduces both glioblastoma cell growth and resistance to temozolomide
by Forte, IM and Indovina, P and Iannuzzi, CA and Cirillo, D and Di Marzo, D and Barone, D and Capone, F and Pentimalli, F and Giordano, A
INTERNATIONAL JOURNAL OF ONCOLOGY, ISSN 1019-6439, 06/2019, Volume 54, Issue 6, pp. 2189 - 2199
Glioblastoma (GB) is the most common and aggressive malignant tumor of the central nervous system. Despite current intensive treatment regimens, consisting of... 
temozolomide | WILD-TYPE P53 | survivin | RITA | SENSITIZES GLIOBLASTOMA | CANCER CELLS | MGMT | INDUCED APOPTOSIS | DNA damage | MGMT EXPRESSION | apoptosis | ANTITUMOR-ACTIVITY | p53 | glioblastoma | ONCOLOGY | MUTANT P53 | MALIGNANT GLIOMAS | RADIOTHERAPY PLUS CONCOMITANT | SMALL-MOLECULE RITA
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5. Full Text SRC Family Kinase Inhibition in Ewing Sarcoma Cells Induces p38 MAP Kinase‐Mediated Cytotoxicity and Reduces Cell Migration
by Indovina, Paola and Casini, Nadia and Forte, Iris Maria and Garofano, Tiziana and Cesari, Daniele and Iannuzzi, Carmelina Antonella and Del Porro, Leonardo and Pentimalli, Francesca and Napoliello, Luca and Boffo, Silvia and Schenone, Silvia and Botta, Maurizio and Giordano, Antonio
Journal of Cellular Physiology, ISSN 0021-9541, 01/2017, Volume 232, Issue 1, pp. 129 - 135
Ewing sarcoma (ES) is a highly aggressive bone and soft tissue cancer, representing the second most common primary malignant bone tumor in children and... 
STANDARD CHEMOTHERAPY | APOPTOSIS | GENOMIC LANDSCAPE | PHYSIOLOGY | GROWTH IN-VITRO | DEATH | DIFFERENTIATION | UP-REGULATION | TUMORS | CHILDRENS ONCOLOGY GROUP | LINES | CELL BIOLOGY | Cell Survival - drug effects | Apoptosis - drug effects | Humans | src-Family Kinases - antagonists & inhibitors | Sarcoma, Ewing - pathology | Pyrimidines - pharmacology | Bone Neoplasms - pathology | Pyrimidines - chemistry | Bone Neoplasms - metabolism | Cell Movement - drug effects | Pyrazoles - chemistry | src-Family Kinases - metabolism | Cell Line, Tumor | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Pyrazoles - pharmacology | Chemotherapy | Pyrimidines | Sarcoma | Resveratrol | Metastasis | Mitogens | Protein kinases | Cancer
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6. Full Text SRC Family Kinase Inhibition in Ewing Sarcoma Cells Induces p38 MAP Kinase-Mediated Cytotoxicity and Reduces Cell Migration
: SFK INHIBITION IN EWING SARCOMA CELLS
by Indovina, Paola and Casini, Nadia and Forte, Iris Maria and Garofano, Tiziana and Cesari, Daniele and Iannuzzi, Carmelina Antonella and Del Porro, Leonardo and Pentimalli, Francesca and Napoliello, Luca and Boffo, Silvia and Schenone, Silvia and Botta, Maurizio and Giordano, Antonio
Journal of Cellular Physiology, ISSN 0021-9541, 01/2017, Volume 232, Issue 1, pp. 129 - 135
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7. Full Text Pharmacological targeting of p53 through RITA is an effective antitumoral strategy for malignant pleural mesothelioma
by Di Marzo, Domenico and Forte, Iris Maria and Indovina, Paola and Di Gennaro, Elena and Rizzo, Valeria and Giorgi, Francesca and Mattioli, Eliseo and Iannuzzi, Carmelina Antonella and Budillon, Alfredo and Giordano, Antonio and Pentimalli, Francesca
Cell Cycle, ISSN 1538-4101, 02/2014, Volume 13, Issue 4, pp. 652 - 665
Malignant mesothelioma, a very aggressive tumor associated to asbestos exposure, is expected to increase in incidence, and unfortunately, no curative modality... 
