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Journal of Religious History, ISSN 0022-4227, 09/2012, Volume 36, Issue 3, pp. 434 - 435
Journal Article
FRONTIERS IN IMMUNOLOGY, ISSN 1664-3224, 03/2019, Volume 10, p. 325
The Interferon regulatory factors (IRFs) are a family of transcription factors that play pivotal roles in many aspects of the immune response, including immune... 
TRANSCRIPTION FACTORS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | E3 ligase | DENDRITIC CELLS | I INTERFERON INDUCTION | CELLULAR ANTIVIRAL RESPONSE | IMMUNOLOGY | interferon | ubiquitin | NEGATIVE REGULATION | DIFFERENTIAL ACTIVATION | GENE-EXPRESSION | microRNA | monocyte | NF-KAPPA-B | MACROPHAGE POLARIZATION
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2007, Volume 104, Issue 9, pp. 3372 - 3377
Toll-like receptors (TLRs)-2 and -4 are important proteins in innate immunity, recognizing microbial products and eliciting host defense responses. Both use... 
Receptors | HEK293 cells | Plasmids | Peritoneal macrophages | Antibodies | Reporter genes | Ligands | Mice | Physiological regulation | Macrophages | Signaling | Toll-like receptor | APOPTOSIS | signaling | MICE DEFICIENT | MULTIDISCIPLINARY SCIENCES | ESCHERICHIA-COLI | ENDOTOXEMIA | CYTOKINE | IPAF | INTERLEUKIN-1-BETA CONVERTING-ENZYME | ASC | INFLAMMATORY CASPASES | toll-like receptor | IL-1-BETA | Biological Sciences
Journal Article
Rheumatology, ISSN 1462-0332, 04/2015
Journal Article
Current rheumatology reports, 07/2019, Volume 21, Issue 9, p. 46
The systemic inflammatory nature of systemic lupus erythematosus (SLE) is patent not only in the diverse clinical manifestations of the disease but also in the... 
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 7, p. e11776
Ro52 is a member of the TRIM family of single-protein E3 ligases and is also a target for autoantibody production in systemic lupus erythematosus and Sjogren's... 
E3 LIGASE | UBIQUITINATION | IFN REGULATORY FACTOR-3 | BIOLOGY | INNATE IMMUNE-RESPONSE | GENE-EXPRESSION | AUTOANTIGEN RO52 | MEDIATED DEGRADATION | TRIM FAMILY PROTEINS | INTERFERON-ALPHA INDUCTION | I INTERFERON | Toll-Like Receptor 7 - genetics | Cell Line | Interferon-alpha - metabolism | Immunoprecipitation | Toll-Like Receptor 7 - metabolism | Humans | Toll-Like Receptor 9 - genetics | Ribonucleoproteins - metabolism | Interferon Regulatory Factor-7 - genetics | Interferon Regulatory Factor-7 - metabolism | Mice, Knockout | Ubiquitination | Animals | Toll-Like Receptors - genetics | Protein Binding | Ribonucleoproteins - genetics | Mice | HeLa Cells | Toll-Like Receptors - metabolism | Toll-Like Receptor 9 - metabolism | Ubiquitin | Autoimmunity | Antiviral agents | Autoantibodies | Ligases | Protein binding | Sjogren's syndrome | Imiquimod | Stimulation | Infections | Feedback loops | Kinases | Macrophages | Degradation | Proteins | Receptors | Systemic lupus erythematosus | Bone marrow | Toll-like receptors | Drug dosages | CpG islands | Immune system | Lupus | Immune response | TLR7 protein | Stability | Cytokines | Gene expression | TLR3 protein | TLR9 protein | Interferon regulatory factor 7 | Signaling | Chronic conditions | Interferon regulatory factor 3 | Proteasomes | Interferon | Autoimmune diseases | Viral infections | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e85834
Bacterial Lipopolysaccharide (LPS) is a strong inducer of inflammation and does so by inducing polarization of macrophages to the classic inflammatory M1... 
SYSTEM | GAMMA | BRUTONS TYROSINE KINASE | DENDRITIC CELLS | CELL-DEVELOPMENT | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | X-LINKED AGAMMAGLOBULINEMIA | GENE-EXPRESSION | NF-KAPPA-B | ALTERNATIVE ACTIVATION | Inflammation - pathology | Transcription, Genetic - drug effects | Hypersensitivity - enzymology | Protein-Tyrosine Kinases - metabolism | Hypersensitivity - complications | Protein-Tyrosine Kinases - deficiency | Macrophages, Peritoneal - cytology | Inflammation - complications | Cell Polarity - drug effects | Phosphorylation - drug effects | Macrophages, Peritoneal - drug effects | Mice, Inbred C57BL | Macrophages, Peritoneal - enzymology | Macrophages - cytology | Toll-Like Receptor 4 - metabolism | Macrophages - enzymology | Phenotype | Animals | Signal Transduction - drug effects | Hypersensitivity - pathology | Models, Biological | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | Inflammation - enzymology | Inflammation | Genetic transcription | DNA binding proteins | Macrophages | Mitogens | Polarization | Animal models | Transcription factors | Phosphorylation | Disease | Genes | AKT protein | Infections | Activation | Kinases | Eggs | Lipopolysaccharides | Recruitment | Proteins | Genotype & phenotype | Rodents | Transcription activation | Toll-like receptors | Bacteria | Stat1 protein | NF-κB protein | Cytokines | Dendritic cells | Hypersensitivity | Chitin | TLR4 protein | Dentistry | Gene expression | Bruton's tyrosine kinase | Nitric-oxide synthase | Studies | Immunosuppression | Stat6 protein | Inositol-1,4,5-trisphosphate 5-phosphatase | Regulatory proteins | Stimuli
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, p. e103609
Journal Article
Immunity, ISSN 1074-7613, 01/2013, Volume 38, Issue 1, pp. 66 - 78
Journal Article