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1. Full Text Human CD14dim Monocytes Patrol and Sense Nucleic Acids and Viruses via TLR7 and TLR8 Receptors
by Cros, Jérôme and Cagnard, Nicolas and Woollard, Kevin and Patey, Natacha and Zhang, Shen-Ying and Senechal, Brigitte and Puel, Anne and Biswas, Subhra K and Moshous, Despina and Picard, Capucine and Jais, Jean-Philippe and D'Cruz, David and Casanova, Jean-Laurent and Trouillet, Céline and Geissmann, Fréderic
Immunity, ISSN 1074-7613, 09/2010, Volume 33, Issue 3, pp. 375 - 386
Monocytes are effectors of the inflammatory response to microbes. Human CD14 monocytes specialize in phagocytosis and production of reactive oxygen species and... 
Lipopolysaccharide Receptors - physiology | Reactive Oxygen Species - metabolism | Humans | GPI-Linked Proteins | Viruses - immunology | Animals | Antigen Presentation | Nucleic Acids - physiology | Receptors, IgG - analysis | Toll-Like Receptor 7 - physiology | Toll-Like Receptor 8 - physiology | Lupus Erythematosus, Systemic - immunology | Myeloid Differentiation Factor 88 - physiology | HLA-DR Antigens - analysis | Mice | Monocytes - physiology | Cytokines - biosynthesis | Studies | Flow cytometry | Cytokines | Acids | Principal components analysis | Genomes | Gene expression | Experiments | Viral infections
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2. Full Text IDH2 mutations are frequent in angioimmunoblastic T-cell lymphoma
by Cairns, Rob A and Iqbal, Javeed and Lemonnier, François and Kucuk, Can and De Leval, Laurence and Jais, Jean-Philippe and Parrens, Marie and Martin, Antoine and Xerri, Luc and Brousset, Pierre and Chan, Li Chong and Chan, Wing-Chung and Gaulard, Philippe and Mak, Tak W
Blood, ISSN 0006-4971, 02/2012, Volume 119, Issue 8, pp. 1901 - 1903
Mutations in isocitrate dehydrogenase 1 (IDH1) and isocitrate dehydrogenase 2 (IDH2) occur in most grade 2 and 3 gliomas, secondary glioblastomas, and a subset... 
GLIOMAS | PHENOTYPE | LEUKEMIA | HEMATOLOGY | CANCER | Prognosis | Immunoblastic Lymphadenopathy - genetics | Gene Frequency | Humans | Kaplan-Meier Estimate | Lymphoma, T-Cell, Peripheral - genetics | Genotype | Isocitrate Dehydrogenase - genetics | Male | Mutation Rate | Immunoblastic Lymphadenopathy - enzymology | Lymphoma, T-Cell - genetics | Lymphoma, T-Cell, Peripheral - enzymology | Lymphoma, T-Cell - enzymology | Lymphoma, T-Cell, Peripheral - pathology | Lymphoma, T-Cell - pathology | Female | Immunoblastic Lymphadenopathy - pathology | Aged | Mutation | Lymphoid Neoplasia | Brief Report
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3. Full Text SUVmax reduction improves early prognosis value of interim positron emission tomography scans in diffuse large B-cell lymphoma
by Casasnovas, René-Olivier and Meignan, Michel and Berriolo-Riedinger, Alina and Bardet, Stéphane and Julian, Anne and Thieblemont, Catherine and Vera, Pierre and Bologna, Serge and Brière, Josette and Jais, Jean-Philippe and Haioun, Corinne and Coiffier, Bertrand and Morschhauser, Franck and GELA and Groupe d'étude des lymphomes de l'adulte (GELA) and on behalf of the Groupe d'etude des lymphomes de l'adulte (GELA)
Blood, ISSN 0006-4971, 07/2011, Volume 118, Issue 1, pp. 37 - 43
The prognostic value of interim positron emission tomography (PET) interpreted according to visual criteria is a matter of debate in diffuse large B-cell... 
