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Prostate, ISSN 0270-4137, 03/2006, Volume 66, Issue 4, pp. 421 - 429
Journal Article
American Journal of Clinical Pathology, ISSN 0002-9173, 2006, Volume 125, Issue 2, pp. 209 - 216
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2012, Volume 7, Issue 2, p. e31919
The progression of prostate cancers (PCs) to locally invasive, androgen-independent and metastatic disease states is generally associated with treatment... 
GROWTH-FACTOR RECEPTOR | SIDE POPULATION | BONE METASTASES | MULTIDISCIPLINARY SCIENCES | MACROPHAGE INHIBITORY CYTOKINE-1 | IN-VIVO | TARGETING THERAPIES | STEM/PROGENITOR CELLS | ANDROGEN-INDEPENDENCE | PROGRESSION | TUMOR-INITIATING CELLS | In Situ Nick-End Labeling | Prostatic Neoplasms - metabolism | Neoplastic Stem Cells - cytology | Cell Proliferation | Peptides | Antigens, CD - biosynthesis | Humans | Gene Expression Regulation, Neoplastic | Epidermal Growth Factor - metabolism | Male | NF-kappa B - metabolism | AC133 Antigen | Microscopy, Confocal - methods | Disease Progression | Glycoproteins - biosynthesis | Immunohistochemistry - methods | Receptor, Epidermal Growth Factor - metabolism | Microscopy, Fluorescence - methods | Growth Differentiation Factor 15 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Adenocarcinoma | Disintegration | Cytotoxicity | Biochemistry | Metastasis | Kinases | Medical diagnosis | Phosphatase | Macrophages | Cancer therapies | Metastases | Proteins | Disease resistance | Signal transduction | Epidermal growth factor | Bioindicators | NF-κB protein | Antigens | Cytokines | Epidermal growth factor receptors | Invasiveness | Therapeutic applications | Medical treatment | Endocrine therapy | Caspase | Patients | Disease control | Medicine | Signaling | Androgens | Chemotherapy | Macrophage inhibitory cytokine-1 | Stem cells | Minimum inhibitory concentration | Pheochromocytoma cells | Biomarkers | Diagnostic systems | Molecular biology | Immunofluorescence | Viability | Prostate cancer | Prostate | Apoptosis | Tumors
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e44299
Alternative survival pathways are commonly seen to be upregulated upon inhibition of receptor tyrosine kinases (RTK), including Her-2. It is established that... 
GROWTH-FACTOR RECEPTOR | TARGETING SRC | DEPENDENT PLASMINOGEN ACTIVATION | MESSENGER-RNA | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | PROTEIN EXPRESSION | GENE-EXPRESSION | CELL-LINES | T-PA | II OVEREXPRESSION | Signal Transduction | Cell Survival | Humans | Receptor, ErbB-2 - metabolism | Collagen - chemistry | Drug Resistance, Neoplasm | Breast Neoplasms - drug therapy | Proteoglycans - chemistry | Immunohistochemistry - methods | Phenotype | Receptor, Epidermal Growth Factor - metabolism | Breast Neoplasms - pathology | Antibodies, Monoclonal, Humanized - pharmacology | src-Family Kinases - metabolism | Cell Line, Tumor | Female | Laminin - chemistry | Ligands | Annexin A2 - metabolism | Drug Combinations | Trastuzumab | Apoptosis | Cell Movement | Antimitotic agents | Tyrosine | Epidermal growth factor | Analysis | Breast cancer | Annexins | Antineoplastic agents | Health aspects | Cell proliferation | Regulators | Phosphorylation | Activation | Biochemistry | Kinases | Cancer therapies | Cell adhesion & migration | Proteins | Genotype & phenotype | Signal transduction | Endocytosis | Pathways | Tumorigenesis | Inhibition | Coordination compounds | Growth factors | Translocation | Epidermal growth factor receptors | siRNA | Src protein | Ribonucleic acid--RNA | ErbB-2 protein | Substrates | Intermediates | Signaling | Chemotherapy | Cell lines | Biomarkers | Breast | Viability | Cell migration | Cancer | RNA | Ribonucleic acid
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 04/2010, Volume 70, Issue 8 Supplement, pp. 3075 - 3075
Journal Article
Cancer Letters, ISSN 0304-3835, 2012, Volume 323, Issue 1, pp. 29 - 40
Abstract Pancreatic tumors are resistant to conventional chemotherapies. The present study was aimed at evaluating the potential of a novel plant-derived... 
Hematology, Oncology and Palliative Medicine | Natural product | Therapy | Cancer metabolism | Pancreatic cancer | APOPTOSIS | GROWTH-INHIBITION | HYPOXIA | ANTITUMOR | GEMCITABINE | NECROSIS | ONCOLOGY | ANNONA-MURICATA | MUC4 MUCIN | EXPRESSION | ACETOGENINS | Pancreatic Neoplasms - metabolism | Plant Extracts - pharmacology | Humans | Blotting, Western | Necrosis | Neoplasms, Experimental - pathology | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Microscopy, Confocal | Animals | Signal Transduction - drug effects | Mice, Nude | Annona - chemistry | Cell Line, Tumor | Female | Antineoplastic Agents - pharmacology | Mice | Neoplasms, Experimental - metabolism | Phytotherapy - methods | Neoplasms, Experimental - drug therapy | Real-Time Polymerase Chain Reaction | Glucose metabolism | Chemotherapy | Care and treatment | Oncology, Experimental | Physiological aspects | Metastasis | Research | Gene expression | Cells | Tumors | Cancer | Cell culture | Surgical implants | Disease | Toxicity | Clinical trials | Cytotoxicity | Cancer therapies | Protein turnover | Metastases | Cell cycle | Biocompatibility | Inhibition | Drug dosages | NF-κB protein | Cell survival | Pharmacology | Breast cancer | Tumorigenicity | Plants | Metabolism | Chemical compounds | Biological activity | Studies | Properties (attributes) | Natural products | Medical prognosis | Glycolysis | Pheochromocytoma cells | Hypoxia | Diabetes | cancer metabolism | therapy | natural product
Journal Article
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 2010, Volume 295, Issue 1, pp. 69 - 84
Journal Article