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1. Full Text ROS-Generating Mitochondrial DNA Mutations Can Regulate Tumor Cell Metastasis
by Kaori Ishikawa and Keizo Takenaga and Miho Akimoto and Nobuko Koshikawa and Aya Yamaguchi and Hirotake Imanishi and Kazuto Nakada and Yoshio Honma and Jun-Ichi Hayashi
Science, ISSN 0036-8075, 5/2008, Volume 320, Issue 5876, pp. 661 - 664
Mutations in mitochondrial DNA (mtDNA) occur at high frequency in human tumors, but whether these mutations alter tumor cell behavior has been unclear. We used... 
Tumor cell line | Myeloid cells | Reactive oxygen species | NIH 3T3 cells | Cell lines | HeLa cells | Reports | Mitochondrial DNA | Metastasis | Genetic mutation | Tumors | LUNG-CARCINOMA CELLS | APOPTOSIS | MTDNA | MULTIDISCIPLINARY SCIENCES | MOUSE | PREVENTION | PARAGANGLIOMA | TUMORIGENICITY | BINDING | CANCER | GENOME | Free Radical Scavengers - pharmacology | Reactive Oxygen Species - metabolism | Humans | Hybrid Cells | DNA, Mitochondrial | NADH Dehydrogenase - genetics | NADH Dehydrogenase - metabolism | Electron Transport Complex I - metabolism | Neoplasm Metastasis - genetics | Animals | Electron Transport Complex I - genetics | DNA, Neoplasm | Acetylcysteine - pharmacology | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Mice | HeLa Cells | Mutation | Oxygen | Gene mutations | Influence | Genetic aspects | Research | Properties | Oncology | Cells
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2. Full Text Epigenetic regulation of the nuclear-coded GCAT and SHMT2 genes confers human age-associated mitochondrial respiration defects
by Hashizume, Osamu and Ohnishi, Sakiko and Mito, Takayuki and Shimizu, Akinori and Iashikawa, Kaori and Nakada, Kazuto and Soda, Manabu and Mano, Hiroyuki and Togayachi, Sumie and Miyoshi, Hiroyuki and Okita, Keisuke and Hayashi, Jun-Ichi
Scientific Reports, ISSN 2045-2322, 05/2015, Volume 5, Issue 1, p. 10434
Age-associated accumulation of somatic mutations in mitochondrial DNA (mtDNA) has been proposed to be responsible for the age-associated mitochondrial... 
PLURIPOTENT STEM-CELLS | AGING PHENOTYPES | COMPLEMENTATION | MULTIDISCIPLINARY SCIENCES | MICE | DYSFUNCTION | FUNCTIONAL INTEGRITY | DNA MUTATIONS | Acyltransferases - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Oligonucleotide Array Sequence Analysis | Epigenesis, Genetic | Humans | Infant | DNA, Mitochondrial - analysis | Acyltransferases - metabolism | Acyltransferases - genetics | Cellular Reprogramming | Lipase - antagonists & inhibitors | RNA Interference | Aged, 80 and over | Aging | Cell Differentiation | Induced Pluripotent Stem Cells - cytology | Child | Real-Time Polymerase Chain Reaction | Fibroblasts - metabolism | Induced Pluripotent Stem Cells - metabolism | Cell Line | Glycine Hydroxymethyltransferase - genetics | Oxygen Consumption | Glycine - biosynthesis | Gene Dosage | Mitochondria - metabolism | Lipase - metabolism | Glycine Hydroxymethyltransferase - metabolism | Sequence Analysis, DNA | Phenotype | Fibroblasts - cytology | Lipase - genetics | RNA, Small Interfering - metabolism | Genomes | Mitochondrial DNA | Glycine | Defects | DNA microarrays | Stem cells | Fibroblasts | Epigenetics | Mutation | Electron transport | Respiration | Age | Pluripotency | Inhibitory postsynaptic potentials | Geriatrics
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3. Full Text Technique for differentiating alveolar soft part sarcoma from other tumors in paraffin-embedded tissue: comparison of immunohistochemistry for TFE3 and CD147 and of reverse transcription polymerase chain reaction for ASPSCR1-TFE3 fusion transcript
by Tsuji, Kaori, DVM and Ishikawa, Yuichi, MD, PhD and Imamura, Tetsuo, MD
Human Pathology, ISSN 0046-8177, 2012, Volume 43, Issue 3, pp. 356 - 363
Summary The diagnosis of alveolar soft part sarcoma is commonly based on characteristic histology and distinctive periodic acid–Schiff–positive crystals;... 