FDG-PET | CRITERIA | EARLY F-18-FDG PET | INTERNATIONAL WORKSHOP | HODGKINS-LYMPHOMA | RITUXIMAB | RESPONSE ASSESSMENT | FLUORODEOXYGLUCOSE | MEDICINE | HEMATOLOGY | CHEMOTHERAPY | Doxorubicin - therapeutic use | Predictive Value of Tests | Lymphoma, Large B-Cell, Diffuse - drug therapy | Prognosis | Prospective Studies | Humans | Middle Aged | Rituximab | Male | Antineoplastic Agents - therapeutic use | Positron-Emission Tomography - methods | Cyclophosphamide - therapeutic use | Lymphoma, Large B-Cell, Diffuse - diagnostic imaging | Bleomycin - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Adolescent | Vincristine - therapeutic use | Vindesine - therapeutic use | Adult | Female | Fluorodeoxyglucose F18 | Radiopharmaceuticals | Prednisone - therapeutic use | Antibodies, Monoclonal, Murine-Derived - therapeutic use
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4. Full Text Recurrent TET2 mutations in peripheral T-cell lymphomas correlate with T FH-like features and adverse clinical parameters
by Lemonnier, François and Couronné, Lucile and Parrens, Marie and Jaïs, Jean-Philippe and Travert, Marion and Lamant, Laurence and Tournillac, Olivier and Rousset, Therese and Fabiani, Bettina and Cairns, Rob A and Mak, Tak and Bastard, Christian and Bernard, Olivier A and De Leval, Laurence and Gaulard, Philippe
Blood, ISSN 0006-4971, 08/2012, Volume 120, Issue 7, pp. 1466 - 1469
Inactivating mutations of the Ten-Eleven Translocation 2 (TET2) gene were first identified in myeloid malignancies and more recently in peripheral T-cell... 
TRANSFORMATION | IDH2 MUTATIONS | FREQUENT | 5-METHYLCYTOSINE | GENES | LEADS | MYELOID MALIGNANCIES | DIFFERENTIATION | PROTEINS | HEMATOLOGY | EXPRESSION
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5. Full Text Incidence, presentation, and prognosis of malignancies in ataxia-telangiectasia: A report from the french national registry of primary immune deficiencies
by Suarez, Felipe and Mahlaoui, Nizar and Canioni, Danielle and Andriamanga, Chantal and D'Enghien, Catherine Dubois and Brousse, Nicole and Jais, Jean-Philippe and Fischer, Alain and Hermine, Olivier and Stoppa-Lyonnet, Dominique
Journal of Clinical Oncology, ISSN 0732-183X, 10/2015, Volume 33, Issue 2, pp. 202 - 208
Purpose Biallelic mutations in ATM cause ataxia-telangiectasia (AT), a rare inherited disease with a high incidence of cancer. Precise estimates of the risk,... 
MOLECULAR CHARACTERIZATION | INACTIVATION | OXIDATIVE STRESS | ONCOLOGY | KINASE | ATM GENE | LEUKEMIA | MUTATIONS | CANCER | IMMUNODEFICIENCY | LYMPHOID MALIGNANCIES | Ataxia Telangiectasia - mortality | Prognosis | Age Factors | Humans | Middle Aged | Male | Neoplasms - diagnosis | Incidence | Immunologic Deficiency Syndromes | Neoplasms - therapy | Neoplasms - genetics | Adult | Female | Registries | Retrospective Studies | Ataxia Telangiectasia - complications | Leukemia - epidemiology | Lymphoma - epidemiology | Acute Disease | France - epidemiology | Neoplasms - mortality | Hodgkin Disease - epidemiology | Lymphoma, Non-Hodgkin - epidemiology | Aged | Mutation | Neoplasms - epidemiology
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6. Full Text Effect of acute and chronic aldosterone exposure on the retinal pigment epithelium-choroid complex in rodents
by Canonica, Jérémie and Mehanna, Chadi and Bonnard, Benjamin and Jonet, Laurent and Gelize, Emmanuelle and Jais, Jean-Philippe and Jaisser, Frederic and Zhao, Min and Behar-Cohen, Francine
Experimental Eye Research, ISSN 0014-4835, 10/2019, Volume 187, p. 107747
Preclinical and clinical evidences show that aldosterone and/or mineralocorticoid receptor (MR) over-activation by glucocorticoids can be deleterious to the... 
