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by Chen, Q and Cai, JQ and Wang, QX and Wang, YF and Liu, MY and Yang, JX and Zhou, JH and Kang, CS and Li, M and Jiang, CL
CLINICAL CANCER RESEARCH, ISSN 1078-0432, 02/2018, Volume 24, Issue 3, pp. 684 - 695
Purpose: Long noncoding RNAs have been implicated in gliomagenesis, but theirmechanisms of action are mainly undocumented. Through public glioma mRNA... 
HOTAIR | TARGET | LOCALIZATION | ONCOLOGY | PROLIFERATION | HOXA11-AS | MUTATIONS | BETA-CATENIN | EXPRESSION | CANCER | ICAT | Wnt protein | Epidermal growth factor receptors | Glioblastoma | Stat3 protein | Gene expression | Ribonucleic acid--RNA | Nuclear transport | β-catenin | Cell growth | Experimental design | Scaffolding | Glioma cells | Tumorigenesis | Methylation
Journal Article
Current Medicinal Chemistry, ISSN 0929-8673, 2017, Volume 24, Issue 13, pp. 1365 - 1381
Gliomas are the most common primary malignant brain tumors, which have a universally fatal outcome. Current standard treatment for glioma patients is surgical... 
Clinical trials | Glioma stem cell | Glioma | Targeted therapy | Cell signal transduction pathway | Immunotherapy | targeted therapy | STEM-CELLS | CHEMISTRY, MEDICINAL | NEWLY-DIAGNOSED GLIOBLASTOMA | PROGRESSION-FREE SURVIVAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECURRENT GLIOBLASTOMA | cell signal transduction pathway | BRAIN-TUMORS | MALIGNANT GLIOMA | GROWTH-FACTOR RECEPTOR | PHASE-II TRIAL | glioma stem cell | IN-VIVO | PHARMACOLOGY & PHARMACY | clinical trials | HIGH-GRADE GLIOMA | Humans | Brain Neoplasms - pathology | Antibodies, Monoclonal - therapeutic use | Phosphatidylinositol 3-Kinases - metabolism | Stem Cells - cytology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Stem Cells - metabolism | Brain Neoplasms - metabolism | Alkylating Agents - therapeutic use | Glioma - metabolism | Stem Cell Transplantation | Protein Kinase Inhibitors - chemistry | Alkylating Agents - toxicity | Glioma - pathology | Glioma - therapy | Angiogenesis Inhibitors - therapeutic use | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Antibodies, Monoclonal - immunology | Receptor Protein-Tyrosine Kinases - metabolism | Alkylating Agents - chemistry | Angiogenesis Inhibitors - toxicity | Signal Transduction - drug effects | Protein Kinase Inhibitors - therapeutic use | Brain Neoplasms - therapy | Protein Kinase Inhibitors - toxicity | Angiogenesis Inhibitors - chemistry | Tyrosine | Brain | Epidermal growth factor receptors | Brain tumors | Brain cancer | MAP kinase | Raf protein | AKT protein | Radiation therapy | Patients | 1-Phosphatidylinositol 3-kinase | Receptors | Chemotherapy | Surgery | Stem cells | Protein-tyrosine kinase
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 02/2018, Volume 24, Issue 3, pp. 684 - 695
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, p. e0151815
Background Glioblastoma multiform (GBM) is the most common malignant primary brain tumor in adults. Radiotherapy plus concomitant and adjuvant TMZ chemotherapy... 
