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Journal of Neuroscience, ISSN 0270-6474, 2014, Volume 34, Issue 1, pp. 249 - 259
Familial Parkinson disease is associated with mutations in alpha-synuclein (alpha-syn), a presynaptic protein that has been localized not only to the cytosol,... 
ALZHEIMER-DISEASE | OXIDATIVE STRESS | FUSION | PROTEIN | INHIBITION | VESICLES | ENDOPLASMIC-RETICULUM | TOXICITY | FAMILIAL PARKINSONS-DISEASE | FISSION | NEUROSCIENCES | Endoplasmic Reticulum - chemistry | Animals | alpha-Synuclein - analysis | Humans | Cells, Cultured | Female | Male | Mice, Transgenic | Mice | HeLa Cells | Mitochondria - chemistry | Mice, Knockout
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2010, Volume 107, Issue 1, pp. 378 - 383
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2008, Volume 283, Issue 36, pp. 24770 - 24780
Accumulation of indigestible lipofuscin and decreased mitochondrial energy production are characteristic age-related changes of post-mitotic retinal pigment... 
LIPOFUSCIN FLUOROPHORE | RAT | DYSTROPHY | DNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | MACULAR DEGENERATION | COMPONENT | RPE CELLS | N-RETINYLIDENE ETHANOLAMINE | BINDING | PHOTORECEPTORS | Retinoids - metabolism | Epithelial Cells - metabolism | Lipofuscin - pharmacology | Antioxidants - metabolism | Humans | Lysosomes - genetics | Rats, Long-Evans | Adenosine Triphosphate - genetics | Phagocytosis - genetics | Membrane Potential, Mitochondrial - drug effects | Mitosis - genetics | Oxidative Phosphorylation - drug effects | DNA, Mitochondrial - genetics | Lysosomes - metabolism | Aging - genetics | Cell Death - genetics | Mitochondria - genetics | Adenosine Triphosphate - metabolism | Membrane Potential, Mitochondrial - genetics | Pyruvic Acid - metabolism | Lysosomes - pathology | Cell Death - drug effects | Cell Line | Phagocytosis - drug effects | DNA, Mitochondrial - metabolism | Rats | Epithelial Cells - pathology | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - pathology | Aging - pathology | Pigment Epithelium of Eye - metabolism | Macular Degeneration - metabolism | Point Mutation | Pyridinium Compounds - pharmacology | Animals | Mitosis - drug effects | Macular Degeneration - genetics | Glucose - metabolism | Pigment Epithelium of Eye - pathology | Pyridinium Compounds - metabolism | Lipofuscin - metabolism | Retinoids - pharmacology | Aging - metabolism | Macular Degeneration - pathology | Index Medicus | Molecular Basis of Cell and Developmental Biology
Journal Article
European Journal of Immunology, ISSN 0014-2980, 08/2011, Volume 41, Issue 8, pp. 2323 - 2332
Adiponectin (APN), a cytokine constitutively produced in fat tissue, has been shown to exert anti-inflammatory effects in various disease models. While the... 
T cells | Immunomodulation | Adiponectin receptors | Adiponectin | ALLOGRAFT-REJECTION | LEPTIN-DEFICIENT MICE | MACROPHAGES | IMMUNOLOGY | VIRAL MYOCARDITIS | INFLAMMATION | CTLA-4 | MEDIATORS | RECEPTORS | EXPRESSION | INTRACELLULAR TRAFFICKING | Tumor Necrosis Factor-alpha - metabolism | Antigens, CD - immunology | Clathrin-Coated Vesicles - immunology | Humans | Receptors, Adiponectin - immunology | Receptors, Adiponectin - metabolism | Male | Interferon-gamma - metabolism | Vacuolar Proton-Translocating ATPases - metabolism | Clathrin-Coated Vesicles - metabolism | Antigens, CD - metabolism | Vacuolar Proton-Translocating ATPases - immunology | Coxsackievirus Infections - virology | Lymphocyte Activation - immunology | Flow Cytometry | T-Lymphocytes - metabolism | Adiponectin - genetics | Tumor Necrosis Factor-alpha - immunology | Adiponectin - pharmacology | Gene Expression | Jurkat Cells | Mice, Inbred C57BL | Cells, Cultured | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | CTLA-4 Antigen | Coxsackievirus Infections - genetics | Animals | Interferon-gamma - immunology | K562 Cells | Antigens - immunology | T-Lymphocytes - immunology | Cell Proliferation - drug effects | Mice | Adiponectin - immunology | Coxsackievirus Infections - immunology | Microscopy, Fluorescence | Receptors, Adiponectin - genetics | Medical research | T cell receptors | Lymphocytes | Rodents
Journal Article
Brain, ISSN 0006-8950, 2012, Volume 135, Issue 9, pp. 2865 - 2874
Amyotrophic lateral sclerosis is a devastating neurodegenerative disorder that is more prevalent in males than in females. A similar gender difference has been... 
oestrogen | calcium | cyclophilin D | transgenic mice | mitochondria | amyotrophic lateral sclerosis | SOD1 | MOTOR-NEURONS | PERMEABILITY TRANSITION PORE | POPULATION | OXIDATIVE STRESS | ALS | NEUROSCIENCES | CLINICAL NEUROLOGY | MOUSE MODEL | DISEASE | CA2+ UPTAKE | DYSFUNCTION | Neurons - pathology | Superoxide Dismutase - genetics | Spinal Cord - drug effects | Spinal Cord - metabolism | Calcium - metabolism | Cyclophilins - metabolism | Membrane Potential, Mitochondrial - drug effects | Brain - metabolism | Neuroprotective Agents - pharmacology | Mitochondria - genetics | Spinal Cord - pathology | Neurons - metabolism | Neurons - drug effects | Estradiol - pharmacology | Superoxide Dismutase - metabolism | Amyotrophic Lateral Sclerosis - genetics | Cyclophilins - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Mice, Knockout | Brain - drug effects | Amyotrophic Lateral Sclerosis - pathology | Phenotype | Animals | Amyotrophic Lateral Sclerosis - metabolism | Brain - pathology | Mice | Superoxide Dismutase-1 | Brain | Glutamic acid | Motor neurons | Neuroprotection | Animal models | Peptidylprolyl isomerase | Spinal cord | Calcium | Neurodegenerative diseases | Toxicity | Cortex | Transgenic mice | Amyotrophic lateral sclerosis | Sex differences | Superoxide dismutase | Calcium (mitochondrial) | Mitochondria | Mutation | Original
Journal Article
DMM Disease Models and Mechanisms, ISSN 1754-8403, 12/2017, Volume 10, Issue 12, pp. 1529 - 1538
Friedreich ataxia (FRDA) is an autosomal recessive neurodegenerative disorder with progressive ataxia that affects both the peripheral and central nervous... 
Parallel fiber synapse | KIKO | Friedreich ataxia | VGLUT1 | Cerebellar circuit | PURKINJE-CELLS | FRATAXIN | STRUCTURAL PLASTICITY | MITOCHONDRIAL BIOGENESIS | TRANSCRIPTION | ELIMINATION | PATHOLOGY | IRON ACCUMULATION | CELL BIOLOGY | TRIPLET-REPEAT EXPANSION | DEFECTIVE AXONAL-TRANSPORT | POSTNATAL-DEVELOPMENT | 302
Journal Article
Antioxidants and Redox Signaling, ISSN 1523-0864, 07/2009, Volume 11, Issue 7, pp. 1615 - 1626
Journal Article
Journal Article