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Cellular and molecular life sciences : CMLS, 06/2017, Volume 74, Issue 12, p. 2151
The mitochondrial H -ATP synthase is a primary hub of cellular homeostasis by providing the energy required to sustain cellular activity and regulating the... 
Neoplasms - metabolism | Animals | Cell Nucleus - metabolism | Proteins - metabolism | Humans | Hormesis | Mitochondria - metabolism | Neoplasms - pathology | Mitochondrial Proton-Translocating ATPases - metabolism
Journal Article
Cellular and Molecular Life Sciences, ISSN 1420-682X, 06/2017, Volume 74, Issue 12, p. 2151
The mitochondrial H+-ATP synthase is a primary hub of cellular homeostasis by providing the energy required to sustain cellular activity and regulating the... 
Homeostasis
Journal Article
Frontiers in oncology, ISSN 2234-943X, 2018, Volume 8, p. 53
Cancer cells reprogram energy metabolism by boosting aerobic glycolysis as a main pathway for the provision of metabolic energy and of precursors for anabolic... 
Journal Article
Diabetologia, ISSN 0012-186X, 10/2017, Volume 60, Issue 10, p. 2052
Aims/hypothesis Mitochondria are important regulators of the metabolic phenotype in type 2 diabetes. A key factor in mitochondrial physiology is the H+-ATP... 
Obesity | Myotubes | Fasting | Hydrogen | Diabetes mellitus | Homeostasis | Glucose | Metabolism | Fatty acids | Insulin | Skeletal muscle | Carnitine | Musculoskeletal system | Signal transduction | ATP synthase | Mitochondria | Biopsy | Tumor necrosis factor | L-Carnitine | Oxidation | Diabetes | Lipid metabolism | Adenosine triphosphatase | Metabolic disorders
Journal Article
Journal Article
Cell Reports, ISSN 2211-1247, 09/2015, Volume 12, Issue 12, pp. 2143 - 2155
Journal Article
The EMBO Journal, ISSN 0261-4189, 04/2014, Volume 33, Issue 7, pp. 762 - 778
Journal Article
Journal Article
BBA - Bioenergetics, ISSN 0005-2728, 07/2014, Volume 1837, pp. e57 - e57
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2017, Volume 12, Issue 6, pp. e0178376 - e0178376
Objective Charcot-Marie-Tooth 1A (CMT1A) disease is the most common inherited neuropathy that lacks of therapy and of molecular markers to assess disease... 
RELAPSING MULTIPLE-SCLEROSIS | FREE-TRYPTOPHAN | ORAL FINGOLIMOD | CMT NEUROPATHY SCORE | MULTIDISCIPLINARY SCIENCES | DISEASE | PHENOTYPE | CHAIN AMINO-ACIDS | PLACEBO-CONTROLLED TRIAL | CANCER | EXERCISE | Biomarkers - metabolism | Metabolomics | Prospective Studies | Skin - metabolism | Humans | Middle Aged | Biomarkers - analysis | Metabolome | Proteins - analysis | Biomarkers - blood | Charcot-Marie-Tooth Disease - pathology | Proteins - metabolism | Energy Metabolism | Adult | Charcot-Marie-Tooth Disease - metabolism | Charcot-Marie-Tooth Disease - blood | Skin - pathology | Development and progression | Skin | Diagnosis | Health aspects | Charcot-Marie-Tooth disease | Therapy | Plasma | Energy metabolism | Multiple sclerosis | Nuclear magnetic resonance--NMR | Lipids | Amino acids | Leucine | Neuropathy | Protein turnover | Proteins | Coupling (molecular) | Sarcopenia | Mitochondria | Metabolites | Energy | Water analysis | Classification | Longitudinal studies | Bioindicators | Lipid metabolism | Discriminant analysis | Markers | Mass spectroscopy | Liquid chromatography | Metabolism | Patients | Studies | Protein arrays | Depletion | Biopsy | Protein synthesis | Correlation analysis | Biomarkers | Catabolism | Mass spectrometry | Index Medicus | Malalties hereditàries | Amiotròfia neural progressiva de Charcot-Marie-Tooth | Genetic diseases | Nuclear magnetic resonance | NMR
Journal Article
JOURNAL OF PROTEOME RESEARCH, ISSN 1535-3893, 08/2019, Volume 18, Issue 8, pp. 3142 - 3155
The role played by protein turnover in metabolic reprogramming is unknown. Herein, using a SILAC approach, we have studied the changes in the half-life of 266... 
CANCER-CELLS | UBIQUITIN-PROTEASOME SYSTEM | mitochondrial ROS | prolyl hydroxylases | glycolysis | BIOCHEMICAL RESEARCH METHODS | TRANSLATION INITIATION | SILAC | HYPOXIA-INDUCIBLE FACTORS | ATP synthase | GENE-EXPRESSION | DEGRADATION | BIOENERGETIC SIGNATURE | ENERGY-METABOLISM | mTORC1 | ENRICHMENT ANALYSIS | Index Medicus
Journal Article