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Current Drug Targets, ISSN 1389-4501, 2006, Volume 7, Issue 11, pp. 1421 - 1434
Journal Article
Brain Behavior and Immunity, ISSN 0889-1591, 10/2018, Volume 73, pp. 180 - 191
Increasing evidence indicates that multiple actions of the immune system are closely intertwined with the development of depression and subsequent recovery... 
Depression | Th17
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 10/2018, Volume 73, p. 180
Journal Article
Nature Immunology, ISSN 1529-2908, 08/2005, Volume 6, Issue 8, pp. 777 - 784
Journal Article
by Jope, RS and Roh, MS
CURRENT DRUG TARGETS, ISSN 1389-4501, 11/2006, Volume 7, Issue 11, pp. 1421 - 1434
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 12/2003, Volume 87, Issue 6, pp. 1427 - 1435
We describe here a new component of the phosphatidylinositol 3‐kinase/Akt signaling pathway that directly impacts mitochondria. Akt (protein kinase B) was... 
insulin‐like growth factor‐1 | ATP synthase | Akt | glycogen synthase kinase‐3β | mitochondria | phosphatidylinositol 3‐kinase | Insulin-like growth factor-1 | Mitochondria | Phosphatidylinositol 3-kinase | Glycogen synthase kinase-3β | SURVIVAL | TRANSLOCATION | CELLS | LOCALIZATION | APOPTOSIS | PROTEIN-KINASE-B | glycogen synthase kinase-3 beta | PHOSPHORYLATION | MEMBRANE | BIOCHEMISTRY & MOLECULAR BIOLOGY | insulin-like growth factor-1 | NEUROSCIENCES | GLYCOGEN-SYNTHASE KINASE-3-BETA | WITHDRAWAL | phosphatidylinositol 3-kinase | Mitochondria - enzymology | Insulin-Like Growth Factor I - pharmacology | Embryo, Mammalian | Humans | Neuroblastoma - enzymology | Protein-Serine-Threonine Kinases | Cytosol - drug effects | Porins - metabolism | Glycogen Synthase Kinase 3 beta | Phosphatidylinositol 3-Kinases - metabolism | Threonine - metabolism | Kidney | Cell Nucleus - enzymology | Electron Transport Complex IV - metabolism | Immunoblotting - methods | Cytosol - enzymology | Drug Interactions | Time Factors | Carbonyl Cyanide m-Chlorophenyl Hydrazone - pharmacology | Cell Membrane - metabolism | Phosphorylation - drug effects | Cell Membrane - drug effects | Ionophores - pharmacology | Superoxide Dismutase - metabolism | Proto-Oncogene Proteins - metabolism | Cell Line | Brain Neoplasms - enzymology | Cytochromes c - metabolism | Cell Fractionation - methods | Enzyme Inhibitors - pharmacology | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization - methods | Mitochondria - drug effects | Enzyme Activation - drug effects | Glycogen Synthase Kinase 3 - metabolism | Serine - metabolism | Mitochondrial Proton-Translocating ATPases - metabolism | Precipitin Tests - methods | Proto-Oncogene Proteins c-akt | Androstadienes - pharmacology | Voltage-Dependent Anion Channel 1 | Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization - instrumentation | Cell Nucleus - drug effects | Index Medicus | glycogen synthase kinase-3β
Journal Article
Journal Article
Biological Psychiatry, ISSN 0006-3223, 2014, Volume 75, Issue 3, pp. 198 - 206
Background Identifying feasible therapeutic interventions is crucial for ameliorating the intellectual disability and other afflictions of fragile X syndrome... 
Psychiatry | Cognition | learning | glycogen synthase kinase-3 | synaptic plasticity | fragile X syndrome | long-term potentiation (LTP) | PROTEIN | NEUROSCIENCES | PATTERN SEPARATION | DORSAL HIPPOCAMPUS | MEMORY | CA3 | MOUSE MODEL | DENTATE GYRUS | LITHIUM | FMR1 KNOCKOUT MICE | Disks Large Homolog 4 Protein | Electric Stimulation | Fragile X Syndrome - pathology | Synapses - genetics | Synapses - pathology | Fragile X Syndrome - drug therapy | Long-Term Potentiation - drug effects | Lithium Chloride - pharmacology | Cognition Disorders - etiology | Membrane Proteins - metabolism | Disease Models, Animal | Fragile X Syndrome - genetics | Synapses - drug effects | Lithium Chloride - therapeutic use | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Mice, Transgenic | Excitatory Amino Acid Antagonists - pharmacology | Hippocampus - pathology | Long-Term Potentiation - genetics | Cognition - drug effects | Cognition Disorders - drug therapy | Glycogen Synthase Kinase 3 - metabolism | Fragile X Syndrome - complications | Patch-Clamp Techniques | Animals | Protein Kinase Inhibitors - therapeutic use | Guanylate Kinases - metabolism | Fragile X Mental Retardation Protein - genetics | Mice | Protein Kinase Inhibitors - pharmacology | In Vitro Techniques | Glucose metabolism | Synthesis | Seizures (Medicine) | Glycogen | Analysis | Index Medicus | LTP | Fragile X syndrome | cognition
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 2003, Volume 278, Issue 49, pp. 48872 - 48879
The recent discovery of direct interactions between two important regulators of cell fate, the tumor suppressor p53 and glycogen synthase kinase-3beta... 
Tumor Suppressor Protein p53 - physiology | Transcription, Genetic - physiology | Humans | Tumor Suppressor Protein p53 - metabolism | Cell Line, Tumor | Protein Binding | Glycogen Synthase Kinase 3 beta | Apoptosis - physiology | Glycogen Synthase Kinase 3 - metabolism | Index Medicus
Journal Article
Biological Psychiatry, ISSN 0006-3223, 2009, Volume 66, Issue 5, pp. 494 - 502
Journal Article