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New Phytologist, ISSN 0028-646X, 1/2013, Volume 197, Issue 2, pp. 454 - 467
Strawberry ( × ) fruits contain high concentrations of flavonoids. In unripe strawberries, the flavonoids are mainly represented by proanthocyanidins (PAs),... 
Full papers | Flavonoids | Transcriptional regulatory elements | Fruiting | Genes | Biosynthesis | Gene expression regulation | Plants | Regulator genes | Genotypes | Fruits | ragaria | yeast‐two‐hybrid | HLH | MYB | strawberry | 40‐repeat | flavonoid biosynthesis | ananassa | WD40-repeat | Flavonoid biosynthesis | Yeast-two-hybrid | BHLH | Strawberry (Fragaria × ananassa) | FACTOR FAMILY | ARABIDOPSIS-THALIANA | R2R3-MYB TRANSCRIPTION FACTOR | GENE ENCODES | ANTHOCYANIN BIOSYNTHESIS | DOMAIN PROTEIN | bHLH | yeast-two-hybrid | NEGATIVE REGULATOR | PLANT SCIENCES | MYB DOMAIN | strawberry (Fragaria x ananassa) | EXPRESSION | Genes, Plant | Flavonols - metabolism | Transcriptome - genetics | Phylogeny | Mutation - genetics | Fragaria - growth & development | Genetic Complementation Test | Metabolome - genetics | Arabidopsis Proteins - metabolism | Bryopsida - metabolism | Saccharomyces cerevisiae - metabolism | Sequence Homology, Amino Acid | Fruit - growth & development | Plant Proteins - genetics | Protoplasts - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Plants, Genetically Modified | Fragaria - genetics | Gene Expression Regulation, Plant | Proanthocyanidins - biosynthesis | Plant Proteins - metabolism | Fruit - genetics | Cluster Analysis | Crosses, Genetic | Fruit | Physiological aspects | Chemical properties | Index Medicus | Life Sciences | negative regulator | r2r3-myb transcription factor | myb domain | expression | arabidopsis-thaliana | domain protein | gene encodes | factor family | anthocyanin biosynthesis
Journal Article
The Plant Journal, ISSN 0960-7412, 2014, Volume 80, Issue 4, pp. 695 - 708
Journal Article
The Plant Journal, ISSN 0960-7412, 11/2014, Volume 80, Issue 4, pp. 695 - 708
Journal Article
by Klionsky, Daniel J and Abdelmohsen, Kotb and Abe, Akihisa and Abedin, Md Joynal and Abeliovich, Hagai and Acevedo Arozena, Abraham and Adachi, Hiroaki and Adams, Christopher M and Adams, Peter D and Adeli, Khosrow and Adhihetty, Peter J and Adler, Sharon G and Agam, Galila and Agarwal, Rajesh and Aghi, Manish K and Agnello, Maria and Agostinis, Patrizia and Aguilar, Patricia V and Aguirre-Ghiso, Julio and Airoldi, Edoardo M and Ait-Si-Ali, Slimane and Akematsu, Takahiko and Akporiaye, Emmanuel T and Al-Rubeai, Mohamed and Albaiceta, Guillermo M and Albanese, Chris and Albani, Diego and Albert, Matthew L and Aldudo, Jesus and Algül, Hana and Alirezaei, Mehrdad and Alloza, Iraide and Almasan, Alexandru and Almonte-Beceril, Maylin and Alnemri, Emad S and Alonso, Covadonga and Altan-Bonnet, Nihal and Altieri, Dario C and Alvarez, Silvia and Alvarez-Erviti, Lydia and Alves, Sandro and Amadoro, Giuseppina and Amano, Atsuo and Amantini, Consuelo and Ambrosio, Santiago and Amelio, Ivano and Amer, Amal O and Amessou, Mohamed and Amon, Angelika and An, Zhenyi and Anania, Frank A and Andersen, Stig U and Andley, Usha P and Andreadi, Catherine K and Andrieu-Abadie, Nathalie and Anel, Alberto and Ann, David K and Anoopkumar-Dukie, Shailendra and