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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 02/2018, Volume 314, Issue 2, pp. H255 - H267
Heart failure (HF) secondary to myocardial infarction (MI) is linked to kidney complications that comprise cellular, structural, functional, and survival... 
Temporal echocardiography | Ischemia injury | Fibrosis | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MYOCARDIAL-INFARCTION | ischemia injury | RESOLVING RESPONSE | MOUSE | ISCHEMIA | INFLAMMATION | DISEASE | PERIPHERAL VASCULAR DISEASE | fibrosis | MICE | KIDNEY | temporal echocardiography | Spleen - physiopathology | Kidney - pathology | Ventricular Function, Left | Cardio-Renal Syndrome - metabolism | Heart Failure - physiopathology | Male | Myocardial Infarction - diagnostic imaging | Cardio-Renal Syndrome - pathology | Kidney - metabolism | Ventricular Remodeling | Time Factors | Myocardial Infarction - pathology | Inflammation Mediators - metabolism | Ventricular Dysfunction, Left - pathology | Myocardial Infarction - physiopathology | Heart Failure - diagnostic imaging | Spleen - pathology | Cardio-Renal Syndrome - diagnostic imaging | Cardio-Renal Syndrome - physiopathology | Kidney - physiopathology | Disease Models, Animal | Heart - physiopathology | Echocardiography | Mice, Inbred C57BL | Heart Failure - metabolism | Myocardial Infarction - metabolism | Disease Progression | Ventricular Dysfunction, Left - diagnostic imaging | Ventricular Dysfunction, Left - physiopathology | Heart - diagnostic imaging | Ventricular Dysfunction, Left - metabolism | Collagen - metabolism | Animals | Spleen - metabolism | Chronic Disease | Heart failure | Development and progression | Health aspects | Heart ventricle, Left | Myocardial infarction | Heart | Networks | Remodeling | Rodents | Bioindicators | Heart diseases | Function analysis | Spleen | Cell survival | Kidneys | Complications | Inflammation | Histology | Functional analysis | Pathology | Collagen | Myocardium | Biomarkers | Mice | Infarction | Kidney diseases | Ventricle | Cellular structure | Structure-function relationships | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2017, Volume 313, Issue 1, pp. H89 - H102
Journal Article
Circulation, ISSN 0009-7322, 11/2016, Volume 134, Issue Suppl_1 Suppl 1, pp. A16952 - A16952
Introduction12/15lipoxygenase (12/15LOX) metabolites and cyclooxygenase-2(COX-2)-derived prostaglandin E2 (PGE2) are key mediators of the inflammatory response... 
Journal Article
Circulation, ISSN 0009-7322, 11/2016, Volume 134, Issue Suppl_1 Suppl 1, pp. A12625 - A12625
IntroductionAfter myocardial infarction (MI) self-healing is prominent in mice but not in humans. MI-induced innate inflammatory and resolving response is... 
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 84, pp. 24 - 35
Abstract Unresolved inflammation is a major contributor to the development of heart failure following myocardial infarction (MI). Pro-resolving lipid... 
Cardiovascular | Myocardial infarction | Splenic remodeling | Resolution of inflammation | Metabololipidomics | Resolvin D1 | Neutrophils | SURVIVAL | CHRONIC HEART-FAILURE | PROTECTIVE ACTIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | RESOLUTION | TISSUE | RECEPTOR | LIPID MEDIATORS | CELL BIOLOGY | THERAPY | PATHWAY | DISEASE | Cell Count | Male | Myocardial Infarction - diagnostic imaging | Spleen - drug effects | Extracellular Matrix - genetics | Inflammation - complications | Pulmonary Edema - physiopathology | Inflammation - drug therapy | Pulmonary Edema - complications | Receptors, Formyl Peptide - metabolism | Ultrasonography | Myocardial Infarction - physiopathology | Cell Polarity - drug effects | Spleen - pathology | Neutrophil Infiltration - drug effects | Ventricular Function - drug effects | Docosahexaenoic Acids - therapeutic use | Arachidonate 5-Lipoxygenase - metabolism | Docosahexaenoic Acids - chemistry | Extracellular Matrix - drug effects | Mice, Inbred C57BL | Cardiomegaly - diagnostic imaging | Cardiomegaly - drug therapy | Cardiomegaly - physiopathology | Pulmonary Edema - drug therapy | Cardiomegaly - complications | Collagen - metabolism | Docosahexaenoic Acids - pharmacology | Myocardial Infarction - complications | Macrophages - metabolism | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Prostaglandin-Endoperoxide Synthases - metabolism | Macrophages - drug effects | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects | Inflammation | Heart attack | Index Medicus | metabololipidomics | liposomes | myocardial infarction | splenic remodeling | neutrophils | resolution of inflammation
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 02/2018, Volume 314, Issue 2, pp. H160 - H169
Journal Article
SCIENCE SIGNALING, ISSN 1945-0877, 03/2018, Volume 11, Issue 520, p. eaao1818
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2015, Volume 308, Issue 4, pp. H269 - H280
Polyunsaturated fatty acid (PUFA) intake has increased over the last 100 yr, contributing to the current obesogenic environment. Obesity and aging are... 
