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humans (17) 17
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ions (2) 2
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1. Full Text Adapting to stress — chaperome networks in cancer
by Joshi, Suhasini and Wang, Tai and Araujo, Thaís L. S and Sharma, Sahil and Brodsky, Jeffrey L and Chiosis, Gabriela
Nature Reviews Cancer, ISSN 1474-175X, 09/2018, Volume 18, Issue 9, pp. 562 - 575
In this Opinion article, we aim to address how cells adapt to stress and the repercussions chronic stress has on cellular function. We consider acute and... 
HSP90 INHIBITORS | ONCOLOGY | MOLECULAR CHAPERONES | PLANT-SPECIFIC THIOREDOXIN | POSTTRANSLATIONAL MODIFICATIONS | CONFORMATIONAL DYNAMICS | ANTITUMOR-ACTIVITY | STEROID-RECEPTOR | RIBULOSE-BISPHOSPHATE CARBOXYLASE | HISTONE CHAPERONES | HEAT-SHOCK-PROTEIN | Stress, Physiological - physiology | Adaptation, Physiological - physiology | Molecular Chaperones - physiology | Humans | Neoplasms | Care and treatment | Research | Molecular chaperones | Stress (Psychology) | Analysis | Cancer | Proteins | Stresses | Drug delivery | Chaperones
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2. Full Text Graviola: A novel promising natural-derived drug that inhibits tumorigenicity and metastasis of pancreatic cancer cells in vitro and in vivo through altering cell metabolism
by Torres, María P and Rachagani, Satyanarayana and Purohit, Vinee and Pandey, Poomy and Joshi, Suhasini and Moore, Erik D and Johansson, Sonny L and Singh, Pankaj K and Ganti, Apar K and Batra, Surinder K
Cancer Letters, ISSN 0304-3835, 2012, Volume 323, Issue 1, pp. 29 - 40
Abstract Pancreatic tumors are resistant to conventional chemotherapies. The present study was aimed at evaluating the potential of a novel plant-derived... 
Hematology, Oncology and Palliative Medicine | Natural product | Therapy | Cancer metabolism | Pancreatic cancer | APOPTOSIS | GROWTH-INHIBITION | HYPOXIA | ANTITUMOR | GEMCITABINE | NECROSIS | ONCOLOGY | ANNONA-MURICATA | MUC4 MUCIN | EXPRESSION | ACETOGENINS | Pancreatic Neoplasms - metabolism | Plant Extracts - pharmacology | Humans | Blotting, Western | Necrosis | Neoplasms, Experimental - pathology | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Microscopy, Confocal | Animals | Signal Transduction - drug effects | Mice, Nude | Annona - chemistry | Cell Line, Tumor | Female | Antineoplastic Agents - pharmacology | Mice | Neoplasms, Experimental - metabolism | Phytotherapy - methods | Neoplasms, Experimental - drug therapy | Real-Time Polymerase Chain Reaction | Glucose metabolism | Chemotherapy | Care and treatment | Oncology, Experimental | Physiological aspects | Metastasis | Research | Gene expression | Cells | Tumors | Cancer | Cell culture | Surgical implants | Disease | Toxicity | Clinical trials | Cytotoxicity | Cancer therapies | Protein turnover | Metastases | Cell cycle | Biocompatibility | Inhibition | Drug dosages | NF-κB protein | Cell survival | Pharmacology | Breast cancer | Tumorigenicity | Plants | Metabolism | Chemical compounds | Biological activity | Studies | Properties (attributes) | Natural products | Medical prognosis | Glycolysis | Pheochromocytoma cells | Hypoxia | Diabetes | cancer metabolism | therapy | natural product
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3. Full Text Abstract 1256: MUC4 is negatively regulated by hypoxia in ROS-dependent manner in pancreatic cancer
by Joshi, Suhasini and Kumar, Sushil and Ponnusamy, Moorthy P and Batra, Surinder K
Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 15 Supplement, pp. 1256 - 1256
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4. Full Text WITHDRAWN: Recent Advances in Angiopoietin Biology in Cancer - A Review
by Joshi, Suhasini and Tiwari, Ashish
Clinical oncology (Royal College of Radiologists (Great Britain)), ISSN 0936-6555, 03/2011
This article has been withdrawn at the request of the author(s) and/or editor. The Publisher apologizes for any inconvenience this may cause. The full Elsevier... 