Pathology | Immunohistochemistry | ASPSCR1-TFE3 fusion transcript | Differential diagnosis | Alveolar soft part sarcoma | RT-PCR | GENE FUSIONS | IMMUNOREACTIVITY | NEOPLASMS | PATHOLOGY | Diagnosis, Differential | Oncogene Proteins, Fusion - metabolism | Humans | Middle Aged | Gene Fusion | Male | Paraffin Embedding | Sarcoma, Alveolar Soft Part - genetics | Young Adult | Cell Nucleus - metabolism | Oncogene Proteins, Fusion - genetics | Adolescent | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Basigin - metabolism | Adult | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Female | Sarcoma, Alveolar Soft Part - metabolism | Sarcoma, Alveolar Soft Part - diagnosis | Polymerase chain reaction | Sarcoma | Comparative analysis | Methods | Cancer | Proteins | Tumors
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4. Full Text Effects of maternal depressive symptomatology during pregnancy and the postpartum period on infant–mother attachment
by Ohoka, Harue and Koide, Takayoshi and Goto, Setsuko and Murase, Satomi and Kanai, Atsuko and Masuda, Tomoko and Aleksic, Branko and Ishikawa, Naoko and Furumura, Kaori and Ozaki, Norio
Psychiatry and Clinical Neurosciences, ISSN 1323-1316, 08/2014, Volume 68, Issue 8, pp. 631 - 639
Aim Postnatal depression has demonstrated long‐term consequences on child cognitive and emotional development; however, the link between maternal and child... 
bonding disorder | Mother‐to‐Infant Bonding Scale | Edinburgh Postnatal Depression Scale | post‐partum period | maternal mood | Mother-to-Infant Bonding Scale | post-partum period | CHILDBEARING | PSYCHIATRY | POSTNATAL DEPRESSION | DISORDERS | COGNITIVE-DEVELOPMENT | JAPANESE WOMEN | NEUROSCIENCES | CLINICAL NEUROLOGY | IMPACT | MOOD | INVENTORY | CHILDBIRTH | BONDING SCALE | Pregnancy | Pregnancy Complications - psychology | Young Adult | Mother-Child Relations - psychology | Object Attachment | Depression, Postpartum - psychology | Prospective Studies | Humans | Self Report | Depression - psychology | Adult | Female | Postpartum depression | Child development | Pregnant women | Analysis | Postpartum period
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5. Full Text Improvement in PET/CT Image Quality with a Combination of Point-Spread Function and Time-of-Flight in Relation to Reconstruction Parameters
by Akamatsu, G and Ishikawa, K and Mitsumoto, K and Taniguchi, T and Ohya, N and Baba, S and Abe, K and Sasaki, M
JOURNAL OF NUCLEAR MEDICINE, ISSN 0161-5505, 11/2012, Volume 53, Issue 11, pp. 1716 - 1722
The aim of this study was to investigate the effects of the point-spread function (PSF) and time-of-flight (TOF) on improving F-18-FDG PET/CT images in... 
TOMOGRAPHY | noise-equivalent counts | DOPED LUTETIUM OXYORTHOSILICATE | point-spread function | NOISE | image quality | LUNG-CANCER | IMPACT | POSITRON-EMISSION | SCAN DURATION | time-of-flight | PET/CT | F-18-FDG | PATIENT WEIGHT | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | EQUIVALENT | Time Factors | Humans | Middle Aged | Multimodal Imaging - methods | Tomography, X-Ray Computed | Colorectal Neoplasms - diagnostic imaging | Positron-Emission Tomography | Image Processing, Computer-Assisted - methods | Phantoms, Imaging | Quality Control | Studies | Algorithms | Fluorides | Noise | Scanners
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6. Full Text Nationwide epidemiological survey of Leber hereditary optic neuropathy in Japan
by Ueda, Kaori and Morizane, Yuki and Shiraga, Fumio and Shikishima, Keigo and Ishikawa, Hitoshi and Wakakura, Masato and Nakamura, Makoto
Journal of Epidemiology, ISSN 0917-5040, 09/2017, Volume 27, Issue 9, pp. 447 - 450
Leber hereditary optic neuropathy (LHON) is a maternally inherited optic neuropathy that leads to central loss of vision, predominantly in young males. Most... 