Lipidomics | Inflammation | Lipid mediators | Polyunsaturated fatty acids | Neutrophils | Proteomics | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ACID | proteomics | LONG-CHAIN OMEGA-3-FATTY-ACIDS | HIGH-FAT-DIET | INDUCE | polyunsaturated fatty acids | HEART-FAILURE | RESOLUTION | HUMAN SERUM | lipid mediators | inflammation | INSULIN-RESISTANCE | DISEASE | PERIPHERAL VASCULAR DISEASE | SATURATED FAT | neutrophils | lipidomics | 12-Hydroxy-5,8,10,14-eicosatetraenoic Acid - metabolism | Arachidonic Acid - metabolism | Heme Oxygenase-1 - metabolism | Fatty Acids, Omega-3 - metabolism | Macrophage Inflammatory Proteins - metabolism | Mice, Inbred C57BL | Ventricular Function | Diet, High-Fat - adverse effects | Obesity - physiopathology | Myocardial Infarction - metabolism | Wound Healing | Neutrophil Infiltration | Myocardial Infarction - immunology | Obesity - metabolism | Myocardial Infarction - complications | Inflammation - metabolism | Lipoxygenase - metabolism | Animals | CD40 Antigens - metabolism | Cyclooxygenase 2 - metabolism | Myocardial Infarction - physiopathology | Obesity - etiology | Mice | Aging - metabolism | Aging | Obesity | Health aspects | Fatty acids | Heart attack | Risk factors | COX-2 inhibitors | Unsaturated fatty acids | Analysis | Enzymes | Heart attacks | Rodents | Physiology | Index Medicus | Call for Papers
Journal Article
Analytical and Bioanalytical Chemistry, ISSN 1618-2642, 3/2018, Volume 410, Issue 7, pp. 1965 - 1976
Myocardial infarction (MI) and subsequent progressive heart failure pathology is the major cause of death worldwide; however, the mechanism of this pathology... 
Biochemistry, general | Myocardial infarction | Ischemic myocardium | Chemistry | Analytical Chemistry | Food Science | Monitoring/Environmental Analysis | Resolution of inflammation | Characterization and Evaluation of Materials | Lipids | Laboratory Medicine | Bioactive lipid molecules | IMAGING MASS-SPECTROMETRY | CHEMISTRY, ANALYTICAL | MYOCARDIAL-INFARCTION | TISSUE | BIOCHEMICAL RESEARCH METHODS | FATTY-ACIDS | FAILURE | INFLAMMATION | DISEASE | OXIDIZED PHOSPHOLIPIDS | MICE | MEDIATORS | Heart - physiopathology | Inflammation - pathology | Mice, Inbred C57BL | Lipid Metabolism | Male | Myocardium - pathology | Myocardial Infarction - metabolism | Docosahexaenoic Acids - analysis | Inflammation - metabolism | Animals | Myocardial Infarction - pathology | Myocardium - metabolism | Lipids - analysis | Mass Spectrometry - methods | Docosahexaenoic Acids - metabolism | Heart failure | Physiological aspects | Liquid chromatography | Methods | Heart attack | Spatial discrimination | Inflammatory response | Heart diseases | Failure | Spectroscopy | Lipoxin A4 | Contractility | Principal components analysis | Ions | Mass spectroscopy | Inflammation | Muscle contraction | Biological activity | Pathology | Spatial distribution | Ionization | Myocardium | Infarction | Scientific imaging | Ventricle | Mass spectrometry | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 1 - 14
Following myocardial infarction (MI), overactive inflammation remodels the left ventricle (LV) leading to heart failure coinciding with reduced levels of... 
Myocardial infarction | Creatinine | Heart attacks | Kidneys | Lipoxin A4 | Body weight | Coronary artery | Leukocytes (neutrophilic) | Inflammation | Macrophages | Cell activation | Rodents | Ventricle | Heart diseases | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 05/2018, Volume 118, pp. 70 - 80
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2015, Volume 309, Issue 11, pp. H1827 - H1836
The mammalian circadian clock consists of multiple transcriptional regulators that coordinate biological processes in a time-of-day-dependent manner.... 
Circadian clock | Aging | Diastolic dysfunction | Extracellular matrix | Inflammation | Bmal1 | PRESSURE-OVERLOAD | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | HEART-FAILURE | circadian clock | BLOOD-PRESSURE | HYPERTROPHY | inflammation | GENES | diastolic dysfunction | ACUTE MYOCARDIAL-INFARCTION | PERIPHERAL VASCULAR DISEASE | aging | RHYTHM | EXPRESSION | extracellular matrix | AGE | Age Factors | Diastole | Ventricular Function, Left | Extracellular Matrix - metabolism | Male | Smad3 Protein - metabolism | RNA, Messenger - metabolism | Extracellular Matrix - genetics | ARNTL Transcription Factors - deficiency | Inflammation - metabolism | Ventricular Remodeling | Hypertrophy, Left Ventricular - genetics | Time Factors | Ventricular Dysfunction, Left - genetics | Hypertrophy, Left Ventricular - pathology | Inflammation Mediators - metabolism | Ventricular Dysfunction, Left - pathology | Transcription, Genetic | ARNTL Transcription Factors - genetics | Signal Transduction | Hypertrophy, Left Ventricular - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Smad2 Protein - metabolism | Genotype | Disease Progression | Ventricular Dysfunction, Left - physiopathology | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Myocytes, Cardiac - pathology | Phenotype | Animals | Fibrosis | Inflammation - genetics | Myocytes, Cardiac - metabolism | Hypertrophy, Left Ventricular - physiopathology | Transforming Growth Factor beta - metabolism | Gene expression | Collagen | Circadian rhythms | Heart cells | Genetic aspects | Cardiovascular diseases | Health aspects | Cardiomyocytes | Circadian rhythm | Rodents | Cells | Index Medicus | Call for Papers
Journal Article