RNA sequencing | Retina | Inflammation | Mineralocorticoid receptor
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7. Full Text Activating mutations in genes related to TCR signaling in angioimmunoblastic and other follicular helper T-cell-derived lymphomas
by Vallois, David and Dobay, Maria Pamela D and Morin, Ryan D and Lemonnier, François and Missiaglia, Edoardo and Juilland, Mélanie and Iwaszkiewicz, Justyna and Fataccioli, Virginie and Bisig, Bettina and Roberti, Annalisa and Grewal, Jasleen and Bruneau, Julie and Fabiani, Bettina and Martin, Antoine and Bonnet, Christophe and Michielin, Olivier and Jais, Jean-Philippe and Figeac, Martin and Bernard, Olivier A and Delorenzi, Mauro and Haioun, Corinne and Tournilhac, Olivier and Thome, Margot and Gascoyne, Randy D and Gaulard, Philippe and De Leval, Laurence
Blood, ISSN 0006-4971, 09/2016, Volume 128, Issue 11, pp. 1490 - 1502
Angioimmunoblastic T-cell lymphoma (AITL) and other lymphomas derived from follicular T-helper cells (TFH) represent a large proportion of peripheral T-cell... 
STAT3 MUTATIONS | PROLYMPHOCYTIC LEUKEMIA | NK CELLS | LANDSCAPE | RECURRENT MUTATIONS | KINASE | MISSENSE MUTATION | SEZARY-SYNDROME | HEMATOLOGY | EXPRESSION | RHOA | Prognosis | Follow-Up Studies | Signal Transduction | Immunoblastic Lymphadenopathy - genetics | Lymphoma, Follicular - pathology | Humans | Lymphoma, T-Cell, Peripheral - genetics | rhoA GTP-Binding Protein - genetics | Mutation - genetics | Lymphoma, Follicular - genetics | Lymphoma, Follicular - immunology | T-Lymphocytes, Helper-Inducer - immunology | Lymphoma, T-Cell, Peripheral - pathology | Genes, T-Cell Receptor - genetics | Immunoblastic Lymphadenopathy - pathology | Biomarkers, Tumor - genetics | High-Throughput Nucleotide Sequencing - methods | Immunoblastic Lymphadenopathy - immunology | Neoplasm Staging | Cohort Studies | Lymphoma, T-Cell, Peripheral - immunology | Life Sciences | Cancer
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8. Full Text MYC-IG rearrangements are negative predictors of survival in DLBCL patients treated with immunochemotherapy: A GELA/LYSA study
by Copie-Bergman, Christiane and Cuillière-Dartigues, Peggy and Baia, Maryse and Briere, Josette and Delarue, Richard and Canioni, Danielle and Salles, Gilles and Parrens, Marie and Belhadj, Karim and Fabiani, Bettina and Recher, Christian and Petrella, Tony and Ketterer, Nicolas and Peyrade, Frederic and Haioun, Corinne and Nagel, Inga and Siebert, Reiner and Jardin, Fabrice and Leroy, Karen and Jais, Jean-Philippe and Tilly, Herve and Molina, Thierry Jo and Gaulard, Philippe
Blood, ISSN 0006-4971, 11/2015, Volume 126, Issue 22, pp. 2466 - 2474
Diffuse large B-cell lymphoma (DLBCL) with MYC rearrangement (MYC-R) carries an unfavorable outcome. We explored the prognostic value of the MYC translocation... 
