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Journal of Clinical Investigation, ISSN 0021-9738, 03/2006, Volume 116, Issue 3, pp. 783 - 796
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2009, Volume 123, Issue 2, pp. 342 - 351
Background Peanut allergy is the most common food-related cause of lethal anaphylaxis and, unlike other food allergies, typically persists into adulthood.... 
Allergy and Immunology | C3a | Peanut | macrophages | histamine | shock | anaphylaxis | platelet-activating factor | basophils | complement | mast cells | MAST-CELLS | IMMUNOLOGY | mart cells | CUTTING EDGE | EFFECTOR PHASE | TARGETED DISRUPTION | ALLERGY | ACQUIRED-IMMUNITY | CELL WALL COMPONENTS | IN-VIVO | ALLERGIC-ASTHMA | IGE-BINDING EPITOPES | BETA-BLOCKERS | Humans | Platelet Activating Factor - immunology | Histamine - immunology | Complement Activation - immunology | Complement C3a - genetics | Immunoglobulin E - blood | Receptors, IgG - metabolism | Adrenergic beta-Antagonists - pharmacology | Histamine - metabolism | Interleukin-4 - pharmacology | Propranolol - pharmacology | Receptors, IgG - genetics | Allergens - immunology | Basophils - immunology | Mast Cells - metabolism | Anaphylaxis - immunology | Toll-Like Receptors - metabolism | Complement C3a - metabolism | Macrophages - immunology | Mast Cells - immunology | Chymases - blood | Anaphylaxis - etiology | Toll-Like Receptors - immunology | Arachis - metabolism | Peanut Hypersensitivity - immunology | Receptors, IgG - immunology | Cell Degranulation - immunology | Platelet Activating Factor - metabolism | Macrophages - metabolism | Animals | Peanut Hypersensitivity - metabolism | Arachis - immunology | Mice | Mice, Inbred BALB C | Complement C3a - immunology | Basophils - metabolism | Allergens | Medical colleges | Food allergy | Anaphylaxis | Dendritic cells | Proteases | Emergency medicine | Medical law | Platelet activating factor | Human subjects | Food allergies | Plasma | Peanuts | Rodents | Allergies | Binding sites | PAF | peanut
Journal Article
Journal Article
Frontiers in immunology, ISSN 1664-3224, 2019, Volume 10, p. 994
[This corrects the article DOI: 10.3389/fimmu.2019.00543.]. 
Infection | trauma | complement activation | hemorrhagic shock | complement dysregulation | MODS | sepsis
Journal Article
European Journal of Immunology, ISSN 0014-2980, 09/2011, Volume 41, Issue 9, pp. 2741 - 2752
Journal Article
FRONTIERS IN IMMUNOLOGY, ISSN 1664-3224, 03/2019, Volume 10, p. 543
Exposure to traumatic or infectious insults results in a rapid activation of the complement cascade as major fluid defense system of innate immunity. The... 
trauma | clinical trial | complement dysregulation | hemorrhagic shock | complement therapeutics | ACUTE LUNG INJURY | ACTIVATION PRODUCTS | IMMUNOLOGY | ANAPHYLATOXIN C5A | complement activation | HEMORRHAGIC-SHOCK | NEUTROPHIL DYSFUNCTION | C1 INHIBITOR | C5A RECEPTOR | TRAUMATIC BRAIN-INJURY | MODS | SEPTIC SHOCK | INFLAMMATORY RESPONSE | sepsis
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2017, Volume 12, Issue 2, p. e0172446
C5a drives airway constriction and inflammation during the effector phase of allergic asthma, mainly through the activation of C5a receptor 1 (C5aR1). Yet,... 
NEUTROPHIL DYSFUNCTION | TH2 RESPONSES | HUMAN EOSINOPHILS | COMPLEMENT | ANAPHYLATOXIN RECEPTORS | DENDRITIC CELLS | MULTIDISCIPLINARY SCIENCES | AIRWAY INFLAMMATION | LYMPHOID-CELLS | T-CELLS | LUNG INFLAMMATION | Lung - pathology | CD11b Antigen - immunology | Macrophages - pathology | Ovalbumin - immunology | Receptor, Anaphylatoxin C5a - analysis | Dendritic Cells - immunology | Dendritic Cells - pathology | CD4-Positive T-Lymphocytes - pathology | Eosinophils - immunology | CD4-Positive T-Lymphocytes - immunology | Animals | CD11b Antigen - analysis | Myeloid Cells - immunology | Asthma - immunology | Receptor, Anaphylatoxin C5a - immunology | Eosinophils - pathology | Mice | Myeloid Cells - pathology | Asthma - pathology | Immunity, Cellular | Mediastinum - pathology | Lung - immunology | Macrophages - immunology | Care and treatment | Monocytes | Inflammation | Research | G proteins | Asthma | Helper cells | Effector cells | Fluorescence | Activation | Mucus | Lymphocytes T | Macrophages | Lymph nodes | Respiratory tract | Genotype & phenotype | Signal transduction | CD103 antigen | Lymphocytes | Compartments | Respiratory tract diseases | Immune system | Lymphoid cells | Myeloid cells | CD11b antigen | Dendritic cells | Leukocytes (eosinophilic) | Neutrophils | Leukocytes (neutrophilic) | Allergies | CD4 antigen | Lymphocytes B | Lungs | Green fluorescent protein | Regulation | Eosinophils | Lymph
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2013, Volume 123, Issue 9, pp. 3788 - 3796
Journal Article