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by Kain, E
LITERATUR UND KRITIK, ISSN 0024-466X, 11/2015, Issue 499, pp. 103 - 110
Journal Article
Methods in Molecular Biology, ISSN 1064-3745, 2017, Volume 1508, pp. 185 - 193
The detection of fungal elements and their characterization in patient specimens provides fundamental information. On histological sections fungi are most... 
Schiffs’ reaction | PAS stain | Silver stain | Histochemistry | Mycoses - diagnosis | Mycoses - microbiology | Tissue Embedding | Humans | Mycoses - pathology | Staining and Labeling | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2008, Volume 14, Issue 10, pp. 1088 - 1096
Journal Article
Nephrology Dialysis Transplantation, ISSN 0931-0509, 05/2016, Volume 31, Issue suppl_1, pp. i351 - i352
Journal Article
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 06/2018, Volume 217, Issue 6, pp. 2205 - 2221
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 07/2017, Volume 377, Issue 1, pp. 52 - 61
CD55 prevents convertase enzyme formation in the complement cascade, acting as a brake on complement activation. Inactivating mutations in CD55 result in... 
MEDICINE, GENERAL & INTERNAL | ACTIVATION | INFLAMMATORY-BOWEL-DISEASE | HEMOLYTIC-UREMIC SYNDROME | CELL RESPONSES | COMPLEMENT REGULATORY PROTEIN | INAB PHENOTYPE | DECAY-ACCELERATING FACTOR | PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA | MEMBRANOPROLIFERATIVE GLOMERULONEPHRITIS | T-CELLS | Intestine, Small - pathology | Humans | CD55 Antigens - genetics | Child, Preschool | Infant | Male | Protein-Losing Enteropathies - genetics | Syndrome | Complement System Proteins - metabolism | CD55 Antigens - blood | Complement Activation - genetics | Homozygote | T-Lymphocytes - metabolism | Complement Inactivating Agents - pharmacology | Pedigree | Protein-Losing Enteropathies - complications | Statistics, Nonparametric | Immunoglobulin A - blood | Female | Thrombosis - genetics | Mutation | Child | Complement Activation - drug effects | Proteins | Molecular targeted therapy | Gene mutations | Gastrointestinal diseases | Analysis | Homeostasis | Research | Thrombosis | Blood clot | Gastrointestinal tract diseases | Complement component C5a | Lymphocytes T | Cell activation | Immunology | Pain | Intestine | Hepatology | Gastroenterology | Malabsorption | Thromboembolism | Digestive tract | Age | Edema | Medical research | Lymphatic system | Hypersensitivity | Diarrhea | Inflammation | Heredity | Protein deficiency | Patients | Hereditary diseases | Complement activation | Infectious diseases | Decay-accelerating factor | Index Medicus | Abridged Index Medicus
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 07/2019, Volume 9, Issue 1, pp. 1 - 11
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2017, Volume 199, Issue 2, pp. 531 - 546
Journal Article
Journal Article
Biology Open, ISSN 2046-6390, 10/2016, Volume 5, Issue 10, pp. 1516 - 1529
Autophagy is an evolutionarily conserved process used for removing surplus and damaged proteins and organelles from the cytoplasm. The unwanted material is... 
VAMP8 | STX17 | LAMP-2 | Lysosome | Autophagosome | LC3 | Macroautophagy | CONTACT SITES | DANON-DISEASE | CHOLESTEROL | CHAPERONE-MEDIATED AUTOPHAGY | MEMBRANE-PROTEIN LAMP-2 | CARDIOMYOPATHY | MATURATION | TRANSPORT | TETHERING COMPLEXES | BIOLOGY | DEGRADATION
Journal Article
Oncogene, ISSN 0950-9232, 12/2000, Volume 19, Issue 52, pp. 6043 - 6052
Eph receptor tyrosine kinases and their ephrin ligands have been implicated in embryonic vascular development and in in vivo models of angiogenesis. Eph... 
Eph receptor tyrosine kinase | Angiogenesis | Endothelial cells | VASCULAR DEVELOPMENT | RESPONSE GENE | endothelial cells | angiogenesis | BIOCHEMISTRY & MOLECULAR BIOLOGY | BLOOD-VESSEL FORMATION | PROTEIN-TYROSINE KINASE | Eph receptor tyrosine kinases | CELL-LINES | ECK | CELL BIOLOGY | CARDIOVASCULAR DEVELOPMENT | ONCOLOGY | GENETICS & HEREDITY | MELANOMA PROGRESSION | B61 | Neoplasms - metabolism | Neoplasm Transplantation | Receptor, EphA2 | Ephrin-A1 | Sarcoma, Kaposi - metabolism | Humans | Gene Expression Regulation, Neoplastic | Transplantation, Heterologous | Neovascularization, Pathologic - blood | Breast Neoplasms - metabolism | Neovascularization, Pathologic - pathology | Phosphotyrosine - metabolism | Laminin | Female | Tumor Cells, Cultured | Capillaries - metabolism | Hyperplasia - metabolism | Proteoglycans | Signal Transduction | Cells, Cultured | Neoplasms - blood supply | Sarcoma, Kaposi - blood supply | Breast Neoplasms - blood supply | Receptor Protein-Tyrosine Kinases - blood | Receptor Protein-Tyrosine Kinases - metabolism | Capillaries - growth & development | Blotting, Western | Sarcoma, Kaposi - pathology | Hyperplasia - pathology | Animals | Proteins - metabolism | Receptor Protein-Tyrosine Kinases - genetics | Breast Neoplasms - pathology | Endothelium, Vascular - metabolism | Mice, Nude | Collagen | Fluorescent Antibody Technique | Endothelium, Vascular - pathology | Ligands | Neovascularization, Pathologic - genetics | Mice | Neovascularization, Pathologic - metabolism | Neoplasms - pathology | Drug Combinations | Physiological aspects | Neovascularization | Research | Protein kinases | Cancer cells | Endothelium | Index Medicus
Journal Article
Journal Article