RANDOMIZED PHASE-3 TRIAL | RITUXIMAB PLUS CYCLOPHOSPHAMIDE | BURKITTS-LYMPHOMA | INTENSIFIED CHEMOTHERAPY | TRANSLOCATIONS | POOR-PROGNOSIS | NON-HODGKINS-LYMPHOMA | SINGLE CENTERS EXPERIENCE | HEMATOLOGY | B-CELL-LYMPHOMA | ELDERLY-PATIENTS | Translocation, Genetic | Cyclophosphamide - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Humans | Middle Aged | Antibodies, Monoclonal, Murine-Derived - administration & dosage | Male | Immunoglobulins - genetics | Lymphoma, Large B-Cell, Diffuse - metabolism | Immunoglobulins - metabolism | Aged, 80 and over | Vincristine - administration & dosage | Adult | Female | Doxorubicin - administration & dosage | Prednisone - administration & dosage | Lymphoma, Large B-Cell, Diffuse - drug therapy | Survival Rate | Proto-Oncogene Proteins c-myc - metabolism | Lymphoma, Large B-Cell, Diffuse - mortality | Disease-Free Survival | Gene Rearrangement | Adolescent | Aged | Proto-Oncogene Proteins c-myc - genetics | Lymphoma, Large B-Cell, Diffuse - genetics | Life Sciences | Cancer
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9. Full Text Preoperative circulating tumor cell detection using the isolation by size of epithelial tumor cell method for patients with lung cancer is a new prognostic biomarker
by Hofman, Véronique and Bonnetaud, Christelle and Ilie, Marius I and Vielh, Philippe and Vignaud, Jean Michel and Fléjou, Jean Fraņcois and Lantuejoul, Sylvie and Piaton, Eric and Mourad, Nadine and Butori, Catherine and Selva, Eric and Poudenx, Michel and Sibon, Stéphanie and Kelhef, Sabrina and Vénissac, Nicolas and Jais, Jean-Philippe and Mouroux, Jérôme and Molina, Thierry Jo and Hofman, Paul
Clinical Cancer Research, ISSN 1078-0432, 02/2011, Volume 17, Issue 4, pp. 827 - 835
Purpose: Pathologic TNM staging is currently the best prognostic factor for non-small cell lung carcinoma (NSCLC). However, even in early-stage NSCLC, the... 
SURVIVAL | POLYMERASE-CHAIN-REACTION | MESSENGER-RNA | ONCOLOGY | INTERNATIONAL SYSTEM | RESECTION | MARKER | RT-PCR | ENDOTHELIAL-CELLS | PERIPHERAL-BLOOD | EXPRESSION | Carcinoma, Non-Small-Cell Lung - pathology | Multivariate Analysis | Neoplastic Cells, Circulating - pathology | Lung Neoplasms - mortality | Humans | Middle Aged | Kaplan-Meier Estimate | Lung Neoplasms - pathology | Male | Carcinoma, Non-Small-Cell Lung - diagnosis | Lung Neoplasms - therapy | Carcinoma, Non-Small-Cell Lung - mortality | Case-Control Studies | Biomarkers, Tumor - blood | Disease-Free Survival | Carcinoma, Non-Small-Cell Lung - therapy | Aged, 80 and over | Adult | Female | Aged | Lung Neoplasms - diagnosis | Neoplasm Staging | Preoperative Period
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10. Full Text Intensified chemotherapy with ACVBP plus rituximab versus standard CHOP plus rituximab for the treatment of diffuse large B-cell lymphoma (LNH03-2B): an open-label randomised phase 3 trial
by Récher, Christian, Prof and Coiffier, Bertrand, Prof and Haioun, Corinne, Prof and Molina, Thierry Jo, Prof and Fermé, Christophe, MD and Casasnovas, Olivier, MD and Thiéblemont, Catherine, Prof and Bosly, André, Prof and Laurent, Guy, Prof and Morschhauser, Franck, MD and Ghesquières, Hervé, MD and Jardin, Fabrice, Prof and Bologna, Serge, MD and Fruchart, Christophe, MD and Corront, Bernadette, MD and Gabarre, Jean, MD and Bonnet, Christophe, MD and Janvier, Maud, MD and Canioni, Danielle, Prof and Jais, Jean-Philippe, MD and Salles, Gilles, Prof and Tilly, Hervé, Dr, Prof and Grp Etud Lymphomes Adulte and Groupe d'Etude des Lymphomes de l'Adulte
Lancet, The, ISSN 0140-6736, 2011, Volume 378, Issue 9806, pp. 1858 - 1867
Summary Background The outcome of diffuse large B-cell lymphoma has been substantially improved by the addition of the anti-CD20 monoclonal antibody rituximab... 
