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1. Minguo kong jun
by Jiang, Genjin
2017, ISBN 7503483873
Book
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2. Qiang xing jun
by Jiang, Muliang, -1973
2012, Minguo ji cui, 1, 202
Book
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3. Jun zhu Chang Jiang wei
: liang zhu wen ming zhan
by Lu, Wenbao and Wang, Yi and Jiang, Weidong
2013, ISBN 9787220087486, 1 v.
Book
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4. Qin Hua Ri jun di liu shi tuan Nanjing zhan yi ji bao xing shi lu
by Jiang, Zichen
2017, ISBN 9787229124069
Book
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5. Full Text Exendin-4 Protects β-Cells From Interleukin-1β–Induced Apoptosis by Interfering With the c-Jun NH2-Terminal Kinase Pathway
by Mourad Ferdaoussi and Saida Abdelli and Jiang-Yan Yang and Marion Cornu and Guy Niederhauser and Dimitri Favre and Christian Widmann and Romano Regazzi and Bernard Thorens and Gérard Waeber and Amar Abderrahmani
Diabetes (New York, N.Y.), ISSN 0012-1797, 05/2008, Volume 57, Issue 5, pp. 1205 - 1215
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Biological and medical sciences | Endocrinopathies | Etiopathogenesis. Screening. Investigations. Target tissue resistance | Diabetes. Impaired glucose tolerance | Medical sciences | Endocrine pancreas. Apud cells (diseases) | Insulin-Secreting Cells - physiology | JNK Mitogen-Activated Protein Kinases - drug effects | Cell Line | Interleukin-1beta - pharmacology | JNK Mitogen-Activated Protein Kinases - biosynthesis | Cells, Cultured | Rats | JNK Mitogen-Activated Protein Kinases - metabolism | Hypoglycemic Agents - pharmacology | Peptides - pharmacology | Animals | Insulin-Secreting Cells - drug effects | Islets of Langerhans - cytology | Enzyme Induction - drug effects | Insulin-Secreting Cells - cytology | Mice | Venoms - pharmacology | Insulin-Secreting Cells - enzymology | Index Medicus | Abridged Index Medicus
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6. Full Text FGF21 impedes peripheral myelin development by stimulating p38 MAPK/c‐Jun axis
by Zhang, Yunzhong and Jiang, Ketao and Xie, Guoqing and Ding, Jie and Peng, Su and Liu, Xiaoyu and Sun, Cheng and Tang, Xin
Journal of cellular physiology, ISSN 0021-9541, 02/2021, Volume 236, Issue 2, pp. 1345 - 1361
fibroblast growth factor 21 | c‐Jun | p38 MAPK | peripheral myelination | Schwann cells | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Genetic engineering | Fibroblast growth factors | Gene expression | Mitogens | Protein kinases | Peripheral nervous system | Neonates | Fibroblast growth factor | Krox-20 protein | Myelin | Central nervous system | Antibodies | Extracellular signal-regulated kinase | Homeostasis | MAP kinase | Nervous system | Kinases | Energy balance | Infusion | Myelination | Protein kinase | Nerves | Gene manipulation | Peripheral myelin protein 22 | Growth factors | Index Medicus
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7. Full Text MiR-194, commonly repressed in colorectal cancer, suppresses tumor growth by regulating the MAP4K4/c-Jun/MDM2 signaling pathway
by Wang, Bo and Shen, Zhan-long and Gao, Zhi-dong and Zhao, Gang and Wang, Chun-you and Yang, Yang and Zhang, Ji-zhun and Yan, Yi-chao and Shen, Chao and Jiang, Ke-wei and Ye, Ying-jiang and Wang, Shan
Cell cycle (Georgetown, Tex.), ISSN 1538-4101, 04/2015, Volume 14, Issue 7, pp. 1046 - 1058
c-Jun | colorectal cancer | proliferation | MDM2 | apoptosis | miR-194 | MAP4K4 | MiR-194 | Colorectal cancer | Proliferation | C-Jun | Apoptosis | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Multivariate Analysis | Neoplasm Transplantation | Proto-Oncogene Proteins c-mdm2 - genetics | Cell Proliferation | Prognosis | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | RNA Interference | Base Sequence | G1 Phase Cell Cycle Checkpoints | Female | 3' Untranslated Regions | Binding Sites | Intracellular Signaling Peptides and Proteins - genetics | Proto-Oncogene Proteins c-mdm2 - metabolism | Colorectal Neoplasms - metabolism | Protein-Serine-Threonine Kinases - metabolism | Colorectal Neoplasms - mortality | Promoter Regions, Genetic | Gene Expression | Signal Transduction | Down-Regulation | Kaplan-Meier Estimate | Proportional Hazards Models | Protein-Serine-Threonine Kinases - genetics | Animals | Mice, Nude | Proto-Oncogene Proteins c-jun - metabolism | Cell Line, Tumor | Protein Binding | Mice, Inbred BALB C | MicroRNAs - genetics | Colorectal Neoplasms - pathology | Index Medicus
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8. Full Text Oncofetal HLF transactivates c-Jun to promote hepatocellular carcinoma development and sorafenib resistance
by Xiang, Dai-Min and Sun, Wen and Zhou, Tengfei and Zhang, Cheng and Cheng, Zhuo and Li, Shi-Chao and Jiang, Weiqi and Wang, Ruoyu and Fu, Gongbo and Cui, Xiuliang and Hou, Guojun and Jin, Guang-Zhi and Li, Hengyu and Hou, Caiying and Liu, Hui and Wang, Hongyang and Ding, Jin