Annual incidence | Leber hereditary optic neuropathy | Penetrance | Mitochondrial DNA | Gender proportion | POPULATION | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | CLINICAL-FEATURES | MUTATIONS | MITOCHONDRIAL-DNA | Japan - epidemiology | DNA, Mitochondrial - genetics | Health Surveys | Humans | Optic Atrophy, Hereditary, Leber - epidemiology | Adult | Female | Male | Optic Atrophy, Hereditary, Leber - genetics | Mutation | Incidence | Confidence intervals | Optic neuropathy | Mathematical analysis | Neuropathy | Males | Females | Epidemiology | Eyes & eyesight | Deoxyribonucleic acid--DNA
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7. Full Text Selective ablation of basophils in mice reveals their nonredundant role in acquired immunity against ticks
by Wada, Takeshi and Ishiwata, Kenji and Koseki, Haruhiko and Ishikura, Tomoyuki and Ugajin, Tsukasa and Ohnuma, Naotsugu and Obata, Kazushige and Ishikawa, Ryosuke and Yoshikawa, Soichiro and Mukai, Kaori and Kawano, Yohei and Minegishi, Yoshiyuki and Yokozeki, Hiroo and Watanabe, Naohiro and Karasuyama, Hajime
Journal of Clinical Investigation, ISSN 0021-9738, 08/2010, Volume 120, Issue 8, pp. 2867 - 2875
Ticks are ectoparasitic arthropods that can transmit a variety of microorganisms to humans and animals during blood feeding, causing serious infectious... 
MEDICINE, RESEARCH & EXPERIMENTAL | MOUSE BASOPHIL | MAST CELL-DEFICIENT | CHRONIC ALLERGIC INFLAMMATION | IN-VIVO | TRYPTASE MMCP-6 | HAEMAPHYSALIS-LONGICORNIS TICKS | GUINEA-PIGS | PATHOGEN TRANSMISSION | HOST-RESISTANCE | DERMACENTOR-VARIABILIS | Receptors, IgE - physiology | Animals | Mice, Inbred C57BL | Basophils - physiology | Mast Cells - physiology | Mice | Immunoglobulin E - blood | Tick Infestations - immunology | Tryptases - physiology | Prevention | Lyme disease | Proteases | Basophils | Cellular immunity | Research | Properties | Risk factors
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8. Full Text Global metabolic reprogramming of colorectal cancer occurs at adenoma stage and is induced by MYC
by Satoh, Kiyotoshi and Yachida, Shinichi and Sugimoto, Masahiro and Oshima, Minoru and Nakagawa, Toshitaka and Akamoto, Shintaro and Tabata, Sho and Saitoh, Kaori and Kato, Keiko and Sato, Saya and Igarashi, Kaori and Aizawa, Yumi and Kajino-Sakamoto, Rie and Kojima, Yasushi and Fujishita, Teruaki and Enomoto, Ayame and Hirayama, Akiyoshi and Ishikawa, Takamasa and Taketo, Makoto Mark and Kushida, Yoshio and Haba, Reiji and Okano, Keiichi and Tomita, Masaru and Suzuki, Yasuyuki and Fukuda, Shinji and Aoki, Masahiro and Soga, Tomoyoshi
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 09/2017, Volume 114, Issue 37, pp. E7697 - E7706
Cancer cells alter their metabolism for the production of precursors of macromolecules. However, the control mechanisms underlying this reprogramming are... 