Internal Medicine | MEDICINE, GENERAL & INTERNAL | MULTICENTER | RESPONSE CRITERIA | AGGRESSIVE LYMPHOMAS | POOR-PROGNOSIS | NON-HODGKINS-LYMPHOMA | REGIMEN | YOUNG-PATIENTS | ELDERLY-PATIENTS | DES-LYMPHOMES | TRANSPLANTATION | Cyclophosphamide - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Prospective Studies | Follow-Up Studies | Humans | Middle Aged | Antibodies, Monoclonal, Murine-Derived - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Male | Cyclophosphamide - adverse effects | Dose-Response Relationship, Drug | Young Adult | Vindesine - adverse effects | Adult | Female | Prednisolone | Doxorubicin - administration & dosage | Severity of Illness Index | Prednisone - administration & dosage | Lymphoma, Large B-Cell, Diffuse - drug therapy | Prednisone - adverse effects | Drug Administration Schedule | Risk Assessment | Rituximab | Treatment Outcome | Bleomycin - administration & dosage | Bleomycin - adverse effects | Vincristine | Lymphoma, Large B-Cell, Diffuse - mortality | Disease-Free Survival | Lymphoma, Large B-Cell, Diffuse - diagnosis | Maximum Tolerated Dose | Adolescent | Survival Analysis | Vindesine - administration & dosage | Doxorubicin - adverse effects | Chemotherapy | Physiological aspects | Lymphomas | B cells | Drug therapy | Methods | Cancer | Dehydrogenases | Toxicity | Population studies | Doxorubicin | Hepatitis | Randomization | Bleomycin | Motivation | Drug dosages | Neutropenia | Cell survival | Hematology | Prednisone | Survival | Patients | Lymphoma | Cyclophosphamide | Lymphocytes B | Monoclonal antibodies | CD20 antigen | Tumors | B-cell lymphoma | Clinical trials | Public speaking | Grants | Clinical medicine | Side effects | lymphoma | survival | International standardization | chemotherapy
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11. Full Text Mutation of RRM2B, encoding p53-controlled ribonucleotide reductase (p53R2), causes severe mitochondrial DNA depletion
by Bourdon, Alice and Minai, Limor and Serre, Valérie and Jais, Jean-Philippe and Sarzi, Emmanuelle and Aubert, Sophie and Chrétien, Dominique and De Lonlay, Pascale and Paquis-Flucklinger, Véronique and Arakawa, Hirofumi and Nakamura, Yusuke and Munnich, Arnold and Rötig, Agnès
Nature Genetics, ISSN 1061-4036, 06/2007, Volume 39, Issue 6, pp. 776 - 780
Mitochondrial DNA (mtDNA) depletion syndrome (MDS; MIM 251880) is a prevalent cause of oxidative phosphorylation disorders characterized by a reduction in... 
