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2008
Book
2002, Di 1 ban., Wen xin ming bian ji ming ji zhe cong shu, ISBN 7806760903, 10, 2, 468 p., [1] leaf of plates
Book
2003, Di 1 ban., ISBN 7109082601, 3, 3, 5, 229
Book
2000, Di 1 ban., Xin min wen ku. Ye guang bei wen cong, ISBN 9787805318622, 12, 814
Book
2000, Di 1 ban., Xin min wen ku. Ye guang bei wen cong, ISBN 9787805316284, 9, 2, 484
Book
1990, Di 1 ban., ISBN 9787500510642, 7, 496
Book
1984, Di 1 ban., 2, 4, 348 p., [1] folded leaf of plates
Book
2003, Di 1 ban., ISBN 7504929913, 4, 2, 299
Book
LEPROSY REVIEW, ISSN 0305-7518, 12/2013, Volume 84, Issue 4, pp. 328 - 329
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 09/2017, Volume 377, Issue 10, pp. 923 - 935
Journal Article
Endocrine, Metabolic and Immune Disorders - Drug Targets, ISSN 1871-5303, 06/2008, Volume 8, Issue 2, pp. 77 - 77(1)
Type 2 diabetes, obesity, metabolic syndrome, and their cardiovascular complications represent a major public health problem around the world. Strategies for... 
Journal Article
2017, ISBN 9787515406961, 1 v.
Book
Science, ISSN 0036-8075, 12/2004, Volume 306, Issue 5703, pp. 1937 - 1940
Journal Article
Scientific Reports, ISSN 2045-2322, 09/2016, Volume 6, Issue 1, pp. 33732 - 33732
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2014, Volume 9, Issue 4, pp. e95399 - e95399
Insulin degrading enzyme (IDE) is a potential drug target in the treatment of type 2 diabetes (T2D). IDE controls circulating insulin through a... 
CAFETERIA DIET | CLEARANCE | INHIBITION | MULTIDISCIPLINARY SCIENCES | RESISTANCE | PPAR-GAMMA | HYPERINSULINEMIA | FATTY-ACIDS | MULTIPLE SERINE KINASES | TNF-ALPHA | NF-KAPPA-B | Liver - pathology | Palmitic Acid - pharmacology | Gene Expression - drug effects | Obesity - drug therapy | Diet, High-Fat - adverse effects | Hepatocytes - metabolism | RNA, Messenger - metabolism | Obesity - genetics | Hepatocytes - cytology | Insulysin - antagonists & inhibitors | Liver - drug effects | Obesity - etiology | Thiazolidinediones - pharmacology | Hepatocytes - drug effects | Insulin - pharmacology | Insulysin - metabolism | Liver - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Glucagon - pharmacology | Hypoglycemic Agents - pharmacology | Obesity - pathology | Tumor Necrosis Factor-alpha - pharmacology | Insulin - metabolism | Animals | Cell Line, Tumor | Insulysin - genetics | Mice | Blood Glucose - metabolism | Care and treatment | RNA | Analysis | Metabolic diseases | Diagnosis | Insulin | Health aspects | Risk factors | Regulations | Biomedical research | Enzyme activity | Glucagon | Laboratories | Pathogenesis | Liver | mRNA | Adipocytes | Hepatoma | Kinases | Palmitic acid | Experiments | Degradation | Proteins | Antioxidants | Control | Reduction | Enzymatic activity | Rodents | Animal tissues | Tumor necrosis factor-TNF | Obesity | Enzymes | Fasting | Pioglitazone | Diabetes mellitus | Tumor necrosis factor-α | Fatty acids | Diet | Hepatocytes | Insulin resistance | Diabetes | Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 07/2009, Volume 58, Issue 7, pp. 1509 - 1517
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 2008, Volume 374, Issue 3, pp. 405 - 408
The nuclear receptor PPARγ is a lipid sensor that regulates lipid metabolism through gene transcription. Inhibition of PPARγ activity by TNF-α is involved in... 
TNF-α | Nuclear corepressor | PPARγ | NUCLEAR HORMONE RECEPTORS | TNF-alpha | BIOCHEMISTRY & MOLECULAR BIOLOGY | nuclear corepressor | PROLIFERATOR-ACTIVATED RECEPTOR | FIBROBLAST-GROWTH-FACTOR | TRANSCRIPTIONAL COACTIVATOR | BINDING PROTEIN | PPAR gamma | TUMOR-NECROSIS-FACTOR | BIOPHYSICS | GENE-EXPRESSION | ADIPOCYTE DIFFERENTIATION | DEPENDENT REPRESSION | NF-KAPPA-B
Journal Article
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