Metabolomics | Metabolism | MYC | Colorectal cancer | Omics | CELLS | colorectal cancer | MITOCHONDRIAL BIOGENESIS | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | TUMORS | omics | REPRESSION | GENE | metabolomics | MUTATION | metabolism | CARCINOMA | EXPRESSION | Adenoma - genetics | Colorectal Neoplasms - genetics | Humans | Carcinogenesis - genetics | Cell Proliferation - physiology | Transcriptome | Male | Genes, myc | Pyrimidines - biosynthesis | Carcinogenesis - metabolism | Metabolomics - methods | Proto-Oncogene Proteins c-myc - metabolism | Adenoma - metabolism | Animals | Female | Mice | Proto-Oncogene Proteins c-myc - genetics | Colorectal Neoplasms - metabolism | Disease Models, Animal | Physiological aspects | Health aspects | Methods | Cancer cells | Biological Sciences | PNAS Plus
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9. Full Text Emergence of nontrivial magnetic excitations in a spin-liquid state of kagomé volborthite
by Daiki Watanabe and Kaori Sugii and Masaaki Shimozawa and Yoshitaka Suzuki and Takeshi Yajima and Hajime Ishikawa and Zenji Hiroi and Takasada Shibauchi and Yuji Matsuda and Minoru Yamashita
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2016, Volume 113, Issue 31, pp. 8653 - 8657
When quantum fluctuations destroy underlying long-range ordered states, novel quantum states emerge. Spin-liquid (SL) states of frustrated quantum... 
FIELDS | thermal transport | CAF2 | MULTIDISCIPLINARY SCIENCES | THERMAL HALL CONDUCTIVITY | PHONON-SCATTERING | spin liquid | QUANTUM MAGNET | frustrated magnetism | LATTICE | TB3GA5O12 | Physics - Strongly Correlated Electrons | Physical Sciences
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10. Full Text Smartphone-Based Pedestrian’s Avoidance Behavior Recognition towards Opportunistic Road Anomaly Detection
by Ishikawa, Tsuyoshi and Fujinami, Kaori
ISPRS International Journal of Geo-Information, ISSN 2220-9964, 10/2016, Volume 5, Issue 10, p. 182
Road anomalies, such as cracks, pits and puddles, have generally been identified by citizen reports made by e-mail or telephone; however, it is difficult for... 
road anomaly | smartphone | opportunistic sensing | avoidance | behavior recognition
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11. Full Text Acquired expression of mutant mitofusin 2 causes progressive neurodegeneration and abnormal behavior
by Ishikawa, Kaori and Yamamoto, Satoshi and Hattori, Satoko and Nishimura, Naoya and Tani, Haruna and Mito, Takayuki and Matsumoto, Hirokazu and Miyakawa, Tsuyoshi and Nakada, Kazuto
Journal of Neuroscience, ISSN 0270-6474, 02/2019, Volume 39, Issue 9, pp. 1588 - 1604
Neurons have high plasticity in developmental and juvenile stages that decreases in adulthood. Mitochondrial dynamics are highly important in neurons to... 
MFN2 | Progressive neurodegeneration | Hyperactivity | Mitochondrial dynamics | Cognitive impairment | MITOCHONDRIAL FUSION | hyperactivity | PHENOTYPES | NEUROSCIENCES | MOUSE MODELS | mitochondrial dynamics | DRP1 | PATHOGENESIS | DISEASES | DNA | cognitive impairment | MICE | MUTATIONS | progressive neurodegeneration | Lethal effects | Cell survival | Neurodegenerative diseases | Neurons | Abnormalities | Mimicry | Neurological diseases | Learning | Mitochondria | Neurodegeneration | Dependence | Plasticity (developmental) | Brain damage | Brain injury
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12. Full Text Concentration of mitochondrial DNA mutations by cytoplasmic transfer from platelets to cultured mouse cells
by Ishikawa, Kaori and Kobayashi, Kohei and Yamada, Akihito and Umehara, Moe and Oka, Toshihiko and Nakada, Kazuto
PLoS ONE, ISSN 1932-6203, 03/2019, Volume 14, Issue 3, p. e0213283
Accumulation of mutations in mitochondrial DNA (mtDNA) is thought to be responsible for mitochondrial, and other, diseases and biological phenomena, such as... 