apoptosis | TP53 mutations | RITA | mesothelioma | nutlin-3 | p21 | Nutlin-3 | Mesothelioma | Apoptosis | PROTECTS MICE | DOWN-REGULATION | CELL-SURVIVAL | CANCER | CELL BIOLOGY | GENE | GROWTH ARREST | MUTANT P53 | INHIBITOR | EXPRESSION | Cell Survival - drug effects | Mesothelioma - pathology | Apoptosis - drug effects | Furans - pharmacology | Humans | Lung Neoplasms - metabolism | Tumor Suppressor Protein p53 - metabolism | Lung Neoplasms - pathology | Imidazoles - pharmacology | Antineoplastic Agents - administration & dosage | Cisplatin - pharmacology | Piperazines - pharmacology | Cisplatin - administration & dosage | Drug Synergism | Pleural Neoplasms - metabolism | Heterografts | Mesothelioma - metabolism | rho GTP-Binding Proteins - metabolism | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Mutation | Cell Cycle - drug effects | Pleural Neoplasms - pathology
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8. Full Text The Oncolytic Virus dl 922-947 Triggers Immunogenic Cell Death in Mesothelioma and Reduces Xenograft Growth
by Di Somma, Sarah and Iannuzzi, Carmelina Antonella and Passaro, Carmela and Forte, Iris Maria and Iannone, Raffaella and Gigantino, Vincenzo and Indovina, Paola and Botti, Gerardo and Giordano, Antonio and Formisano, Pietro and Portella, Giuseppe and Malfitano, Anna Maria and Pentimalli, Francesca
Frontiers in oncology, ISSN 2234-943X, 2019, Volume 9, p. 564
Malignant pleural mesothelioma (MPM) is an aggressive cancer associated with asbestos exposure that urgently requires effective therapeutic strategies. Current... 
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9. Full Text The Oncolytic Virus dl922-947 Triggers Immunogenic Cell Death in Mesothelioma and Reduces Xenograft Growth
by Di Somma, S and Iannuzzi, CA and Passaro, C and Forte, IM and Iannone, R and Gigantino, V and Indovina, P and Botti, G and Giordano, A and Formisano, P and Portella, G and Malfitano, AM and Pentimalli, F
FRONTIERS IN ONCOLOGY, ISSN 2234-943X, 07/2019, Volume 9
Background: Malignant pleural mesothelioma (MPM) is an aggressive cancer associated with asbestos exposure that urgently requires effective therapeutic... 
dl922-947 | immunogenic cell death | MALIGNANT PLEURAL MESOTHELIOMA | virotherapy | COMBINATION | CANCER | INTERLEUKIN-8 | THERAPY | ONCOLOGY | oncolytic virus | ADENOVIRUSES | HEALTH | mesothelioma | EXPRESSION | ASBESTOS | EPIDEMIOLOGY | Mesothelioma | Biochemistry | Immune response | Cell death | Health aspects | Care and treatment | Genetic aspects | Diagnosis | Research | Medical research | Cancer patients | Prognosis | Medicine, Experimental
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10. Full Text p53 status as effect modifier of the association between pre-treatment fasting glucose and breast cancer outcomes in non diabetic, HER2 positive patients treated with trastuzumab
by Vici, Patrizia and Sperati, Francesca and Maugeri-Saccà, Marcello and Melucci, Elisa and Benedetto, Anna Di and Lauro, Luigi Di and Pizzuti, Laura and Sergi, Domenico and Terrenato, Irene and Esposito, Luca and Iannuzzi, Carmelina Antonella and Pasquale, Raffaella and Botti, Claudio and Fuhrman, Barbara and Giordano, Antonio and Mottolese, Marcella and Barba, Maddalena
Oncotarget, ISSN 1949-2553, 2014, Volume 5, Issue 21, pp. 10382 - 10392
p53 status | HER2 positive breast cancer | Fasting glucose | Trastuzumab
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11. Full Text p53 status as effect modifier of the association between pre-treatment fasting glucose and breast cancer outcomes in non diabetic, HER2 positive patients treated with trastuzumab
by Vici, Patrizia and Sperati, Francesca and Maugeri-Saccà, Marcello and Melucci, Elisa and Di Benedetto, Anna and Di Lauro, Luigi and Pizzuti, Laura and Sergi, Domenico and Terrenato, Irene and Esposito, Luca and Iannuzzi, Carmelina Antonella and Pasquale, Raffaella and Botti, Claudio and Fuhrman, Barbara and Giordano, Antonio and Mottolese, Marcella and Barba, Maddalena
Oncotarget, 11/2014, Volume 5, Issue 21, p. 10382
Mounting evidence supports the role of p53 in metabolic processes involved in breast carcinogenesis. We investigated whether p53 status affects the association... 
