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Scientific reports, ISSN 2045-2322, 2018, Volume 8, Issue 1, pp. 1 - 9
..., Satoru Sasaki3, Yuhei Komiyama3, Takuji Izumikawa4, Marcus Conrad5, Shiro Bannai1 & Hideyo Sato1 System xc− was recently described as the most upstream node... 
Cysteine | Starvation | Cell death | Tumor cells | Cytotoxicity | Synergism | Cisplatin | Cancer | Glutathione | Apoptosis
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Nature communications, ISSN 2041-1723, 2014, Volume 5, Issue 1, p. 3978
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Journal of radiation research, ISSN 1349-9157, 2011, Volume 52, Issue 1, pp. 24 - 31
Journal Article
Cancer science, ISSN 1347-9032, 2018, Volume 109, Issue 1, pp. 215 - 224
Gemcitabine‐based therapy remains the mainstay of treatment for patients with biliary tract cancers (BTCs) with no second‐line treatment(s) established yet.... 
biliary tract cancer | trametinib | refractory | gemcitabine | MEK inhibitor | CHOLANGIOCARCINOMA | MULTICENTER PHASE-II | 2ND-LINE CHEMOTHERAPY | BRAF | COMBINATION | HEPATOCELLULAR-CARCINOMA | INHIBITION | ONCOLOGY | S-1 | MUTATIONS | ASSOCIATION | Administration, Oral | Humans | Middle Aged | Pyrimidinones - adverse effects | Male | Treatment Outcome | Protein Kinase Inhibitors - adverse effects | Transcription Factors - genetics | Whole Exome Sequencing | Protein Kinase Inhibitors - administration & dosage | Pyridones - administration & dosage | Gallbladder Neoplasms - drug therapy | Survival Analysis | Aged, 80 and over | Female | Aged | Pyrimidinones - administration & dosage | Biliary Tract Neoplasms - drug therapy | Nuclear Proteins - genetics | Neurofibromin 1 - genetics | Pyridones - adverse effects | Ampulla of Vater - pathology | Bile Duct Neoplasms - drug therapy | Antimitotic agents | Cancer patients | Enzyme inhibitors | Antineoplastic agents | Biliary tract | Alternative splicing | Gemcitabine | Toxicity | Research funding | MAP kinase | Employees | Metastasis | Kinases | Ampulla of Vater | Cancer therapies | Survival | Patients | Bile duct | Studies | Allosteric properties | Chemotherapy | Medical prognosis | Response rates | Biomarkers | Gallbladder | Mutation | Safety | Pharmaceuticals | Index Medicus | Original
Journal Article
Journal of neurochemistry, ISSN 1471-4159, 2008, Volume 104, Issue 4, pp. 1116 - 1131
Electrophilic compounds are a newly recognized class of redox‐active neuroprotective compounds with electron deficient, electrophilic carbon centers that react... 
neurite outgrowth‐promoting prostaglandin 11 | Keap1 | S‐alkylation | cysteine thiol | Nrf2 | carnosic acid | S-alkylation | Cysteine thiol | Carnosic acid | Neurite outgrowth-promoting prostaglandin 11 | OXIDATIVE STRESS | MITOCHONDRIAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | NEUROSCIENCES | GLUTAMATE TOXICITY | CORTICAL-NEURONS | ANTIOXIDANT | neurite outgrowth-promoting prostaglandin 11 | TRANSCRIPTION FACTOR | CEREBRAL-ISCHEMIA | CELL-LINE | Plant Extracts - chemistry | Plant Extracts - pharmacology | Cercopithecus aethiops | Catechols - chemistry | Male | PC12 Cells | Dose-Response Relationship, Drug | Neuroprotective Agents - pharmacology | Cattle | Catechols - pharmacology | Cysteine - metabolism | Neurons - metabolism | Neurons - drug effects | Kelch-Like ECH-Associated Protein 1 | Neuroprotective Agents - chemistry | Mice, Inbred C57BL | Cells, Cultured | Diterpenes, Abietane - chemistry | Intracellular Signaling Peptides and Proteins | Rats | Rats, Sprague-Dawley | Diterpenes, Abietane - pharmacology | Animals | Proteins - metabolism | Signal Transduction - drug effects | Rosmarinus | NF-E2-Related Factor 2 - metabolism | Alkylation - drug effects | Signal Transduction - physiology | Mice | COS Cells | Neurons | Cysteine | Herbs | Neurosciences | Biochemistry | Oxidation | Chemical compounds
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 11/2011, Volume 26, Issue 11, pp. 2682 - 2694
The aging process correlates with the accumulation of cellular and tissue damage caused by oxidative stress. Although previous studies have suggested that... 
Low‐turnover osteoporosis | Bone fragility | Senescent collagen cross‐links | Copper/zinc superoxide dismutase (CuZn‐SOD OR SOD1) | Reactive oxygen species (ROS) | Senescent collagen cross-links | Low-turnover osteoporosis | Copper/zinc superoxide dismutase (CuZn-SOD OR SOD1) | ANTIOXIDANTS | INCREASES | ACCELERATION | REACTIVE OXYGEN SPECIES (ROS) | BONE FRAGILITY | DISMUTASE | MINERALIZATION | BIOLOGY | ENDOCRINOLOGY & METABOLISM | COPPER/ZINC SUPEROXIDE DISMUTASE (CuZn-SOD OR SOD1) | ELEVATED OXIDATIVE STRESS | MICE | LOW-TURNOVER OSTEOPOROSIS | SENESCENT COLLAGEN CROSS-LINKS | EXPRESSION | OXIDANTS | Bone and Bones - pathology | RANK Ligand - metabolism | Cell Count | Aging - drug effects | Cytoplasm - metabolism | Bone and Bones - drug effects | Osteoporosis - metabolism | Bone and Bones - metabolism | Oxidation-Reduction - drug effects | Superoxides - metabolism | Osteoporosis - physiopathology | Superoxide Dismutase - metabolism | Cell Survival - drug effects | Bone Resorption - physiopathology | Osteoclasts - pathology | Osteoblasts - drug effects | Osteogenesis - drug effects | Ascorbic Acid - administration & dosage | Superoxide Dismutase - deficiency | Bone Resorption - complications | Macrophage Colony-Stimulating Factor - metabolism | Aging - pathology | Cross-Linking Reagents - metabolism | Collagen - metabolism | Osteoblasts - pathology | Bone and Bones - physiopathology | Organ Size - drug effects | Phenotype | Animals | Osteoporosis - pathology | Signal Transduction - drug effects | Osteoporosis - complications | Ascorbic Acid - pharmacology | Bone Resorption - pathology | Mice | Cytoplasm - drug effects | Osteoclasts - drug effects | Oxidative stress | Ascorbic acid | Spine | Intracellular signalling | Superoxide dismutase | Osteoblasts | Antioxidants | Cross-linking | Bones | Copper | TRANCE protein | Bone strength | Vertebrae | Therapeutic applications | Macrophage colony-stimulating factor | Mechanical properties | Stress | Vitamins | Connective tissue | Analysis | Collagen | Osteoclasts | Bone mineral density | Animal subjects | Osteogenesis
Journal Article
INTERNAL MEDICINE, ISSN 0918-2918, 2019, Volume 58, Issue 13, pp. 1865 - 1870
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