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Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e87871
Caveolin-1 is a scaffolding/regulatory protein that interacts with diverse signaling molecules. Caveolin-1 null mice have marked metabolic abnormalities, yet... 
CELLS | ACTIVATED PROTEIN-KINASE | REACTIVE OXYGEN | PULMONARY-HYPERTENSION | NITRIC-OXIDE SYNTHASE | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | MITOCHONDRIA | HYDROGEN-PEROXIDE | MICE | DYSFUNCTION | Caveolin 1 - antagonists & inhibitors | RNA, Small Interfering - genetics | Reactive Oxygen Species - metabolism | Oxidative Stress | Signal Transduction | Glutathione - metabolism | Cells, Cultured | Metabolome | Mitochondria - metabolism | Aorta - metabolism | Mitochondria - pathology | Autophagy | Hydrogen Peroxide - metabolism | Mice, Knockout | Aorta - pathology | Catalase - metabolism | Animals | Cattle | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Mice | Caveolin 1 - physiology | Oxidative stress | Glucose | Fatty acids | Dextrose | Endothelium | Caveolin-1 | Cell culture | Hydrogen peroxide | Laboratories | Homeostasis | Kinases | Experiments | Medical schools | Proteins | Lysates | Signal transduction | Mitochondria | Rodents | Fibroblasts | Physiology | Life sciences | Bioindicators | Heart diseases | Hypertension | Caveolin | Abnormalities | siRNA | Metabolism | Endothelial cells | Switching | Intermediates | Medicine | Signaling | Hospitals | Molecular modelling | Scaffolding | Nitric oxide | Glycolysis | Biomarkers | Insulin resistance | Cardiovascular diseases | Phagocytosis | Cancer
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 08/2005, Volume 25, Issue 16, pp. 6980 - 6989
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2011, Volume 286, Issue 3, pp. 2320 - 2330
The MARCKS protein (myristoylated alanine-rich C kinase substrate) is an actin- and calmodulin-binding protein that is expressed in many mammalian tissues. The... 
Phosphorylation | Wound healing | Caveolae | fluorescence resonance energy transfer | Hydrodynamics | Phospholipids | Phosphatidylinositol 4,5-diphosphate | Insulin | imaging | Endothelial cells | Protein turnover | Endothelium | Signal transduction | Actin | Calcium-binding protein | MARCKS protein | Biosensors | Calmodulin | Cell migration
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2011, Volume 286, Issue 3, pp. 2320 - 2330
The MARCKS protein (myristoylated alanine-rich C kinase substrate) is an actin-and calmodulin-binding protein that is expressed in many mammalian tissues. The... 
CARCINOMA-CELLS | LIPID DOMAINS | SIGNALING PATHWAYS | FILAMENT CROSS-LINKING | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUBCELLULAR-LOCALIZATION | NITRIC-OXIDE | KINASE-C | N-WASP | ACTIN CYTOSKELETON | PLASMA-MEMBRANE | Hypoglycemic Agents - metabolism | Myristoylated Alanine-Rich C Kinase Substrate | Actin-Related Protein 2-3 Complex - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Cell Movement - physiology | Gene Knockdown Techniques | Dose-Response Relationship, Drug | Phosphatidylinositol 4,5-Diphosphate - metabolism | Actin-Related Protein 2-3 Complex - metabolism | Time Factors | Cattle | Membrane Proteins - metabolism | Wiskott-Aldrich Syndrome Protein, Neuronal - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Insulin - pharmacology | Phosphatidylinositol 4,5-Diphosphate - genetics | Caveolae - metabolism | Endothelial Cells - metabolism | Membrane Proteins - genetics | Hypoglycemic Agents - pharmacology | Cell Movement - drug effects | Insulin - metabolism | Animals | Signal Transduction - drug effects | Endothelial Cells - cytology | Biosensing Techniques - methods | Signal Transduction - physiology | Wiskott-Aldrich Syndrome Protein, Neuronal - genetics | Cell Migration | Phosphatidylinositol Signaling | Protein Phosphorylation | Signal Transduction | Actin | Endothelium
Journal Article
Pflügers Archiv - European Journal of Physiology, ISSN 0031-6768, 12/2007, Volume 455, Issue 3, pp. 465 - 477
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2011, Volume 108, Issue 38, pp. 15792 - 15797
Nitric oxide (NO) and hydrogen peroxide (H₂O₂) are synthesized within cardiac myocytes and play key roles in modulating cardiovascular signaling. Cardiac... 