Antonioli, Manuela and Aoki, Hiroshi and Apostolova, Nadezda and Aquila, Saveria and Aquilano, Katia and Araki, Koichi and Arama, Eli and Aranda, Agustin and Araya, Jun and Arcaro, Alexandre and Arias, Esperanza and Arimoto, Hirokazu and Ariosa, Aileen R and Armstrong, Jane L and Arnould, Thierry and Arsov, Ivica and Asanuma, Katsuhiko and Askanas, Valerie and Asselin, Eric and Atarashi, Ryuichiro and Atherton, Sally S and Atkin, Julie D and Attardi, Laura D and Auberger, Patrick and Auburger, Georg and Aurelian, Laure and Autelli, Riccardo and Avagliano, Laura and Avantaggiati, Maria Laura and Avrahami, Limor and Awale, Suresh and Azad, Neelam and Bachetti, Tiziana and Backer, Jonathan M and Bae, Dong-Hun and Bae, Jae-sung and Bae, Ok-Nam and Bae, Soo Han and Baehrecke, Eric H and Baek, Seung-Hoon and Baghdiguian, Stephen and Bagniewska-Zadworna, Agnieszka and ... and Medicinska fakulteten and Region Östergötland and Linköpings universitet and Institutionen för klinisk och experimentell medicin and Diagnostikcentrum and Klinisk patologi och klinisk genetik and Institutionen för medicin och hälsa and Avdelningen för läkemedelsforskning and Avdelningen för cellbiologi
Autophagy, ISSN 1554-8627, 01/2016, Volume 12, Issue 1, pp. 1 - 222
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1488 - 15
Type 1 diabetes mellitus (T1DM) is due to the selective destruction of islet beta cells by immune cells. Current therapies focused on repressing the immune... 
LIVER RECEPTOR HOMOLOG-1 | RISK-FACTORS | DENDRITIC CELLS | ALPHA-CELLS | MACROPHAGES | REGENERATION | MULTIDISCIPLINARY SCIENCES | BETA-CELLS | REGULATORY T-CELLS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | MICE | Islets of Langerhans - drug effects | Apoptosis - drug effects | Streptozocin | Diabetes Mellitus, Type 2 - genetics | Humans | Diabetes Mellitus, Experimental - genetics | Male | Transplantation, Heterologous | Diabetes Mellitus, Experimental - therapy | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Receptors, Cytoplasmic and Nuclear - immunology | Diabetes Mellitus, Type 2 - immunology | Islets of Langerhans Transplantation | Female | Diabetes Mellitus, Experimental - chemically induced | Macrophages - immunology | Cell Survival - drug effects | Islets of Langerhans - pathology | Macrophages - pathology | Mice, Inbred C57BL | Gene Expression Regulation | Islets of Langerhans - immunology | Phenalenes - pharmacology | Receptors, Cytoplasmic and Nuclear - agonists | Receptors, Cytoplasmic and Nuclear - genetics | Immunity, Innate | Insulin-Secreting Cells - immunology | Hypoglycemic Agents - pharmacology | Diabetes Mellitus, Experimental - immunology | Insulin - metabolism | T-Lymphocytes, Regulatory - drug effects | Animals | Insulin-Secreting Cells - drug effects | Cell Communication - drug effects | Macrophages - drug effects | Mice | Diabetes Mellitus, Type 2 - pathology | Insulin-Secreting Cells - pathology | Repressing | Animal models | Cell survival | Target recognition | Secretion | Liver | Diabetes mellitus | Organs | Homology | Diabetes mellitus (insulin dependent) | Glucose | Insulin | Beta cells | Immune systems | Regeneration | Hyperglycemia | Hepatocytes | Islet cells | Diabetes | Islets of Langerhans | Pancreas | Apoptosis | Index Medicus
Journal Article