Prognosis | Humans | Middle Aged | Receptor, ErbB-2 - metabolism | Carcinoma - mortality | Antineoplastic Agents - administration & dosage | Tumor Suppressor Protein p53 - genetics | Antibodies, Monoclonal, Humanized - administration & dosage | DNA Mutational Analysis | Carcinoma - epidemiology | Adult | Female | Breast Neoplasms - epidemiology | Effect Modifier, Epidemiologic | Diagnosis, Differential | Blood Glucose - analysis | Carcinogenesis - genetics | Carcinoma - diagnosis | Treatment Outcome | Disease Progression | Carcinogenesis - drug effects | Survival Analysis | Breast Neoplasms - mortality | Breast Neoplasms - diagnosis | Neoplasm Staging | Trastuzumab
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12. Full Text NONO ubiquitination is mediated by FBW7 and GSK3 via a degron lost upon chromosomal rearrangement in cancer
by Alfano, L and Caporaso, A and Altieri, A and Costa, C and Forte, IM and Iannuzzi, CA and Barone, D and Esposito, L and Giordano, A and Pentimalli, F
JOURNAL OF CELLULAR PHYSIOLOGY, ISSN 0021-9541, 05/2018, Volume 233, Issue 5, pp. 4338 - 4344
NONO is an RNA-binding protein involved in transcription, mRNA splicing, DNA repair, and checkpoint activation in response to UV radiation. NONO expression has... 
PSF | PHYSIOLOGY | FUSION | RNA | PHOSPHORYLATION | NONO | papillary renal carcinoma | TFE3 | C-MYC | CELL BIOLOGY | PROTEIN-DEGRADATION | P54(NRB) | TUMOR-SUPPRESSOR | E3 ubiquitin ligase | CELL-CYCLE | FBW7 | LIGASE | Ubiquitin | Deregulation | Analysis | Melanoma | Radiation | Genetic aspects | Genetic transcription | Cancer | Protein binding | Transcription | Splicing | U.V. radiation | Gene expression | DNA repair | Proteins | Ultraviolet radiation | RNA-binding protein | Ubiquitination | Ribonucleic acids | Colon | Fusion protein | Bioinformatics | Prostate | Clear cell-type renal cell carcinoma | Deoxyribonucleic acid--DNA
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13. Full Text NONO ubiquitination is mediated by FBW7 and GSK3 β via a degron lost upon chromosomal rearrangement in cancer
by Alfano, Luigi and Caporaso, Antonella and Altieri, Angela and Costa, Caterina and Forte, Iris M and Iannuzzi, Carmelina A and Barone, Daniela and Esposito, Luca and Giordano, Antonio and Pentimalli, Francesca
Journal of Cellular Physiology, ISSN 0021-9541, 05/2018, Volume 233, Issue 5, pp. 4338 - 4344
NONO is an RNA‐binding protein involved in transcription, mRNA splicing, DNA repair, and checkpoint activation in response to UV radiation. NONO expression has... 
E3 ubiquitin ligase | FBW7 | NONO | papillary renal carcinoma | TFE3
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