TUMOR INVASION | MESSENGER-RNA | GELATINASE-A | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | CLASSIFICATION | MATRIX-METALLOPROTEINASE-9 EXPRESSION | INHIBITORS | RADIOTHERAPY | IDENTIFICATION | ADJUVANT TEMOZOLOMIDE | Glioblastoma - enzymology | Prognosis | Dacarbazine - therapeutic use | Humans | RNA, Messenger - genetics | Kaplan-Meier Estimate | Proportional Hazards Models | Treatment Outcome | RNA, Messenger - metabolism | Disease Progression | Matrix Metalloproteinase 9 - metabolism | Neoplasm Grading | Glioblastoma - genetics | Dacarbazine - pharmacology | Matrix Metalloproteinase 9 - genetics | Dacarbazine - analogs & derivatives | Survival Analysis | Biomarkers, Tumor - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Temozolomide | Glioblastoma - drug therapy | DNA Methylation - drug effects | Complications and side effects | Patient outcomes | Dosage and administration | Genetic aspects | Research | Drug therapy | Glioblastoma multiforme | Brain | Correlation | Matrix metalloproteinases | Brain tumors | Brain cancer | Glioblastoma | Biology | Genomes | Neurosurgery | Matrix metalloproteinase | Cancer therapies | Modulators | Metastases | Datasets | Regression models | Extracellular matrix | Metalloproteinase | Medical research | Hazards | Regression analysis | Radiation therapy | Gene expression | Patients | Survival | Zinc | Gelatinase B | Studies | Chemotherapy | Brain research | Hospitals | Glioma | Medical prognosis | Biomarkers | Adults | Methylation | Comparative analysis | Tumors
Journal Article
Cancer Letters, ISSN 0304-3835, 2016, Volume 375, Issue 2, pp. 263 - 273
Journal Article
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 1 - 15
Long noncoding RNAs (lncRNAs) have emerged as new regulatory molecules implicated in diverse biological processes, including therapeutic resistance. However,... 
Binding | Talc | Therapeutic applications | Methylguanine | Glioblastoma | Stat3 protein | Event-related potentials | AKT protein | Gene expression | Biological activity | O6-methylguanine-DNA methyltransferase | Chemotherapy | DNA microarrays | DNA methyltransferase | Acetylation | Temozolomide | Deoxyribonucleic acid--DNA | FOXO3 protein
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, p. e0126022
Background Glioblastomas (GBM) are comprised of a heterogeneous population of tumor cells, immune cells, and extracellular matrix. Interactions among these... 
MOLECULAR CLASSIFICATION | GROWTH-FACTOR-BETA | GLIOMA PATIENTS | TGF-BETA | RNA-SEQ | MICROGLIA | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | VACCINATION | TUMOR-ASSOCIATED MACROPHAGES | SUPPRESSOR-CELLS | Receptors, CCR2 - genetics | Prognosis | Humans | Middle Aged | Brain Neoplasms - pathology | Transcriptome | Receptors, Interleukin-17 - genetics | Young Adult | DNA Methylation | Glioblastoma - genetics | Aged, 80 and over | Microglia - pathology | Adult | Brain Neoplasms - mortality | Interleukin-10 Receptor beta Subunit - genetics | Cytokines - genetics | Glioblastoma - diagnosis | Macrophages - pathology | Brain Neoplasms - diagnosis | Risk Factors | Proportional Hazards Models | Brain Neoplasms - genetics | Interleukin-17 - genetics | Chemokine CCL2 - genetics | Phenotype | Chemokine CXCL10 - genetics | Glioblastoma - pathology | Adolescent | Survival Analysis | Aged | Glioblastoma - mortality | Cytokines | Analysis | Genetic aspects | Research | Glioblastoma multiforme | Cancer | CC chemokine receptors | Brain tumors | Brain cancer | Genes | Glioblastoma | Genomes | Neurosurgery | Macrophages | Data bases | Gene sequencing | Cell growth | Databases | Immunology | CCR2 protein | Interleukin 1 | Gangrene | Extracellular matrix | Growth factors | Immune system | Risk groups | Tumor cells | Inflammation | Regression analysis | Risk analysis | Gene expression | Survival | Patients | Microglia | Survival analysis | Hospitals | Interleukin 17 receptors | Glioma | Medical prognosis | CXCL10 protein | Interleukin 10 | Predictions | Monocyte chemoattractant protein 1 | Tumors
Journal Article
International Journal of Cancer, ISSN 0020-7136, 07/2019, Volume 145, Issue 2, pp. 517 - 530
Mutualistic and dynamic communication between tumour cells and the surrounding microenvironment accelerates the initiation, progression, chemoresistance and... 
microenvironment | immunosuppression | glioblastoma | LGALS1 | prognosis | STEM-CELLS | TGF-BETA | SUPPRESSION | GALECTIN-1 | ONCOLOGY | TUMOR-ASSOCIATED MACROPHAGES | EXPRESSION | T-CELLS | PROGRESSION | LYMPHOCYTES | Dendritic cells | Gliomas | Analysis | Immunotherapy | Cytokines | Genes | Genomics | Glioblastoma | Chemoresistance | Malignancy | Gene expression | Macrophages | Suppressor cells | Heterogeneity | Immunosuppression | Algorithms | Glioma | Tumors | Immune system
Journal Article