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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2006, Volume 103, Issue 42, pp. 15491 - 15496
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2002, Volume 99, Issue 17, pp. 11393 - 11398
Vascular endothelial growth factor (VEGF) plays a critical role during normal embryonic angiogenesis and also in the pathological angiogenesis that occurs in a... 
Angiogenesis | Biological Sciences | Receptors | Humans | Antibodies | Monoclonal antibodies | Animal traps | Pharmacokinetics | Dosage | Tumors | Cancer | REGRESSION | INHIBITION | METASTASIS | ANGIOPOIETINS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MONOCLONAL-ANTIBODY | TUMOR ANGIOGENESIS | NEOVASCULARIZATION | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Vascular Endothelial Growth Factor A | Phosphorylation | Vascular Endothelial Growth Factors | Melanoma, Experimental - drug therapy | Antineoplastic Agents - therapeutic use | Endothelial Growth Factors - pharmacology | Endothelial Growth Factors - immunology | Endothelium, Vascular - physiology | Vascular Endothelial Growth Factor Receptor-1 | Immunoglobulin Constant Regions - genetics | Rhabdomyosarcoma - blood supply | Bone Neoplasms - blood supply | Cell Division | Extracellular Matrix - physiology | Lymphokines - antagonists & inhibitors | Drug Design | Protein Engineering | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Rhabdomyosarcoma - drug therapy | Antineoplastic Agents - pharmacology | Bone Neoplasms - drug therapy | Lymphokines - pharmacology | Proto-Oncogene Proteins - antagonists & inhibitors | Melanoma, Experimental - blood supply | Umbilical Veins | Endothelial Growth Factors - antagonists & inhibitors | Animals | Immunoglobulin G - genetics | Lymphokines - immunology | Mice | Mice, Inbred BALB C | 3T3 Cells | Growth | Physiological aspects | Causes of | Neovascularization | Vascular endothelial growth factor | Diseases
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 05/2017, Volume 16, Issue 5, pp. 861 - 870
Journal Article
Journal Article
MOLECULAR CANCER THERAPEUTICS, ISSN 1535-7163, 11/2019, Volume 18, Issue 11, pp. 2051 - 2062
In the tumor microenvironment, multiple inhibitory checkpoint receptors can suppress T-cell function, thereby enabling tumor immune evasion. Blockade of one of... 
RESPONSES | ONCOLOGY | LAG-3 | INHIBITORY RECEPTORS | IMMUNE CHECKPOINT | LYMPHOCYTE-ACTIVATION GENE-3 | PD-1 | EXPRESSION | CANCER | CD8(+) T-CELLS | MOLECULES
Journal Article
Genes and Development, ISSN 0890-9369, 05/2004, Volume 18, Issue 9, pp. 1060 - 1071
Journal Article
International Journal of Oncology, ISSN 1019-6439, 2009, Volume 34, Issue 1, pp. 79 - 87
Journal Article
The American Journal of Pathology, ISSN 0002-9440, 2002, Volume 160, Issue 5, pp. 1683 - 1693
Diabetic retinopathy remains a leading cause of irreversible blindness. A critical early pathology in the disease is the adhesion of leukocytes to the retinal... 
ADHESION MOLECULE-1 | ICAM-1 EXPRESSION | MESSENGER-RNA | VEGF | IN-VIVO | NEOVASCULARIZATION | PATHOLOGY | CELL-SURVIVAL | ENDOTHELIAL GROWTH-FACTOR | PLASMA LEAKAGE | TIE2 RECEPTOR | Retina - drug effects | Diabetic Retinopathy - drug therapy | Vascular Endothelial Growth Factor A | Endothelial Growth Factors - metabolism | Retina - metabolism | Vascular Endothelial Growth Factors | Protein-Serine-Threonine Kinases | Rats, Long-Evans | Nitric Oxide Synthase - drug effects | Angiopoietin-1 | Endothelium, Vascular - drug effects | Male | Diabetic Retinopathy - pathology | RNA, Messenger - metabolism | Blood-Retinal Barrier - drug effects | Nitric Oxide Synthase Type II | Dose-Response Relationship, Drug | Lymphokines - genetics | Cattle | Lymphokines - metabolism | Nitric Oxide Synthase - biosynthesis | Leukocytes - cytology | RNA, Messenger - drug effects | Proto-Oncogene Proteins - metabolism | Membrane Glycoproteins - pharmacology | Mice, Inbred C57BL | RNA, Messenger - genetics | Nitric Oxide Synthase Type III | Rats | Cell Adhesion - drug effects | Enzyme Activation - drug effects | Diabetic Retinopathy - metabolism | Proto-Oncogene Proteins c-akt | Intercellular Adhesion Molecule-1 - metabolism | Animals | Membrane Glycoproteins - therapeutic use | Endothelial Growth Factors - genetics | Intercellular Adhesion Molecule-1 - genetics | Endothelium, Vascular - pathology | Mice | Nitric Oxide - metabolism | Leukocytes - metabolism | Mitogen-Activated Protein Kinases - drug effects | Retina - pathology | Mitogen-Activated Protein Kinases - metabolism | Regular
Journal Article
The FASEB Journal, ISSN 0892-6638, 11/1995, Volume 9, Issue 14, pp. 1436 - 1444
Glycosyltransferases (Glyc‐T's) catalyze the synthesis of the carbohydrate portions of glycoproteins, glycolipids, and proteoglycans. Most Glyc‐T's transfer... 
cell surface | glycosylation | carbohydrate function | mammalian mutants | MAMMALIAN MUTANTS | CELL SURFACE | OLIGOSACCHARIDES | GENE | HAMSTER OVARY CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BIOLOGY | CARBOHYDRATE FUNCTION | GLYCOSYLATION | MOLECULAR-BASIS | CELL BIOLOGY
Journal Article