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Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 361 - 365
Angiotensin II (AngII) mediates progression of aortic aneurysm, but the relative contribution of its type 1 (AT1) and type 2 (AT2) receptors remains unknown.... 
Connective tissues | Receptors | Root growth | Medical treatment | REPORTS | Aneurysms | Placebos | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | ACTIVATION | MECHANISM | MULTIDISCIPLINARY SCIENCES | MARFAN-SYNDROME | MOUSE MODEL | GROWTH | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | EXPRESSION | Aortic Aneurysm - metabolism | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Aortic Rupture - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Enalapril - therapeutic use | MAP Kinase Signaling System | Marfan Syndrome - drug therapy | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Aortic Rupture - prevention & control | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Receptor, Angiotensin, Type 2 - genetics | Angiotensin II - metabolism | Signal Transduction | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Losartan - pharmacology | Aortic Rupture - pathology | Disease Progression | Mice, Knockout | Animals | Enalapril - pharmacology | Receptor, Angiotensin, Type 2 - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Aortic Aneurysm - drug therapy | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Aortic aneurysms | Development and progression | Genetic aspects | Health aspects | Angiotensin | Signal transduction | Peptides | Cellular biology | Coronary vessels | Rodents
Journal Article
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 358 - 361
Transforming growth factor—β (TGFβ) signaling drives aneurysm progression in multiple disorders, including Marfan syndrome (MFS), and therapies that inhibit... 
Transcriptional regulatory elements | Echocardiography | Aortic diseases | Root growth | REPORTS | Placebos | Aneurysms | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | CELLS | ACTIVATION | VALSALVA | MULTIDISCIPLINARY SCIENCES | N-TERMINAL KINASE | MOUSE MODEL | NOONANS-SYNDROME | RECEPTOR | MUTATIONS | INHIBITOR | Diphenylamine - pharmacology | Aortic Aneurysm - metabolism | Mitogen-Activated Protein Kinase 8 - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Anthracenes - therapeutic use | MAP Kinase Signaling System | Diphenylamine - analogs & derivatives | Marfan Syndrome - drug therapy | Smad4 Protein - genetics | Transforming Growth Factor beta - antagonists & inhibitors | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Transforming Growth Factor beta - immunology | Diphenylamine - therapeutic use | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Mitogen-Activated Protein Kinase 8 - metabolism | Aortic Aneurysm - physiopathology | Losartan - pharmacology | Smad2 Protein - metabolism | Sulfonamides - pharmacology | Anthracenes - pharmacology | Disease Progression | Aorta - pathology | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Sulfonamides - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Protein Kinase Inhibitors - pharmacology | Smad4 Protein - deficiency | Enzyme Activation | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article
Science, ISSN 0036-8075, 4/2006, Volume 312, Issue 5770, pp. 117 - 121
Aortic aneurysm and dissection are manifestations of Marfan syndrome (MFS), a disorder caused by mutations in the gene that encodes fibrillin-1. Selected... 
Airspace | Receptors | Architecture | Dilatation | Root growth | Placebos | Reports | Mice | Aortic aneurysm | Elastic tissue | Marfan syndrome | PATHOGENESIS | GROWTH-FACTOR-BETA | ACTIVATION | FIBRILLIN | MULTIDISCIPLINARY SCIENCES | RATS | RECEPTOR | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | DILATATION | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Propranolol - therapeutic use | Neutralization Tests | Aortic Aneurysm - etiology | Lung Diseases - drug therapy | Marfan Syndrome - drug therapy | Propranolol - administration & dosage | Antibodies - immunology | Transforming Growth Factor beta - antagonists & inhibitors | Female | Marfan Syndrome - complications | Microfilament Proteins - genetics | Adrenergic beta-Antagonists - administration & dosage | Adrenergic beta-Antagonists - therapeutic use | Angiotensin II Type 1 Receptor Blockers - administration & dosage | Aortic Aneurysm - prevention & control | Disease Models, Animal | Transforming Growth Factor beta - immunology | Fibrillin-1 | Lung - pathology | Pulmonary Alveoli - pathology | Signal Transduction | Fibrillins | Pregnancy Complications - drug therapy | Aorta - pathology | Pregnancy | Elastic Tissue - pathology | Animals | Receptor, Angiotensin, Type 1 - metabolism | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Losartan - administration & dosage | Lung Diseases - pathology | Mutation | Transforming Growth Factor beta - metabolism | Complications and side effects | Aortic aneurysms | Genetic aspects | Research | Risk factors
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2014, Volume 124, Issue 1, pp. 448 - 460
Loeys-Dietz syndrome (LDS) is a connective tissue disorder that is characterized by a high risk for aneurysm and dissection throughout the arterial tree and... 
Connective tissue diseases | Angiotensin | Physiological aspects | Development and progression | Genetic aspects | Cellular signal transduction | Research | Transforming growth factors | Studies | Substance abuse treatment | Rodents | Genes | Cloning | Mutation | Kinases | Dissection | Age
Journal Article
Science, ISSN 0036-8075, 04/2006, Volume 312, Issue 5770, p. 117
Aortic aneurysm and dissection are manifestations of Marfan syndrome (MFS), a disorder caused by mutations in the gene that encodes fibrillin-1. Selected... 
Signal transduction | Genetic disorders | Antagonist drugs | Rodents | Pharmacology | Models
Journal Article
Journal Article
JOURNAL OF CLINICAL INVESTIGATION, ISSN 0021-9738, 01/2014, Volume 124, Issue 1, pp. 448 - 460
Journal Article
Science, ISSN 0036-8075, 04/2011, Volume 332, Issue 6027, pp. 358 - 361
Activation of transforming growth factor-β (TGF-β) signaling promotes the development of aortic aneurysms in the connective tissue disorder Marfan syndrome... 
Development and progression | Aortic aneurysms | Genetic aspects | Marfan syndrome | Aneurysms | Signal transduction | Cytokines | Cellular biology | Rodents | Coronary vessels
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 01/2014, Volume 25, Issue 1, pp. 81 - 91
Journal Article
Nature Medicine, ISSN 1078-8956, 02/2007, Volume 13, Issue 2, p. 204
  Skeletal muscle has the ability to achieve rapid repair in response to injury or disease. Many individuals with Marfan syndrome (MFS), caused by a deficiency... 
Musculoskeletal system | Signal transduction | Medical research | Rodents | Muscular dystrophy
Journal Article
Science (Washington), ISSN 0036-8075, 04/2006, Volume 312, Issue 5770, pp. 117 - 121
Aortic aneurysm and dissection are manifestations of Marfan syndrome (MFS), a disorder caused by mutations in the gene that encodes fibrillin-1. Selected... 
Journal Article
SCIENCE TRANSLATIONAL MEDICINE, ISSN 1946-6234, 05/2019, Volume 11, Issue 490, p. eaat4822
Women with Marfan syndrome (MFS) are at high risk for pregnancy-associated aortic dissection. Pathogenic models that singularly invoke hemodynamic stress are... 
MEDICINE, RESEARCH & EXPERIMENTAL | ROOT REPLACEMENT | WOMEN | GROWTH | VASOPRESSIN | MICE | UTERINE CONTRACTILITY | CONTRIBUTES | EXPRESSION | CARDIOVASCULAR PROBLEMS | ANEURYSM PROGRESSION | CELL BIOLOGY
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2014, Volume 124, Issue 1, pp. 448 - 448
Loeys-Dietz syndrome (LDS) is a connective tissue disorder that is characterized by a high risk for aneurysm and dissection throughout the arterial tree and... 
Journal Article