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5. Full Text Genetically engineered mucin mouse models for inflammation and cancer
by Joshi, Suhasini and Kumar, Sushil and Bafna, Sangeeta and Rachagani, Satyanarayana and Wagner, Kay-Uwe and Jain, Maneesh and Batra, Surinder K
Cancer and Metastasis Reviews, ISSN 0167-7659, 12/2015, Volume 34, Issue 4, pp. 593 - 609
Mucins are heavily O-glycosylated proteins primarily produced by glandular and ductal epithelial cells, either in membrane-tethered or secretory forms, for... 
Biomedicine | Mucins | Synteny | Knockout models | Cancer Research | Oncology | Vaccines | Transgenic mice models | Biomedicine general | Cancer | CHROMOSOMAL LOCALIZATION | PANCREATIC-CANCER | TISSUE-SPECIFIC EXPRESSION | TUMOR-ASSOCIATED MUCIN | OVARIAN-CANCER | BOWEL-DISEASE | GROWTH-FACTOR RECEPTOR | ONCOLOGY | GENE-EXPRESSION | MOLECULAR-CLONING | ABERRANT EXPRESSION | Antigens, Neoplasm - genetics | Inflammation - pathology | Prognosis | Humans | Disease Progression | Mice, Knockout | Mucous Membrane - metabolism | Animals | Genetic Engineering | Mice | Neoplasms - pathology | Mucins - genetics | Disease Models, Animal | Analysis | Genetically modified organisms | Physiological aspects | Models | Genetic engineering | Inflammation
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6. Full Text Screening of over 1000 Indian patients with breast and/or ovarian cancer with a multi-gene panel: prevalence of BRCA1/2 and non-BRCA mutations
by Singh, Jaya and Thota, Nishita and Singh, Suhasini and Padhi, Shila and Mohan, Puja and Deshwal, Shivani and Sur, Soumit and Ghosh, Mithua and Agarwal, Amit and Sarin, Ramesh and Ahmed, Rosina and Almel, Sachin and Chakraborti, Basumita and Raina, Vinod and DadiReddy, Praveen K and Smruti, B K and Rajappa, Senthil and Dodagoudar, Chandragouda and Aggarwal, Shyam and Singhal, Manish and Joshi, Ashish and Kumar, Rajeev and Kumar, Ajai and Mishra, Deepak K and Arora, Neeraj and Karaba, Aarati and Sankaran, Satish and Katragadda, Shanmukh and Ghosh, Arunabha and Veeramachaneni, Vamsi and Hariharan, Ramesh and Mannan, Ashraf U
Breast Cancer Research and Treatment, ISSN 0167-6806, 7/2018, Volume 170, Issue 1, pp. 189 - 196
Breast and/or ovarian cancers are among the most common cancers in women across the world. In the Indian population, the healthcare burden of breast and/or... 
