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by Nik-Zainal, Serena and Davies, Helen and Staaf, Johan and Ramakrishna, Manasa and Glodzik, Dominik and Zou, Xueqing and Martincorena, Inigo and Alexanov, Ludmil B and Martin, Sancha and Wedge, David C and van Loo, Peter and Ju, Young Seok and Smid, Marcel and Brinkman, Arie B and Morganella, Sano and Aure, Miriam R and Lingjærde, Ole Christian and Langerød, Anita and Ringnér, Markus and Ahn, Sung-Min and Boyault, Sanine and Brock, Jane E and Broeks, Annegien and Butler, Adam and Desmedt, Christine and Dirix, Luc and nov, Serge and Fatima, Aquila and Foekens, John A and Gerstung, Moritz and Hooijer, Gerrit K. J and Jang, Se Jin and Jones, David R and Kim, Hyung-Yong and King, Tari A and Krishnamurthy, Savitri and Lee, Hee Jin and Lee, Jeong-Yeon and Li, Yilong and McLaren, Stuart and Menzies, Anew and Mustonen, Ville and O'Meara, Sarah and Pauporté, Iris and Pivot, Xavier and Purdie, Colin A and Raine, Keiran and Ramakrishnan, Kamna and Roíguez-González, F. Germán and Romieu, Gilles and Sieuwerts, Anieta M and Simpson, Peter T and Shepherd, Rebecca and Stebbings, Lucy and Stefansson, Olafur A and Teague, Jon and Tommasi, Stefania and Treilleux, Isabelle and van den Eynden, Gert G and Vermeulen, Peter and Vincent-Salomon, Anne and Yates, Lucy and Caldas, Carlos and van't Veer, Laura and Tutt, Anew and Knappskog, Stian and Tan, Benita Kiat Tee and Jonkers, Jos and Borg, Åke and Ueno, Naoto T and Sotiriou, Christos and Viari, Alain and Futreal, P. Anew and Campbell, Peter J and Span, Paul N and van Laere, Steven and Lakhani, Sunil R and Eyfjord, Jorunn E and Thompson, Alastair M and Birney, Ewan and Stunnenberg, Henik G and van de Vijver, Marc J and Martens, John W. M and Børresen-Dale, Anne-Lise and Richardson, Anea L and Kong, Gu and Thomas, Gilles and Stratton, Michael R and Los Alamos National Lab. (LANL), Los Alamos, NM (United States) and Onkologi och Patologi, MV and Familial Breast Cancer and Lund University and MEMEG and Oncology and Pathology, MV and BioCARE: Biomarkers in Cancer Medicine improving Health Care, Education and Innovation and Lunds universitet and Familjär bröstcancer
Nature, ISSN 0028-0836, 2016, Volume 534, Issue 7605, pp. 47 - +
Journal Article
PLoS Genetics, ISSN 1553-7390, 05/2012, Volume 8, Issue 5, p. e1002723
The epithelial-mesenchymal transition (EMT) is an embryonic transdifferentiation process consisting of conversion of polarized epithelial cells to motile... 
MIDDLE-T | TO-MESENCHYMAL TRANSITION | GENE | GROWTH | GENETICS & HEREDITY | PROTEIN PHOSPHATASE 2A | IDENTIFICATION | EXPRESSION | CANCER STEM-CELLS | TWIST | SNAIL | Epithelial Cells - metabolism | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Epithelial-Mesenchymal Transition - genetics | Mammary Glands, Human - metabolism | Breast Neoplasms - metabolism | Claudins - metabolism | Cell Transformation, Neoplastic - genetics | Female | Telomerase - metabolism | Cell Differentiation | Epithelial Cells - cytology | Cell Line | Claudins - genetics | Mammary Glands, Human - cytology | Protein Phosphatase 2 - antagonists & inhibitors | Retinoblastoma Protein - metabolism | Tumor Suppressor Protein p53 - metabolism | Mice, Transgenic | Transcription Factors - genetics | Cell Transformation, Neoplastic - metabolism | Homeodomain Proteins - genetics | Mammary Glands, Animal - cytology | Transcription Factors - metabolism | Animals | Breast Neoplasms - genetics | Mammary Glands, Animal - metabolism | Protein Phosphatase 2 - metabolism | Twist-Related Protein 1 - genetics | Mice | Genes, ras | Twist-Related Protein 1 - metabolism | Zinc Finger E-box-Binding Homeobox 1 | Transcription factors | Physiological aspects | Development and progression | Breast cancer | Genetic aspects | Research | Cell differentiation | Risk factors | Proteins | Rodents | Colonies & territories | Gene expression | Experiments | Tumors | Apoptosis | Life Sciences | Cancer
Journal Article
Journal Article
Nature Genetics, ISSN 1061-4036, 09/2015, Volume 47, Issue 10, pp. 1200 - 1205
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2011, Volume 6, Issue 4, p. e19234
Journal Article
Journal Article
Journal Article
Breast Cancer Research and Treatment, ISSN 0167-6806, 2/2012, Volume 132, Issue 1, pp. 29 - 39
Understanding how cancer genes are mutated in individual tumors is an important issue with potential clinical and therapeutic impact. This is especially... 
Oncology | PI3K pathway | Breast cancer | Medicine & Public Health | Mutation screening | TP53 mutation | PTEN LOSS | PIK3CA MUTATIONS | COLORECTAL CANCERS | ANGIOGENESIS | EXPRESSION PROFILES | CONTINGENCY-TABLES | ONCOLOGY | CARCINOMAS | TUMORIGENESIS | CORRELATE | PTEN/MMAC1 | Carcinoma, Ductal, Breast - genetics | Receptors, Steroid - metabolism | Humans | Middle Aged | Carcinoma, Ductal, Breast - mortality | Carcinoma, Lobular - mortality | Proto-Oncogene Proteins c-akt - genetics | Tumor Suppressor Protein p53 - genetics | Breast Neoplasms - metabolism | Carcinoma, Lobular - genetics | DNA Mutational Analysis | Adult | Female | Cadherins - genetics | Carcinoma, Ductal, Breast - metabolism | PTEN Phosphohydrolase - genetics | Genetic Association Studies | Kaplan-Meier Estimate | Signal Transduction - genetics | Phosphatidylinositol 3-Kinases - genetics | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Breast Neoplasms - mortality | Aged | Carcinoma, Lobular - metabolism | Criminal investigation | Gene mutations | Genetic aspects | Chemical properties | Diagnosis | Tumor proteins | Health aspects | Cancer | Signal Transduction | Carcinoma, Lobular | Breast Neoplasms | Phosphatidylinositol 3-Kinases | Proto-Oncogene Proteins c-akt | Life Sciences | Receptors, Steroid | Tumor Suppressor Protein p53 | PTEN Phosphohydrolase | Cadherins | Carcinoma, Ductal, Breast
Journal Article