SWISS-MODEL | CLUSPRO | CELLS | GENE | DNTP POOLS | DOCKING | KINASE | GENETICS & HEREDITY | R2 PROTEIN | ENCEPHALOMYOPATHY | DAMAGE | Mitochondrial Diseases - pathology | Ribonucleotide Reductases - physiology | Mitochondria, Muscle | Humans | Molecular Sequence Data | Male | Lod Score | Tumor Suppressor Protein p53 - genetics | Mitochondrial Diseases - etiology | DNA, Mitochondrial - genetics | DNA Mutational Analysis | Gene Deletion | Cell Cycle Proteins - genetics | Fibroblasts | Female | Infant, Newborn | Cells, Cultured | Tumor Suppressor Protein p53 - metabolism | Mutation - genetics | Mice, Knockout | Homozygote | Animals | Pedigree | Mice | Ribonucleotide Reductases - genetics | Cell Cycle Proteins - physiology | Mitochondrial DNA | Research | Gene mutations | Properties | Ribonucleoside reductase | Genetics | Oxidation | Mutation | Rodents
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12. Full Text Next-generation sequencing in diffuse large B-cell lymphoma highlights molecular divergence and therapeutic opportunities: A LYSA study
by Dubois, Sydney and Viailly, Pierre-Julien and Mareschal, Sylvain and Bohers, Elodie and Bertrand, Philippe and Ruminy, Philippe and Maingonnat, Catherine and Jais, Jean-Philippe and Peyrouze, Pauline and Figeac, Martin and Molina, Thierry J and Desmots, Fabienne and Fest, Thierry and Haioun, Corinne and Lamy, Thierry and Copie-Bergman, Christiane and Brière, Josette and Petrella, Tony and Canioni, Danielle and Fabiani, Bettina and Coiffier, Bertrand and Delarue, Richard and Peyrade, Frédéric and Bosly, André and André, Marc and Ketterer, Nicolas and Salles, Gilles and Tilly, Hervé and Leroy, Karen and Jardin, Fabrice
Clinical Cancer Research, ISSN 1078-0432, 06/2016, Volume 22, Issue 12, pp. 2919 - 2928
Purpose: Next-generation sequencing (NGS) has detailed the genomic characterization of diffuse large B-cell lymphoma (DLBCL) by identifying recurrent somatic... 
FREQUENT | BURKITT-LYMPHOMA | ONCOLOGY | RITUXIMAB | TUMOR-SUPPRESSOR GENE | TARGETS | HODGKIN LYMPHOMA | PROGNOSTIC-SIGNIFICANCE | EXPRESSION | SOMATIC MUTATIONS | GENOME | Doxorubicin - therapeutic use | Oncogene Proteins - genetics | Prospective Studies | Humans | Antineoplastic Agents - therapeutic use | Gene Expression Profiling | Cyclophosphamide - therapeutic use | Cell Cycle Proteins - genetics | B-Lymphocytes - pathology | Molecular Targeted Therapy - methods | GTP-Binding Protein alpha Subunits, Gq-G11 - genetics | Lymphoma, Large B-Cell, Diffuse - drug therapy | Phosphotransferases (Alcohol Group Acceptor) - genetics | Receptors, Cytoplasmic and Nuclear - genetics | DNA-Binding Proteins - genetics | Whole Exome Sequencing | Tumor Necrosis Factor alpha-Induced Protein 3 - genetics | B-Lymphocytes - immunology | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Vincristine - therapeutic use | Karyopherins - genetics | High-Throughput Nucleotide Sequencing | Lymphoma, Large B-Cell, Diffuse - genetics | Precision Medicine - methods | Prednisone - therapeutic use | Antibodies, Monoclonal, Murine-Derived - therapeutic use | Life Sciences | Human health and pathology | Immunology | Hematology | Cancer
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13. Full Text Event-free survival at 24 months is a robust end point for disease-related outcome in diffuse large B-cell lymphoma treated with immunochemotherapy
by Maurer, Matthew J and Maurer, Matthew J and Ghesquières, Hervé and Ghesquières, Hervé and Jais, Jean-Philippe and Jais, Jean-Philippe and Witzig, Thomas E and Witzig, Thomas E and Haioun, Corinne and Haioun, Corinne and Thompson, Carrie A and Thompson, Carrie A and Delarue, Richard and Delarue, Richard and Micallef, Ivana N and Micallef, Ivana N and Peyrade, Frédéric and Peyrade, Fréd́eric and Macon, William R and Macon, William R and Molina, Thierry Jo and Jo Molina, Thierry and Ketterer, Nicolas and Ketterer, Nicolas and Syrbu, Sergei I and Syrbu, Sergei I and Fitoussi, Olivier and Fitoussi, Olivier and Kurtin, Paul J and Kurtin, Paul J and Allmer, Cristine and Allmer, Cristine and Nicolas-Virelizier, Emmanuelle and Nicolas-Virelizier, Emmanuelle and Slager, Susan L and Slager, Susan L and Habermann, Thomas M and Habermann, Thomas M and Link, Brian K and Link, Brian K and Salles, Gilles and Salles, Gilles and Tilly, Hervé and Tilly, Hervé and Cerhan, James R and Cerhan, James R
Journal of Clinical Oncology, ISSN 0732-183X, 2014, Volume 32, Issue 10, pp. 1066 - 1073
Purpose Studies of diffuse large B-cell lymphoma (DLBCL) are typically evaluated by using a time-to-event approach with relapse, re-treatment, and death... 