Multi-gene panel | Next-generation sequencing | Medicine & Public Health | Oncology | Breast cancer | BRCA1 | BRCA2 | DIAGNOSIS | MANAGEMENT | SERIES | HEREDITARY BREAST | RISK | SUSCEPTIBILITY GENES | CARE | ONCOLOGY | FREQUENCY | GERMLINE MUTATIONS | Genetic Predisposition to Disease | Early Detection of Cancer | Fanconi Anemia Complementation Group N Protein - genetics | Ovarian Neoplasms - diagnosis | Humans | Middle Aged | Ovarian Neoplasms - pathology | India - epidemiology | Tumor Suppressor Protein p53 - genetics | Ovarian Neoplasms - epidemiology | Ovarian Neoplasms - genetics | BRCA1 Protein - genetics | Breast Neoplasms - genetics | Mass Screening | Breast Neoplasms - pathology | Germ-Line Mutation | Adult | Female | Aged | Breast Neoplasms - diagnosis | Breast - pathology | BRCA2 Protein - genetics | Breast Neoplasms - epidemiology | Genes | Genetic aspects | Tumor proteins | Genetic screening | Ovarian cancer | Cancer
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7. Full Text HSP90-incorporating chaperome networks as biosensor for disease-related pathways in patient-specific midbrain dopamine neurons
by Kishinevsky, Sarah and Kishinevsky, Sarah and Wang, Tai and Wang, Tai and Rodina, Anna and Rodina, Anna and Chung, Sun Young and Chung, Sun Young and Xu, Chao and Xu, Chao and Philip, John and Philip, John and Taldone, Tony and Taldone, Tony and Joshi, Suhasini and Joshi, Suhasini and Alpaugh, Mary L and Alpaugh, Mary L and Bolaender, Alexander and Bolaender, Alexander and Gutbier, Simon and Sandhu, Davinder and Sandhu, Davinder and Fattahi, Faranak and Fattahi, Faranak and Zimmer, Bastian and Zimmer, Bastian and Shah, Smit K and Shah, Smit K and Chang, Elizabeth and Chang, Elizabeth and Inda, Carmen and Inda, Carmen and Koren, John and Koren, John and Saurat, Nathalie G and Saurat, Nathalie G and Leist, Marcel and Gross, Steven S and Gross, Steven S and Seshan, Venkatraman E and Seshan, Venkatraman E and Klein, Christine and Tomishima, Mark J and Tomishima, Mark J and Erdjument-Bromage, Hediye and Erdjument-Bromage, Hediye and Neubert, Thomas A and Neubert, Thomas A and Henrickson, Ronald C and Henrickson, Ronald C and Chiosis, Gabriela and Chiosis, Gabriela and Studer, Lorenz and Studer, Lorenz
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 4345 - 15
Environmental and genetic risk factors contribute to Parkinson's Disease (PD) pathogenesis and the associated midbrain dopamine (mDA) neuron loss. Here, we... 
ONSET PARKINSONISM | PINK1 | PROTEIN | MITOCHONDRIAL | MOLECULAR CHAPERONES | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | PARKINSON-DISEASE | NF-KAPPA-B | CANCER | STEM | Mesencephalon - metabolism | Dopaminergic Neurons - metabolism | HSP90 Heat-Shock Proteins - physiology | Biosensing Techniques | Stress, Physiological | HSP90 Heat-Shock Proteins - metabolism | NF-kappa B - metabolism | STAT3 Transcription Factor - metabolism | Mesencephalon - pathology | Chemical sensors | Networks | Hsp90 protein | Parkinson's disease | Mesencephalon | Change detection | Pathogenesis | Innovations | Activation | Tyrosine 3-monooxygenase | Risk factors | Signal transduction | Pathways | Sensors | Movement disorders | Stresses | Tyrosine | NF-κB protein | Dopamine | Neurodegenerative diseases | Neurons | Hydroxylase | Stat3 protein | Health risks | Pharmacology | Risk analysis | Stress | Neurological diseases | Organic chemistry | Signaling | Stem cells | Biosensors | Viability | Pluripotency | Index Medicus
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8. Full Text Altered Mucins (MUC) trafficking in Benign and malignant conditions
by Joshi, Suhasini and Kumar, Sushil and Choudhury, Amit and Ponnusamy, Moorthy P and Batra, Surinder K
Oncotarget, ISSN 1949-2553, 2014, Volume 5, Issue 17, pp. 7272 - 7284
Endocytosis | Trafficking | Mucins | Plasma membrane | Cancer
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9. Full Text Bile acids-mediated overexpression of MUC4 via FAK-dependent c-Jun activation in pancreatic cancer
by Joshi, Suhasini and Cruz, Eric and Rachagani, Satyanarayana and Guha, Sushovan and Brand, Randall E and Ponnusamy, Moorthy P and Kumar, Sushil and Batra, Surinder K
Molecular Oncology, ISSN 1574-7891, 08/2016, Volume 10, Issue 7, pp. 1063 - 1077
The majority of pancreatic cancer (PC) patients are clinically presented with obstructive jaundice with elevated levels of circulatory bilirubin and alkaline... 