APOPTOSIS | HSP60 | DISEASES | CLONAL EXPANSION | MULTIDISCIPLINARY SCIENCES | MICE | GENERATION | EXPLAINS | DYSFUNCTION | DRIFT | MTDNA MUTATIONS | Mitochondrial diseases | Genetic aspects | Mitochondrial DNA | Research | Gene mutations | Cell lines | Animal models | Complex formation | Cybrids | Disease | DNA polymerase | Homology | Mutation rates | Tissues | Defects | Proteins | Polyethylene glycol | Aging | Deoxyribonucleic acid--DNA | Polypeptides | Neurodegenerative diseases | Abnormalities | Diabetes mellitus | Organs | Diseases | Neurological diseases | Environmental science | Mutagenesis | Quality control | Mutation | Respiration | Platelets | Cancer | Deoxyribonucleic acid | DNA
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13. Full Text Mice deficient in the Shmt2 gene have mitochondrial respiration defects and are embryonic lethal
by Tani, Haruna and Ohnishi, Sakiko and Shitara, Hiroshi and Mito, Takayuki and Yamaguchi, Midori and Yonekawa, Hiromichi and Hashizume, Osamu and Ishikawa, Kaori and Nakada, Kazuto and Hayashi, Jun-Ichi
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 425 - 8
Accumulation of somatic mutations in mitochondrial DNA (mtDNA) has been proposed to be responsible for human aging and age-associated mitochondrial respiration... 
Translation | tRNA fMet | Serine | Gene regulation | Lethality | Mitochondrial DNA | Embryos | Defects | Hypotheses | Fibroblasts | Aging | Epigenetics | Glycine C-acetyltransferase | Acetyltransferase | Mutation | Electron transport | Respiration | Age | Geriatrics
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14. Full Text Effects of prenatal stress and neonatal handling on anxiety, spatial learning and serotonergic system of male offspring mice
by Akatsu, Shigemi and Ishikawa, Chihiro and Takemura, Kaori and Ohtani, Akiko and Shiga, Takashi
Neuroscience Research, ISSN 0168-0102, 12/2015, Volume 101, pp. 15 - 23
Environmental factors during perinatal period have various effects on behavior. The present study examined the effects of prenatal stress and neonatal handling... 
Neonatal handling | Anxiety | Serotonin receptor | Spatial learning | Prenatal stress | BRAIN-REGIONS | 5-HT1A RECEPTORS | BEHAVIOR | RECEPTOR EXPRESSION | GESTATIONAL STRESS | POSTNATAL STIMULATION | NEUROSCIENCES | HIPPOCAMPUS | MATERNAL STRESS | RAT-BRAIN | GRANULE CELLS | Animals, Newborn | Frontal Lobe - metabolism | Prenatal Exposure Delayed Effects - psychology | Mice, Inbred C57BL | Receptor, Serotonin, 5-HT2C - metabolism | Anxiety - physiopathology | Male | Prenatal Exposure Delayed Effects - physiopathology | RNA, Messenger - metabolism | Prenatal Exposure Delayed Effects - metabolism | Receptors, Serotonin - metabolism | Spatial Learning - physiology | Pregnancy | Hippocampus - metabolism | Animals | Stress, Psychological - metabolism | Maternal Behavior | Female | Mice | Handling (Psychology) | Receptor, Serotonin, 5-HT2A - metabolism | Anxiety - metabolism | Receptor, Serotonin, 5-HT1A - metabolism | Stress, Psychological - physiopathology | Infants (Newborn) | Serotonin | Pregnant women
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15. Full Text Disruption of the mouse Shmt2 gene confers embryonic anaemia via foetal liver-specific metabolomic disorders
by Tani, H and Mito, T and Velagapudi, V and Ishikawa, K and Umehara, M and Nakada, K and Suomalainen, A and Hayashi, JI
SCIENTIFIC REPORTS, ISSN 2045-2322, 11/2019, Volume 9, Issue 1, pp. 1 - 11
In a previous study, we proposed that age-related mitochondrial respiration defects observed in elderly subjects are partially due to age-associated... 
MITOCHONDRIAL-DNA MUTATIONS | NUCLEAR | METABOANALYST | DISEASES | NEURAL-TUBE DEFECTS | MULTIDISCIPLINARY SCIENCES | MICE | PHENOTYPES | DYSFUNCTION | DEFICIENCY | DELETION | Taurine | Liver diseases | Growth rate | Anemia | Serine | Cell division | Lethality | Embryo fibroblasts | Nucleotides | Embryos | Defects | Mitochondria | Erythroblasts | Rodents | Metabolic pathways | Electron transport | Respiration | Age | Geriatrics
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