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Cancer research, ISSN 0008-5472, 6/2018, Volume 78, Issue 12, pp. 3321 - 3336
The high mortality of pancreatic cancer demands that new therapeutic avenues be developed. The orally available small molecule inhibitor AT13148 potently... 
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 510, Issue 7504, pp. 298 - 302
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2013, Volume 110, Issue 22, pp. 8882 - 8887
Journal Article
Nature, ISSN 0028-0836, 09/2014, Volume 513, Issue 7519, p. 574
Journal Article
Nature, ISSN 0028-0836, 08/2016, Volume 536, Issue 7617, pp. 401 - 402
The cancer pancreatic ductal adenocarcinoma (PDAC) has a poor prognosis, and is an area of intense biomedical research. Previous studies1,2 have shown that... 
Cell growth | Mitochondria | Biomedical research | Medical prognosis | Pancreatic cancer | Amino acids | Biosynthesis | Metabolism | Autophagy | Tumors
Journal Article
Cancer Cell, ISSN 1535-6108, 01/2015, Volume 27, Issue 1, pp. 57 - 71
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2012, Volume 7, Issue 7, pp. e41831 - e41831
mTOR inhibitors are used clinically to treat renal cancer but are not curative. Here we show that autophagy is a resistance mechanism of human renal cell... 
COMPLEX | TRANSCRIPTION FACTOR NRF2 | MULTIDISCIPLINARY SCIENCES | TARGETING AUTOPHAGY | PROGRAMMED NECROSIS | DEATH MECHANISMS | METABOLIC STRESS | MAMMALIAN-CELLS | CANCER | RENAL-CELL CARCINOMA | MOUSE MODELS | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Cell Survival - drug effects | Carcinoma, Renal Cell - pathology | Antioxidants - metabolism | Humans | Mitochondria - metabolism | Basal Metabolism - drug effects | Mitochondria - drug effects | Autophagy - drug effects | Necrosis | TOR Serine-Threonine Kinases - antagonists & inhibitors | Xenograft Model Antitumor Assays | Chloroquine - pharmacology | Animals | Cell Nucleus - metabolism | Active Transport, Cell Nucleus - drug effects | NF-E2-Related Factor 2 - metabolism | Oxidation-Reduction - drug effects | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Mice | Protein Kinase Inhibitors - pharmacology | Cell Nucleus - drug effects | TOR protein | Biotechnology | Oxidative stress | Reactive oxygen species | Syngeneic grafts | Genomics | Leukemia | Kinases | Autophagy | Antioxidants | Toxicology | Mitochondria | Cell growth | Allografts | Xenografts | Gangrene | Tumorigenesis | Inhibition | Growth factors | Cytomegalovirus | Cell survival | Bioavailability | Tumor cell lines | Dentistry | Survival | Medicine | Inhibitors | Medical prognosis | Cell lines | Hypoxia | Lymphomas | Mutation | Phagocytosis | Clear cell-type renal cell carcinoma | Kidney transplantation | Cancer | Apoptosis | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 10/2015, Volume 17, Issue 10, pp. 1317 - 1326
Journal Article
Genes and Development, ISSN 0890-9369, 03/2011, Volume 25, Issue 5, pp. 460 - 470
Autophagy is a catabolic pathway used by cells to support metabolism in response to starvation and to clear damaged proteins and organelles in response to... 
Mitochondria | Metabolism | Ras | Autophagy | p62 | Cancer | autophagy | mitochondria | DEVELOPMENTAL BIOLOGY | MEDIATE | SUPPRESSION | IMPAIRMENT | CELL BIOLOGY | PARKIN | DISEASE | GENETICS & HEREDITY | cancer | metabolism | MICE | PROMOTES | Cell Line | Starvation | Cell Proliferation