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Nature, ISSN 0028-0836, 02/2018, Volume 554, Issue 7693, pp. 544 - 548
Therapeutic antibodies that block the programmed death-1 (PD-1)-programmed death-ligand 1 (PD-L1) pathway can induce robust and durable responses in patients... 
METASTATIC MELANOMA | CTLA-4 BLOCKADE | BLADDER-CANCER | THERAPY | LANDSCAPE | SUBTYPES | UROTHELIAL CARCINOMA | MULTIDISCIPLINARY SCIENCES | GENES | ADAPTIVE IMMUNE RESISTANCE | MPDL3280A | CD8-Positive T-Lymphocytes - cytology | Humans | Antibodies, Monoclonal - therapeutic use | Urothelium - pathology | Urologic Neoplasms - pathology | Neoplasm Metastasis | Urologic Neoplasms - drug therapy | Urologic Neoplasms - genetics | Immunotherapy | Antibodies - immunology | Antigens, Neoplasm - metabolism | Transforming Growth Factor beta - antagonists & inhibitors | Antigens, Neoplasm - analysis | Disease Models, Animal | Fibroblasts - metabolism | Antibodies - therapeutic use | Antigens, Neoplasm - immunology | Antibodies, Monoclonal - pharmacology | Treatment Outcome | B7-H1 Antigen - immunology | Urologic Neoplasms - immunology | Antibodies - pharmacology | Tumor Microenvironment - immunology | Collagen - metabolism | Phenotype | Animals | B7-H1 Antigen - antagonists & inhibitors | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | CD8-Positive T-Lymphocytes - drug effects | Urothelium - immunology | Mice | Mutation | CD8-Positive T-Lymphocytes - immunology | Urothelium - drug effects | Transforming Growth Factor beta - metabolism | Cohort Studies | Drug Resistance, Neoplasm - drug effects | PD-1 protein | CD8 antigen | Transforming growth factor-a | Antibodies | Parenchyma | Lymphocytes T | Metastasis | Immunity | Metastases | Genotype & phenotype | Signal transduction | Lymphocytes | Cell cycle | Fibroblasts | Growth factors | Urothelial cancer | Phenotypes | Cytokines | Melanoma | Blocking | Gene expression | Patients | Generalized linear models | Signaling | Stromal cells | Medical prognosis | Collagen | PD-L1 protein | Ligands | Determinants | Infiltration | Cancer | Tumors | Apoptosis | Index Medicus | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Cancer and Oncology | Medicin och hälsovetenskap | Cancer och onkologi
Journal Article
Nature Immunology, ISSN 1529-2908, 07/2010, Volume 11, Issue 7, pp. 635 - 643
It is now established that the transcription factors E2A, EBF1 and Foxo1 have critical roles in B cell development. Here we show that E2A and EBF1 bound... 
LOOP-HELIX PROTEIN | PROGENITORS | CHROMATIN SIGNATURES | HUMAN GENOME | COMMITMENT | DISTINCT | GENE-EXPRESSION | DIFFERENTIATION | SPECIFICATION | IMMUNOLOGY | LINEAGE | Precursor Cells, B-Lymphoid - metabolism | Lymphopoiesis - genetics | Precursor Cells, B-Lymphoid - immunology | Gene Regulatory Networks | TCF Transcription Factors - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Precursor Cells, B-Lymphoid - pathology | Gene Expression Regulation, Developmental | Trans-Activators - genetics | B-Lymphocytes - pathology | B-Lymphocytes - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | Mice, Inbred C57BL | Cells, Cultured | Transcription Factor 7-Like 1 Protein | Forkhead Transcription Factors - genetics | Mice, Knockout | Cell Lineage | Animals | B-Lymphocytes - immunology | Trans-Activators - metabolism | Forkhead Box Protein O1 | Mice | Histones - metabolism | Methylation | TCF Transcription Factors - genetics | Regulatory Elements, Transcriptional - genetics | Physiological aspects | Transcription factors | Genetic aspects | Cellular signal transduction | B cells | Research | Regulators | Cell survival | Cell fate | Lymphocytes B | FOXO1 protein | Lysine | Regulatory sequences | Loci | Histone H3 | Index Medicus | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN
Journal Article
Nature Medicine, ISSN 1078-8956, 06/2018, Volume 24, Issue 6, pp. 749 - 757
We describe results from IMmotion150, a randomized phase 2 study of atezolizumab (anti-PD-L1) alone or combined with bevacizumab (anti-VEGF) versus sunitinib... 
MEDICINE, RESEARCH & EXPERIMENTAL | MULTICENTER | PD-L1 EXPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHASE-III | SINGLE-ARM | CANCER | CELL BIOLOGY | PATHWAY | SUPPRESSOR-CELLS | NIVOLUMAB | TUMOR-GROWTH | ENDOTHELIAL GROWTH-FACTOR | Sunitinib - pharmacology | Bevacizumab - pharmacology | Kidney Neoplasms - genetics | Bevacizumab - therapeutic use | Bevacizumab - adverse effects | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Gene Expression Regulation, Neoplastic | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Carcinoma, Renal Cell - genetics | Antibodies, Monoclonal - therapeutic use | Male | Gene Expression Profiling | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Aged, 80 and over | Adult | Female | Carcinoma, Renal Cell - drug therapy | Antibodies, Monoclonal - pharmacology | Kaplan-Meier Estimate | Treatment Outcome | Mutation - genetics | Sunitinib - adverse effects | Sunitinib - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Aged | Kidney Neoplasms - drug therapy | Development and progression | Dosage and administration | Angiogenesis inhibitors | Carcinoma, Renal cell | Comparative analysis | Drug therapy | Cell survival | Inflammation | Gene expression | Molecular chains | Bevacizumab | Metastases | Confidence intervals | Angiogenesis | Immune checkpoint | Immunotherapy | γ-Interferon | PD-L1 protein | Biomarkers | Bioindicators | Vascular endothelial growth factor | Clear cell-type renal cell carcinoma | Index Medicus
Journal Article
Journal Article
Proteomics, ISSN 1615-9853, 01/2017, Volume 17, Issue 1-2, p. 1770010
  DOI: 10.1002/pmic.201600061 MHCI and MHCII peptides are presented on the surface of all nucleated cells in mammals and provide the immune system with a... 
Peptides | Researchers
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, pp. e0166438 - e0166438
Journal Article
Cell Reports, ISSN 2211-1247, 04/2019, Volume 27, Issue 1, pp. 269 - 281.e4
Myeloid-derived suppressor cells (MDSCs) are found in most cancer malignancies and support tumorigenesis by suppressing immunity and promoting tumor growth.... 
CBP | bromodomain | EP300 | tumors | myeloid-derived suppressive cells | H3K27ac | INHIBITION | P300 | EXPANSION | CBP/P300 BROMODOMAIN | SUBSETS | MECHANISMS | SUPPRESSOR-CELLS | CANCER | EXPRESSION | CELL BIOLOGY
Journal Article