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Chinese Journal of Cancer Prevention and Treatment, ISSN 1673-5269, 05/2013, Volume 20, Issue 9, pp. 650 - 655
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e53906
Background: MicroRNAs are short regulatory RNAs that negatively modulate protein expression at a post-transcriptional and/or translational level and are deeply... 
BREAST-CANCER | LUNG-CANCER | SMALL RNAS | INDUCED APOPTOSIS | GASTRIC-CANCER | CYCLIN D1 | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER CELLS | TUMOR-SUPPRESSOR PTEN | MESENCHYMAL TRANSITION | EXPRESSION | MicroRNAs - antagonists & inhibitors | Apoptosis - drug effects | Humans | MicroRNAs - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Bone Neoplasms - pathology | Proto-Oncogene Proteins c-akt - genetics | Bone Neoplasms - metabolism | Luciferases | Female | Antineoplastic Agents - pharmacology | Bone Neoplasms - genetics | Gene Expression Regulation, Neoplastic - drug effects | 3' Untranslated Regions | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Genes, Reporter | Osteosarcoma - metabolism | PTEN Phosphohydrolase - genetics | Enzyme Inhibitors - pharmacology | Morpholines - pharmacology | PTEN Phosphohydrolase - metabolism | Cisplatin - pharmacology | Phosphatidylinositol 3-Kinases - genetics | Drug Resistance, Neoplasm - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | MicroRNAs - genetics | Osteosarcoma - genetics | Cell Cycle - drug effects | Osteosarcoma - pathology | Drug Resistance, Neoplasm - drug effects | MicroRNA | Osteosarcoma | Analysis | Genes | Luciferase | Genetic research | Genetic transcription | Cisplatin | Apoptosis | Immunohistochemistry | Post-transcription | Biotechnology | Pathogenesis | Oncology | AKT protein | Retailing | Metastasis | Kinases | Phosphatase | Tissues | Assaying | Osteoblasts | Bone surgery | Cancer therapies | Proteins | Liver cancer | Cell growth | Bioaccumulation | Cell cycle | Biocompatibility | Bioinformatics | Cell survival | MiRNA | Breast cancer | Gene expression | Real time | Ribonucleic acid--RNA | Survival | 1-Phosphatidylinositol 3-kinase | Bone cancer | Chemotherapy | Inhibitors | 3' Untranslated regions | MicroRNAs | Orthopedics | Cell lines | Prostate | Viability | PTEN protein | RNA | Ribonucleic acid
Journal Article
Journal Article
American journal of physiology. Gastrointestinal and liver physiology, ISSN 0193-1857, 06/2019, Volume 316, Issue 6, pp. G816 - G825
Intra-acinar trypsinogen activation occurs in the earliest stages of pancreatitis and is believed to play important roles in pancreatitis pathogenesis.... 
mouse models | pancreatic disorders | digestive enzyme | cell death | inflammation
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5
[...]the histograms in panel C and D have changed slightly. (2013) Correction: MicroRNA-221 Induces Cell Survival and Cisplatin Resistance through PI3K/Akt... 
Bone cancer | Cell survival | Osteosarcoma | MicroRNAs | MiRNA | Biocompatibility | AKT protein | Ribonucleic acid--RNA | Cisplatin | 1-Phosphatidylinositol 3-kinase | RNA | Ribonucleic acid
Journal Article
Gastroenterology, ISSN 0016-5085, 05/2019, Volume 156, Issue 6, pp. S-334 - S-334
Journal Article
Gastroenterology, ISSN 0016-5085, 05/2019, Volume 156, Issue 6, pp. S-320 - S-320
Journal Article
by Wu, G and Liu, C and Sun, LG and Wang, Q and Sun, BA and Han, B and Kai, JJ and Luan, JH and Liu, CT and Cao, K and Lu, Y and Cheng, LZ and Lu, J
NATURE COMMUNICATIONS, ISSN 2041-1723, 11/2019, Volume 10, Issue 1, pp. 1 - 8
Journal Article
Gut, ISSN 0017-5749, 09/2012, Volume 61, Issue 9, pp. 1315 - 1322
Background and AimsPancreatic ductal adenocarcinoma (PDAC) is the fourth leading cause of cancer death in the USA. Surgical resection is the only effective... 