Gut, ISSN 0017-5749, 10/2019, Volume 68, Issue 10, pp. 1858 - 1871
Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Transcription factors | Prognosis | Leukemia | Oct-4 protein | Hepatocellular carcinoma | Hepatoma | Kinases | Medical diagnosis | Cancer therapies | Proteins | Liver cancer | Signal transduction | Hepatology | Surgery | Xenografts | Therapeutic applications | Fetuses | c-Jun protein | Gene expression | Patients | Hospitals | Medical prognosis | Stem cells | Biomarkers | Clinical medicine | Tumors | Index Medicus | Abridged Index Medicus
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9. Full Text Traumatic scratch injury in astrocytes triggers calcium influx to activate the JNK/c‐Jun/AP‐1 pathway and switch on GFAP expression
by Gao, Kai and Wang, Chen Ran and Jiang, Feng and Wong, Ann Yuen Kwan and Su, Na and Jiang, Jiao Hua and Chai, Rui Chao and Vatcher, Greg and Teng, Junlin and Chen, Jianguo and Jiang, Yu‐Wu and Yu, Albert Cheung Hoi
Glia, ISSN 0894-1491, 12/2013, Volume 61, Issue 12, pp. 2063 - 2077
calcium wave | astrogliosis | glial scar | glial fibrillary acidic protein | JNK | Astrogliosis | Calcium wave | Glial scar | Glial fibrillary acidic protein | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Astrocytes - cytology | Gliosis - genetics | MAP Kinase Signaling System - physiology | Calcium - metabolism | Transcription Factor AP-1 - genetics | Gliosis - metabolism | Transcriptional Activation | Cells, Cultured | Glial Fibrillary Acidic Protein - metabolism | Cerebral Cortex - cytology | Transcription Factor AP-1 - metabolism | Cerebral Cortex - metabolism | Mice, Inbred ICR | Genes, jun - physiology | Animals | Mice | Calcium Signaling - genetics | Astrocytes - metabolism | Physiological aspects | Brain | Intermediate filament proteins | Analysis | Injuries | Calcium | Brain damage | Index Medicus | Activator protein 1
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10. Full Text TGF-β Down-regulates Apolipoprotein M Expression through the TAK-1-JNK-c-Jun Pathway in HepG2 Cells
by Ren, Kun and Ren, Kun and Mo, Zhong-Cheng and Mo, Zhong-Cheng and Liu, Xing and Liu, Xing and Tang, Zhen-Li and Tang, Zhen-Li and Jiang, Yue and Jiang, Yue and Peng, Xiao-Shan and Peng, Xiao-Shan and Zhang, Qing-Hai and Zhang, Qing-Hai and Shi, Jin-Feng and Shi, Jin-Feng and Yi, Guang-Hui and Yi, Guang-Hui
Lipids, ISSN 0024-4201, 2/2017, Volume 52, Issue 2, pp. 109 - 117
Life Sciences | Neurochemistry | Medicinal Chemistry | Lipidology | ApoM | Nutrition | Microbial Genetics and Genomics | JNK | Medical Biochemistry | TGF-β | TAK-1 | C-Jun | TGF‐β | C‐Jun | TAK‐1 | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Nutrition & Dietetics | Science & Technology | Lipocalins - metabolism | Promoter Regions, Genetic | Apolipoproteins - metabolism | Humans | MAP Kinase Kinase Kinases - metabolism | Anthracenes - pharmacology | Lactones - pharmacology | Hep G2 Cells | Dose-Response Relationship, Drug | Resorcinols - pharmacology | Transforming Growth Factor beta - pharmacology | Apolipoproteins M | MAP Kinase Signaling System - drug effects | Time Factors | Apolipoproteins - genetics | Lipocalins - genetics | Proto-Oncogene Proteins c-jun - metabolism | Gene Expression Regulation, Neoplastic - drug effects | Index Medicus
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11. Taiping Yang jun shi di li
by Jiang, Zhenhua
2012, Minguo ji cui, 2, 4, 14, 2, 334
Pan-Pacific relations
Book
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12. Taiwan di qu wu shi nian jun shi shi
: 1949-2006
by Jiang, Tingyu
2013, ISBN 9787506565196, 1 v.
Book
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13. Full Text The Jun/miR-22/HuR regulatory axis contributes to tumourigenesis in colorectal cancer
by Liu, Yanqing and Chen, Xiaorui and Cheng, Rongjie and Yang, Fei and Yu, Mengchao and Wang, Chen and Cui, Shufang and Hong, Yeting and Liang, Hongwei and Liu, Minghui and Zhao, Chihao and Ding, Meng and Sun, Wu and Liu, Zhijian and Sun, Feng and Zhang, Chenyu and Zhou, Zhen and Jiang, Xiaohong and Chen, Xi
Molecular cancer, ISSN 1476-4598, 01/2018, Volume 17, Issue 1, pp. 11 - 11
Jun | Proliferation | HuR | MiR-22 | Migration | Colorectal cancer | Biochemistry & Molecular Biology | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Proliferation | Colorectal Neoplasms - genetics | Humans | ELAV-Like Protein 1 - genetics | Gene Expression Regulation, Neoplastic | Databases, Genetic | Animals | Heterografts | RNA Interference | Cell Transformation, Neoplastic - genetics | Models, Biological | Cell Line, Tumor | Transcription, Genetic | Mice | MicroRNAs - genetics | 3' Untranslated Regions | Genes, Reporter | Oncogenes | Disease Models, Animal | Genes, jun | Cell proliferation | Prevention | Care and treatment | Prognosis | MicroRNA | Analysis | Research | Risk factors | Index Medicus | miR-22
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