FAK | FXR | c-Jun | Bile acids | MUC4 | Pancreatic cancer | c‐Jun | ONCOLOGY | ESOPHAGUS | Pancreatic Neoplasms - blood | Transcription, Genetic - drug effects | Bile Acids and Salts - pharmacology | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Mucin-4 - metabolism | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - enzymology | Mice, Transgenic | Pancreatic Neoplasms - genetics | Protein Transport - drug effects | Enzyme Activation - drug effects | Up-Regulation - drug effects | Animals | Cell Nucleus - metabolism | Signal Transduction - drug effects | Proto-Oncogene Proteins c-jun - metabolism | Base Sequence | Cell Line, Tumor | Gene Expression Regulation, Neoplastic - drug effects | Cell Nucleus - drug effects | Receptors, Cytoplasmic and Nuclear - metabolism | Medical colleges | Phosphatases | Promoters (Genetics) | Analysis | Oncology, Experimental | Mucins | Metastasis | Bilirubin | Research | Cancer | Jaundice | Cholecystectomy | Animal models | Gallbladder diseases | Kinases | Patients | Metastases | Studies | Acids | Transcription activation | Pheochromocytoma cells | Focal adhesion kinase | Bile | Tumors
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10. Full Text Influence of filament distribution on transverse tow permeability: Model predictions and experimental validation
by Godbole, Mayur G and Purandare, Rahul and Harshe, Rahul and Hood, Avinash and Gururaja, Suhasini and Joshi, Makarand and Advani, Suresh
Composites Part A, ISSN 1359-835X, 03/2019, Volume 118, pp. 150 - 161
In the past, various analytical and semi-analytical permeability models have been implemented for process modeling of flow through single scale fabric with... 
Filament distribution | SEM | Liquid composite molding | Transverse tow permeability | RESIN | REINFORCEMENTS | WOVEN FIBER PREFORMS | UNSATURATED FLOW | LIQUID | COMPRESSIBILITY | MATERIALS SCIENCE, COMPOSITES | RTM | FLOW PERMEABILITY | ENGINEERING, MANUFACTURING | IMPREGNATION | Textile fabrics | Models | Permeability | Mechanical engineering | Analysis | Aerospace engineering
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11. Full Text Tumor suppressor FOXO3 mediates signals from the EGF receptor to regulate proliferation of colonic cells
by Wentao Qi and Christopher R. Weber and Kaarin Wasland and Hemant Roy and Ramesh Wali and Suhasini Joshi and Suzana D. Savkovic
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 02/2011, Volume 300, Issue 2, pp. 264 - 272
Epithelial proliferation, critical for homeostasis, healing, and colon cancer progression, is in part controlled by epidermal growth factor receptor (EGFR).... 