RANDOMIZED PHASE-3 TRIAL | ACVBP | ONCOLOGY | PROGNOSIS | ERA | OLDER | RISK | CHOP CHEMOTHERAPY | CHEMOTHERAPY PLUS RITUXIMAB | ELDERLY-PATIENTS | Recurrence | Lymphoma, Large B-Cell, Diffuse - drug therapy | Humans | Middle Aged | Antibodies, Monoclonal, Murine-Derived - administration & dosage | Kaplan-Meier Estimate | Rituximab | Male | Clinical Trials as Topic | Case-Control Studies | Lymphoma, Large B-Cell, Diffuse - mortality | Young Adult | Disease-Free Survival | Time Factors | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Adolescent | Aged, 80 and over | Adult | Female | Aged | Bios4 | Bios3 | ORIGINAL REPORTS | Hema10
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14. Full Text Sex- and diet-specific changes of imprinted gene expression and dna methylation in mouse placenta under a high-fat diet
by Gallou-Kabani, Catherine and Gabory, Anne and Tost, Jörg and Karimi, Mohsen and Mayeur, Sylvain and Lesage, Jean and Boudadi, Elsa and Gross, Marie-Sylvie and Taurelle, Julien and Vigé, Alexandre and Breton, Christophe and Reusens, Brigitte and Remacle, Claude and Vieau, Didier and Ekström, Tomas J and Jais, Jean-Philippe and Junien, Claudine
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 12, p. e14398
Background: Changes in imprinted gene dosage in the placenta may compromise the prenatal control of nutritional resources. Indeed monoallelic behaviour and... 
BOVINE BLASTOCYSTS | MURINE PLACENTA | ORGANIC CATION TRANSPORTERS | NUTRIENT TRANSPORT | ADAPTIVE RESPONSES | TROPHOBLAST DIFFERENTIATION | MULTIDISCIPLINARY SCIENCES | FETAL-GROWTH | EPIGENETIC REGULATION | MATERNAL UNDERNUTRITION | HUMAN CHORIONIC-GONADOTROPIN | Pregnancy, Animal | Dietary Fats - metabolism | Genomic Imprinting | Epigenesis, Genetic | Gene Expression Regulation | Male | Gene Dosage | Placenta - metabolism | Pregnancy | DNA Methylation | Animals | Alleles | Sex Factors | Female | Mice | Animal Feed | Epigenetic inheritance | Chromatin | Genes | Genomics | Genomes | DNA binding proteins | Genetic transcription | Epidemiology | Diet | Pregnant women | DNA | Physiological aspects | Genetic research | Book publishing | Comparative analysis | Methylation | Neurosciences | Deregulation | Transcription factors | Breastfeeding & lactation | Laboratories | Biological evolution | Sex | Cardiovascular disease | Dimorphism | Gestation | High fat diet | Rodents | DNA methylation | Clusters | Life sciences | Sexual dimorphism | Chromosomes | Chromosome 17 | Deoxyribonucleic acid--DNA | CpG islands | Hypertension | Gene dosage | Obesity | Sensitivity analysis | Nutrition | Cloning | Metabolism | Gene expression | Embryos | Chromatin remodeling | Studies | Offspring | Hypotheses | Sensitivity | Placenta | Womens health | Sexes | Preeclampsia | Quality control | Epigenetics | Binding sites | Deoxyribonucleic acid
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