Heart | Protein isoforms | Phosphorylation | Hydrogen | Fluorescence | Antibodies | Oxides | Peroxides | Modulated signal processing | Endothelial cells | Signal transduction | Nitric oxide synthase | Angiotensin II | Biosensors | ACTIVATED PROTEIN-KINASE | SPECIFICITY | signal transduction | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | RECEPTOR | biosensors | ANGIOTENSIN-II | SYNTHASE | HEART | REACTIVE OXYGEN | CONTRACTION | angiotensin II | ENDOTHELIAL-CELLS | nitric oxide synthase | AMP-Activated Protein Kinases - metabolism | Myocardial Contraction - drug effects | Immunoblotting | Myocytes, Cardiac - enzymology | Copper - chemistry | Nitric Oxide Synthase Type III - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Pyrazoles - pharmacology | Vasoconstrictor Agents - pharmacology | Fluorescent Dyes - chemistry | Angiotensin II - pharmacology | Myocytes, Cardiac - cytology | Nitric Oxide - chemistry | AMP-Activated Protein Kinases - antagonists & inhibitors | Cell Size - drug effects | Mice, Inbred C57BL | Cells, Cultured | Hydrogen Peroxide - pharmacology | Adrenergic beta-Agonists - pharmacology | Pyrimidines - pharmacology | Nitric Oxide Synthase Type III - genetics | Hydrogen Peroxide - metabolism | Mice, Knockout | Animals | Myocytes, Cardiac - drug effects | Isoproterenol - pharmacology | Nitric Oxide Synthase Type I - metabolism | Mice | Nitric Oxide Synthase Type I - genetics | Nitric Oxide - metabolism | Microscopy, Fluorescence | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Physiological aspects | Hydrogen peroxide | Research | Nitric oxide | Angiotensin | Biological Sciences
Journal Article
Nature Communications, ISSN 2041-1723, 11/2015, Volume 6, Issue 1, pp. 8966 - 8966
Glia play critical roles in maintaining the structure and function of the nervous system; however, the specific contribution that astroglia make to... 
SYNTHASE | S-NITROSYLATION | DROSOPHILA-MELANOGASTER | FIBRILLARY ACIDIC PROTEIN | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MOUSE MODEL | NEURONAL CELL-DEATH | STRESS | NADPH-DIAPHORASE | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2006, Volume 103, Issue 50, pp. 19093 - 19098
Regional alveolar hypoxia causes local vasoconstriction in the lung, shifting blood flow from hypoxic to normoxic areas, thereby maintaining gas exchange. This... 
Pulmonary alveoli | Lungs | Vasoconstriction | Fluorescence | Hypoxia | Cell membranes | Pulmonary hypertension | Pulmonary artery | Pulmonary gas exchange | Vasoconstrictors | Arterial hypoxemia | Precapillary pulmonary arterial smooth-muscle cells | Transient receptor potential channel 6-deficient mouse model | Hypoxia-induced diacylglycerol accumulation | pulmonary hypertension | MULTIDISCIPLINARY SCIENCES | hypoxia-incluced diacylglycerol accumulation | CAPACITATIVE CA2+ ENTRY | OPERATED CA2 | RECEPTOR POTENTIAL CHANNELS | INHIBITION | SMOOTH-MUSCLE-CELLS | ENDOTHELIN-1 | precapillary pulmonary arterial smooth-muscle cells | HYPERTENSION | ARTERIAL MYOCYTES | transient receptor potential channel 6-deficient mouse model arterial hypoxemia | NONSELECTIVE CATION CHANNELS | OXYGEN-SENSING MECHANISMS | Acute Disease | Gene Expression | Cations - chemistry | Lung Diseases - metabolism | Cations - metabolism | TRPC Cation Channels - deficiency | Aorta - metabolism | Hypoxia - metabolism | Mice, Knockout | Hypoxia - genetics | Lung Diseases - genetics | Animals | Diglycerides - metabolism | Membrane Potentials | TRPC Cation Channels - metabolism | Pulmonary Gas Exchange | Mice | TRPC Cation Channels - genetics | Smooth muscle | Research | Diacylglycerol | hypoxia-induced diacylglycerol accumulation | transient receptor potential channel 6-deficient mouse model | Biological Sciences | arterial hypoxemia
Journal Article
Journal Article