FOXO3 | EGFR | Colon | PHYSIOLOGY | GROWTH-FACTOR-RECEPTOR | CANCER CELLS | PHOSPHATIDYLINOSITOL 3-KINASE | DOWN-REGULATION | CYCLE ARREST | BETA-CATENIN | INTESTINAL EPITHELIAL-CELLS | P27(KIP1) | FORKHEAD TRANSCRIPTION FACTORS | IN-VITRO | colon | GASTROENTEROLOGY & HEPATOLOGY | Intestinal Mucosa - metabolism | Phosphorylation | Cell Proliferation | Signal Transduction | Colon - pathology | Down-Regulation | Humans | Phosphatidylinositol 3-Kinases - metabolism | Colon - metabolism | Colonic Neoplasms - metabolism | Mice, Knockout | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Receptor, Epidermal Growth Factor - metabolism | Animals | Cell Cycle | Forkhead Transcription Factors - metabolism | Colonic Neoplasms - pathology | Cell Line, Tumor | Cytosol - metabolism | Mice | Forkhead Box Protein O3 | Proto-Oncogene Proteins c-akt - metabolism | Forkhead Transcription Factors - deficiency | Physiological aspects | Development and progression | Care and treatment | Colon cancer | Epidermal growth factor | Mucosal Biology
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12. Full Text Genetic variants of mucins: Unexplored conundrum
by Kumar, Sushil and Cruz, Eric and Joshi, Suhasini and Patel, Asish and Jahan, Rahat and KBatra, Surinder and Jain, Maneesh
Carcinogenesis, ISSN 0143-3334, 07/2017, Volume 38, Issue 7, pp. 671 - 679
Alternative gene splicing, occurring ubiquitously in multicellular organisms can produce several protein isoforms with putatively different functions. The... 
BREAST-CANCER | PREFERENTIAL EXPRESSION | EXTRACELLULAR-DOMAIN | PROTEIN | MUC1 SPLICE VARIANTS | ONCOLOGY | GENOMIC ORGANIZATION | EPIDERMAL-GROWTH-FACTOR | OVARIAN-CANCER | DRY EYE PATIENTS | PANCREATIC-CANCER CELLS | Multigene Family - genetics | Inflammation - pathology | Cell Proliferation - genetics | Alternative Splicing - genetics | Humans | Gene Expression Regulation, Neoplastic | Exons - genetics | Cell Movement - genetics | Mucins - biosynthesis | Neoplasms - genetics | Neoplasm Invasiveness - pathology | Inflammation - genetics | Neoplasm Invasiveness - genetics | Neoplasms - pathology | Mucins - genetics | Protein Isoforms - genetics | Index Medicus | Reviews
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13. A Chemical Biology Approach to the Chaperome in Cancer-HSP90 and Beyond
by Taldone, Tony and Wang, Tai and Rodina, Anna and Pillarsetty, Naga Vara Kishore and Digwal, Chander S and Sharma, Sahil and Yan, Pengrong and Joshi, Suhasini and Pagare, Piyusha P and Bolaender, Alexander and Roboz, Gail J and Guzman, Monica L and Chiosis, Gabriela
Cold Spring Harbor perspectives in biology, 04/2019
Cancer is often associated with alterations in the chaperome, a collection of chaperones, cochaperones, and other cofactors. Changes in the expression levels... 
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14. Full Text Novel HER3/MUC4 oncogenic signaling aggravates the tumorigenic phenotypes of pancreatic cancer cells
by Lakshmanan, Imayavaramban and Seshacharyulu, Parthasarathy and Haridas, Dhanya and Rachagani, Satyanarayana and Gupta, Suprit and Joshi, Suhasini and Guda, Chittibabu and Yan, Ying and Jain, Maneesh and Ganti, Apar K and Ponnusamy, Moorthy P and Batra, Surinder K
Oncotarget, ISSN 1949-2553, 2015, Volume 6, Issue 25, pp. 21085 - 21099
Several studies have demonstrated that MUC4 is involved in progression and metastasis of pancreatic cancer (PC). Here, we report that HER3/MUC4 interaction in... 
HER2 | KPC model | MUC4 | Pancreatic cancer | HER3 | Neoplasm Transplantation | Phosphorylation | Receptor, ErbB-3 - metabolism | Cell Proliferation | Pancreatic Neoplasms - metabolism | Humans | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Molecular Sequence Data | Phosphatidylinositol 3-Kinases - metabolism | Carcinogenesis | Neoplasm Metastasis | Female | Amino Acid Sequence | Signal Transduction | Mucin-4 - metabolism | Pancreatic Neoplasms - pathology | Proto-Oncogene Proteins c-myc - metabolism | Disease Progression | Sequence Homology, Amino Acid | Phenotype | Animals | Cell Cycle | Mice, Nude | Mice
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