LOCALIZATION | DIAGNOSIS | CELLS | ADENOCARCINOMA | FNA | INTRAEPITHELIAL NEOPLASIA | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | PROBE | PROGRESSION | Pancreatic cancer | Cathepsins | Physiological aspects | Development and progression | Research | Diagnosis | Cancer | Medical imaging | Disease | Rodents | Biomarkers | Metastasis | Endoscopy | Localization | Tumors
Journal Article
Gastrointestinal Endoscopy, ISSN 0016-5107, 12/2017, Volume 86, Issue 6, pp. 1157 - 1159
Journal Article
Gastroenterology, ISSN 0016-5085, 05/2019, Volume 156, Issue 6, pp. S-166 - S-167
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 03/2001, Volume 280, Issue 3, pp. 465 - 472
In the cholecystokinin (CCK) hyperstimulation model of acute pancreatitis, two early intracellular events, activation of trypsinogen and activation of nuclear... 
Inflammation | Cholecystokinin | Nuclear factor-κB | Pancreatitis | CYTOKINES | PHYSIOLOGY | MOB-1 | cholecystokinin | nuclear factor-kappa B | B/REL ACTIVATION | CELL BIOLOGY | PROTEASE INHIBITOR | FUT-175 | inflammation | GENE-EXPRESSION | REQUIRES | pancreatitis
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2012, Volume 122, Issue 4, pp. 1519 - 1528
Genetic mutations that give rise to active mutant forms of Ras are oncogenic and found in several types of tumor. However, such mutations are not clear... 
SIGNAL-TRANSDUCTION | PROTEASE INHIBITOR | MEDICINE, RESEARCH & EXPERIMENTAL | PANCREATIC DUCTAL ADENOCARCINOMA | ACTIVATION | K-RAS | ONCOGENIC KRAS | MOUSE MODEL | IN-VIVO | ACINAR-CELLS | CANCER | Inflammation - chemically induced | Pancreatic Neoplasms - metabolism | Pancreatic Neoplasms - etiology | Humans | Carcinoma, Pancreatic Ductal - metabolism | Sincalide - toxicity | Pancreatitis, Chronic - chemically induced | Proto-Oncogene Proteins p21(ras) - physiology | Gabexate - toxicity | I-kappa B Kinase - physiology | Carcinoma, Pancreatic Ductal - genetics | Cyclooxygenase 2 - biosynthesis | Inflammation - complications | Inflammation - metabolism | Cell Transformation, Neoplastic - genetics | Inflammation Mediators - metabolism | Carcinoma, Pancreatic Ductal - etiology | Gene Expression Regulation, Neoplastic - physiology | I-kappa B Kinase - deficiency | Lipopolysaccharides - toxicity | Cyclooxygenase 2 Inhibitors - pharmacology | Neoplasm Proteins - biosynthesis | Pancreatic Neoplasms - pathology | Ceruletide - toxicity | Enzyme Induction | Cyclooxygenase 2 - physiology | Gabexate - analogs & derivatives | Mice, Transgenic | Pancreatic Neoplasms - genetics | I-kappa B Kinase - genetics | Pancreas - metabolism | Pancreatitis, Chronic - genetics | Carcinoma, Pancreatic Ductal - pathology | Precancerous Conditions - genetics | Proto-Oncogene Proteins p21(ras) - biosynthesis | Gene Knock-In Techniques | Animals | NF-kappa B - physiology | Inflammation - genetics | Mice | Feedback, Physiological - physiology | Precancerous Conditions - chemically induced | Genes, ras | Pancreatitis, Chronic - metabolism
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 12/2008, Volume 14, Issue 24, pp. 8143 - 8151
Purpose: Nuclear factor κB (NFκB) activity may increase survival and protect cancer cells from chemotherapy. Therefore, NFκB activity may be prognostic, and... 
chemoresistance | pancreatic cancer | siRNA | liposomes | SOY ISOFLAVONE GENISTEIN | ACTIVATION | CHEMOTHERAPEUTIC-AGENTS | INHIBITION | CARCINOMA CELLS | ONCOLOGY | RESISTANCE | ORTHOTOPIC MODEL | ANTITUMOR-ACTIVITY | SUPPRESSION | EXPRESSION
Journal Article